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1                                              S. marcescens does not normally colonize human skin, but
2                                              S. marcescens expresses prodigiosin, a bright red and ce
3                                              S. marcescens induced neutrophil recruitment to the corn
4                                              S. marcescens induces corneal inflammation by activation
5                                              S. marcescens isolates were compared using restriction-e
6                                              S. marcescens oxyR mutants were severely impaired in bio
7                                              S. marcescens was identified from a pharmacy water fauce
8 isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and
9 ene knockout mice was abraded, and 1 x 10(7) S. marcescens were added in the presence of a silicone h
10                  It has been reported that a S. marcescens chimera, SM : rS5'ec, in which five diverg
11         The activity of the fraction against S. marcescens was explained by (R)-(-)-mellein alone, an
12 tic (R)-(-)-mellein and micromolide, against S. marcescens and a Gram-positive bacterium, Staphylococ
13 tion of bronchoscopes with P. aeruginosa and S. marcescens and possible infection of patients at a co
14 ducted an investigation of P. aeruginosa and S. marcescens isolates related to bronchoscopy at a comm
15 aerogenes, K. pneumoniae, P. aeruginosa, and S. marcescens) became more susceptible.
16  with Acinetobacter spp., P. aeruginosa, and S. marcescens, 5/6 with Citrobacter spp., 13/14 with Ent
17  seven of these had Enterobacter cloacae and S. marcescens in the same culture.
18 nce birth, but gut colonization with GBS and S. marcescens occurred closer to time of bloodstream inf
19                        Cases were defined as S. marcescens BSIs in patients receiving PN from the pha
20  its transporter ShlB resulted in attenuated S. marcescens strains that failed to cause profound weig
21 eement was observed, excluding A. baumannii, S. marcescens, and S. pneumoniae, for which >/=4-fold di
22 n foreskin fibroblasts was also inhibited by S. marcescens secretomes indicating that the effect is n
23 , that is located upstream of NucC-dependent S. marcescens promoters and the late promoters of P2-rel
24 P, and prodigiosin concentration changes for S. marcescens during cultivation in batch culture.
25                     Several risk factors for S. marcescens infection were identified, but hospital an
26                            Purified LPS from S. marcescens, but not from Escherichia coli or S. marce
27 roduce SlpB as a new cytotoxic protease from S. marcescens.
28 nd interleukin-1 receptor type 1 (IL-1R1) in S. marcescens-induced corneal inflammation and infection
29 itro cytotoxic activity commonly observed in S. marcescens culture filtrates.
30 nergy-dependent and Sec-dependent pathway in S. marcescens.
31 echanism for the second step of secretion in S. marcescens.
32                            Tobramycin-killed S. marcescens induced corneal inflammation in C57BL/6 mi
33 encia burhodogranaria, is gram negative like S. marcescens.
34       R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. muenchen) do
35 less, the temperature response of the native S. marcescens ATCase suggests a strong entropic effect t
36 ase treatments abrogated the cytotoxicity of S. marcescens culture filtrates towards HeLa cells, sugg
37                             LPS depletion of S. marcescens secretomes with polymyxin B agarose render
38    Despite multiple clinical descriptions of S. marcescens nosocomial pneumonia, little is known rega
39 ective means to control the dissemination of S. marcescens, an in-depth analysis of the population st
40 ential therapeutic targets for inhibition of S. marcescens-induced corneal inflammation.
41              This study describes a model of S. marcescens pneumonia that mimics known clinical featu
42      A case was defined as the occurrence of S. marcescens bacteremia in any patient in the surgical
43                               An outbreak of S. marcescens and E. cloacae bacteremia in a surgical in
44 ding standards contributed to an outbreak of S. marcescens BSIs.
45 tify any environmental or staff reservoir of S. marcescens.
46 in which OxyR contributes to early stages of S. marcescens biofilm formation by influencing fimbrial
47  first reports applying PFGE to the study of S. marcescens, and this method was a useful marker of st
48 istant clones suggests that the treatment of S. marcescens infections will become increasingly diffic
49 marcescens, but not from Escherichia coli or S. marcescens strains with mutations in the waaG and waa
50 colonized with provisionally matching GBS or S. marcescens.
51 for Fis are 100% identical in K. pneumoniae, S. marcescens, E. coli, and S. typhimurium and 96 to 98%
52 ematic collection of antimicrobial-resistant S. marcescens associated with bloodstream infections in
53 what is observed in other bacterial species, S. marcescens OxyR is required for oxidative stress resi
54 eal aspiration model of lethal and sublethal S. marcescens pneumonia in BALB/c mice and extensively c
55 ability of the tigecycline Etest for testing S. marcescens, Acinetobacter spp., and S. pneumoniae is
56 or in vitro cytotoxic activity revealed that S. marcescens mutant strains that are deficient in produ
57                        Our results show that S. marcescens is a diverse species with a high level of
58             Together these data suggest that S. marcescens LPS is sufficient for inhibition of epithe
59       Finally, recombinant expression of the S. marcescens 56-kDa metalloprotease conferred a cytotox
60 CTP stimulates the catalytic activity of the S. marcescens ATCase and CTP/UTP inhibitory synergism ha
61  measured differences in processivity of the S. marcescens chitinases.
62                Bioinformatic analysis of the S. marcescens Db11 genome revealed three additional open
63                      One crystal form of the S. marcescens enzyme displays a bound pyruvate as well a
64 93-r97) of the regulatory polypeptide of the S. marcescens enzyme have been replaced with their E. co
65 ) retained 455 out of 460 amino acids of the S. marcescens enzyme, it possessed characteristics simil
66 n E. coli for extracellular secretion of the S. marcescens nuclease.
67  proteins are the original substrates of the S. marcescens T6SS, before horizontal acquisition of oth
68 yperthermophile Sulfolobus solfataricus, the S. marcescens structure shows similar subunit structures
69                          NucC binding to the S. marcescens nuclease promoter P(nucA) and to the seque
70 ues of E. coli have been replaced with their S. marcescens counterpart, lost both heterotrophic and h
71                      Literature pertinent to S. marcescens-mediated necrotizing fasciitis is also rev
72 (-/-), and TLR4/5(-/-) corneas infected with S. marcescens had significantly increased CFU, indicatin
73 itis of the chest wall due to infection with S. marcescens that initially manifested as bilateral bre
74 ed insect survival after oral infection with S. marcescens.
75 ording to univariate analysis, patients with S. marcescens bacteremia stayed in the surgical intensiv
76                     Twenty-six patients with S. marcescens bacteremia were identified; eight (31 perc
77 tify risk factors, we compared patients with S. marcescens bacteremia with randomly selected controls
78 were positive for P. aeruginosa also yielded S. marcescens.
79 nyl infusions from two case patients yielded S. marcescens and E. cloacae.

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