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1 h a subset are not suggestive of stalling by RNA polymerase III.
2 nscription of the 5 S rRNA and tRNA genes by RNA polymerase III.
3 POLR3A and POLR3C encode subunits of RNA polymerase III.
4 , U2), whereas the U6 gene is transcribed by RNA polymerase III.
5 r TFIIIB plays key roles in transcription by RNA polymerase III.
6 occurs in the internal promoter regions for RNA polymerase III.
7 elocation to the nucleus, where it represses RNA polymerase III.
8 biogenesis of noncoding RNAs transcribed by RNA polymerase III.
9 rase II promoter rather than from ubiquitous RNA polymerase III.
10 es at the transcription initiation region of RNA polymerase III.
11 the up-stream region of genes transcribed by RNA polymerase III.
12 nent in basal and regulated transcription by RNA polymerase III.
13 plasmid DNA by the function of the cellular RNA polymerase III.
14 g and accurately initiating transcription by RNA polymerase III.
15 ion initiation sites of genes transcribed by RNA polymerase III.
16 mid-borne 5S and U6 RNA genes transcribed by RNA polymerase III.
17 achieve in the case of genes transcribed by RNA polymerase III.
18 efective transcription of SNR6 (U6 snRNA) by RNA polymerase III.
19 ation/reinitiation of transcription by human RNA polymerase III.
20 n and tagetitoxin, suggesting involvement of RNA polymerase III.
21 factors that mediate promoter selectivity by RNA polymerase III.
22 ion initiation sites of genes transcribed by RNA polymerase III.
23 h DNA binding factors, and directly recruits RNA polymerase III.
24 ession of transcription that is dependent on RNA polymerase III.
25 ly promotes SINE expression and occupancy by RNA polymerase III.
26 ubunit factors required for transcription by RNA polymerase III.
27 ements is transcribed into non-coding RNA by RNA polymerase III.
28 late methylation impedes U6 transcription by RNA polymerase III.
29 hed at telomeres and at genes transcribed by RNA polymerase III.
30 ion initiation sites of genes transcribed by RNA polymerase III.
31 ter is critical for optimal transcription by RNA polymerase III.
32 tRNAs, and other transcripts synthesized by RNA polymerase III and facilitates their maturation, whi
33 ong type I IFN (IFN-I) response triggered by RNA polymerase III and melanoma differentiation-associat
37 results provide a characterization of human RNA polymerase III and show that the RPC5 subunit is ess
38 e of thymines, which is the pause signal for RNA polymerase III and thus could potentially reduce tra
39 recognition particle (SRP) is transcribed by RNA polymerase III, and most steps in SRP biogenesis occ
42 was also associated with the absence of anti-RNA polymerase III antibodies and presence of anti-U1 RN
49 al in that they are initially transcribed by RNA polymerase III as a single, approximately 122-nt pri
50 tein include all nascent transcripts made by RNA polymerase III as well as certain small RNAs synthes
52 ults reveal an unexpected role for tDNAs and RNA polymerase III-associated proteins in establishment
53 of polyglutamine, MOAG-2/LIR-3 regulates the RNA polymerase III-associated transcription of small non
54 ons have demonstrated an association between RNA polymerase III autoantibodies and a close temporal r
56 ternal DNA scaffold is needed for TFIIIB and RNA polymerase III binding, and that productive initiati
57 rate that L11 suppresses c-Myc-dependent and RNA polymerase III-catalyzed transcription of 5 S rRNA a
59 elements, or in extragenic loci that inhibit RNA polymerase III complex assembly, reduce barrier acti
60 have partially purified and characterized a RNA polymerase III complex that can direct transcription
61 ting a hierarchy that favors assembly of the RNA polymerase III complex versus assembly of adjacent a
65 levels coincided with hypoxic inhibition of RNA polymerase III-dependent MYC target genes, which MYC
67 ncing, SINE-seq), which selectively profiles RNA Polymerase III-derived SINE RNA, thereby identifying
71 evidence suggests that genes transcribed by RNA polymerase III exhibit multiple functions within a c
74 omplexes including RNA splicing proteins and RNA polymerase III from yeast, have been undertaken.
