ライフサイエンスコーパス: Ringer

1 Ringer injections had no effects.

2 Ringer made hypertonic by the addition of 2.5 M sucrose

3 Ringer solution and 214 microM VIP(10-28) were each perf

4 Ringer solution containing enzyme was injected into the

5 Ringer solution was infused into the knee joint cavity o

6 Ringer's solution composition changes on the retina-faci

7 ng and 10 older subjects for infusion of (1) Ringer solution (control), (2) 0.5 mm L-tyrosine, (3) 5

8 older (O) human subjects for infusion of (1) Ringer solution (control), (2) 5 mM BH(4), (3) 5 mM BH(4

9 .086 min[-1]) and in K+-free (0.062 min[-1]) Ringer's solution, or when the fibers were suspended in

10 crease if cells were wounded in a low Ca(2+) Ringer's solution that inhibited both membrane resealing

11 en cells were wounded twice in normal Ca(2+) Ringer's solution, decreases in tension at the second wo

12 at, for fibroblasts wounded in normal Ca(2+) Ringer's solution, the membrane tension decreased dramat

13 uld restore membrane resealing in low Ca(2+) Ringer's solution.

14 its replacement by an isotonic Ca(2+)-Mg(2+) Ringer solution and cooling sharply reduced such access.

15 ) was induced by exposure to CO(2)-HCO(3)(-) Ringer's and the opposing outward flux by returning to H

16 Exposure to CO(2)-HCO(3)(-) Ringer's led to a transient decrease in pH(i) (component

17 However, when the CO(2)-HCO(3)(-) Ringer's was removed, the pH(i) increased transiently (c

18 g lumen and bath perfusion with an HCO(3)(-)-Ringer solution.

19 onium (TEA), (3) EMLA + TEA (Combo), and (4) Ringer solution (Control).

20 (L-NAME), (3) Keto + l-NAME (Combo), and (4) Ringer solution (Control).

21 Oxygen tension of a pH 7.5 Ringer's solution is measured in an airtight chamber tha

22 , (4) anti-bFGF antibody, (5) ngIgG, and (6) Ringer's solution.

23 sterior surface is continually bathed with a Ringer's solution in equilibrium with a CO2-gas air mixt

24 lished when cells were bathed in an alkaline Ringer solution.

25 When the cell was bathed in Ca2+-free Ba2+ Ringer solution, the K+ currents were blocked and large

26 h retina superfused with a bicarbonate-based Ringer solution in the subjective day and night; that is

27 se to 40 mM lactate in bicarbonate free (BF) Ringer's that was inhibited by niflumic acid and by MCT

28 was superfused with glutathione bicarbonate Ringer's solution (GBR); with GBR and 10 nM, 100 nM, or

29 ded by replacing bilateral Krebs bicarbonate Ringer (KBR) with Hepes-buffered Ringer solution exhibit

30 the conjunctiva with Na(+)-free bicarbonated Ringer's solution (BRS) were used to estimate contributi

31 to changes in pH(o) in bicarbonate-buffered Ringer solution.

32 bicarbonate Ringer (KBR) with Hepes-buffered Ringer solution exhibited basolateral, but not apical, r

33 vo loop studies HCO3 (-)-Ringer and butyrate-Ringer exhibit similar rates of water absorption in norm

34 Lumen butyrate-Ringer incubation activated NHE3-mediated Na(+) absorpti

35 in DSS-induced inflammation luminal butyrate-Ringer reversed water secretion observed with HCO3 (-)-R

36 intradermal microdialysis sites: control (C, Ringer solution), NO synthase inhibited (NOS-I, 10 mm l-

37 mmHg) men and women to serve as: control (C, Ringer solution), NOS inhibited (NOS-I, 10 mM L-NAME), A

38 ears) human subjects, serving as control (C, Ringer solution), NOS-inhibited (10.0 mM NG-nitro-L-argi

39 xposed to 110 mM hydroxylamine in a low-Ca2+ Ringer solution for a period of 10-50 s beginning 10-17

40 th EGTA or by bathing such cells in low-Ca2+ Ringer's solution.

