ライフサイエンスコーパス: Rasmussen

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1 Rasmussen and Svensson correctly point out that there is

2 Rasmussen's encephalitis (RE) is a rare disease of the c

3 Rasmussen's encephalitis (RE), a childhood disease chara

4 Rasmussen's encephalitis is a childhood disease resultin

5 Rasmussen's encephalitis is a rare chronic neurological

6 Rasmussen's encephalitis is a rare progressive pediatric

7 Rasmussen's syndrome is a progressive and intractable fo

8 Seventy-six subjects with a Rasmussen Disease Score (RDS) of 6 or higher were random

9 gton's diseases, schizophrenia, epilepsy and Rasmussen's encephalitis to stroke.

10 to pathological states such as epilepsy and Rasmussen's encephalitis.

11 covered a 50-cm-thick peat deposit near Cape Rasmussen (65.2 degrees S), in the maritime Antarctic.

12 In this issue of Developmental Cell, Rasmussen et al. investigate the morphogenesis of the Ca

13 nsult) and progressive in six children (four Rasmussen encephalitis, two Sturge-Weber syndrome).

14 In this issue of the Journal, Gorst-Rasmussen et al. discuss one such approach.

15 uctural alterations were also found in human Rasmussen encephalitis, a CTL-mediated human autoimmune

16 roglia, suggesting a window for treatment if Rasmussen's encephalitis can be diagnosed early.

17 s seen with MRI might be a good biomarker in Rasmussen's encephalitis.

18 onal glutamate receptor subunit 3 (GluR3) in Rasmussen's encephalitis, a severe form of pediatric epi

19 Astrocyte death may occur in Rasmussen's encephalitis, and destruction of this cell t

20 slow rather than halt disease progression in Rasmussen's encephalitis, without changing the eventual

21 emonstrate that the local immune response in Rasmussen's syndrome includes restricted T cell populati

22 In this issue of the Journal, Rasmussen-Torvik et al. have highlighted this problem by

23 s have been implicated in the development of Rasmussen's encephalitis, a rare neurodegenerative disea

24 tors is implicated in the pathophysiology of Rasmussen's encephalitis.

25 in part determine the nature and severity of Rasmussen's encephalitis and other complement-dependent

26 ies support the sequence comparison study of Rasmussen et al., in which there is a catalytic metal-bi

27 al left hemispherectomy for the treatment of Rasmussen's encephalitis.

28 ter left hemidecorticectomy for treatment of Rasmussen's syndrome.

29 GluR3 autoantibodies are found in some Rasmussen's encephalitis patients, and GluR3 antibodies

30 mmunological studies support the notion that Rasmussen's encephalitis is probably driven by a T-cell

31 Shibani Pati and Todd Rasmussen summarize progress in preclinical research on

32 appearing neocortex obtained from cases with Rasmussen encephalitis, cystic-gliotic encephalopathy, a

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