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1                                              SAF also significantly lowered fasting glucose (P = 0.03
2                                              SAF is a potentially valuable clinical screening tool fo
3                                              SAF partially mediated the association between T2DM and
4                                              SAF was classified into two types: type 1, which was fil
5                                              SAF-1 activity was detected in the chondrocytes of OA ca
6                                              SAF-1 contains several negative and positively functioni
7                                              SAF-1 DNA-binding activity is increased in both cytokine
8                                              SAF-1 is a 477-amino acid protein with six zinc fingers.
9                                              SAF-1, a zinc finger transcription factor, is activated
10                                              SAF-1-overexpressing mice spontaneously developed AA amy
11                                              SAF-A oligomerization decompacts large-scale chromatin s
12                                              SAFs for photons were generated for mildly and severely
13                                              SAFs of W-CIN cells were remarkably similar to those ind
14                                              SAFs studied between the obese phantoms and the 50th per
15 role of serum amyloid A-activating factor 1 (SAF-1) in MMP-1 expression was assessed by transient tra
16 factor, serum amyloid A-activating factor 1 (SAF-1), has been shown to regulate several genes, includ
17  factor serum amyloid A-activating factor-1 (SAF-1) has been identified as a regulator of a number of
18         Serum amyloid A-activating factor-1 (SAF-1) is a zinc finger transcription factor that is act
19   Serum amyloid A (SAA) activating factor-1 (SAF-1) is an inducible transcription factor that plays a
20 amyloid A-activating transcription factor-1 (SAF-1) plays a major role in regulating transcription of
21 factor, serum amyloid A activating factor-1 (SAF-1), leading to markedly higher levels of angiogenesi
22        Of the three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of
23 ulated with various adjuvants: QS-21, IL-12, SAF-1, CD40L, and GM-CSF were compared.
24   These include NF-kappaB, C/EBP, YY1, AP-2, SAF and Sp1.
25  cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of homology to MAZ/ZF
26 nesis of AA amyloidosis, we have developed a SAF-1 transgenic mouse model.
27 ly higher DNA binding activity than either a SAF-1 homodimer or a c-Fos/c-Jun heterodimer.
28                Scaffold attachment factor A (SAF-A), also called heterogenous nuclear ribonuclear pro
29 we report that scaffold attachment factor A (SAF-A), originally identified as a structural nuclear pr
30 oantigens, and scaffold attachment factor A (SAF-A).
31 hosphorylation sites created a highly active SAF-1 protein with high DNA-binding ability.
32  that reentry is important to maintain acute SAF.
33 important in maintaining stretch-induced AF (SAF).
34                                     Although SAF is expressed in all lymphocytes, immunofluorescence
35                                     Although SAF-1 transgenic mice do not spontaneously develop arthr
36 e SAA promoter in association with SAF as an SAF.Sp1 heteromeric complex.
37                                To develop an SAF teledermatology workflow within the Epic system, the
38 transfected cells increased expression of an SAF-regulated promoter.
39 her levels of matrix metalloproteinase-1 and SAF-1 in the inflamed joints of SAF-1 transgenic mice co
40 on responsive transcription factors AP-1 and SAF-1 synergistically regulate transcriptional induction
41                            QS-21, IL-12, and SAF-1 biased the humoral immune response toward Th1-type
42 gy to MAZ/ZF87/Pur-1 protein while SAF-5 and SAF-8 isoforms are unique and are related to SAF-1/MAZ/Z
43 he three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of homology t
44  same slides by using the FLIP algorithm and SAF score, blinded to their first evaluation.
45 tains the tumor suppressors SAFB1, BRG1, and SAF-A.
46 MP-dependent protein kinase (PKA-Calpha) and SAF-1.
47 ctural component of articular cartilage, and SAF-1 in both SAF-1 transgenic and nontransgenic mice.
48                 Supplementation with CLA and SAF exerted different effects on BMI, total and trunk ad
49  benefits of beam spinning EW excitation and SAF detection and provides the conditions for quantitati
50 P-1 family of proteins, c-Fos and c-Jun, and SAF-1 form a ternary protein complex, which has markedly
51                The combination of SP-PCR and SAF microlens array allows for on-chip highly sensitive
52 ed transactivation potential of both Sp1 and SAF as a consequence of a phosphorylation event.
53 own to regulate microtubule organization and SAFs known to promote microtubule assembly such as Maski
54 ntaining either a functional or an antisense SAF-1 complementary DNA.
