ライフサイエンスコーパス: Rickettsia

1 g sites (VBSs) that are conserved across all Rickettsia.

2 s with respect to the parasitic lifestyle of Rickettsia.

3 ity to clone this obligate intragranulocytic rickettsia.

4 solate genotypically characterized as an SFG rickettsia.

5 ously unidentified spotted-fever-group (SFG) rickettsia.

6 rity, or a direct result of infection by the Rickettsia.

7 a tick-associated bacterium) and a pea-aphid Rickettsia.

8 ith E. chaffeensis and a spotted fever group rickettsia.

9 hogens in the genera Listeria, Shigella, and Rickettsia.

10 esumably facilitates the persistence of this rickettsia.

11 dothelium-targeting intracellular bacterium, Rickettsia.

12 d 6 were infected with a spotted fever group rickettsia; 1 patient had evidence of coinfection with E

13 All four, a Rickettsia, a Spiroplasma and two different strains of W

14 Within the Rickettsia, ABM is confined to members of the spotted fe

15 lies are sufficient to explain the spread of Rickettsia across the southwestern United States.

16 t was closely related to Rickettsia parkeri, Rickettsia africae, and Rickettsia sibirica.

17 initiated studies to characterize macrophage-Rickettsia akari and -Rickettsia typhi interactions and

18 ckettsia rickettsii, Rickettsia parkeri, and Rickettsia akari are the most common causes of spotted f

19 M18dRGA plasmid that originally derives from Rickettsia amblyommatis and encodes the expression of GF

20 Three distinct plasmids were demonstrated in Rickettsia amblyommii AaR/SC by Southern analysis using

21 ra to R. rickettsii, Rickettsia parkeri, and Rickettsia amblyommii antigens.

22 Strains of the tick-borne rickettsia Anaplasma marginale differ markedly in transm

23 The rickettsia Anaplasma marginale is the most prevalent tic

24 variant protein in the outer membrane of the rickettsia Anaplasma marginale.

25 d bacterial intracellular parasites, such as Rickettsia and Chlamydia, to import ATP from the cytosol

26 cally proteobacteria of the genera Brucella, Rickettsia and Coxiella, and corona-, calici- and lyssav

27 ereas, certain microorganisms, including all Rickettsia and other pathogens, use an alternative thyX-

28 he abdomen, described for the first time for Rickettsia and this host order.

29 One matriline was co-infected with Rickettsia and Wolbachia and produced offspring with a r

30 are infected with the endosymbiotic bacteria Rickettsia and Wolbachia.

31 infected with various Ehrlichia, Bartonella, Rickettsia, and Babesia species.

32 in-like cross-reactive enzymes in Chlamydia, Rickettsia, and Xanthomonas.

33 yrin repeat domain-containing protein, named Rickettsia ankyrin repeat protein 1 (RARP-1), and identi

34 racellular parasitic bacteria, Chlamydia and Rickettsia, apparently from plants, and proteases that m

35 hylogenetically distant psocopteran species, Rickettsia are shown to be associated with four transiti

36 athogenic species of the spotted fever group Rickettsia are subjected to repeated exposures to the ho

37 mice to infection with Rickettsia conorii or Rickettsia australis was significantly greater than in w

38 the citrate synthase gene, we compared ELB, Rickettsia australis, R. rickettsii, and R. akari with t

39 Similarly, spleen cells from Rickettsia australis-immune mice exerted specific CTL ac

40 sma, Ehrlichia, Candidatus Neoehrlichia, and Rickettsia bacteria in mosquitoes, comprising nine docum

41 FISH analysis indicated the presence of Rickettsia bacteria in ovary tissue, indicating their ma

42 Also of note was that Ehrlichia and Rickettsia bacteria were detected in each life stage of

43 w biochemical analysis has revealed that the Rickettsia bacterial protein Sca2--recently shown to be

44 und that different strains of a male-killing Rickettsia bacterium infecting the beetle Adalia bipunct

45 We have identified a Rickettsia bacterium that is associated with sex ratio d

46 a novel Rickettsia, most closely related to Rickettsia bellii (a tick-associated bacterium) and a pe

47 ilar to that of the tra genes encoded in the Rickettsia bellii and R. massiliae genomes.

