ライフサイエンスコーパス: R. solanacearum

1 R. solanacearum RpoS (RpoS(Rso)) was demonstrated to fun

2 R. solanacearum synthesized putrescine via a SpeC ornith

3 ogy (IVET), we screened a library of 133 200 R. solanacearum strain K60 promoter fusions and isolated

4 An R. solanacearum mutant lacking the pathogen's two extrac

5 has in recent years led to the concept of an R. solanacearum species complex.

6 ion accelerated wilt symptom development and R. solanacearum growth and systemic spread.

7 hich includes R. eutropha, R. pickettii, and R. solanacearum.

8 Cloned R. solanacearum aer1 and aer2 genes restored aerotaxis t

9 lcaligenes eutrophus) that fully complements R. solanacearum phcA mutants.

10 uce an extracellular factor that complements R. solanacearum mutants deficient in production of the 3

11 Two site-directed R. solanacearum mutants lacking either CheA or CheW, whi

12 psI operon is the major virulence factor for R. solanacearum.

13 is not a sole carbon or nitrogen source for R. solanacearum, was enriched 76-fold to 37 microM in R.

14 serve as sole carbon or nitrogen sources for R. solanacearum.

15 gesting that the main location in tomato for R. solanacearum during pathogenesis is iron replete.

16 ng one of the exo-PGs, pehB, was cloned from R. solanacearum K60.

17 file of ipx genes suggests that in its host, R. solanacearum confronts and overcomes a stressful and

18 lomics identified 22 metabolites enriched in R. solanacearum-infected sap.

19 The chromosomal pehB genes in R. solanacearum wild-type strain K60 and in an endo-PG P

20 ided no clues as to the role of acyl-HSLs in R. solanacearum gene regulation.

21 o investigate the role of these acyl-HSLs in R. solanacearum virulence gene expression, transposon mu

22 cearum, was enriched 76-fold to 37 microM in R. solanacearum-infected sap.

23 regulation of flagellum-mediated motility in R. solanacearum.

24 SS2(bp) genes, as well as their orthologs in R. solanacearum.

25 production, whereas inactivation of phcA in R. solanacearum increases siderophore production.

26 Mutation of rpoS(Rso) in R. solanacearum reduced survival during starvation and l

27 e acyl-HSL-dependent autoinduction system in R. solanacearum is part of a more complex autoregulatory

28 Therefore, in R. solanacearum the acyl-HSL-dependent autoinduction sys

29 quired for a wild-type level of virulence in R. solanacearum although its individual role in wilt dis

30 ildtype rescued DeltaspeC growth, indicating R. solanacearum produced and exported putrescine to xyle

31 scopy revealed that during tomato infection, R. solanacearum forms biofilm-like masses in xylem vesse

32 ssion of many ipx genes was subject to known R. solanacearum virulence regulators.

33 While Phc is present in most R. solanacearum strains, it is apparently absent from ot

34 contributes significantly to the ability of R. solanacearum to locate and effectively interact with

35 and a naturally acyl-HSL-defective strain of R. solanacearum to produce acyl-HSLs.

36 actor (VEF) produced by wild-type strains of R. solanacearum.

37 s the gene encoding the catalytic subunit of R. solanacearum's sole assimilatory nitrate reductase, d

38 erophores present in culture supernatants of R. solanacearum, R. metallidurans, and Bacillus megateri

39 ontributes substantially to the virulence of R. solanacearum.

40 The predicted R. solanacearum FliM closely resembled motor switch prot

41 y suggest that nitrate assimilation promotes R. solanacearum virulence by enhancing root attachment,

42 K60 generally resembles R. solanacearum CFBP2957, a Caribbean tomato isolate, bu

43 To locate and infect host plant roots R. solanacearum needs taxis, the ability to move toward

44 wledge, this is the first demonstration that R. solanacearum forms biofilms in plant xylem vessels, a

45 em vessels of infected plants, we found that R. solanacearum is essentially nonmotile in planta, alth

46 We found that R. solanacearum manipulates its host to increase nutrien

47 graphy, and mass spectroscopy indicated that R. solanacearum produces staphyloferrin B rather than sc

48 The R. solanacearum genome encodes two putative aerotaxis tr

49 characterized, and mutated two genes in the R. solanacearum flagellar biosynthetic pathway.

50 The identification of the R. solanacearum enzyme enables us to propose that the an

51 rtholog of HrpB, the master regulator of the R. solanacearum T3SS (T3SS(rso)) and its secreted effect

52 ositively activates motility, in contrast to R. solanacearum where it represses motility.

53 of unknown function, and 13% were unique to R. solanacearum.

54 lates virulence gene expression in wild-type R. solanacearum.