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1 contribute to the thrombogenic potential of S. sanguis.
2 we have also investigated the F-ATPase from S. sanguis.
3 gesting additional mechanisms of response to S. sanguis.
4 und to hydroxyapatite to promote adhesion of S. sanguis.
5 model of platelet aggregation in response to S. sanguis.
6 s with strains of Streptococcus gordonii and S. sanguis.
7 nctional adhesion epitopes on the surface of S. sanguis.
8 uced to the same extent in the nonpathogenic S. sanguis.
10 ms by Western blot indicated that S. oralis, S. sanguis, A. viscosus, A. odontolyticus, and A. israel
14 hypothesis that aggregation occurs in vivo, S. sanguis (Agg+ or Agg- suspension) was infused intrave
15 in salivary films to form binding sites for S. sanguis, an in vitro model of saliva-coated teeth was
17 y is induced under acidic conditions in both S. sanguis and S. mutans; however, it is not induced to
18 harides occurred primarily on the strains of S. sanguis and S. oralis while G-containing polysacchari
21 mutans and Streptococcus sanguinis (formerly S. sanguis), as a model to probe the possible mechanisms
24 c activity expressed by S. mutans VA-29R and S. sanguis ATCC 10556 against X-Pro4-nitroanilide and X-
25 s provide evidence that S. mutans VA-29R and S. sanguis ATCC 10556 possess a pathway for the complete
28 mmune arthritis, DBA/1J pups were given live S. sanguis, CB11, or type II collagen intragastrically.
30 atpD deletion strain of E. coli showed that S. sanguis-E. coli hybrid enzymes were able to degrade A
32 specific antibodies or peptides block PAAP, S. sanguis fails to induce platelet aggregation in vitro
33 are consistent with a thrombogenic role for S. sanguis in human disease, contributing to the develop
36 Analysis of starch degradation products from S. sanguis IUOM-11M and A. viscosus IUOM-62 demonstrated
38 ose inhibited sucrose-dependent synthesis of S. sanguis IUOM-11M insoluble polysaccharide and both pr
39 We therefore hypothesized that circulating S. sanguis might cause coronary thrombosis and signs of
42 xhibited by selected strains of S. gordonii, S. sanguis, S. mutans, S. mitis, and S. oralis but only
44 um, P. gingivalis, P. intermedia, S. mutans, S. sanguis, Selenomonas sputigena, T. denticola, and T.
47 roliferated more when incubated with PAAP(+) S. sanguis than with PAAP(-) Streptococcus gordonii or t
48 reparations from S. aureus, B. subtilis, and S. sanguis were not able to induce NO from lipopolysacch
49 region with 10 tandem repeats of a 20-mer in S. sanguis, which is replaced by a similar but less repe
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