41 e stretch modulation persists in a zero Ca2+ Ringer and, hence, is not dependent on Ca2+ influx throu

42 cally paced myocytes (0.5 Hz, 1 mmol/L Ca2+, Ringer's solution [pH 7.4], 22 degrees C).

43 um proteins when incubated in buffered chick Ringer's solution.

44 helial 36Cl fluxes in bicarbonate-containing Ringer's were determined.

45 ger solution in the bath and K(+)-containing Ringer solution in the pipette, both currents were selec

46 With Na(+)-containing Ringer solution in the bath and K(+)-containing Ringer s

47 P < 0.05) and restored the NO contribution (Ringer: 44 +/- 3 % CVCmax vs. AA: 59 +/- 6 % CVCmax; P <

48 , produced cellular acidification in control Ringer.

49 e other 24 degrees C site served as control (Ringer solution + cooling).

50 e HS diet, AA improved the plateau % CVCmax (Ringer: 80 +/- 2 % CVCmax vs. AA: 89 +/- 3 % CVCmax; P <

51 nhibitor) in 50/50 dimethyl sulfoxide (DMSO)/Ringer's solution, 300 KIU aprotinin (a serine protease

52 on of neurones with zero calcium (1 mM EGTA) Ringer solution inhibited depolarization-induced calcium

53 compare the effects of treatment with either Ringer's lactate solution or ethyl pyruvate solution on

54 plantation, kidneys were flushed with either Ringer's solution or CRS at 35-37 degrees C or were not

55 H2O-1 (mean +/- s.e.m.), was 1/39th that for Ringer solution (1.94 +/- 0.01 microliter 2O-1 ).

56 outward flux by returning to HCO(3)(-)-free Ringer's.

57 rslips and then perfused with HCO(3)(-)-free Ringer's.

58 B(OH)(4)(-) (2.5-10 mM) in bicarbonate-free Ringer induced a rapid small acidification (0.01 pH unit

59 ts in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA AM-pretreated preparations;

60 he currents observed in divalent cation-free Ringer's solution were due to Cx46 hemichannel opening,

61 ifts in E(m) in fibres studied in Cl(-)-free Ringer solution consistent with the Goldman-Hodgkin-Katz

62 rfusion bath with a low-HCO(3)(-) Cl(-)-free Ringer's solution (2.85 mM; pH 6.5), in the presence or

63 re perfused with a HCO(3)(-)-rich Cl(-)-free Ringer's solution (28.5 mM; pH 7.5).

64 efflux when cells were bathed in Cl(-)-free Ringer's.

65 d bathing solutions were iso-osmotic Cl-free Ringer's solutions modified using N-methyl-D-glucamine a

66 seen when returning immediately to IBMX-free Ringer solution.

67 studied in Cl(-)-free, normal and Na(+)-free Ringer solutions and in the presence of bumetanide, chlo

68 n isotonic Cl(-)-free, normal and Na(+)-free Ringer solutions showed similar E(m) values consistent w

69 In Na+-free Ringer, apical H2DIDS produced a transient alkalinizatio

70 delivered by pressure ejections of odor-free Ringer solution.

71 Compartments #1, #2 and #5 contained frog Ringer solution, #4 was filled with Vaseline and formed

72 LY evoked similar effects in normal frog Ringer's solution, Ca-free Ringer's solution, and BAPTA

73 Vessels, microperfused with frog Ringer solutions containing 0.1% bovine serum albumin an

74 exposure of muscles to a hypertonic glycerol-Ringer solution, its replacement by an isotonic Ca(2+)-M

75 Fluid challenges at 6 h (Ringer's solution at 80 ml x kg(-1) given over a period

76 In in vivo loop studies HCO3 (-)-Ringer and butyrate-Ringer exhibit similar rates of wate

77 ersed water secretion observed with HCO3 (-)-Ringer to fluid absorption.

78 as incubated for 30 minutes in 25 mM HCO3(-)-Ringer with agents promoting corneal deturgescence or co

79 In the control medium, 25 mM HCO3(-)-Ringer, 86Rb+ was accumulated to yield a T-M ratio of 6.

80 In contrast, fibres exposed to hypertonic Ringer solutions of normal ionic composition showed no s

81 etal muscle fibres by exposure to hypertonic Ringer solutions.

