ライフサイエンスコーパス: Rickettsia

1 dothelium-targeting intracellular bacterium, Rickettsia.

2 g sites (VBSs) that are conserved across all Rickettsia.

3 s with respect to the parasitic lifestyle of Rickettsia.

4 ity to clone this obligate intragranulocytic rickettsia.

5 solate genotypically characterized as an SFG rickettsia.

6 ously unidentified spotted-fever-group (SFG) rickettsia.

7 rity, or a direct result of infection by the Rickettsia.

8 a tick-associated bacterium) and a pea-aphid Rickettsia.

9 esumably facilitates the persistence of this rickettsia.

10 The Rickettsia ~1800-amino-acid autotransporter protein surf

11 infection due to the intracellular pathogen Rickettsia 364D (also known by the proposed name "Ricket

12 ssay for the detection and identification of Rickettsia 364D suitable for ecological and diagnostic a

13 This work describes the development of a Rickettsia 364D-specific TaqMan assay to simplify and ac

14 per 4-mul sample and is highly specific for Rickettsia 364D.

15 bitats had significantly lower prevalence of Rickettsia (8.7% and 4.6% for BUB and UBB sites, respect

16 All four, a Rickettsia, a Spiroplasma and two different strains of W

17 Within the Rickettsia, ABM is confined to members of the spotted fe

18 lies are sufficient to explain the spread of Rickettsia across the southwestern United States.

19 t was closely related to Rickettsia parkeri, Rickettsia africae, and Rickettsia sibirica.

20 initiated studies to characterize macrophage-Rickettsia akari and -Rickettsia typhi interactions and

21 ckettsia rickettsii, Rickettsia parkeri, and Rickettsia akari are the most common causes of spotted f

22 M18dRGA plasmid that originally derives from Rickettsia amblyommatis and encodes the expression of GF

23 Three distinct plasmids were demonstrated in Rickettsia amblyommii AaR/SC by Southern analysis using

24 ra to R. rickettsii, Rickettsia parkeri, and Rickettsia amblyommii antigens.

25 Strains of the tick-borne rickettsia Anaplasma marginale differ markedly in transm

26 The rickettsia Anaplasma marginale is the most prevalent tic

27 variant protein in the outer membrane of the rickettsia Anaplasma marginale.

28 many new secondary endosymbiotic strains of Rickettsia and Arsenophonus were found, increasing the k

29 d bacterial intracellular parasites, such as Rickettsia and Chlamydia, to import ATP from the cytosol

30 cally proteobacteria of the genera Brucella, Rickettsia and Coxiella, and corona-, calici- and lyssav

31 ereas, certain microorganisms, including all Rickettsia and other pathogens, use an alternative thyX-

32 he abdomen, described for the first time for Rickettsia and this host order.

33 One matriline was co-infected with Rickettsia and Wolbachia and produced offspring with a r

34 are infected with the endosymbiotic bacteria Rickettsia and Wolbachia.

35 in-like cross-reactive enzymes in Chlamydia, Rickettsia, and Xanthomonas.

36 ly greater numbers of R. parkeri than of "Ca Rickettsia andeanae" rickettsiae were present in tick sa

37 ected with Rickettsia parkeri or "Candidatus Rickettsia andeanae," a quantitative PCR (qPCR) assay wa

38 identified localized R. parkeri, but not "Ca Rickettsia andeanae," in the vertebrate host.

39 d to R. parkeri than in those exposed to "Ca Rickettsia andeanae." The specific factors that contribu

40 yrin repeat domain-containing protein, named Rickettsia ankyrin repeat protein 1 (RARP-1), and identi

41 racellular parasitic bacteria, Chlamydia and Rickettsia, apparently from plants, and proteases that m

42 Rickettsia are obligate intracellular bacteria that evad

43 hylogenetically distant psocopteran species, Rickettsia are shown to be associated with four transiti

44 athogenic species of the spotted fever group Rickettsia are subjected to repeated exposures to the ho

45 rarer flea genotype had an 83% incidence of Rickettsia asembonensis, a recently described bacterium