48 e of Sao Paulo, PCR analysis detected DNA of Rickettsia bellii in 16 ticks (40%), and 3 other ticks (

49 Electroporation of Rickettsia parkeri and Rickettsia bellii with pRAM18/Rif/GFPuv yielded GFPuv-ex

50 Rickettsia belong to a family of Gram-negative obligate

51 Considering the movement of SFG Rickettsia between vertebrate and invertebrate hosts, th

52 One variable component of Rickettsia biology involves arthropod vectors: for insta

53 ssical alpha-proteobacteria and closely with Rickettsia but significantly worse with the rickettsial

54 report that the cell surface antigen sca4 of Rickettsia co-localizes with vinculin in cells at sites

55 y to disseminated endothelial infection with Rickettsia conorii (Malish 7 strain), these T cell subse

56 wn that sca2 is transcribed and expressed in Rickettsia conorii and have used a heterologous gain-of-

57 y demonstrated that polyclonal antibodies to Rickettsia conorii and monoclonal antibodies to outer me

58 C3H/HeN mice infected with 3 x 10(5) PFU of Rickettsia conorii developed an acute progressive diseas

59 Spleen cells from mice immune to Rickettsia conorii exerted specific major histocompatibi

60 mounts of interferon (IFN)-gamma on day 1 of Rickettsia conorii infection, which was associated with

61 Here, we describe the transformation of Rickettsia conorii Malish 7 with the plasmid pRam18dRGA[

62 tibility of MyD88(-/-)mice to infection with Rickettsia conorii or Rickettsia australis was significa

63 The 1.2-kb DNA fragment of the Rickettsia conorii outer membrane protein B gene (OmpB(4

64 Rickettsia conorii stimulation of BMDCs caused significa

65 s in the lungs of C3H/HeN mice infected with Rickettsia conorii with the purpose of identifying evide

66 anism of killing of obligately intracellular Rickettsia conorii within human target cells, mainly end

67 ologic agent of Mediterranean spotted fever, Rickettsia conorii, is susceptible to complement-mediate

68 By exploring the molecular pathogenesis of Rickettsia conorii, the agent of Mediterranean spotted f

69 ndothelial cell lining of blood vessels with Rickettsia conorii, the causative agent of Mediterranean

70 Rickettsia conorii-infected BMDCs from resistant mice ha

71 mice following intravenous inoculation with Rickettsia conorii.

72 a potential role of antibody in immunity to Rickettsia conorii.

73 ngll, the Ehrlichia phagocytophila group and Rickettsia conorii.

74 The genus Rickettsia consists of intracellular bacteria that cause

75 The tick-borne rickettsia Cowdria ruminantium has been propagated conti

76 Curiously, spotted fever group Rickettsia differ from other pathogens in possessing two

77 an emerging tick-borne disease caused by the rickettsia Ehrlichia chaffeensis.

78 an emerging tick-borne disease caused by the rickettsia Ehrlichia chaffeensis.

79 ehrlichiosis is caused by a tick-transmitted rickettsia, Ehrlichia chaffeensis.

80 ligate intracellular organisms in the genera Rickettsia, Ehrlichia, and Anaplasma, persists in ticks

81 t the genome of a previously uncharacterized Rickettsia endosymbiont from Culicoides newsteadi (RiCNE

82 We present the draft genome for the Rickettsia endosymbiont of Ixodes scapularis (REIS), a s

83 l metabolism critical for a lineage-specific Rickettsia entry mechanism.

84 mids have been identified in most species of Rickettsia examined, with some species maintaining multi

85 Like many intracellular pathogens, Rickettsia exploit the cytoskeleton to enter and spread

86 ing number of recent reports have implicated Rickettsia felis as a human pathogen, paralleling the in

87 emerged recently (e.g., Bartonella henselae, Rickettsia felis), and their mechanisms of transmission

88 Bioinformatic analyses of several rickettsia genomes revealed the presence of a cohort of

89 that sca1, a gene present in nearly all SFG rickettsia genomes, is actively transcribed and expresse

90 contributes to a limited synteny with other Rickettsia genomes.