82 In Ringer solution containing both Cl- and HCO3-, the magni

83 In Ringer solution, the Ca2+ in the light-releasable pool c

84 In Ringer's solution, leupeptin delayed globulization witho

85 The proteolytic activity of fiber cells in Ringer's solution containing 10(-)(6) M and 2 x 10(-)(3)

86 ight of much lower intensity if delivered in Ringer solution but not if delivered in 0 Ca(2+), 0 Na(+

87 m or amplitude when rods were pre-exposed in Ringer solution to light which was bright enough to supp

88 Compared with that in fibers in Ringer's solution, the rate of increase of [Ca2+]i in fi

89 llumination, whether the bleach was given in Ringer solution or in 0Ca(2+)/0Na(+) solution.

90 The [Ca2+]i in the globules in Ringer's solution, determined using Calcium Green-2, was

91 fluid had a similar effect to hyaluronan in Ringer solution.

92 i which normally accompanies illumination in Ringer solution.

93 troqinoxaline-2,3-dione (CNQX; 10 microM) in Ringer solution containing physiological concentrations

94 Mortality in Ringer's acetate, stroma-free hemoglobin, and 10% pentas

95 distilled water, in Ringer's solution or in Ringer's solution with added 1 M sucrose.

96 mesial TLE (MTLE) were immediately placed in Ringer's lactate; stearate indicator microelectrodes wer

97 s, which could be observed even from rods in Ringer solution.

98 PLP-null and control white matter soaked in Ringer remained largely compact.

99 stilling an isosmolar 5% albumin solution in Ringer's lactate with 3 microCi 125I-albumin.

100 th the oscillation period when stimulated in Ringer solution.

101 lined at a rate that was much slower than in Ringer solution and consistent with previous physiologic

102 ed to a third or less of its normal value in Ringer solution.

103 ontrol mice overnight in distilled water, in Ringer's solution or in Ringer's solution with added 1 M

104 The model used was the isolated Ringer perfused sheep choroid plexus.

105 , 6 min, 4 mM), challenge with elevated K(+) Ringer caused a dose-dependent DeltaDC in the range 10-1

106 A brief application (8 seconds) of high K(+) Ringer elicited a robust cytosolic Ca(2+) increase at th

107 ptor agonists had no effect on the high K(+) Ringer-elicited cytosolic Ca(2+) signal at OPL.

108 -40.0 +/- 4.1 mV (n = 14) and in 140 mM K(+) Ringer's it depolarized to -7.4 +/- 1.8 mV.

109 Application of Ba(2+) in 5 mM K(+) Ringer's produced a concentration-dependent depolarizati

110 In 5 mM K(+) Ringer's, the V(m) of cell clusters averaged -40.0 +/- 4

111 Challenge with 100 mM [K(+)] Ringer was used to assess the K(+) permeability of the p

112 C on subsequent challenge with 100 mM [K(+)] Ringer, indicating no effect on perineurial K(+) permeab

113 els was evoked with long exposure to high K+ Ringer's solution.

114 g sympathetic ganglion neurones in 2.0 mM K+ Ringer solution.

115 um, changing from normal Ringer to high [K+] Ringer (100 mM, KCl replacing NaCl) for 2 min caused neg

116 C) in response to challenge with 100 mM [K+] Ringer was used to assess the K+ permeability of the per

117 The inward current was observed in a KCl Ringer's bath and was almost nonexistent in a NaCl bath.

118 ainage period, animals received either Krebs Ringer Henseleit (the bile-depleted group), or sodium ta

119 exposed and continuously suffused with Krebs Ringer bicarbonate warmed to 37 degrees C.

120 t hepatocytes were incubated in anoxic Krebs-Ringer-HEPES buffer at pH 6.2 for 4 hours and reoxygenat

121 In Ca2+-free Krebs-Ringer bicarbonate buffer containing 2.8 mmol/l glucose,

122 e isolated by collagenase digestion in Krebs-Ringer bicarbonate (KRB) buffer at 37 degrees C.