77 ative DNA binding with another member of the RNA polymerase III general transcription machinery, TFII
78 s is the synthesis of RNA molecules, certain RNA polymerase III genes also function as genomic landma
79 We first used STAGE in yeast to confirm that RNA polymerase III genes are the most prominent targets
80 resenting the first quantitative analysis of RNA polymerase III genes in situ by electron microscopy.
81 IIIB generally required for transcription of RNA polymerase III genes, and the second is hBRFU, one o
83 romyces cerevisiae RNA polymerase III, human RNA polymerase III has not been entirely characterized.
84 vitro transcription of the rat vRNA gene by RNA polymerase III has previously been shown to be depen
86 encoding a putative RPC5-like subunit of the RNA Polymerase III in a model species Nicotiana benthami
88 the founding members of those recognized by RNA polymerase III in which all control elements for ini
89 ence that methylated SINE DNA is occupied by RNA polymerase III, including the use of high-throughput
93 eri and Maraia (2015) demonstrate that yeast RNA polymerase III integrates inputs from both strands o
95 We have found that the synthesis of tRNA by RNA polymerase III is also inhibited in response to ARF.
99 ether the general transcription machinery or RNA polymerase III is preferentially phosphorylated.
102 d polyuridine sequence of viral and cellular RNA polymerase III non-coding transcripts is critical fo
103 peaks within the domains occur frequently at RNA-polymerase-III-occupied transfer RNA (tRNA) genes, w
106 ranscription factor occurs in the absence of RNA polymerase III or polymerase II but requires specifi
109 e upon the stability and accumulation of its RNA polymerase III (Pol III) directed transcripts was de
110 transcription regulation that involves both RNA polymerase III (Pol III) elements and CCCTC binding
114 tivators that regulate the activity of human RNA polymerase III (Pol III) in the context of chromatin
115 sents a limiting step in the assembly of the RNA polymerase III (pol III) initiation factor TFIIIB.
120 Short hairpin RNAs (shRNAs) transcribed by RNA polymerase III (Pol III) promoters can trigger seque
122 h the sweet orange MAF1 (CsMAF1) protein, an RNA polymerase III (Pol III) repressor that controls rib
123 s or oncogenes, directly associates with the RNA polymerase III (pol III) subunit RPC32 and enhances
126 e chromatin immunoprecipitation assay in the RNA polymerase III (pol III) system that allowed us to m
127 A genes and regulates their transcription by RNA polymerase III (pol III) through direct binding and
133 es cerevisiae gene BDP1 encodes a subunit of RNA polymerase III (Pol III) transcription factor (TFIII
134 e chromosomal locations bound in vivo by the RNA polymerase III (Pol III) transcription factor III C
137 vation and various stress conditions repress RNA polymerase III (Pol III) transcription in S. cerevis
139 hat allow productive binding in vitro of the RNA polymerase III (Pol III) transcription initiation fa
141 minal repeat retrotransposon Ty3 is found at RNA polymerase III (Pol III) transcription start sites o
144 evisiae U6 RNA gene, SNR6, is transcribed by RNA polymerase III (Pol III), but lacks the intragenic B
145 anscription of small non-coding RNA genes by RNA polymerase III (Pol III), but the precise role of th
146 ns and a subset of small RNAs transcribed by RNA polymerase III (pol III), including the signal recog
147 e central transcription initiation factor of RNA polymerase III (pol III), is composed of three subun
149 ituted with recombinant factors and purified RNA polymerase III (pol III), pol III must be treated wi
150 about the initial phase of transcription by RNA polymerase III (Pol III), the enzyme that synthesize
153 charomyces cerevisiae integrates upstream of RNA polymerase III (Pol III)-transcribed genes, yet the
155 o evidence of the production of noncanonical RNA polymerase III (Pol III)-transcribed viral microRNAs
164 n led to the identification of DNA-dependent RNA polymerase III (Pol-III) as the enzyme responsible f
165 molecular epitope spreading in patients with RNA polymerase III (POLR3) and the minor spliceosome spe
166 dontia and Hypogonadotropic Hypogonadism) or RNA polymerase III (POLR3)-related leukodystrophy cases
167 c La proteins, p43 does not bind strongly to RNA polymerase III precursor transcripts and does not ex
168 ities such as processing and/or transport of RNA polymerase III precursor transcripts and translation