91 uch as Coxiella, Buchnera and members of the Rickettsia genus.

92 An emphasis on cellular immunity against Rickettsia has led to neglect of analysis of the role of

93 Rickettsia has therefore "rediscovered" formin-like acti

94 ormatic analysis of genomic DNA sequences of Rickettsia identified putative lysine methyltransferases

95 We detected Rickettsia in all of the parthenogenetic individuals we

96 We found that a robust Rickettsia-induced innate response in resistant mice cle

97 hermore, the secretion levels of IL-1beta by Rickettsia-infected BMDCs and in the sera of infected mi

98 (70%), and Rickettsia spp. in 19 ticks (8%); Rickettsia-infected ticks contained R. rhipicephali (16

99 Compared with uninfected whiteflies, Rickettsia-infected whiteflies produced more offspring,

100 ed significantly increased susceptibility to Rickettsia infection compared with NK cell-sufficient Ra

101 ignaling molecules involved in the uptake of Rickettsia into mammalian and Drosophila cells have been

102 equired to establish whether the presence of Rickettsia is linked to asexual reproduction in Liposcel

103 Scientific analysis of the genus Rickettsia is undergoing a rapid period of change with t

104 parkeri, a member of the spotted fever group Rickettsia, is the causative agent of American boutonneu

105 o the observed patterns of mtDNA variation-a rickettsia-like microorganism, Wolbachia pipientis, whic

106 and provide a paradigm for understanding how Rickettsia-like pathogens are maintained within vectors.

107 and the invasive bacteria within the genera Rickettsia, Listeria, and Shigella.

108 differences between the ABM of Listeria and Rickettsia may indicate fundamental differences in the m

109 In addition, the oxyhemoglobin restored the rickettsia-mediated, rapid killing of the treated RAW264

110 18dRGA (13.3 - 28.1 copies), and R. parkeri, Rickettsia monacensis and Rickettsia montanensis contain

111 nic isolates (Rickettsia rickettsii Hip2 and Rickettsia montana M5/6) with respect to their putative

112 ), and R. parkeri, Rickettsia monacensis and Rickettsia montanensis contained 9.9, 5.5 and 7.5 copies

113 s to determine the tick proteins involved in Rickettsia montanensis infection of tick-derived cells f

114 rate that DvKPI limits host cell invasion by Rickettsia montanensis, possibly through an association

115 lis with the spotted fever group rickettsia, Rickettsia montanensis, results in sustained D. variabil

116 rDNA sequence indicates that this is a novel Rickettsia, most closely related to Rickettsia bellii (a

117 ic formins to assemble actin comet tails for Rickettsia motility.

118 Obligate intracellular bacteria of the genus Rickettsia must adhere to and invade the host endotheliu

119 obacteria, Bordetella, Leishmania, Borrelia, Rickettsia, Neisseria, and Bacillus anthracis.

120 There was no antigenic cross-reactivity with Rickettsia or Ehrlichia spp. or other related organisms.

121 Electroporation of Rickettsia parkeri and Rickettsia bellii with pRAM18/Rif

122 each protein directs an independent mode of Rickettsia parkeri motility at different times during in

123 Here, we show that SFG species Rickettsia parkeri typically lack actin tails during spr

124 Rickettsia parkeri, a member of the spotted fever group

125 Rickettsia parkeri, a recently recognized pathogen of hu

126 Rickettsia rickettsii, Rickettsia parkeri, and Rickettsia akari are the most co

127 ctivity of the paired sera to R. rickettsii, Rickettsia parkeri, and Rickettsia amblyommii antigens.

128 ges suggested that it was closely related to Rickettsia parkeri, Rickettsia africae, and Rickettsia s

129 they suggest important roles for vinculin in Rickettsia pathogenesis.

130 eria will permit forward genetic analysis of Rickettsia pathogenesis.

