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1 (S.D.+/-0.7) in the 15 to 24 age group; 3.0 (S.D.+/-1.5) in the 45 to 54 age group; and 3.02 (S.D.+/-
6 he scaling exponents (from 0.73 +/- 0.11 (+/-S.D., session 1), 0.72 +/- 0.10 (session 2) and 0.75 +/-
12 ight-handed men (mean age 30.5, range 18-37, S.D. 6.5 years) given the same olfactory nerve stimuli i
13 women (mean age 25.3 years old, range 20-44, S.D. 8.3 years) were compared with those of 8 right-hand
14 uated increase in HR (7 +/- 4% vs. 13 +/- 7% S.D.), CO (10 +/- 6% vs. 18 +/- 8%) and femoral blood fl
15 NA increased from supine to upright (17+/-8 (S.D.) versus 38+/-12 bursts min-1; P<0.01), and continue
17 metabolism) in 11 old (71 +/- 9 years of age S.D.) and 11 young (24 +/- 2 years of age) healthy subje
18 normotensive Caucasian adults (mean age (+/- S.D.) = 29 +/- 6 years), who were either Arg16 (n = 18)
21 .5 ata had better mean+/-standard deviation (S.D.) behavioral scores (14+/-2; p<0.05) than control (1
23 h more variable beat-to-beat intervals (mean S.D. of beat-to-beat intervals was 0.027 s/beat in contr
26 hot plate test (52 degrees C): the mean (+/-S.D.) hind paw lick latency of rats in the high anti-NGF
27 he intracellular pH, 7.54 +/- 0.03 (mean +/- S.D., n = 15), determined with the Neutral red method, o
28 engths s-1): type I, 0.54 +/- 0.05 (mean +/- S.D., n = 3); type IIA, 1.23 +/- 0.34 (n = 27); type alp
33 x)) was 16 871 +/- 2941 mmHg s(-1) (mean +/- S.D.) and the relaxation time constant was 3.9 +/- 0.8 m
35 d a PCr/NTP ratio of 0.93 +/- 0.11 (mean +/- S.D.) using a 2 s interpulse interval, a value very clos
36 ns) (neurobehavior index 43 +/- 19, mean +/- S.D.) compared to Group 1 rats, in which only spontaneou
37 out 1) and 40.7 +/- 8.4 s (bout 2) (mean +/- S.D.) and (b) the magnitude of the later slow component
38 ifferentiation by 87 +/- 7% (n = 2, mean +/- S.D.) but had no effect on 1,25-(OH)2D3 inhibition of ce
39 ticulum (ER) (pH(ER) = 7.4 +/- 0.2, mean +/- S.D.) to Golgi (pH(G) = 6.2 +/- 0.4) to mature secretory
41 intrinsic stiffness is 91 +/- 23 % (mean +/- S.D.) of that necessary to provide minimal stabilisation
46 both breath amplitude (152 +/- 73%; mean +/- S.D.; P = 0.004) and frequency (46 +/- 37%; P = 0.049).
47 lderly subjects were studied with a mean +/- S.D. age and body mass index (BMI) of 71 +/- 5 years and
50 4.8 kg and height 163.4 +/- 9.1 cm, mean +/- S.D.) and control (n = 9; age 67.1 +/- 2 years, body mas
52 CAO; 31 +/- 3.7 mm3 1 h after MCAO (mean +/- S.D.) when administered intracranially up to 60 min post
53 rd resting levels by 20 +/- 15 min (mean +/- S.D.) post-transection, except in regions of the axon wi
55 aseline [Na+]i was 14.6 +/- 4.9 mM (mean +/- S.D.) in CO2/HCO3(-)-containing saline with 3 mM K+.
58 ecording, E(GABA) was -25 +/- 5 mV (mean +/- S.D.), and was stable between embryonic day 17 and post-
61 nstant, tau fast, of 0.9 +/- 0.2 s (mean +/- S.D.) and a later, slow time constant, tau slow, of 4.2
62 ase (p < 0.05) in diabetic samples (mean +/- S.D., 313.8 +/- 52.7 pmol/mg protein) when compared with
64 male subjects (age 29 +/- 7 years, mean +/- S.D.) performed three transitions to a work rate (WR) co
65 ive subjects (age, 40 +/- 10 years: mean +/- S.D.), orthostatic tolerance was assessed using graded l
66 ated 3.9+/-1.7 mm and 6.1+/-3.4 mm2 (mean +/-S.D.) of linear bone height and bone area, respectively.
67 und after versus before activation (mean =/- S.D.; n = 3) was: for AP7.3, 9.8 =/- 0.6; for PAC1.1, 8.
68 veloped larger infarcts (40.1+/-1.0%, mean+/-S.D., p<0.001) than MS- or aCSF-treated controls (34.3+/
69 infarct volume of 178.7+/-84.2 mm(3) (mean+/-S.D.) with a large coefficient of variation (47%) and ra
70 (178.7+/-84.2 vs. 243.3+/-50.1 mm(3), mean+/-S.D., Student's t-test P=0.046) and showed a greater var
72 n CBF of the SHR (1.5+/-0.2 ml/g/min, mean+/-S.D.) was approximately 25% higher than that of the WKY
73 Similarly, total infarct volumes (mean+/-S.D.) were smaller in animals receiving HBO at 2.5 ata (
74 ntrol subjects aged 15.2+/-0.5 years (mean+/-S.D.) and eighteen IA subjects aged 15.1+/-1.4 years wer
76 haemorrhage for 18.0 +/- 4.6 days (means +/- S.D.) during which time the haematocrit was reduced from
78 eter of 1.6 microm (range = 0.11-7.4 microm, S.D. = 1.01, n = 311) with a small percentage (8%) being
80 ed seizure latency from about 17+/-8 min (+/-S.D.) in controls to 5+/-1 and 6+/-2 min, respectively.
81 The mean distance CEJ-bone level was 1.4 mm (S.D.+/-0.7) in the 15 to 24 age group; 3.0 (S.D.+/-1.5)
86 trated to be highly reproducible since the R.S.D. for 10 successive amperometric calibrations using t
88 averaging: tauV(O(2))(,on) 35 +/- 14 s (+/- S.D.), tau[PCr](on) 33 +/- 12 s, tauV(O(2))(,off) 50 +/-
95 - 8 dendritic profiles (group mean value +/- S.D., five animals), whereas clusters in layer 2 contain
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