169 itiation complexes on U1 to U5 promoters but RNA polymerase III preinitiation complexes on U6 promote
170 Its activity in mice is correlated with its RNA polymerase III promoter activity and its orientation
171 RNA sequence has two elements which fit the RNA polymerase III promoter consensus sequence, and show
172 arrying silencing cassettes consisting of an RNA polymerase III promoter expressing short hairpin RNA
173 ng achieved with an shRNAi expressed from an RNA polymerase III promoter in transient transfection wa
174 ts of breaks in the individual strands of an RNA polymerase III promoter on initiation of transcripti
175 se data establish that RNA polymerase II and RNA polymerase III promoter sequences are superimposed f
176 ptional control region contains an efficient RNA polymerase III promoter, in addition to the well cha
177 ion of a loxP-flanked neomycin cassette into RNA polymerase III promoter, which controls a vector-bas
178 randed oligonucleotides or plasmids encoding RNA polymerase III promoter-driven small hairpin RNA.
180 s first provided by the properties of TFIIIB-RNA polymerase III-promoter complexes assembled with del
182 Thus, in fruit flies, different classes of RNA polymerase III promoters differentially utilize TBP
183 s) for CRISPR-TFs can only be expressed from RNA polymerase III promoters in human cells, limiting th
184 sized exogenously or can be transcribed from RNA polymerase III promoters in vivo, thus permitting th
185 cifically, MHV68 miRNAs are transcribed from RNA polymerase III promoters located within adjacent vir
187 gene and up to four sgRNAs from independent RNA polymerase III promoters that are incorporated into
189 kbones expressing various RIG-I ligands from RNA polymerase III promoters were screened in a cell cul
190 anscription of both TATA-less and snRNA-type RNA polymerase III promoters, and a factor equally relat
191 tor IIIB (TFIIIB), an activity that binds to RNA polymerase III promoters, generally through protein-
192 with specificities for different classes of RNA polymerase III promoters, have evolved in human cell
199 nced mating loci, and regions transcribed by RNA polymerase III, providing evidence that the enzymati
202 n the transcription of tRNA and 5 S genes by RNA polymerase III, recruitment of the transcription fac
204 ll nuclear RNA (snRNA) gene transcription by RNA polymerase III requires cooperative promoter binding
205 mplex transition.Transcription initiation by RNA polymerase III requires TFIIIB, a complex formed by
206 n U6 small nuclear RNA gene transcription by RNA polymerase III requires the general transcription fa
208 ity of viral microRNAs (miRNAs) derived from RNA polymerase III (RNAP III) transcribed precursors.
210 nd on common host transcripts transcribed by RNA polymerase III (RNAP III), yet how these transcripts
211 RNA) genes (tDNAs), which are transcribed by RNA polymerase III (RNAPIII) and encode RNA molecules re
214 IIIC transcription factor but do not recruit RNA polymerase III, show specific intranuclear positioni
215 an activator of snRNA promoters, and in the RNA polymerase III snRNA promoters, with TATA-binding pr
216 haromyces cerevisiae tau55, a subunit of the RNA polymerase III-specific general transcription factor
220 ereupon it primes the phosphorylation of the RNA polymerase III subunit Rpc53 by a specific GSK-3 fam
222 f a concerted mechanism involving TFIIIB and RNA polymerase III subunits for the closed to open pre-i
224 ition and the properties of E2E RNAs made by RNA polymerase III suggest that the function of this vir
225 ieve recruitment of Saccharomyces cerevisiae RNA polymerase III, TBP is associated with two additiona
226 expressed human vRNA genes even though a new RNA polymerase III termination sequence has evolved betw
227 ' unique end after the A-tail and before the RNA polymerase III terminator, and random mutations foun
228 icipates in two steps of promoter opening by RNA polymerase III that are comparable to the successive
229 of retrotransposons that are transcribed by RNA polymerase III, thus generating exclusively noncodin
230 he bovine leukemia virus, a retrovirus, uses RNA polymerase III to directly transcribe the pre-miRNA
231 the world's experts on RNA polymerase I and RNA polymerase III to highlight and share their latest r
234 s the EBV latent replication origin OriP and RNA polymerase III-transcribed EBV-encoded RNA genes.
