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1 the presence of basolateral bicarbonate-rich Ringer solution.
2 ed to a third or less of its normal value in Ringer solution.
3 delivered by pressure ejections of odor-free Ringer solution.
4 seen when returning immediately to IBMX-free Ringer solution.
5 ontrol and received a continuous infusion of Ringer solution.
6 lished when cells were bathed in an alkaline Ringer solution.
7 s, which could be observed even from rods in Ringer solution.
8 to changes in pH(o) in bicarbonate-buffered Ringer solution.
9 i which normally accompanies illumination in Ringer solution.
10 th the oscillation period when stimulated in Ringer solution.
11 extran 2000 reduced flows in comparison with Ringer solution.
12 g lumen and bath perfusion with an HCO(3)(-)-Ringer solution.
13 g sympathetic ganglion neurones in 2.0 mM K+ Ringer solution.
14 fluid had a similar effect to hyaluronan in Ringer solution.
15 etal muscle fibres by exposure to hypertonic Ringer solutions.
17 control (90% propylene glycol + 10% lactated Ringer solution); (2) 20 mm capsazepine to inhibit TRPV-
18 Compartments #1, #2 and #5 contained frog Ringer solution, #4 was filled with Vaseline and formed
19 her in contralateral joints expanded by 2 ml Ringer solution (5.80 +/- 0.84 micrograms h-1, n = 5, P
21 , instead, the outer segment was returned to Ringer solution after the bleach, thereby allowing [Ca2+
25 lined at a rate that was much slower than in Ringer solution and consistent with previous physiologic
26 its replacement by an isotonic Ca(2+)-Mg(2+) Ringer solution and cooling sharply reduced such access.
27 ng VIP(10-28) at the three concentrations or Ringer solution and perfusion was continued for 45-60 mi
28 esponses had a longer latency than in normal Ringer solution and were blocked by [D-pGlu1, D-Phe2, D-
29 studied in Cl(-)-free, normal and Na(+)-free Ringer solutions and in the presence of bumetanide, chlo
30 ide and replacement of Na+ by choline in the Ringer solution, and irreversibly by both fetal and mate
31 nto the knees of anaesthetized rabbits, with Ringer solution as control in the contralateral joint.
33 ight of much lower intensity if delivered in Ringer solution but not if delivered in 0 Ca(2+), 0 Na(+
34 ifts in E(m) in fibres studied in Cl(-)-free Ringer solution consistent with the Goldman-Hodgkin-Katz
37 troqinoxaline-2,3-dione (CNQX; 10 microM) in Ringer solution containing physiological concentrations
39 vessels were perfused via micropipettes with Ringer solutions containing bovine serum albumin (1 mg m
40 ng and 10 older subjects for infusion of (1) Ringer solution (control), (2) 0.5 mm L-tyrosine, (3) 5
41 older (O) human subjects for infusion of (1) Ringer solution (control), (2) 5 mM BH(4), (3) 5 mM BH(4
42 rodialysis sites were perfused with lactated Ringer solution (Control), 40 pm, 4 nm or 400 nm ET-1; i
43 2 (n = 11) sites were perfused with lactated Ringer solution (Control), 400 nm ET-1, 10 mm N(G) -nitr
44 ion, microdialysis fibres were perfused with Ringer solution (control), a ATP-sensitive potassium cha
45 ays, microdialysis fibres were perfused with Ringer solution (control), a non-specific NO synthase in
48 intradermal microdialysis with: (1) lactated Ringer solution (Control); (2) 10 mm ascorbate (Ascorbat
50 magnitude when the cell was superfused with Ringer solution during the 5 s interval between odour ex
51 bicarbonate Ringer (KBR) with Hepes-buffered Ringer solution exhibited basolateral, but not apical, r
52 xposed to 110 mM hydroxylamine in a low-Ca2+ Ringer solution for a period of 10-50 s beginning 10-17
55 ger solution in the bath and K(+)-containing Ringer solution in the pipette, both currents were selec
56 h retina superfused with a bicarbonate-based Ringer solution in the subjective day and night; that is
57 ked by 20 Hz nerve firing occurred in normal Ringer solution, in ryanodine, and in caffeine with a pe
58 sites were then heated to 42 degrees C, with Ringer solution infused in one probe and N-nitro-L-argin
59 on of neurones with zero calcium (1 mM EGTA) Ringer solution inhibited depolarization-induced calcium
60 exposure of muscles to a hypertonic glycerol-Ringer solution, its replacement by an isotonic Ca(2+)-M
61 also unchanged when the cone was exposed to Ringer solution made up from heavy water, whose solvent
62 intradermal microdialysis sites: control (C, Ringer solution), NO synthase inhibited (NOS-I, 10 mm l-
63 mmHg) men and women to serve as: control (C, Ringer solution), NOS inhibited (NOS-I, 10 mM L-NAME), A
64 ears) human subjects, serving as control (C, Ringer solution), NOS-inhibited (10.0 mM NG-nitro-L-argi
65 In contrast, fibres exposed to hypertonic Ringer solutions of normal ionic composition showed no s
66 s evoked by continuous stimulation in normal Ringer solution or by bursts of stimuli in hexamethonium
68 fluid drainage rate was reduced relative to Ringer solution (P < 0.001, ANOVA) but increased steeply
69 l pH was observed at two different values of Ringer solution pH, indicating that the circadian phenom
71 + solution before the flash and returning to Ringer solution shortly before the normal time of recove
72 n isotonic Cl(-)-free, normal and Na(+)-free Ringer solutions showed similar E(m) values consistent w
74 polarization produced by a high K(+) (40 mM) Ringer solution that was delivered rapidly and briefly t
76 When the cell was bathed in Ca2+-free Ba2+ Ringer solution, the K+ currents were blocked and large
78 m or amplitude when rods were pre-exposed in Ringer solution to light which was bright enough to supp
81 + P), bretylium tosylate (BT), and lactated Ringer solution were infused via intradermal microdialys
84 H (pHi) or Na(+) ([Na(+)](i)) in response to Ringer solutions with/without B(OH)(4)(-) or HCO(3)(-) a
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