131 and an emerging human rickettsial pathogen, Rickettsia philipii, in a population of the Pacific Coas

132 Members of the genus Rickettsia possess the ability to invade host cells and

133 ever, tests which indicate the presence of a Rickettsia, previously found to cause male-killing in an

134 hetic peptide, derivative from the H6PGA4 R. rickettsia protein, homologous to OmpA.

135 udomonas putida, Mycobacterium tuberculosis, Rickettsia prowazakii, Legionella pneumophila, Vibrio ch

136 e the genomes of the intracellular parasites Rickettsia prowazekii and Mycobacterium leprae.

137 report the crystal structures of PKMT1 from Rickettsia prowazekii and PKMT2 from Rickettsia typhi, b

138 Typhus group rickettsiae, including Rickettsia prowazekii and R. typhi, produce visible plaq

139 l demonstration of genetic transformation of Rickettsia prowazekii and represents the initial step in

140 cells revealed the typhus group rickettsiae, Rickettsia prowazekii and Rickettsia typhi, to have no a

141 unique insertions in the gene coding for the Rickettsia prowazekii ATP/ADP translocase (Tlc) was gene

142 he determination of membrane topology of the Rickettsia prowazekii ATP/ADP translocase (Tlc).

143 transmembrane regions I, II, and III of the Rickettsia prowazekii ATP/ADP translocase to the structu

144 ribution of transmembrane region VIII of the Rickettsia prowazekii ATP/ADP translocase to the structu

145 We have determined the accessibility of the Rickettsia prowazekii ATP/ADP translocase transmembrane

146 Recent studies have demonstrated that Rickettsia prowazekii can regulate transcription of sele

147 An invasion gene homolog, invA, of Rickettsia prowazekii has recently been identified to en

148 The obligate intracellular bacterium Rickettsia prowazekii has recently been shown to transpo

149 Rickettsia prowazekii is an obligate intracellular patho

150 Rickettsia prowazekii is an obligate intracytosolic path

151 to determine which of the selected genes of Rickettsia prowazekii mediate the escape process.

152 The obligate intracytoplasmic pathogen Rickettsia prowazekii relies on the transport of many es

153 exposed in vitro to trimethyltransferases of Rickettsia prowazekii RP027-028 and of R. typhi RT0101 a

154 Here we have characterized the Rickettsia prowazekii RP534 protein, a homologue of the

155 e (NOS) pathway in inhibiting the ability of Rickettsia prowazekii to initially infect (invade) mouse

156 group (TG) rickettsiae Rickettsia typhi and Rickettsia prowazekii were characterized by chemical ana

157 ensis, Brucella spp., Burkholderia spp., and Rickettsia prowazekii) and tested by RT-PCR-ESI-MS.

158 a (Mycobacterium leprae, Yersinia pestis and Rickettsia prowazekii) show how an organism can undergo

159 an-disease pathogens Treponema palladium and Rickettsia prowazekii, and the extremely radioresistant

160 many with significant similarity to those of Rickettsia prowazekii, genes predicted to encode differe

161 Rickettsia prowazekii, the causative agent of epidemic t

162 he genetic mechanism of membrane assembly in Rickettsia prowazekii, the causative agent of epidemic t

163 Rickettsia prowazekii, the causative agent of epidemic t

164 Rickettsia prowazekii, the causative agent of epidemic t

165 Rickettsia prowazekii, the causative agent of epidemic t

166 Rickettsia prowazekii, the etiologic agent of epidemic t

167 Rickettsia prowazekii, the etiologic agent of epidemic t

168 Rickettsia prowazekii, the etiological agent of epidemic

169 the obligate endosymbiont and human pathogen Rickettsia prowazekii, the plant pathogen Agrobacterium

170 A spotted fever rickettsia quantitative PCR assay (SQ-PCR) was developed

171 The bacterium Rickettsia quintana was consistently found in the gut an

172 The SFG rickettsia, R. rickettsii, displayed long actin tails (>

173 4.3% of EID events are caused by bacteria or rickettsia, reflecting a large number of drug-resistant

174 is intracellular pathogen, and perhaps other Rickettsia-related microbes of medical importance.