46 We previously showed that Rickettsia australis activates ASC inflammasome in macro

47 Rickettsia australis induced significantly increased lev

48 e, we demonstrated that the concentration of Rickettsia australis was significantly greater in infect

49 mice to infection with Rickettsia conorii or Rickettsia australis was significantly greater than in w

50 Similarly, spleen cells from Rickettsia australis-immune mice exerted specific CTL ac

51 sma, Ehrlichia, Candidatus Neoehrlichia, and Rickettsia bacteria in mosquitoes, comprising nine docum

52 FISH analysis indicated the presence of Rickettsia bacteria in ovary tissue, indicating their ma

53 Also of note was that Ehrlichia and Rickettsia bacteria were detected in each life stage of

54 w biochemical analysis has revealed that the Rickettsia bacterial protein Sca2--recently shown to be

55 und that different strains of a male-killing Rickettsia bacterium infecting the beetle Adalia bipunct

56 We have identified a Rickettsia bacterium that is associated with sex ratio d

57 a novel Rickettsia, most closely related to Rickettsia bellii (a tick-associated bacterium) and a pe

58 ilar to that of the tra genes encoded in the Rickettsia bellii and R. massiliae genomes.

59 e of Sao Paulo, PCR analysis detected DNA of Rickettsia bellii in 16 ticks (40%), and 3 other ticks (

60 Electroporation of Rickettsia parkeri and Rickettsia bellii with pRAM18/Rif/GFPuv yielded GFPuv-ex

61 Rickettsia belong to a family of Gram-negative obligate

62 Considering the movement of SFG Rickettsia between vertebrate and invertebrate hosts, th

63 One variable component of Rickettsia biology involves arthropod vectors: for insta

64 ssical alpha-proteobacteria and closely with Rickettsia but significantly worse with the rickettsial

65 report that the cell surface antigen sca4 of Rickettsia co-localizes with vinculin in cells at sites

66 wn that sca2 is transcribed and expressed in Rickettsia conorii and have used a heterologous gain-of-

67 y demonstrated that polyclonal antibodies to Rickettsia conorii and monoclonal antibodies to outer me

68 C3H/HeN mice infected with 3 x 10(5) PFU of Rickettsia conorii developed an acute progressive diseas

69 Spleen cells from mice immune to Rickettsia conorii exerted specific major histocompatibi

70 mounts of interferon (IFN)-gamma on day 1 of Rickettsia conorii infection, which was associated with

71 Rickettsia conorii infects vascular endothelial cells pr

72 Here, we describe the transformation of Rickettsia conorii Malish 7 with the plasmid pRam18dRGA[

73 tibility of MyD88(-/-)mice to infection with Rickettsia conorii or Rickettsia australis was significa

74 The 1.2-kb DNA fragment of the Rickettsia conorii outer membrane protein B gene (OmpB(4

75 Rickettsia conorii stimulation of BMDCs caused significa

76 s in the lungs of C3H/HeN mice infected with Rickettsia conorii with the purpose of identifying evide

77 anism of killing of obligately intracellular Rickettsia conorii within human target cells, mainly end

78 ologic agent of Mediterranean spotted fever, Rickettsia conorii, is susceptible to complement-mediate

79 a transposon for insertional mutagenesis of Rickettsia conorii, isolating variants defective for rep

80 By exploring the molecular pathogenesis of Rickettsia conorii, the agent of Mediterranean spotted f

81 ndothelial cell lining of blood vessels with Rickettsia conorii, the causative agent of Mediterranean

82 Rickettsia conorii-infected BMDCs from resistant mice ha

83 mice following intravenous inoculation with Rickettsia conorii.

84 a potential role of antibody in immunity to Rickettsia conorii.

85 ngll, the Ehrlichia phagocytophila group and Rickettsia conorii.

86 ction produced by the alpha-proteobacterium, Rickettsia conorii.

87 The genus Rickettsia consists of intracellular bacteria that cause

88 LC3 with a small portion of R. australis and Rickettsia-containing double-membrane-bound vacuoles in

89 The tick-borne rickettsia Cowdria ruminantium has been propagated conti

90 Curiously, spotted fever group Rickettsia differ from other pathogens in possessing two

91 This new Rickettsia distributes throughout the body of its whitef

92 an emerging tick-borne disease caused by the rickettsia Ehrlichia chaffeensis.