123 Sphincter muscle was incubated in Krebs-Ringer bicarbonate buffer in the absence and presence of

124 lated, cultured, and then perifused in Krebs-Ringer bicarbonate buffer with 2 mmol/l glutamine using

125 without endothelium were suspended in Krebs-Ringer bicarbonate solution for isometric tension record

126 eriphery and immersed in an oxygenated Krebs-Ringer buffer.

127 he perfusate from rat liver exposed to Krebs-Ringer bicarbonate buffer only, 0-1mM [3,4-(13)C(2)]-4-h

128 nitially perfused at 37 degrees C with Krebs-Ringer's (KR) solution (in mmol/L: Ca(2+) 2.5, K(+) 5, M

129 Lactated Ringer's solution and 3-hr cold ischemia time were used

130 intradermal microdialysis with: (1) lactated Ringer solution (Control); (2) 10 mm ascorbate (Ascorbat

131 and each randomly assigned as: (1) lactated Ringer's (control); (2) 20 mm Nomega-nitro-l-arginine me

132 control (90% propylene glycol + 10% lactated Ringer solution); (2) 20 mm capsazepine to inhibit TRPV-

133 were resuscitated by administering lactated Ringer's solution intravenously to achieve and maintain

134 after each blood withdrawal, after lactated Ringer's resuscitation, and after infusion of shed blood

135 + P), bretylium tosylate (BT), and lactated Ringer solution were infused via intradermal microdialys

136 ), sildenafil (PDE5 inhibitor), and lactated Ringer's solution (control).

137 tive neurons in CA1 vs. Hextend and lactated Ringer's, and CA3 vs. Hextend (p<.05).

138 normal blood pressure with a blood/lactated Ringer's (1:2) mixture.

139 This was followed by lactated Ringer's solution, infused to a target urine output of 1

140 hloride) and balanced crystalloids (lactated Ringer's solution or Plasma-Lyte A).

141 o receive a 1-hr infusion of either lactated Ringer's solution (n = 6), 0.9% saline (n = 6), 5% dextr

142 posite effects in outcome, favoring Lactated Ringer.

143 ith its occurrence in animals given lactated Ringer's solution.

144 ally and flushed with cold heparin, lactated Ringer's and procaine (HeLP) solution.

145 ne (5 to 10 microg/kg) or hyperoxic lactated Ringer's solution.

146 induced by infusion of 0.2 M HCl in lactated Ringer solution in the acid group.

147 0.9% saline (n = 6), 5% dextrose in lactated Ringer's solution (D5RL) (n = 6), or 5% dextrose in wate

148 Three doses of EP dissolved in lactated Ringer's solution or lactated Ringer's solution (LR) alo

149 ntrols (n = 6) received intravenous lactated Ringer's solution according this dosing schedule: 1.5 mL

150 All groups received intravenous lactated Ringer's solution at 4 mL.kg-1.%burn(-1).24 hrs-1 for re

151 est the hypothesis that intravenous lactated Ringer's solution, infused at a rate used in resuscitati

152 = 9) solution made up exactly like lactated Ringer's solution except for the substitution of either

153 suggests that balanced fluids like Lactated Ringer's solution may be preferable.

154 8 medical ICU patients receiving no lactated Ringer's solution (RL).

155 t analysis suggested that volume of Lactated Ringer and 0.9% saline infused had opposite effects in o

156 Infusion of lactated Ringer's demonstrated no changes in the measured variabl

157 ention group) or an equal volume of lactated Ringer's solution (acid control group).

158 fusion than after administration of lactated Ringer's solution (p < 0.05).

159 e than 50%, while administration of lactated Ringer's solution provoked an approximately 2.5 times gr

160 ly discovered that small amounts of lactated Ringer's solution, which are inadequately cleared from a

161 volume was returned in the form of lactated Ringer's solution.

162 volume was returned in the form of lactated Ringer's solution.

163 volume was returned in the form of lactated Ringer's solution.

164 ients receiving a rapid infusion of lactated Ringer's solution.

165 nd less efficacious than the use of lactated Ringer.

166 of hemorrhage using 3% NaCl (HS) or lactated Ringer's (LR).

167 line (n = 25), Hextend (n = 25), or lactated Ringer's (n = 10).

