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1 d storage (14% survival with CRS vs. 0% with Ringer's solution).
2 ither hypertonic saline, Isosal, or lactated Ringer's solution.
3 HDL or placebo), and then by 1 L of lactated Ringer's solution.
4 n ventilators and resuscitated with lactated Ringer's solution.
5  CAI-dentate area was perfused with modified Ringer's solution.
6 osm/L of 7.5% hypertonic saline, or lactated Ringer's solution.
7 nd 74%, respectively, compared with lactated Ringer's solution.
8 n ventilators and resuscitated with lactated Ringer's solution.
9 ne (5 to 10 microg/kg) or hyperoxic lactated Ringer's solution.
10 th EGTA or by bathing such cells in low-Ca2+ Ringer's solution.
11 um proteins when incubated in buffered chick Ringer's solution.
12  volume was returned in the form of lactated Ringer's solution.
13 uld restore membrane resealing in low Ca(2+) Ringer's solution.
14  volume was returned in the form of lactated Ringer's solution.
15 ith its occurrence in animals given lactated Ringer's solution.
16  and perfused at a rate of 5 microL/min with Ringer's solution.
17 testinal sleeve incubated in a physiological Ringer's solution.
18  volume was returned in the form of lactated Ringer's solution.
19 , (4) anti-bFGF antibody, (5) ngIgG, and (6) Ringer's solution.
20 ients receiving a rapid infusion of lactated Ringer's solution.
21 els was evoked with long exposure to high K+ Ringer's solution.
22 rch solution); 5% human albumin; or lactated Ringer's solution.
23 rfusion bath with a low-HCO(3)(-) Cl(-)-free Ringer's solution (2.85 mM; pH 6.5), in the presence or
24 re perfused with a HCO(3)(-)-rich Cl(-)-free Ringer's solution (28.5 mM; pH 7.5).
25 nhibitor) in 50/50 dimethyl sulfoxide (DMSO)/Ringer's solution, 300 KIU aprotinin (a serine protease
26 - 2 mL/mg) groups compared with the lactated Ringer's solution (35 +/- 5 mL/kg) group.
27 0 mins, pigs were resuscitated with lactated Ringer's solution (40 mL/kg over 30 mins).
28                                    In normal Ringer's solution, 57% of the rods tested could follow r
29 large body surface area burn, using lactated Ringer's solution, 6% hetastarch, and hypertonic saline
30  for Isosal solution) compared with lactated Ringer's solution (75.3 +/- 11.6 mL/kg) (p = .01).
31        Within 5 to 10 minutes of exposure to Ringer's solution, a general decrease in the light trans
32 ntrols (n = 6) received intravenous lactated Ringer's solution according this dosing schedule: 1.5 mL
33 ention group) or an equal volume of lactated Ringer's solution (acid control group).
34 eperfusion compared with albumin or lactated Ringer's solution administration (p < .001).
35 urned to baseline immediately after lactated Ringer's solution administration, while PAOP remained in
36 eased transiently immediately after lactated Ringer's solution administration.
37 , and immediately and 30 mins after lactated Ringer's solution administration.
38 compared with that in fiber cells exposed to Ringer's solution alone (1995+/-461 nM, n = 10).
39 ron chelator deferoxamine (DFO), or lactated Ringer's solution alone (LR) on lung injury parameters a
40 patients were randomized to receive lactated Ringer's solution and 17 to receive HES 130/0.4.
41                                     Lactated Ringer's solution and 3-hr cold ischemia time were used
42  to 145 mmol/L) was measured in the lactated Ringer's solution and hetastarch groups (130 to 133 mmol
43 n nonburned skin compared with both lactated Ringer's solution and hypertonic saline dextran.
44 rs with the endothelium bathed in a modified Ringer's solution and the epithelium bathed with silicon
45 s were similarly perfused with cold lactated Ringer's solution and were followed without immunosuppre
46 ts in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA AM-pretreated preparations;
47 ellate) morphology by exposure to serum-free Ringer's solution; and (3) swelling cells by exposure to
48 tation in all three groups was with lactated Ringer's solution as needed to maintain baseline cardiac
49     All groups received intravenous lactated Ringer's solution at 4 mL.kg-1.%burn(-1).24 hrs-1 for re
50                     Fluid challenges at 6 h (Ringer's solution at 80 ml x kg(-1) given over a period
51 n, during which either PentaLyte or lactated Ringer's solution-based resuscitation was administered.