93 ehrlichiosis is caused by a tick-transmitted rickettsia, Ehrlichia chaffeensis.

94 ligate intracellular organisms in the genera Rickettsia, Ehrlichia, and Anaplasma, persists in ticks

95 pathogens were Borrelia, Babesia, Anaplasma, Rickettsia, Ehrlichia, Bartonella, Francisella, Powassan

96 independent, phylogenetically ancient Torix Rickettsia endosymbiont found constantly in a laboratory

97 t the genome of a previously uncharacterized Rickettsia endosymbiont from Culicoides newsteadi (RiCNE

98 We present the draft genome for the Rickettsia endosymbiont of Ixodes scapularis (REIS), a s

99 l metabolism critical for a lineage-specific Rickettsia entry mechanism.

100 mids have been identified in most species of Rickettsia examined, with some species maintaining multi

101 Like many intracellular pathogens, Rickettsia exploit the cytoskeleton to enter and spread

102 ing number of recent reports have implicated Rickettsia felis as a human pathogen, paralleling the in

103 emerged recently (e.g., Bartonella henselae, Rickettsia felis), and their mechanisms of transmission

104 Bioinformatic analyses of several rickettsia genomes revealed the presence of a cohort of

105 that sca1, a gene present in nearly all SFG rickettsia genomes, is actively transcribed and expresse

106 contributes to a limited synteny with other Rickettsia genomes.

107 uch as Coxiella, Buchnera and members of the Rickettsia genus.

108 An emphasis on cellular immunity against Rickettsia has led to neglect of analysis of the role of

109 Rickettsia has therefore "rediscovered" formin-like acti

110 ormatic analysis of genomic DNA sequences of Rickettsia identified putative lysine methyltransferases

111 We detected Rickettsia in all of the parthenogenetic individuals we

112 We found that a robust Rickettsia-induced innate response in resistant mice cle

113 hermore, the secretion levels of IL-1beta by Rickettsia-infected BMDCs and in the sera of infected mi

114 (70%), and Rickettsia spp. in 19 ticks (8%); Rickettsia-infected ticks contained R. rhipicephali (16

115 Compared with uninfected whiteflies, Rickettsia-infected whiteflies produced more offspring,

116 ed significantly increased susceptibility to Rickettsia infection compared with NK cell-sufficient Ra

117 ignaling molecules involved in the uptake of Rickettsia into mammalian and Drosophila cells have been

118 The bacterium Rickettsia is found widely in phytophagous insects and o

119 equired to establish whether the presence of Rickettsia is linked to asexual reproduction in Liposcel

120 Scientific analysis of the genus Rickettsia is undergoing a rapid period of change with t

121 parkeri, a member of the spotted fever group Rickettsia, is the causative agent of American boutonneu

122 o the observed patterns of mtDNA variation-a rickettsia-like microorganism, Wolbachia pipientis, whic

123 and provide a paradigm for understanding how Rickettsia-like pathogens are maintained within vectors.

124 and the invasive bacteria within the genera Rickettsia, Listeria, and Shigella.

125 18dRGA (13.3 - 28.1 copies), and R. parkeri, Rickettsia monacensis and Rickettsia montanensis contain

126 ), and R. parkeri, Rickettsia monacensis and Rickettsia montanensis contained 9.9, 5.5 and 7.5 copies

127 s to determine the tick proteins involved in Rickettsia montanensis infection of tick-derived cells f

128 rate that DvKPI limits host cell invasion by Rickettsia montanensis, possibly through an association

129 lis with the spotted fever group rickettsia, Rickettsia montanensis, results in sustained D. variabil

130 rDNA sequence indicates that this is a novel Rickettsia, most closely related to Rickettsia bellii (a

131 ic formins to assemble actin comet tails for Rickettsia motility.