168 ed in lactated Ringer's solution or lactated Ringer's solution (LR) alone were given by intravenous i

169 lood + 0.12, 0.24, or 0.36 g/kg) or lactated Ringer's solution (shed blood + 2 x volume of shed blood

170 eperfusion compared with albumin or lactated Ringer's solution administration (p < .001).

171 n, during which either PentaLyte or lactated Ringer's solution-based resuscitation was administered.

172 rch solution); 5% human albumin; or lactated Ringer's solution.

173 in, immunosuppressive treatment, or lactated Ringer's therapy.

174 atients who received HES 130/0.4 or lactated Ringer, respectively (P < 0.038).

175 receive either SAAP with oxygenated lactated Ringer's (LR) solution (n = 6) or SAAP with oxygenated H

176 ood (FWB), (2) SAAP with oxygenated lactated Ringer's (LR), 1,600 mL/2 min, or (3) SAAP with oxygenat

177 blood plus albumin than blood plus lactated Ringer's solution (P < 0.01).

178 scitation with red blood cells plus lactated Ringer's solution (RL) is more effective than RL alone i

179 patients were randomized to receive lactated Ringer's solution and 17 to receive HES 130/0.4.

180 eriod; the untreated group received Lactated Ringer (LR) at the same time points.

181 compared between subjects receiving lactated Ringer's solution vs. subjects receiving normal saline.

182 ate-buffered saline, normal saline, lactated Ringer's solution, dextran, hespan, 5% human albumin, 25

183 r Hextend required less volume than lactated Ringer's (both p<.05).

184 e mean arterial blood pressure than lactated Ringer's or Hextend and confer neuroprotection in a mous

185 the PJ34-treated groups than in the Lactated Ringer group at 7 and 24 hours of reperfusion.

186 ded to the storage solution and the lactated Ringer poststorage rinse solution.

187 The patients in the lactated Ringer's group, however, received more fluid (P < 0.0001

188 her group (n = 8) received only the lactated Ringer's solution (LRS) vehicle.

189 d to guide the infusion rate of the lactated Ringer's.

190 the PJ34-treated group than in the Lactated Ringer-treated mice at 24 hours of reperfusion.

191 ovolemic shock, HSD (250 mL) versus lactated Ringer's solution (LR) as the initial resuscitation flui

192 ed mean arterial blood pressure vs. lactated Ringer's (p<.05).

193 rodialysis sites were perfused with lactated Ringer solution (Control), 40 pm, 4 nm or 400 nm ET-1; i

194 2 (n = 11) sites were perfused with lactated Ringer solution (Control), 400 nm ET-1, 10 mm N(G) -nitr

195 ation than after resuscitation with lactated Ringer's solution (p < 0.05).

196 bsequently either resuscitated with lactated Ringer's solution (three times shed blood volume, n = 18

197 blood was then returned along with lactated Ringer's solution (two times the shed blood volume) to p

198 times the volume of shed blood with lactated Ringer's solution for 60 mins.

199 the volume of maximal bleedout with lactated Ringer's solution over 60 mins.

200 times the volume of shed blood with lactated Ringer's solution over 60 minutes.

201 on, awakened, and resuscitated with lactated Ringer's solution titrated to maintain hematocrit +/- 3%

202 After perfusion with lactated Ringer's solution, extravasated bBSA was detected with i

203 ed rats were then resuscitated with lactated Ringer's solution, four times the maximum shed blood vol

204 n ventilators and resuscitated with lactated Ringer's solution.

205 n ventilators and resuscitated with lactated Ringer's solution.

