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1 fe-threatening, tick-borne disease caused by Rickettsia rickettsii.
2 umbilical vein endothelial cells (HUVEC) by Rickettsia rickettsii.
3 d Rickettsia spp.; the other is specific for Rickettsia rickettsii.
4 om those of several other agents, especially Rickettsia rickettsii.
5 ), Ehrlichia canis, Bartonella henselae, and Rickettsia rickettsii.
6 ct human umbilical vein after infection with Rickettsia rickettsii.
7 about the structure and morphogenesis of the Rickettsia rickettsii actin tail relative to Shigella an
8 dothelial cells consequent to infection with Rickettsia rickettsii, an obligate intracellular gram-ne
9 me host cell apoptosis was explored by using Rickettsia rickettsii, an obligate intracellular Gram-ne
10 vein endothelial cells during infection with Rickettsia rickettsii, an obligate, intracellular bacter
12 veloped for the detection and enumeration of Rickettsia rickettsii and other closely related spotted
13 spotted fever and boutonneuse fever, due to Rickettsia rickettsii and R. conorii, respectively, are
14 The type I signal peptidase lepB genes from Rickettsia rickettsii and Rickettsia typhi, the etiologi
16 infections caused by Ehrlichia chaffeensis, Rickettsia rickettsii, and Coxiella burnetti, no signifi
18 ed fever group rickettsiae (SFGR) other than Rickettsia rickettsii are responsible for spotted fever
19 nstrated IgG or IgM antibodies reactive with Rickettsia rickettsii at a diagnostic titer (i.e., >/=64
20 plasma phagocytophilum, Ehrlichia canis, and Rickettsia rickettsii), but the sample was highly positi
21 cognized by reference human antisera against Rickettsia rickettsii, Chlamydia group positive, Trepone
22 ponse to certain pathogenic organisms (e.g., Rickettsia rickettsii), data documenting endothelial cel
23 steria monocytogenes, Shigella flexneri, and Rickettsia rickettsii, exploit the host cytoskeleton by
25 WB-8-2 and two other nonpathogenic isolates (Rickettsia rickettsii Hip2 and Rickettsia montana M5/6)
28 etion of chemokines and prostaglandins after Rickettsia rickettsii infection of human cerebral, derma
32 otted fever, a tick-borne zoonosis caused by Rickettsia rickettsii, is among the most lethal of all i
33 used by the obligate intracellular bacterium Rickettsia rickettsii, is associated with widespread inf
34 e HGE agent and to either Coxiella burnetii, Rickettsia rickettsii, or Rickettsia typhi was infrequen
35 Ehrlichia canis, E. chaffeensis, E. ewingii, Rickettsia rickettsii, R. conorii, and other spotted fev
37 rs of the spotted fever group (SFG), such as Rickettsia rickettsii, the agent of Rocky Mountain spott
39 nse during infection of endothelial cells by Rickettsia rickettsii, the causative agent of Rocky Moun
40 k Amblyomma cooperi in the enzootic cycle of Rickettsia rickettsii, the etiologic agent of Brazilian
41 potentially fatal human infection caused by Rickettsia rickettsii, the etiologic agent of Rocky Moun
42 l (EC) is a primary target of infection with Rickettsia rickettsii, the etiologic agent of Rocky Moun
44 mutant pairs from two independent strains of Rickettsia rickettsii, the virulent R strain and the avi
46 cies of a mariner-based transposon system in Rickettsia rickettsii were determined using a plaque ass
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