ライフサイエンスコーパス: RS virus

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します

1 ino acid differences between the two group A RS virus G proteins, these animal studies did not reveal

2 Among the group A RS virus isolates, this G-protein variable region had am

3 Group A RS viruses were dominant the first 2 years, followed by

4 Among the group B RS virus G proteins, amino acid differences were as grea

5 The group A and group B RS viruses demonstrated genetic variability between year

6 prototype strains revealed that the group B RS viruses may vary more extensively than was observed o

7 s readthrough of intergenic junctions during RS virus transcription, thereby acting as a transcriptio

8 The M2-1 protein of human RS virus functions as a transcription factor which incre

9 absence of SH/G polycistronic transcripts in RS virus-infected cells.

10 erent gene junctions of RS virus do modulate RS virus transcription termination.

11 r data, the naturally occurring gene ends of RS virus that terminate inefficiently have short U tract

12 tion efficiencies of each of the 10 genes of RS virus are different.

13 re show that the different gene junctions of RS virus do modulate RS virus transcription termination.

14 ate that the mechanism for the regulation of RS virus gene expression is more complex than was previo

15 ono- and dicistronic subgenomic replicons of RS virus by direct metabolic labeling of RNA in the pres

16 Nine dicistronic subgenomic replicons of RS virus were constructed; each contained one of the RS

17 Precise definition of the role of RS virus antigenic variability in the establishment of r

18 d in all human, bovine, and ovine strains of RS virus.

19 tion of the M2-1 protein in transcription of RS virus subgenomic replicons was assayed.

20 hown to bind monocistronic and polycistronic RS virus mRNAs during infection.

21 ing the M2 protein of respiratory syncytial (RS) virus contains two open reading frames (ORFs).

22 ends of the genes of respiratory syncytial (RS) virus direct polyadenylation and termination of vira

23 The M2 gene of respiratory syncytial (RS) virus has two open reading frames (ORFs).

24 The M2-1 protein of respiratory syncytial (RS) virus is a transcriptional processivity and antiterm

25 e two major groups of respiratory syncytial (RS) virus may contribute to reinfections with these viru

26 rse gene junctions of respiratory syncytial (RS) virus to signal the termination of transcription was

27 Respiratory syncytial (RS) viruses isolated over three epidemic periods in a ch

28 were constructed; each contained one of the RS virus gene junctions in its natural upstream and down

29 ted, and has been shown to interact with the RS virus nucleocapsid (N) protein.

30 gainst intranasal challenge by either of the RS viruses.

31 intra-group variability in reinfections with RS virus has not been defined.

WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。