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1 RNA pol I polymerases are generally active only in cells
12 Nevertheless, we show that a nonendogenous RNA pol I promoter drives efficient rescue of influenza
14 ns between the TTD with the Rpa12 subunit of RNA pol I seem to be an integral part of the mechanism.
15 s by specific DNA bound proteins such as the RNA pol I termination factor, Reb1p, or lac repressor.
17 ranscribes the protein-coding genes, whereas RNA pol I transcribes the genes that encode the three RN
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