ライフサイエンスコーパス: RhoA protein

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1 Stattic treatment also caused loss of RhoA protein.

2 ecreases the degradation of existing Ras and RhoA protein.

3 inin (HA)-tagged wild-type or mutant Rac1 or RhoA proteins.

4 d subsequent increases in prenylated Ras and RhoA proteins.

5 Here, we show that RhoA protein accumulates at adherens junctions in the de

6 astatin-dependent increases in RhoB, but not RhoA, protein accumulation.

7 n alginate gel induces a precipitous loss of RhoA protein and a loss of stress fibers concomitant wit

8 RhoA protein and activity were also increased in vessels

9 cells with knockdown of CCM2 have increased RhoA protein and display impaired directed cell migratio

10 r, the ectopic expression of miR-155 reduced RhoA protein and disrupted tight junction formation.

11 logy correlates with a profound induction of RhoA protein and stress fibers.

12 cromass cultures similarly entails a loss of RhoA protein and that expression of dominant negative Rh

13 in neurite growth correlates with increased RhoA protein at the neurite tip, decreased Smurf1 (a pro

14 Therefore, we created RhoA proteins containing phosphomimetic residues in plac

15 Expression of wild-type or mutant RhoA proteins does not alter mAChR-mediated inhibition o

16 ility at least in part by down-regulation of RhoA protein expression and activity through mTORC1-medi

17 Inhibition of RhoA protein expression using small-interfering RNA, how

18 vities and the amounts of prenylated Ras and RhoA proteins in CHO-hIR-WT (but not CHO-DeltaNPEY) cell

19 treatment led to a decrease in the amount of RhoA protein, indicating that the loss of PLD activity i

20 The decreased stability of the RhoA protein is correlated with an increased resistance

21 adenosine diphosphate-ribosylation factor 1, RhoA, protein kinase C (PKC)-beta or PKC-alpha to the pl

22 Since a RhoA/protein kinase C (PKC)-mediated pathway is known to

23 induced phenotypes associated with elevated RhoA protein levels and RhoA/ROCK signaling.

24 elective rescue was dependent upon increased RhoA protein levels in mutant huntingtin-expressing cell

25 , produces a rapid and transient increase in RhoA protein levels.

26 udies with constitutively activated Rac1 and RhoA proteins revealed no negative effects on MyoD-induc

27 the expression of a constitutively activated RhoA protein that disrupts the cell-matrix interaction r

28 ecretion using human cell cultures, Rac1 and RhoA protein variants, and pharmacological inhibitors.

29 RhoA protein was found throughout the retina, including

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