132 Obligate intracellular bacteria of the genus Rickettsia must adhere to and invade the host endotheliu

133 obacteria, Bordetella, Leishmania, Borrelia, Rickettsia, Neisseria, and Bacillus anthracis.

134 Electroporation of Rickettsia parkeri and Rickettsia bellii with pRAM18/Rif

135 Here, we report that the human pathogen Rickettsia parkeri is sensitive to IFN-I and benefits fr

136 each protein directs an independent mode of Rickettsia parkeri motility at different times during in

137 ng Amblyomma maculatum cohorts infected with Rickettsia parkeri or "Candidatus Rickettsia andeanae,"

138 Here, we show that SFG species Rickettsia parkeri typically lack actin tails during spr

139 Rickettsia parkeri, a member of the spotted fever group

140 Rickettsia parkeri, a recently recognized pathogen of hu

141 Rickettsia rickettsii, Rickettsia parkeri, and Rickettsia akari are the most co

142 ctivity of the paired sera to R. rickettsii, Rickettsia parkeri, and Rickettsia amblyommii antigens.

143 ges suggested that it was closely related to Rickettsia parkeri, Rickettsia africae, and Rickettsia s

144 block autophagy recognition of the pathogen Rickettsia parkeri.

145 they suggest important roles for vinculin in Rickettsia pathogenesis.

146 eria will permit forward genetic analysis of Rickettsia pathogenesis.

147 ttsia 364D (also known by the proposed name "Rickettsia philipii").

148 and an emerging human rickettsial pathogen, Rickettsia philipii, in a population of the Pacific Coas

149 Members of the genus Rickettsia possess the ability to invade host cells and

150 hetic peptide, derivative from the H6PGA4 R. rickettsia protein, homologous to OmpA.

151 udomonas putida, Mycobacterium tuberculosis, Rickettsia prowazakii, Legionella pneumophila, Vibrio ch

152 e the genomes of the intracellular parasites Rickettsia prowazekii and Mycobacterium leprae.

153 report the crystal structures of PKMT1 from Rickettsia prowazekii and PKMT2 from Rickettsia typhi, b

154 cells revealed the typhus group rickettsiae, Rickettsia prowazekii and Rickettsia typhi, to have no a

155 unique insertions in the gene coding for the Rickettsia prowazekii ATP/ADP translocase (Tlc) was gene

156 he determination of membrane topology of the Rickettsia prowazekii ATP/ADP translocase (Tlc).

157 transmembrane regions I, II, and III of the Rickettsia prowazekii ATP/ADP translocase to the structu

158 ribution of transmembrane region VIII of the Rickettsia prowazekii ATP/ADP translocase to the structu

159 We have determined the accessibility of the Rickettsia prowazekii ATP/ADP translocase transmembrane

160 An invasion gene homolog, invA, of Rickettsia prowazekii has recently been identified to en

161 The obligate intracellular bacterium Rickettsia prowazekii has recently been shown to transpo

162 Rickettsia prowazekii is an obligate intracellular patho

163 Rickettsia prowazekii is an obligate intracytosolic path

164 to determine which of the selected genes of Rickettsia prowazekii mediate the escape process.

165 The obligate intracytoplasmic pathogen Rickettsia prowazekii relies on the transport of many es

166 exposed in vitro to trimethyltransferases of Rickettsia prowazekii RP027-028 and of R. typhi RT0101 a

167 Here we have characterized the Rickettsia prowazekii RP534 protein, a homologue of the

168 ensis, Brucella spp., Burkholderia spp., and Rickettsia prowazekii) and tested by RT-PCR-ESI-MS.