175 arctic or Afrotropical species revealed that Rickettsia represent a widespread but previously overloo

176 have discovered that the spotted fever group rickettsia Rickettsia montanensis, a relative of R. rick

177 f D. variabilis with the spotted fever group rickettsia, Rickettsia montanensis, results in sustained

178 The intracellular bacteria Rickettsia, Rickettsiella and Diplorickettsia were repor

179 y related symbionts (in the genera Regiella, Rickettsia, Rickettsiella and Spiroplasma).

180 about the structure and morphogenesis of the Rickettsia rickettsii actin tail relative to Shigella an

181 compared the dynamics and behavior of ABM of Rickettsia rickettsii and Listeria monocytogenes.

182 veloped for the detection and enumeration of Rickettsia rickettsii and other closely related spotted

183 spotted fever and boutonneuse fever, due to Rickettsia rickettsii and R. conorii, respectively, are

184 The type I signal peptidase lepB genes from Rickettsia rickettsii and Rickettsia typhi, the etiologi

185 Bartonella vinsonii, and 22 seroconverted to Rickettsia rickettsii antigens.

186 ed fever group rickettsiae (SFGR) other than Rickettsia rickettsii are responsible for spotted fever

187 nstrated IgG or IgM antibodies reactive with Rickettsia rickettsii at a diagnostic titer (i.e., >/=64

188 gulatory proteins in actin-based motility of Rickettsia rickettsii have not been established.

189 WB-8-2 and two other nonpathogenic isolates (Rickettsia rickettsii Hip2 and Rickettsia montana M5/6)

190 Rickettsia rickettsii infection of endothelial cells is

191 etion of chemokines and prostaglandins after Rickettsia rickettsii infection of human cerebral, derma

192 Rickettsia rickettsii infection results in numerous resp

193 Rickettsia rickettsii is an obligate intracellular patho

194 Rickettsia rickettsii is an obligate intracellular patho

195 In addition, Rickettsia rickettsii was detected by PCR/ESI-MS from fo

196 cies of a mariner-based transposon system in Rickettsia rickettsii were determined using a plaque ass

197 plasma phagocytophilum, Ehrlichia canis, and Rickettsia rickettsii), but the sample was highly positi

198 ponse to certain pathogenic organisms (e.g., Rickettsia rickettsii), data documenting endothelial cel

199 dothelial cells consequent to infection with Rickettsia rickettsii, an obligate intracellular gram-ne

200 me host cell apoptosis was explored by using Rickettsia rickettsii, an obligate intracellular Gram-ne

201 vein endothelial cells during infection with Rickettsia rickettsii, an obligate, intracellular bacter

202 te DNA extracted from Ehrlichia chaffeensis, Rickettsia rickettsii, and Bartonella henselae.

203 infections caused by Ehrlichia chaffeensis, Rickettsia rickettsii, and Coxiella burnetti, no signifi

204 cognized by reference human antisera against Rickettsia rickettsii, Chlamydia group positive, Trepone

205 steria monocytogenes, Shigella flexneri, and Rickettsia rickettsii, exploit the host cytoskeleton by

206 otted fever, a tick-borne zoonosis caused by Rickettsia rickettsii, is among the most lethal of all i

207 used by the obligate intracellular bacterium Rickettsia rickettsii, is associated with widespread inf

208 e HGE agent and to either Coxiella burnetii, Rickettsia rickettsii, or Rickettsia typhi was infrequen

209 Ehrlichia canis, E. chaffeensis, E. ewingii, Rickettsia rickettsii, R. conorii, and other spotted fev

210 Rickettsia rickettsii, Rickettsia parkeri, and Rickettsi

211 rs of the spotted fever group (SFG), such as Rickettsia rickettsii, the agent of Rocky Mountain spott

212 Rickettsia rickettsii, the causative agent of Rocky Moun

213 nse during infection of endothelial cells by Rickettsia rickettsii, the causative agent of Rocky Moun

214 k Amblyomma cooperi in the enzootic cycle of Rickettsia rickettsii, the etiologic agent of Brazilian

