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1 S. aureus and E. coli antigens were detected in immune-b
2 S. aureus bacteremia is often associated with an adverse
3 S. aureus biofilms showed less susceptibility to killing
4 S. aureus colonizes the skin of the majority of children
5 S. aureus increases biofilm formation in response to hyp
6 S. aureus infection in the intensive care unit (ICU) mos
7 S. aureus is internalized by Cftr-deficient macrophages
8 S. aureus nasal carriage prevalence was higher among IHO
9 S. aureus strains from clonal complexes 1 and 8 were mor
10 S. aureus toxins and virulence proteases often circulate
11 S. aureus uses secreted cyclic autoinducing peptides (AI
12 S. aureus was isolated from 527 participants (67.0%), an
15 l cells, abundance changes for more than 400 S. aureus proteins were quantified, revealing, e.g., the
17 pid (<1h) detection of P. aeruginosa (6294), S. aureus(LAC), through on-chip electrical sensing of ba
22 loss of a single transporter did not affect S. aureus However, disruption of any two systems signifi
24 latelets participate in host defense against S. aureus both through direct killing of S. aureus and e
25 elets to participate in host defense against S. aureus infection was determined by assessing two poss
29 mily is indispensable for protection against S. aureus infection and its clearance at wound sites.
32 blish long-term protective Ab titers against S. aureus was not a consequence of diminished formation
33 a flavone rich extract "430D-F5" against all S. aureus accessory gene regulator (agr) alleles in the
36 ulate interactions between P. aeruginosa and S. aureus We demonstrate that P. aeruginosa quorum sensi
40 d selectively differentiate both E. coli and S. aureus infections from sterile inflammation in vivo.
41 imilarity of S. aureus colonizing humans and S. aureus in meat from the stores in which those individ
45 mice exhibited abnormal abscess formation at S. aureus-infected skin wound sites and were also more s
46 ted cefoxitin disk diffusion for 37 atypical S. aureus isolates (156 readings) with MHA supplemented
47 (HDM) has shown that Staphylococcus aureus (S. aureus) and Escherichia coli (E. coli) species are ab
48 -inflammatory role in Staphylococcus aureus (S. aureus) and methicillin-resistant S. aureus (MRSA) in
51 zation of the skin by Staphylococcus aureus (S. aureus) is increased in atopic dermatitis and can res
54 geted small interfering RNA silencing before S. aureus exposure blocked the increase in protease acti
55 have previously shown an association between S. aureus carriage and severe allergic disease and aller
58 of S. aureus The downregulation of IP-10 by S. aureus was mediated by components of its cell wall, b
59 , these results demonstrate an adaptation by S. aureus to obesity/T2D with increased expression of cl
62 tic of atopic eczema (AE) is colonization by S. aureus, with exacerbations associated with an increas
65 e trends in the annual rates of infection by S. aureus subtypes and mean antibiotic resistance, we co
66 tients (approximately 38%) colonized only by S. aureus and treated with appropriate antibiotic for at
67 trate that lipoyl-E2-PDH is also released by S. aureus and moonlights as a macrophage immunosuppressa
68 hese observations indicate that Pi uptake by S. aureus differs from established models and that acqui
69 on of peptidoglycan, a mechanism utilized by S. aureus to block bacterial cell wall breakdown, limits
72 Colonization densities of M. catarrhalis, S. aureus, and P. jirovecii are unlikely to be of diagno
73 ionally, neutrophils, essential for clearing S. aureus, demonstrated sex-specific S. aureus bacterici
78 rstand the molecular mechanisms that control S. aureus biofilm formation and the basis for the recalc
79 echanisms by which miR response to cutaneous S. aureus contributes to DFU pathophysiology are unknown
80 ounds infected with an isogenic AT-deficient S. aureus strain was unimpeded, exhibiting efficient bac
81 re challenged with an isogenic SpA-deficient S. aureus mutant, cells proliferated in the BM survival
83 experiments employing alpha-toxin-deficient S. aureus and the corresponding wild-type strain reveal
84 Mice were infected i.v. using 8 different S. aureus strains, and development of the infection was
88 and was also highly effective in eradicating S. aureus biofilm infection when used in a CLS rat centr
89 In galectin-3(+/+) mice, SspB-expressing S. aureus caused larger lesions and resulted in higher b
90 to bacterial virulence in vivo, since fewer S. aureus subsp. aureus NCTC8325-4 DeltasdrD bacteria th
92 provided a nasal swab which was analyzed for S. aureus, methicillin-resistant S. aureus (MRSA), multi
94 formative density cutoffs were not found for S. aureus and M. catarrhalis, and a lack of confirmed ca
95 ere determined to be 0.06 to 0.25 mug/ml for S. aureus ATCC 29213, 0.016 to 0.12 mug/ml for E. faecal
96 on limits as low as 7, 40 and 100 CFU/mL for S. aureus in pure broth culture, and inoculated in food
97 ranges were determined to be 25 to 31 mm for S. aureus ATCC 25923, 25 to 31 mm for S. pneumoniae ATCC
101 y structure of the native 100S ribosome from S. aureus, revealing the molecular mechanism of its form
103 MCL in sepsis caused by G(+) bacteria (e.g., S. aureus) and antibiotic-resistant bacteria (e.g., MRSA