55 ce of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibited interleukin-1-mediated MMP
56 is regulated by a posttranslational event as SAF DNA-binding and transactivation abilities are increa
57 ng the function of spindle matrix-associated SAFs.
58          Non-invasive skin autofluorescence (SAF) measurement serves as a proxy for tissue accumulati
59 ests, and noninvasive skin autofluorescence (SAF; a measure of tissue AGE levels) on people aged >55
60                                   Epic-based SAF teledermatology can improve access to dermatologic c
61             The physical interaction between SAF-1 and AP-1 was demonstrated both in vitro by Far-Wes
62 ealed colocalization and interaction between SAF-1 and PKA-Calpha.
63 efficiently promotes transcription from both SAF-1 and AP-1 sites of human MMP-1 promoter.
64 nt of articular cartilage, and SAF-1 in both SAF-1 transgenic and nontransgenic mice.
65 omised the transactivation potential of both SAF-1 and AP-1.
66 es additional layers of regulation, and both SAFs are only degraded after being released from their i
67 or protein p21 was significantly affected by SAF-1; its expression level was highly induced by cellul
68               Of the three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high de
69               In this report, using a cloned SAF gene in transient transfection assay, we provide evi
70 rvation, increased DNA binding of the cloned SAF and its hyperphosphorylation, in response to okadaic
71                          Of the three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showe
72                                          CMV-SAF-1 transgenic mice, upon B. burgdorferi infection, sh
73                                 Conceivably, SAF-1 could be a potential therapeutic target to control
74                                 In contrast, SAF had no effect on BMI or total adipose mass but reduc
75  health care organizations wanting to create SAF teledermatology workflows within the Epic EHR system
76 ion of SAF-1 protein from either end creates SAF-1 isoforms that are highly transcriptionally active.
77 he most influential variables in determining SAF values in men, whilst in female participants, SR was
78      NUSAP1 contains a conserved SAP domain (SAF-A/B, Acinus, and PIAS).
79  whose expression levels were altered during SAF-1 overexpression.
80            This result suggested that C/EBP, SAF, and NF-kappa B are required for transient acute pha
81                          Photon and electron SAFs were then calculated for relevant organs in all mod
82 sentative plots were made of photon electron SAFs, evaluating the traditional assumption that all ele
83       The DNA binding activity of endogenous SAF was increased by agonists of PKC.
84 MP1 promoter, and interference of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibite
85                Phosphorylation of endogenous SAF-1 in response to IL-1 and IL-6 was markedly inhibite
86  promoter as well as knockdown of endogenous SAF-1 markedly inhibited IL-1beta- and TGF-beta-mediated
87                   Moreover, cells expressing SAF-A in which serine 59 is mutated to alanine have mult
88 , in addition to tissue-specific expression, SAFs, a family of zinc finger transcription factors, und
89    These data show that transcription factor SAF-1 is involved in the regulation of IL-6-mediated ind
90 n that the inflammatory transcription factor SAF-1 is, at least in part, responsible for the marked i
91 inflammation-responsive transcription factor SAF-1 was found to interact with it.
92             Increased SAA-activating factor (SAF) activity was detected in the liver, lung, and brain
93 tor serum amyloid A (SAA)-activating factor (SAF), a family of zinc finger proteins, plays a signific
94 show that serum amyloid A-activating factor (SAF)-1, a novel transcription factor, and the AP-1 famil
95 n factor, serum amyloid A activating factor (SAF)-1, was demonstrated by EMSA.
96 , referred to as silencer-associated factor (SAF), is a member of the helix-turn-helix factor family
97 n, and a nuclear factor, SAS-binding factor (SAF), that interacts with the SAS element has been ident
98  and SAA activating sequence binding factor (SAF).
99 nflammation-responsive transcription factor, SAF (for SAA activating factor), has been implicated in
100 le matrix contains spindle assembly factors (SAFs) such as Eg5 and dynein which are known to regulate
101  of importin-bound spindle assembly factors (SAFs), which stimulate microtubule (MT) nucleation and o
102 id cells and senescence-associated features (SAFs).
103              We use a self-assembling fiber (SAF) system to form structures tens of micrometers long.
104 rization of a self-assembling peptide fiber (SAF) system based on alpha-helical coiled-coil building
105 m fillets and the Sparus aurata fibroblasts (SAF-1) cell-line during an 8day storage period at +4 deg
106 oring system (steatosis, activity, fibrosis [SAF]) allowing the use of an algorithm (fatty liver inhi
107 ation with supercritical-angle fluorescence (SAF) detection efficiently rejects the fluorescence orig
108 -PCR with super critical angle fluorescence (SAF) microlens array embedded in a microchip.