52                      Addition of INS37217 to Ringer's solution bathing the apical membrane transientl
53 the conjunctiva with Na(+)-free bicarbonated Ringer's solution (BRS) were used to estimate contributi
54     LY evoked similar effects in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA
55 le; however, after 15 minutes of exposure to Ringer's solution, [Ca2+]i in fibers from the outer cort
56                                              Ringer's solution composition changes on the retina-faci
57 nd 58.9 +/- 3.9 minutes, respectively) or in Ringer's solution containing 1 mM [Ca2+]o + 50 microM ve
58                               Perfusion with Ringer's solution containing 1 mM [Ca2+]o, caused disint
59   The proteolytic activity of fiber cells in Ringer's solution containing 10(-)(6) M and 2 x 10(-)(3)
60 vely, compared with sucrose-EDTA solution or Ringer's solution containing 10(-)(8) M [Ca(2+)](o).
61                           In the presence of Ringer's solution containing 2 x 10(-)(3) M [Ca(2+)](o)
62         T:(g) of fiber cells superfused with Ringer's solution containing 2 x 10(-)(3) M, 10(-)(6) M,
63 ither background illumination or exposure to Ringer's solution containing low Ca2+ and/or cyclic GMP-
64           Exposure of the isolated fibers to Ringer's solution (containing 2 mM Ca2+) led to a monoex
65  elongated fibers, which, in the presence of Ringer's solution (containing 2 mM Ca2+), underwent disi
66 nutes, the [Ca2+]i of fiber cells exposed to Ringer's solution, containing 2 mM Ni2+ (574.7+/-29 nM;
67                                Compared with Ringer's solution (control), microdialysis delivery of t
68 ), sildenafil (PDE5 inhibitor), and lactated Ringer's solution (control).
69 0.9% saline (n = 6), 5% dextrose in lactated Ringer's solution (D5RL) (n = 6), or 5% dextrose in wate
70 en cells were wounded twice in normal Ca(2+) Ringer's solution, decreases in tension at the second wo
71               The [Ca2+]i in the globules in Ringer's solution, determined using Calcium Green-2, was
72 ate-buffered saline, normal saline, lactated Ringer's solution, dextran, hespan, 5% human albumin, 25
73 emoglobin) or a control infusion of lactated Ringer's solution (each infusion given over a total of 4
74  = 9) solution made up exactly like lactated Ringer's solution except for the substitution of either
75                After perfusion with lactated Ringer's solution, extravasated bBSA was detected with i
76 times the volume of shed blood with lactated Ringer's solution for 60 mins.
77 ed rats were then resuscitated with lactated Ringer's solution, four times the maximum shed blood vol
78  was superfused with glutathione bicarbonate Ringer's solution (GBR); with GBR and 10 nM, 100 nM, or
79  isosal group was lower than in the lactated Ringer's solution group only in the cerebellum.
80 onic saline group compared with the lactated Ringer's solution group.
81  Test solutions (10 mL/kg of either lactated Ringer's solution, hetastarch, or hypertonic saline dext
82 sterior surface is continually bathed with a Ringer's solution in equilibrium with a CO2-gas air mixt
83          The short-term infusion of lactated Ringer's solution in normal adults (hemodynamically stab
84 obules generated from fiber cells exposed to Ringer's solution; in addition, no high molecular weight
85 est the hypothesis that intravenous lactated Ringer's solution, infused at a rate used in resuscitati
86                This was followed by lactated Ringer's solution, infused to a target urine output of 1
87  were resuscitated by administering lactated Ringer's solution intravenously to achieve and maintain
88                   Oxygen tension of a pH 7.5 Ringer's solution is measured in an airtight chamber tha
89           Exposure of the cortical fibers to Ringer's solution led to biphasic changes in the intensi
90 on of the isolated rat lens fiber cells with Ringer's solution led to their globulization in 30 +/- 3
91                                           In Ringer's solution, leupeptin delayed globulization witho
92 ed in lactated Ringer's solution or lactated Ringer's solution (LR) alone were given by intravenous i
93 ovolemic shock, HSD (250 mL) versus lactated Ringer's solution (LR) as the initial resuscitation flui
94 her group (n = 8) received only the lactated Ringer's solution (LRS) vehicle.
95  suggests that balanced fluids like Lactated Ringer's solution may be preferable.