169 a (Mycobacterium leprae, Yersinia pestis and Rickettsia prowazekii) show how an organism can undergo

170 an-disease pathogens Treponema palladium and Rickettsia prowazekii, and the extremely radioresistant

171 many with significant similarity to those of Rickettsia prowazekii, genes predicted to encode differe

172 Rickettsia prowazekii, the causative agent of epidemic t

173 he genetic mechanism of membrane assembly in Rickettsia prowazekii, the causative agent of epidemic t

174 Rickettsia prowazekii, the causative agent of epidemic t

175 Rickettsia prowazekii, the causative agent of epidemic t

176 Rickettsia prowazekii, the etiologic agent of epidemic t

177 Rickettsia prowazekii, the etiologic agent of epidemic t

178 the obligate endosymbiont and human pathogen Rickettsia prowazekii, the plant pathogen Agrobacterium

179 A spotted fever rickettsia quantitative PCR assay (SQ-PCR) was developed

180 The bacterium Rickettsia quintana was consistently found in the gut an

181 The SFG rickettsia, R. rickettsii, displayed long actin tails (>

182 4.3% of EID events are caused by bacteria or rickettsia, reflecting a large number of drug-resistant

183 is intracellular pathogen, and perhaps other Rickettsia-related microbes of medical importance.

184 arctic or Afrotropical species revealed that Rickettsia represent a widespread but previously overloo

185 have discovered that the spotted fever group rickettsia Rickettsia montanensis, a relative of R. rick

186 f D. variabilis with the spotted fever group rickettsia, Rickettsia montanensis, results in sustained

187 The intracellular bacteria Rickettsia, Rickettsiella and Diplorickettsia were repor

188 y related symbionts (in the genera Regiella, Rickettsia, Rickettsiella and Spiroplasma).

189 about the structure and morphogenesis of the Rickettsia rickettsii actin tail relative to Shigella an

190 veloped for the detection and enumeration of Rickettsia rickettsii and other closely related spotted

191 spotted fever and boutonneuse fever, due to Rickettsia rickettsii and R. conorii, respectively, are

192 The type I signal peptidase lepB genes from Rickettsia rickettsii and Rickettsia typhi, the etiologi

193 ed fever group rickettsiae (SFGR) other than Rickettsia rickettsii are responsible for spotted fever

194 nstrated IgG or IgM antibodies reactive with Rickettsia rickettsii at a diagnostic titer (i.e., >/=64

195 gulatory proteins in actin-based motility of Rickettsia rickettsii have not been established.

196 es from 8 Brucella canis IFA-positive and 10 Rickettsia rickettsii IFA-positive dogs.

197 etion of chemokines and prostaglandins after Rickettsia rickettsii infection of human cerebral, derma

198 Rickettsia rickettsii is an obligate intracellular patho

199 m, Borrelia miyamotoi, Borrelia mayonii, and Rickettsia rickettsii The sensitivity for identification

200 In addition, Rickettsia rickettsii was detected by PCR/ESI-MS from fo

201 cies of a mariner-based transposon system in Rickettsia rickettsii were determined using a plaque ass

202 plasma phagocytophilum, Ehrlichia canis, and Rickettsia rickettsii), but the sample was highly positi

203 ponse to certain pathogenic organisms (e.g., Rickettsia rickettsii), data documenting endothelial cel

204 te DNA extracted from Ehrlichia chaffeensis, Rickettsia rickettsii, and Bartonella henselae.

205 hia chaffeensis, Orientia tsutsugamushi, and Rickettsia rickettsii, etc.

206 steria monocytogenes, Shigella flexneri, and Rickettsia rickettsii, exploit the host cytoskeleton by

207 otted fever, a tick-borne zoonosis caused by Rickettsia rickettsii, is among the most lethal of all i

208 used by the obligate intracellular bacterium Rickettsia rickettsii, is associated with widespread inf

209 The causative agent of this disease, Rickettsia rickettsii, is transmitted by several species

210 e HGE agent and to either Coxiella burnetii, Rickettsia rickettsii, or Rickettsia typhi was infrequen

211 Ehrlichia canis, E. chaffeensis, E. ewingii, Rickettsia rickettsii, R. conorii, and other spotted fev

212 Rickettsia rickettsii, Rickettsia parkeri, and Rickettsi

213 rs of the spotted fever group (SFG), such as Rickettsia rickettsii, the agent of Rocky Mountain spott

214 Rickettsia rickettsii, the causative agent of Rocky Moun

215 nse during infection of endothelial cells by Rickettsia rickettsii, the causative agent of Rocky Moun

216 k Amblyomma cooperi in the enzootic cycle of Rickettsia rickettsii, the etiologic agent of Brazilian

217 potentially fatal human infection caused by Rickettsia rickettsii, the etiologic agent of Rocky Moun

218 mutant pairs from two independent strains of Rickettsia rickettsii, the virulent R strain and the avi

219 Here, we investigate whether Rickettsia rickettsii-infected host endothelial cells re

220 d Rickettsia spp.; the other is specific for Rickettsia rickettsii.