235 We show that condensin frequently associates RNA polymerase III-transcribed genes (tRNA and 5S rRNA)
237 se results suggest that different classes of RNA polymerase III-transcribed genes have distinct gener
239 show that RB represses different classes of RNA polymerase III-transcribed genes via distinct mechan
241 on of both RNA polymerase II-transcribed and RNA polymerase III-transcribed snRNA genes and is recogn
242 t ER was associated with a large fraction of RNA polymerase III-transcribed tRNA genes, independent o
243 e majority RNA polymerase II transcript; the RNA polymerase III-transcribed U1 small nuclear RNA has
244 A) is a perfect match to the TATA box of the RNA polymerase III-transcribed U6 small nuclear RNA (SNR
247 icellular organisms beyond the regulation of RNA polymerase III transcription and suggest that Maf1 p
248 The retinoblastoma protein (RB) represses RNA polymerase III transcription effectively both in viv
249 integrase targeted by a synthetic fusion of RNA polymerase III transcription factor IIIB subunits, B
250 the N terminus of Bdp1p, a component of the RNA polymerase III transcription factor TFIIIB, is requi
251 tes map to tRNA and other genes bound by the RNA polymerase III transcription factor TFIIIC, and ribo
252 e key glycolytic enzyme, and La protein, the RNA polymerase III transcription factor, with the cis-ac
253 n physically associates with a subcomplex of RNA polymerase III transcription factors on the tRNA gen
257 We selected the BRF1 gene, which encodes an RNA polymerase III transcription initiation factor subun
258 ts contribute to the definition of the basal RNA polymerase III transcription machinery and show that
261 retinoblastoma (RB) protein represses global RNA polymerase III transcription of genes that encode no
263 itors reduce PNC prevalence in parallel with RNA polymerase III transcription reduction, and a subset
269 In the fruit fly Drosophila melanogaster, RNA polymerase III transcription was found to be depende
270 that tDNAs associate with NPCs to coordinate RNA polymerase III transcription with the nuclear export
272 better understanding of the function of this RNA polymerase III transcription, we have examined the p
279 that components of the RNA polymerase II and RNA polymerase III transcriptional machines compete for
281 protects the UUU(OH) 3' terminii of nascent RNA polymerase III transcripts from exonuclease digestio
283 iated with precursor tRNAs and other nascent RNA polymerase III transcripts while nonphosphorylated (
284 cleolar localization of Misu is dependent on RNA polymerase III transcripts, and knockdown of Misu de
285 functions commonly associated with the core RNA Polymerase III transcripts, but also more diverse ce
292 und on both the RNA polymerase II U1 and the RNA polymerase III U6 promoters as determined by chromat
293 es by either RNA polymerase II (U1 to U5) or RNA polymerase III (U6) is dependent upon a proximal seq
294 NAs by RNA polymerase II (U1, U2, U4, U5) or RNA polymerase III (U6) is dependent upon a unique, posi
295 est that to terminate transcription in vivo, RNA polymerase III uses a mechanism other than hairpin-d
296 t hairpin siRNAs or siRNAs expressed from an RNA polymerase III vector based on the mouse U6 RNA prom
297 ut the period examined, E2E transcription by RNA polymerase III was found to be at least as efficient
298 , ribosome-EF-Tu complex, 20S proteasome and RNA polymerase III, we illustrate how local sharpening c
299 o provides the first nascentome analysis for RNA polymerase III, which indicates that transcription o
300 Bdp1 subunit of the Brf2-TFIIIB complex, and RNA polymerase III, with negative and positive outcomes
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