96 d bathing solutions were iso-osmotic Cl-free Ringer's solutions modified using N-methyl-D-glucamine a
97              Recovery in plasma, compared to Ringer's solution (n = 4), was 103%.
98 o receive a 1-hr infusion of either lactated Ringer's solution (n = 6), 0.9% saline (n = 6), 5% dextr
99 plantation, kidneys were flushed with either Ringer's solution or CRS at 35-37 degrees C or were not
100 ontrol mice overnight in distilled water, in Ringer's solution or in Ringer's solution with added 1 M
101      Three doses of EP dissolved in lactated Ringer's solution or lactated Ringer's solution (LR) alo
102  with kidneys flushed with CRS compared with Ringer's solution or nontreatment.
103 hloride) and balanced crystalloids (lactated Ringer's solution or Plasma-Lyte A).
104 after CRS was significantly higher than with Ringer's solution or without flushing (80% vs. 25% and 1
105 .086 min[-1]) and in K+-free (0.062 min[-1]) Ringer's solution, or when the fibers were suspended in
106 the volume of maximal bleedout with lactated Ringer's solution over 60 mins.
107 times the volume of shed blood with lactated Ringer's solution over 60 minutes.
108 de of the epithelium, was enhanced in simple Ringer's solution over that in tissue culture medium, an
109  blood plus albumin than blood plus lactated Ringer's solution (P < 0.01).
110 ation than after resuscitation with lactated Ringer's solution (p < 0.05).
111 fusion than after administration of lactated Ringer's solution (p < 0.05).
112 s to investigate the effects of ATP added to Ringer's solution perfusing the retinal-facing (apical)
113 cally paced myocytes (0.5 Hz, 1 mmol/L Ca2+, Ringer's solution [pH 7.4], 22 degrees C).
114 ive increase in [Ca2+]i in fibers exposed to Ringer's solution, preceding globulization.
115 e than 50%, while administration of lactated Ringer's solution provoked an approximately 2.5 times gr
116                           Resuscitation with Ringer's solution resulted in a standard base excess, an
117 scitation with red blood cells plus lactated Ringer's solution (RL) is more effective than RL alone i
118 8 medical ICU patients receiving no lactated Ringer's solution (RL).
119 lood + 0.12, 0.24, or 0.36 g/kg) or lactated Ringer's solution (shed blood + 2 x volume of shed blood
120 exposed to 1 mM [Ca2+]o in Na+ - or K+ -free Ringer's solution (tg = 66.7 +/- 5.3 and 58.9 +/- 3.9 mi
121 ter (p < .001) in animals receiving lactated Ringer's solution than in rabbits receiving either collo
122 crease if cells were wounded in a low Ca(2+) Ringer's solution that inhibited both membrane resealing
123 esence of phosphatidylcholine (0.025 g/ml in Ringer's solution), the injured cells initially responde
124                                           In Ringer's solution, the damaged cells stopped contracting
125 at, for fibroblasts wounded in normal Ca(2+) Ringer's solution, the membrane tension decreased dramat
126              Compared with that in fibers in Ringer's solution, the rate of increase of [Ca2+]i in fi
127  When these cells were placed back in normal Ringer's solution, they remained viable and active for s
128 bsequently either resuscitated with lactated Ringer's solution (three times shed blood volume, n = 18
129 on, awakened, and resuscitated with lactated Ringer's solution titrated to maintain hematocrit +/- 3%
130  blood was then returned along with lactated Ringer's solution (two times the shed blood volume) to p
131 compared between subjects receiving lactated Ringer's solution vs. subjects receiving normal saline.
132 studied in those subjects receiving lactated Ringer's solution vs. those persons receiving normal sal
133 0, or 100 mg/kg) or equal volume of lactated Ringer's solution was infused on day 1; the alternate so
134 mL/kg test solution dose was given, lactated Ringer's solution was infused to achieve the same end po
135 ncrease of [Ca2+]i in fiber cells exposed to Ringer's solution was measured, and the effects on the i
136                   In contrast, when lactated Ringer's solution was used, multiple boluses were requir
137 he currents observed in divalent cation-free Ringer's solution were due to Cx46 hemichannel opening,
138 ly discovered that small amounts of lactated Ringer's solution, which are inadequately cleared from a
139  distilled water, in Ringer's solution or in Ringer's solution with added 1 M sucrose.

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