221 unoglobulin G (IgG) antibodies reactive with Rickettsia rickettsii.

222 fe-threatening, tick-borne disease caused by Rickettsia rickettsii.

223 tgun sequencing and annotating the genome of Rickettsia sibirica strain 246, an obligate intracellula

224 uiring North Asian tick typhus (infection by Rickettsia sibirica) during travel to regions of Asia wh

225 Rickettsia parkeri, Rickettsia africae, and Rickettsia sibirica.

226 paired host resistance of CB-17 scid mice to Rickettsia, similar to what was observed in Rag(-/-)gamm

227 Here, we show that Rickettsia sp. nr. bellii swept into a population of an

228 Among Rickettsia species (Alphaproteobacteria: Rickettsiales),

229 Tick-borne spotted fever group (SFG) Rickettsia species are obligate intracellular bacteria c

230 Rickettsia species are obligate intracellular bacteria w

231 Importantly, diverse Rickettsia species are predicted to utilize divergent me

232 Pathogenic Rickettsia species cause high morbidity and mortality, e

233 -time PCR assay for the detection of these 3 Rickettsia species from formalin-fixed, paraffin-embedde

234 i_MEAM1 found earlier in whiteflies, the new Rickettsia species has more gene families and pathways,

235 ver (BSF), this study evaluated infection by Rickettsia species in A. cooperi ticks collected from an

236 en a more virulent spotted fever group (SFG) Rickettsia species is transmitted at higher levels durin

237 lity of both rickettsial pathogens and novel Rickettsia species or strains with unknown pathogenicity

238 a1 sequences from geographically diverse SFG Rickettsia species showed that there are high degrees of

239 Lactobacillus, Pseudomonas, and Rickettsia species were significantly enriched in sample

240 hia equi, 9 with Ehrlichia platys, 20 with a Rickettsia species, 16 with a Bartonella species, and 7

241 various Anaplasma, Borrelia, Erhlichia, and Rickettsia species, as well as on Bartonella henselae an

242 he pathogenesis of spotted fever group (SFG) Rickettsia species, including R. conorii and R. ricketts

243 ate cytosolic bacterial pathogens, including Rickettsia species, interact with innate immunity.

244 include descriptions of novel Ehrlichia and Rickettsia species, recognition of the occurrence and cl

245 as other closely related spotted fever group Rickettsia species.

246 entry are differentially encoded in diverse Rickettsia species.

247 nown about their prevalence and functions in Rickettsia species.

248 suis) and O tsutsugamushi, Rickettsia typhi/Rickettsia spp, and Leptospira spp infections in blood o

249 236/876 [27%]), with 18% (13/71) for R typhi/Rickettsia spp, O tsutsugamushi, and Leptospira spp comb

250 Our data suggest that R typhi/Rickettsia spp, O tsutsugamushi, and Leptospira spp infe

251 ions were caused by O tsutsugamushi, R typhi/Rickettsia spp, or Leptospira spp.

252 R assays were developed for the detection of Rickettsia spp.

253 ing that a mechanism for plasmid transfer in Rickettsia spp. has yet to be proposed.

254 cisella endosymbiont in 174 ticks (70%), and Rickettsia spp. in 19 ticks (8%); Rickettsia-infected ti

255 ognized pathogen of human, is one of several Rickettsia spp. in the United States that causes a spott

256 positive result for a concurrent Orientia or Rickettsia spp. quantitative PCR, and the use of antibio

257 Borrelia burgdorferi sensu lato and Rickettsia spp. were detected in one patient each.

258 rrelia miyamotoi, Anaplasma phagocytophilum, Rickettsia spp., Candidatus Neoehrlichia mikurensis and

259 r variabilis) were tested for Borrelia spp., Rickettsia spp., Ehrlichia spp., and Anaplasma phagocyto

260 la spp., Coxiella burnetii, Leptospira spp., Rickettsia spp., Salmonella enterica and Salmonella ente

261 One assay detects all tested Rickettsia spp.; the other is specific for Rickettsia ri

262 Removal of the Rickettsia stops egg production and reproduction in the

263 ably maintaining a polymorphism of different Rickettsia strains in this species.