215 potentially fatal human infection caused by Rickettsia rickettsii, the etiologic agent of Rocky Moun

216 l (EC) is a primary target of infection with Rickettsia rickettsii, the etiologic agent of Rocky Moun

217 Rickettsia rickettsii, the etiologic agent of Rocky Moun

218 mutant pairs from two independent strains of Rickettsia rickettsii, the virulent R strain and the avi

219 Here, we investigate whether Rickettsia rickettsii-infected host endothelial cells re

220 fe-threatening, tick-borne disease caused by Rickettsia rickettsii.

221 umbilical vein endothelial cells (HUVEC) by Rickettsia rickettsii.

222 om those of several other agents, especially Rickettsia rickettsii.

223 ), Ehrlichia canis, Bartonella henselae, and Rickettsia rickettsii.

224 ct human umbilical vein after infection with Rickettsia rickettsii.

225 d Rickettsia spp.; the other is specific for Rickettsia rickettsii.

226 tgun sequencing and annotating the genome of Rickettsia sibirica strain 246, an obligate intracellula

227 uiring North Asian tick typhus (infection by Rickettsia sibirica) during travel to regions of Asia wh

228 Rickettsia parkeri, Rickettsia africae, and Rickettsia sibirica.

229 paired host resistance of CB-17 scid mice to Rickettsia, similar to what was observed in Rag(-/-)gamm

230 Here, we show that Rickettsia sp. nr. bellii swept into a population of an

231 Among Rickettsia species (Alphaproteobacteria: Rickettsiales),

232 ncluded them in a phylogenetic analysis of 9 Rickettsia species and 10 Rickettsia strains.

233 Both E. canis and an uncharacterized Rickettsia species appeared to result in chronic or recu

234 Tick-borne spotted fever group (SFG) Rickettsia species are obligate intracellular bacteria c

235 Rickettsia species are obligate intracellular bacteria w

236 Importantly, diverse Rickettsia species are predicted to utilize divergent me

237 Pathogenic Rickettsia species cause high morbidity and mortality, e

238 -time PCR assay for the detection of these 3 Rickettsia species from formalin-fixed, paraffin-embedde

239 ver (BSF), this study evaluated infection by Rickettsia species in A. cooperi ticks collected from an

240 a1 sequences from geographically diverse SFG Rickettsia species showed that there are high degrees of

241 Lactobacillus, Pseudomonas, and Rickettsia species were significantly enriched in sample

242 hia equi, 9 with Ehrlichia platys, 20 with a Rickettsia species, 16 with a Bartonella species, and 7

243 various Anaplasma, Borrelia, Erhlichia, and Rickettsia species, as well as on Bartonella henselae an

244 he pathogenesis of spotted fever group (SFG) Rickettsia species, including R. conorii and R. ricketts

245 include descriptions of novel Ehrlichia and Rickettsia species, recognition of the occurrence and cl

246 entry are differentially encoded in diverse Rickettsia species.

247 nown about their prevalence and functions in Rickettsia species.

248 as other closely related spotted fever group Rickettsia species.

249 suis) and O tsutsugamushi, Rickettsia typhi/Rickettsia spp, and Leptospira spp infections in blood o

250 236/876 [27%]), with 18% (13/71) for R typhi/Rickettsia spp, O tsutsugamushi, and Leptospira spp comb

251 Our data suggest that R typhi/Rickettsia spp, O tsutsugamushi, and Leptospira spp infe

252 ions were caused by O tsutsugamushi, R typhi/Rickettsia spp, or Leptospira spp.

253 R assays were developed for the detection of Rickettsia spp.

254 ing that a mechanism for plasmid transfer in Rickettsia spp. has yet to be proposed.