206 pressure>50 mm Hg for 30 min) with lactated Ringer's, Hextend, or PNPH, and then shed blood was rein

207 her in contralateral joints expanded by 2 ml Ringer solution (5.80 +/- 0.84 micrograms h-1, n = 5, P

208 polarization produced by a high K(+) (40 mM) Ringer solution that was delivered rapidly and briefly t

209 rs with the endothelium bathed in a modified Ringer's solution and the epithelium bathed with silicon

210 age by aortic tear to receive 250 mL of MP4, Ringer's acetate, 10% pentastarch, or 4 g/dL of stroma-f

211 th different crystalloids (NaCl 0.9% (NaCl), Ringer's acetate (RA)) or colloids (Gelafundin 4% (Gel),

212 al endothelial monolayers perfused with NO3- Ringer's were exposed to I- pulses under isosmotic and,

213 undamaged perineurium, changing from normal Ringer to high [K+] Ringer (100 mM, KCl replacing NaCl)

214 esponses had a longer latency than in normal Ringer solution and were blocked by [D-pGlu1, D-Phe2, D-

215 s evoked by continuous stimulation in normal Ringer solution or by bursts of stimuli in hexamethonium

216 In normal Ringer's solution, 57% of the rods tested could follow r

217 Acidification of Ringer solution resulted in a reduced antagonism by Zn2+

218 Applications of Ringer solutions with added NH(4)Cl induced rapid reduct

219 crodialysis fibres for the local delivery of Ringer solution and 20 mm ascorbic acid (AA).

220 bleed-out volume was returned in the form of Ringer's lactate (RL).

221 bleed-out volume was returned in the form of Ringer's lactate (RL).

222 r times the shed blood volume in the form of Ringer's lactate during a 60-minute period.

223 imal shed volume was returned in the form of Ringer's lactate solution.

224 r times the shed blood volume in the form of Ringer's lactate).

225 hed blood volume was returned in the form of Ringer's lactate.

226 4 times the shed blood volume in the form of Ringer's lactate.

227 4 times the shed blood volume in the form of Ringer's lactate.

228 ontrol and received a continuous infusion of Ringer solution.

229 oints received intra-articular injections of Ringer vehicle (control) or an activator of classical PK

230 itoneally every 6 hrs for 48 hrs) instead of Ringer's lactate solution starting 2 hrs after the injec

231 elongated fibers, which, in the presence of Ringer's solution (containing 2 mM Ca2+), underwent disi

232 In the presence of Ringer's solution containing 2 x 10(-)(3) M [Ca(2+)](o)

233 l pH was observed at two different values of Ringer solution pH, indicating that the circadian phenom

234 The work of Ringer, Hartmann, and others emphasized the importance o

235 ng VIP(10-28) at the three concentrations or Ringer solution and perfusion was continued for 45-60 mi

236 received an equivalent number of GFP-EPCs or Ringer's saline.

237 (n = 1443; isotonic or hypertonic saline or Ringer lactate solution) for all fluid interventions oth

238 ions, essentially a choice between saline or Ringer's lactate (compound sodium lactate or Hartmann's

239 l survival (at 12 hrs) was 0% with saline or Ringer's vs. 20% with Hextend, p =.05.

240 al time among animals treated with saline or Ringer's was 45% less compared with Hextend-treated anim

241 vely, compared with sucrose-EDTA solution or Ringer's solution containing 10(-)(8) M [Ca(2+)](o).

242 U with either 6% HES 130/0.42 (Tetraspan) or Ringer's acetate at a dose of up to 33 ml per kilogram o

243 testinal sleeve incubated in a physiological Ringer's solution.

244 The contralateral joint received Ringer solution without enzyme as a control.

245 nt therapy, as compared with those receiving Ringer's acetate.

246 alter pHi responses to CO(2)/HCO(3)(-)-rich Ringer, Na(+)-free induced acidification, or the rate of

247 in BCEC in HCO(3)(-)-free or HCO(3)(-)-rich Ringer, with and without niflumic acid (MCT inhibitor),

248 the presence of basolateral bicarbonate-rich Ringer solution.

249 de of the epithelium, was enhanced in simple Ringer's solution over that in tissue culture medium, an

250 After the sucrose/Ringer soak, normal myelin developed foci of decompactio

251 Sydney Ringer would be overwhelmed today by the implications of

252 Experiments using 0.5 nM leptin in the Ringer produced a concentration of leptin in the CSF of

253 ide and replacement of Na+ by choline in the Ringer solution, and irreversibly by both fetal and mate

254 ker, or when the extracellular Ca(2+) in the Ringer was replaced with equal molar EGTA.

255 between Ag/AgCl electrodes connected through Ringer-agar bridges to compartments #3 and #5.