106 enge model with a highly virulent agr type I S. aureus isolate, PP7-AIP1S vaccination reduced pathoge
107 novel control approaches, we have identified S. aureus components that are required for growth in hum
111 taphylococcus aureus protein A, an important S. aureus virulence factor involved in immune evasion an
113 o identify a functional adenylate cyclase in S. aureus and only detected 2',3'-cAMP but not 3',5'-cAM
114 are institutions in Boston, MA, a decline in S. aureus infections has been accompanied by a shift tow
116 ocesses involved in microcolony formation in S. aureus and suggests that these structures originate a
119 e show here that WTA is directly involved in S. aureus strain-specific virulence and provide insight
120 identified a new subset of MDSC (Eo-MDSC) in S. aureus-infected mice that phenotypically resembles eo
121 a programmed cell lysis (PCL) phenomenon in S. aureus leading to the release of cellular polymers th
122 novel information on staphopains present in S. aureus biofilms in vivo, and illustrate the complex i
126 mall dual-function regulatory RNA, RNAIII in S. aureus, that controls expression of key virulence fac
128 ess the further function of SfaA and SbnD in S. aureus fitness, we tested its effect on murine absces
129 -oxide (HQNO) induces multidrug tolerance in S. aureus through respiratory inhibition and reduction o
130 rculosis; mannitol, with selective uptake in S. aureus and E. coli; and sorbitol, accumulating only i
131 nical course, and outcome between individual S. aureus-infected ICU patients remains enigmatic, sugge
133 subsets were more efficient at internalizing S. aureus and B. anthracis compared with E. coli Alveola
134 e current study, we used lethally irradiated S. aureus as a model multicomponent vaccine and showed t
135 otal of 658 Staphylococcus species isolates (S. aureus, 211 isolates; S. lugdunensis, 3 isolates; and
136 inosa to produce virulence factors that kill S. aureus These data could provide important clues regar
137 determined the ability of platelets to kill S. aureus directly; and, second, we tested the possibili
138 iffraction and that it is sufficient to kill S. aureus These results suggest that, in addition to its
142 kill Staphylococcus aureus Further, a mutant S. aureus that is more sensitive to antimicrobial peptid
146 compared the ability of PBP2a and two native S. aureus transpeptidases to cross-link peptidoglycan st
151 siderophore SA contributes to the ability of S. aureus to replicate in abscesses and epithelial cells
154 investigate the time-resolved adaptation of S. aureus to the intracellular niche in human bronchial
158 Results of semiquantitative analyses of S. aureus burden in serial endotracheal-aspirate (ETA) s
159 ant difference in the genetic backgrounds of S. aureus colonizing AE cases versus controls (Fisher ex
160 they reduced the biofilm bacterial burden of S. aureus (CFU cm(-2)) by three logs with no statistical
164 ed cysteine proteases, and the dependence of S. aureus on L27 cleavage by Prp validates the enzyme as
165 form is a powerful tool for the detection of S. aureus as a potential point-of-care diagnostic platfo
166 e this, the origins and genetic diversity of S. aureus colonizing individual patients during AE disea
169 inhibit heme transfer to IsdB and growth of S. aureus, and a ternary complex of IsdB.Hb.Hp was obser
170 receptor CD36 reduced the internalization of S. aureus RN6390 by A549 cells, but the dependence on CD
173 nst S. aureus both through direct killing of S. aureus and enhancing the antimicrobial function of ma
174 ge phagocytosis and intracellular killing of S. aureus In this study we report evidence in support of
175 nosa LasA endopeptidase potentiates lysis of S. aureus by vancomycin, rhamnolipids facilitate proton-
181 g knowledge of the molecular pathogenesis of S. aureus disease, we suggest that the application of mo
182 as suppressed by the concomitant presence of S. aureus The downregulation of IP-10 by S. aureus was m
183 uggestive evidence of a higher prevalence of S. aureus, MRSA, and MDRSA among children living with an
185 l granulopoiesis and effective resolution of S. aureus-infected wounds, revealing a potential antibio
190 The golden pigment, staphyloxanthin, of S. aureus colonies distinguishes it from other staphyloc
192 or (in laboratory and AD clinical strains of S. aureus) inducing barrier integrity impairment and tig
194 We argue that whole-genome surveillance of S. aureus populations could lead to better forecasting o
195 This suggests that intracellular survival of S. aureus in macrophages may allow the pathogen to chron
198 that alpha-toxin, one of the major toxins of S. aureus, induces activation of acid sphingomyelinase a
200 is to summarize our current understanding of S. aureus biofilm development, focusing on the descripti
202 act of galectin-3 and protease expression on S. aureus virulence was studied in a murine skin infecti
204 y, the impact of interspecies interaction on S. aureus antibiotic susceptibility remains poorly under
205 taphylococcus aureus ATCC 25923 (disk only), S. aureus ATCC 29213 (broth only), Enterococcus faecalis
208 ted bacterial killing independently of other S. aureus proteins, since addition of recombinant SdrD p
210 aintain immune equilibrium and decrease PGN, S. aureus and MRSA-triggered inflammatory response.