264 A defining facet of tick-Rickettsia symbioses is the molecular strategy employed

265 Human infection with Candidatus Rickettsia tarasevichiae (CRT) was first reported in nor

266 We have found a mycetomic Rickettsia that is a strict obligatory symbiont of the p

267 an outer membrane protein present in the R. rickettsia, the etiological agent of spotted fever, able

268 Recent studies aimed at elucidating the rickettsia-tick interaction have discovered that the spo

269 xploit two actin assembly pathways may allow Rickettsia to establish an intracellular niche and sprea

270 ribosylation factors (Arfs), is critical for Rickettsia typhi (typhus group rickettsiae) entry but ps

271 xiella burnetti, Francisella tularensis, and Rickettsia typhi also reacted with our recombinant Barto

272 Here, we report that the Rickettsia typhi genome possesses two genes encoding pat

273 haracterize macrophage-Rickettsia akari and -Rickettsia typhi interactions and to determine how ricke

274 Rickettsia typhi is the causative agent of endemic typhu

275 d with successful Orientia tsutsugamushi and Rickettsia typhi isolations from this laboratory over a

276 of (i) recombinantly expressed fragments of Rickettsia typhi OmpB exposed in vitro to trimethyltrans

277 Coxiella burnetii, Rickettsia rickettsii, or Rickettsia typhi was infrequent; however, 74 (52%) of th

278 UV) as a fluorometric marker and transformed Rickettsia typhi with an rpoB-GFPUV fusion construct.

279 RT0522 is the first protein identified from Rickettsia typhi with functional phospholipase A activit

280 plague (Yersinia pestis) and murine typhus (Rickettsia typhi) caused significant numbers of human ca

281 (Orientia tsutsugamushi) and murine typhus (Rickettsia typhi) research to provide an evidence base f

282 tia tsutsugamushi), murine typhus (caused by Rickettsia typhi), and leptospirosis are common causes o

283 henselae, 3.5%; to Seoul virus, 0.5%; and to Rickettsia typhi, 0.0%.

284 T1 from Rickettsia prowazekii and PKMT2 from Rickettsia typhi, both the apo form and in complex with

285 Murine typhus, or infection with Rickettsia typhi, is a global but neglected disease with

286 Rickettsia typhi, the causative agent of murine (endemic

287 Rickettsia typhi, the causative agent of murine typhus,

288 Rickettsia typhi, the causative agent of murine typhus,

289 For Rickettsia typhi, the etiologic agent of murine typhus,

290 gate, intracellular, Gram-negative bacterium Rickettsia typhi, the etiologic agent of murine typhus,

291 se lepB genes from Rickettsia rickettsii and Rickettsia typhi, the etiologic agents of Rocky Mountain

292 For flea-borne Rickettsia typhi, the etiological agent of murine typhus

293 group rickettsiae, Rickettsia prowazekii and Rickettsia typhi, to have no actin tails and short (appr

294 n processing in gram-negative bacteria, from Rickettsia typhi.

295 in the Breinl strain of R. prowazekii and in Rickettsia typhi.

296 lus influenzae, S suis) and O tsutsugamushi, Rickettsia typhi/Rickettsia spp, and Leptospira spp infe

297 terestingly, the endosymbionts Wolbachia and Rickettsia were detected only in Henan, while the Ricket

298 proteins, we examined Anaplasma marginale, a rickettsia with two highly immunogenic outer membrane pr

299 Sequence typing clusters the Rickettsia within the Torix group of the genus, a group

300 hia induced only mild CI when mated with the Rickettsia-Wolbachia females.