256 s to investigate the effects of ATP added to Ringer's solution perfusing the retinal-facing (apical)

257 d with 172 of 400 patients (43%) assigned to Ringer's acetate (relative risk, 1.17; 95% confidence in

258 therapy versus 65 patients (16%) assigned to Ringer's acetate (relative risk, 1.35; 95% CI, 1.01 to 1

259 also unchanged when the cone was exposed to Ringer solution made up from heavy water, whose solvent

260 compared with that in fiber cells exposed to Ringer's solution alone (1995+/-461 nM, n = 10).

261 ncrease of [Ca2+]i in fiber cells exposed to Ringer's solution was measured, and the effects on the i

262 nutes, the [Ca2+]i of fiber cells exposed to Ringer's solution, containing 2 mM Ni2+ (574.7+/-29 nM;

263 obules generated from fiber cells exposed to Ringer's solution; in addition, no high molecular weight

264 le; however, after 15 minutes of exposure to Ringer's solution, [Ca2+]i in fibers from the outer cort

265 Exposure of the isolated fibers to Ringer's solution (containing 2 mM Ca2+) led to a monoex

266 Addition of INS37217 to Ringer's solution bathing the apical membrane transientl

267 fluid drainage rate was reduced relative to Ringer solution (P < 0.001, ANOVA) but increased steeply

268 H (pHi) or Na(+) ([Na(+)](i)) in response to Ringer solutions with/without B(OH)(4)(-) or HCO(3)(-) a

269 When the cilia were returned to Ringer solution after a period in reduced-Na(+) solution

270 Colloids in solutions similar to Ringer's lactate ('balanced solutions') may avoid these

271 of four times the volume of shed blood using Ringer's lactate solution for 60 mins.

272 ly 90 mins, and were then resuscitated using Ringer's lactate.

273 d storage (14% survival with CRS vs. 0% with Ringer's solution).

274 The endothelium was bathed with Ringer's fluid, and the outer surface was covered with s

275 s with non-cerebral trauma should begin with Ringer's lactate solution.

276 sites were then heated to 42 degrees C, with Ringer solution infused in one probe and N-nitro-L-argin

277 on of the isolated rat lens fiber cells with Ringer's solution led to their globulization in 30 +/- 3

278 treated animals (six of seven) compared with Ringer's acetate (two of seven), 10% pentastarch (one of

279 Compared with Ringer's lactate, saline, and saline-based colloids are

280 Compared with Ringer's solution (control), microdialysis delivery of t

281 with kidneys flushed with CRS compared with Ringer's solution or nontreatment.

282 extran 2000 reduced flows in comparison with Ringer solution.

283 were isolated typically after flushing with Ringer's buffer.

284 001), although no correlation was found with Ringer's lactate solution.

285 and perfused at a rate of 5 microL/min with Ringer's solution.

286 t a holding potential of +40 to +60 mV (with Ringer's in the pipette and pseudointracellular solution

287 milar average mass (2000 kDa; n = 7) or with Ringer solution vehicle (n = 2).

288 ion, microdialysis fibres were perfused with Ringer solution (control), a ATP-sensitive potassium cha

289 ays, microdialysis fibres were perfused with Ringer solution (control), a non-specific NO synthase in

290 nto the knees of anaesthetized rabbits, with Ringer solution as control in the contralateral joint.

291 auma sham-shock (T/SS) and resuscitated with Ringer's lactate or hypertonic saline.

292 The animals were resuscitated with Ringer's lactate solution to maintain constant pulmonary

293 ion with that of standard resuscitation with Ringer's lactate.

294 Resuscitation with Ringer's solution resulted in a standard base excess, an

295 magnitude when the cell was superfused with Ringer solution during the 5 s interval between odour ex

296 T:(g) of fiber cells superfused with Ringer's solution containing 2 x 10(-)(3) M, 10(-)(6) M,

297 after CRS was significantly higher than with Ringer's solution or without flushing (80% vs. 25% and 1

298 than in mice with pancreatitis treated with Ringer's lactate solution.

299 Treatment with Ringer's ethyl pyruvate solution ameliorated structural

300 d with four times the shed blood volume with Ringer's lactate solution over 60 minutes.