212 ll-characterized collection of mecC-positive S. aureus isolates (n = 111) was used for evaluation.
214 throughout kidney tissue at early times post-S. aureus infection compared to antibiotic-treated but n
215 mong vaccine recipients in whom postsurgical S. aureus infection developed, emphasizing the potential
216 n why colonization of superantigen-producing S. aureus can induce, under some circumstances, mucosal
219 ibiotic treatment is ineffective in reducing S. aureus colonization in the lower airways and preventi
220 . aureus (MSSA) and HO methicillin-resistant S. aureus (MRSA) BSIs for 2009-2013 at 2 hospitals and u
222 The common USA300 methicillin-resistant S. aureus (MRSA) strains express a number of toxins, suc
223 o S. aureus, including methicillin-resistant S. aureus (MRSA) strains, despite high titers of specifi
224 nalyzed for S. aureus, methicillin-resistant S. aureus (MRSA), multidrug-resistant S. aureus (MDRSA),
225 tion of mecC-harboring methicillin-resistant S. aureus (MRSA), which failed to identify from 0 to 41%
226 e phagocytosis of both methicillin-resistant S. aureus and methicillin-sensitive S. aureus by >70%, a
228 ectrum beta-lactamase, methicillin-resistant S. aureus, and carbapenem-resistant strains was also obs
229 ncluding MDROs such as methicillin-resistant S. aureus, extended-spectrum beta-lactamase-producing, a
230 istant S. aureus (MRSA), multidrug-resistant S. aureus (MDRSA), absence of scn (putative marker of li
232 e measured the survival of wild-type and SCV S. aureus in whole human blood, which contains high numb
233 functional electron transport chains in SCV S. aureus and wild-type E. faecalis results in reduced g
234 esistant S. aureus and methicillin-sensitive S. aureus by >70%, and restricted intracellular growth b
236 of S. aureus colonization, we deep sequenced S. aureus populations from nine children with moderate t
239 riations showed that the ancestor of all ST8 S. aureus most likely emerged in Central Europe in the m
241 Large parts of ST8 methicillin-susceptible S. aureus (MSSA) isolated in Africa represent a symplesi
242 cC-harboring MRSA as methicillin-susceptible S. aureus This study underlines cefoxitin's status as th
245 Taken together, our results demonstrate that S. aureus secretes a unique proteinaceous MPO inhibitor
246 high-fat-diet obese/T2D mice and found that S. aureus infection was more severe, including increases
249 immortalized and primary keratinocytes, that S. aureus protease SspA/V8 is the dominant secreted fact
253 a prospective clinical trial that shows that S. aureus colonization precedes onset of atopic dermatit
255 ) importers, genomic analysis suggested that S. aureus possesses three distinct Pi transporters: PstS
256 of S. aureus biofilm of AD skin detected the S. aureus derived protease staphopain adjacent to the ba
258 associated with the clonal expansion of the S. aureus population, occurring over a period of weeks t
261 s mediated by a sex-specific response to the S. aureus-secreted virulence factor alpha-hemolysin (Hla
265 y of SdrD as an important key contributor to S. aureus survival and the ability to escape the innate
266 reased, further linking fibrin deposition to S. aureus expression of clfA and infection severity.
267 The high frequency of recurring SSSI due to S. aureus, including methicillin-resistant S. aureus (MR
268 on of molecular pathological epidemiology to S. aureus infection can usher in a new era of highly foc
271 ency in mice restores protective immunity to S. aureus infection, and adjuvancy with a staphylococcal
272 vated to expand PMN numbers in proportion to S. aureus abundance in a manner regulated by TLR2 and IL
273 keratinocyte Myd88 signaling in response to S. aureus PSMalpha drives an IL-17-mediated skin inflamm
274 10 in a TLR2-dependent manner in response to S. aureus, and adoptive transfer of B1a cells was protec
277 us (S. aureus) carriage and sensitization to S. aureus enterotoxins (SEs) have been associated with a
279 ave potential as therapeutic agents to treat S. aureus infections, and purification of the transmembr
282 oresis (PFGE) for typing S. aureus Forty-two S. aureus isolates from three outbreaks and 12 reference
284 ficiently by IFN-beta than was the wild-type S. aureus, and immunoblotting showed that IFN-beta inter
285 -field gel electrophoresis (PFGE) for typing S. aureus Forty-two S. aureus isolates from three outbre
286 nversion of approximately 25% of the in vivo S. aureus mono-culture essential genes to non-essential.
288 rall, our study suggests a mechanism whereby S. aureus modulates cytokines critical for induction of
289 efines a previously unknown pathway by which S. aureus epicutaneous exposure promotes skin inflammati
291 ns, thus expanding the environments in which S. aureus can successfully obtain Pi Consistent with thi
295 odeficient MyD88-knockout mice infected with S. aureus experienced lethal sepsis that was reversed by
296 ls under conditions mimicking infection with S. aureus conferred responsiveness to IL-20 that manifes
298 hly susceptible to pulmonary infections with S. aureus and fail to clear the pathogen during infectio
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