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2 f relapse was 40% for secondary AML, 40% for RAEB-T, 26% for RAEB, and 0% for RA or RARS (P =.0009).
3 JMML, 31% +/- 26%; RA and RAEB, 29% +/- 16%; RAEB-T, 30% +/- 18%; antecedent MDS, 50% +/- 25%; and de
4 fractory anemia with excess blasts [RAEB] or RAEB-t, 62 patients) to receive fludarabine + ara-C + id
5 es for patients with RAEB in transformation (RAEB-T) (69%) or antecedent MDS (81%) were similar to de
7 ransformation or acute myelogenous leukemia (RAEB-T/AML), and 2 had chronic myelomonocytic leukemia (
8 RAEB, essentially identical to the rates in RAEB-t and AML, but event-free survival (EFS) from CR an
9 anemia with excess blasts in transformation (RAEB-t), and especially patients with RAEB, receive chem
13 anemia with excess blasts in transformation [RAEB-T] and tAML [MDS transformed into AML]), colony num
15 tics, a diagnosis of RAEB rather than AML or RAEB-t had no effect on EFS from start of Rx, EFS from C
16 anemia with excess blasts in transformation (RAEB-T) had apoptosis levels in lymphocytes similar to t
17 ition, transplantation before progression to RAEB-T, if possible, may reduce the risk of relapse.
19 anemia with excess blasts in transformation (RAEB-t), or refractory anemia with excess blasts (RAEB).
21 anaemia with excess blasts (RAEB)-1, RAEB-2, RAEB-t, or chronic myelomonocytic leukaemia based on loc
22 (CFU)-leukemia formation in 9 of 15 CMML and RAEB-t patient specimens, whereas VEGF stimulated leukem
27 analyses suggested a trend for patients with RAEB-t to have better EFS from start of Rx than patients
30 dministering AML Rx to patients with RAEB or RAEB-t who have poor prognosis without treatment is iden
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