255 cisella endosymbiont in 174 ticks (70%), and Rickettsia spp. in 19 ticks (8%); Rickettsia-infected ti

256 ognized pathogen of human, is one of several Rickettsia spp. in the United States that causes a spott

257 ed in the 190-kDa surface antigen protein of Rickettsia spp., as well as one of the major capsid glyc

258 la spp., Coxiella burnetii, Leptospira spp., Rickettsia spp., Salmonella enterica and Salmonella ente

259 One assay detects all tested Rickettsia spp.; the other is specific for Rickettsia ri

260 Removal of the Rickettsia stops egg production and reproduction in the

261 ably maintaining a polymorphism of different Rickettsia strains in this species.

262 etic analysis of 9 Rickettsia species and 10 Rickettsia strains.

263 A defining facet of tick-Rickettsia symbioses is the molecular strategy employed

264 Human infection with Candidatus Rickettsia tarasevichiae (CRT) was first reported in nor

265 We have found a mycetomic Rickettsia that is a strict obligatory symbiont of the p

266 an outer membrane protein present in the R. rickettsia, the etiological agent of spotted fever, able

267 Recent studies aimed at elucidating the rickettsia-tick interaction have discovered that the spo

268 xploit two actin assembly pathways may allow Rickettsia to establish an intracellular niche and sprea

269 ribosylation factors (Arfs), is critical for Rickettsia typhi (typhus group rickettsiae) entry but ps

270 xiella burnetti, Francisella tularensis, and Rickettsia typhi also reacted with our recombinant Barto

271 We observed 32 differences between Rickettsia typhi and ELB.

272 isolated from typhus group (TG) rickettsiae Rickettsia typhi and Rickettsia prowazekii were characte

273 Here, we report that the Rickettsia typhi genome possesses two genes encoding pat

274 haracterize macrophage-Rickettsia akari and -Rickettsia typhi interactions and to determine how ricke

275 Rickettsia typhi is the causative agent of endemic typhu

276 of (i) recombinantly expressed fragments of Rickettsia typhi OmpB exposed in vitro to trimethyltrans

277 topes were present on both R. prowazekii and Rickettsia typhi SPAs.

278 Coxiella burnetii, Rickettsia rickettsii, or Rickettsia typhi was infrequent; however, 74 (52%) of th

279 UV) as a fluorometric marker and transformed Rickettsia typhi with an rpoB-GFPUV fusion construct.

280 RT0522 is the first protein identified from Rickettsia typhi with functional phospholipase A activit

281 plague (Yersinia pestis) and murine typhus (Rickettsia typhi) caused significant numbers of human ca

282 tia tsutsugamushi), murine typhus (caused by Rickettsia typhi), and leptospirosis are common causes o

283 henselae, 3.5%; to Seoul virus, 0.5%; and to Rickettsia typhi, 0.0%.

284 T1 from Rickettsia prowazekii and PKMT2 from Rickettsia typhi, both the apo form and in complex with

285 Rickettsia typhi, the causative agent of murine (endemic

286 Rickettsia typhi, the causative agent of murine typhus,

287 Rickettsia typhi, the causative agent of murine typhus,

288 For Rickettsia typhi, the etiologic agent of murine typhus,

289 gate, intracellular, Gram-negative bacterium Rickettsia typhi, the etiologic agent of murine typhus,

290 se lepB genes from Rickettsia rickettsii and Rickettsia typhi, the etiologic agents of Rocky Mountain

291 For flea-borne Rickettsia typhi, the etiological agent of murine typhus

292 group rickettsiae, Rickettsia prowazekii and Rickettsia typhi, to have no actin tails and short (appr

293 n processing in gram-negative bacteria, from Rickettsia typhi.

294 in the Breinl strain of R. prowazekii and in Rickettsia typhi.

295 pression of 120- and 17-kDa antigen genes in Rickettsia typhi.

296 lus influenzae, S suis) and O tsutsugamushi, Rickettsia typhi/Rickettsia spp, and Leptospira spp infe

297 terestingly, the endosymbionts Wolbachia and Rickettsia were detected only in Henan, while the Ricket

298 proteins, we examined Anaplasma marginale, a rickettsia with two highly immunogenic outer membrane pr

299 Sequence typing clusters the Rickettsia within the Torix group of the genus, a group

300 hia induced only mild CI when mated with the Rickettsia-Wolbachia females.