109 To identify possible activation partners for SAF-1, we used a yeast two-hybrid system that detected i
110 where most of the cells stained positive for SAF-1.
111 sis suggests that L(pro) contains a SAP (for SAF-A/B, Acinus, and PIAS) domain, a protein structure a
112 which one serves as the DNA-binding site for SAF-1 transcription factor.
113                  External store-and-forward (SAF) teledermatology systems operate separately from the
114 e of obesity on specific absorbed fractions (SAFs) and dose factors in adults.
115 ed to study how specific absorbed fractions (SAFs) vary with changes in adult body size, for persons
116 es to calculate specific absorbed fractions (SAFs) with internal photon and electron sources.
117 ta by first computing site allele frequency (SAF) likelihood for each site (i.e. the likelihood a sit
118 the task's speed-accuracy tradeoff function (SAF), which prevented us from falsely interpreting varia
119                                 Furthermore, SAF-1 transgenic mice rapidly developed severe AA amyloi
120 iotropic role of SAF-1 in vivo, we generated SAF-1 transgenic mice, in which CMV immediate-early prom
121                                      Greater SAF, T2DM, and cognitive impairment were each associated
122             T2DM was associated with greater SAF.
123  12 male patients; mean age, 20.1 years) had SAF type 1 (mean width, 5.2 x 4.5 mm).
124  46 male patients; mean age, 37.8 years) had SAF type 2 (mean width, 5.1 x 4.7 mm).
125                              To evaluate how SAF is involved in SAA promoter activation, we have inve
126 sent in the atherosclerotic plaque implicate SAF-1 as a key regulator of MMP-14 gene induction in mac
127 sly unreported and significant difference in SAF values between men and women, with median (range) va
128         Two distinct but adjacent domains in SAF-1 are involved in protein-protein contact with c-Fos
129 vidence that VEGF expression is increased in SAF-1-transgenic mice and that SAF-1 induces VEGF transc
130                               Differences in SAFs for organs irradiating themselves were between 0.5%
131                               Differences in SAFs for organs outside the trunk were not greater than
132 ther splicing regulatory proteins, including SAF-B, hnRNP G, YB-1, and p72.
133                    Also, during inflammation SAF-1 transgenic mice exhibited a prolonged acute phase
134                      Development of internal SAF workflows within existing electronic health records
135                                Although many SAFs are non-motile MT-associated proteins, such as NuMA
136                                     The mean SAF of the study cohort was 2.06 (SD = 0.57) arbitrary u
137                   We fabricated miniaturized SAF microlens array as part of a microfluidic chamber in
138                                          MPN-SAF TSS results significantly differed among MPN disease
139                                          MPN-SAF TSS was calculable for 1,408 of 1,433 patients with
140                           A total of 402 MPN-SAF surveys were administered (English [25%], Italian [4
141     Serial administration of the English MPN-SAF among 53 patients showed that most MPN-SAF items are
142 rative Neoplasm Symptom Assessment Form [MPN-SAF], coadministered with the Brief Fatigue Inventory) t
143 N-SAF among 53 patients showed that most MPN-SAF items are well correlated (r > 0.5, P < .001) and hi
144 tomatic elements represented on both the MPN-SAF and the European Organisation for Research and Treat
145                                      The MPN-SAF is a comprehensive and reliable instrument that is a
146                                      The MPN-SAF TSS had excellent internal consistency (Cronbach's a
147                                      The MPN-SAF TSS is a concise, valid, and accurate assessment of
148 nderlying construct, indicating that the MPN-SAF TSS is an appropriate, unified scoring method.
149                                      The MPN-SAF TSS strongly correlated with overall quality of life
150 h increase could be detected with the mutant SAF-1 proteins.
151 ograms (n = 6) or 250 micrograms (n = 20) of SAF-m.
152           Our findings identify serine 59 of SAF-A as a new target of both PLK1 and PP2A in mitosis a
153 ether, these results show that activation of SAF-1 in response to IL-1 and -6 is mediated via MAP kin
154 tribution of MAP kinase in the activation of SAF-1, we prepared two independent mutant proteins in wh
155 tion is involved in functional activation of SAF-1.
156 which PKA increases functional activities of SAF-1.
157                                  Activity of SAF is regulated by a phosphorylation event involving se
158                       Functional activity of SAF is regulated by a posttranslational event as SAF DNA
159 lation increases transcriptional activity of SAF-1 by unmasking the DNA-binding domain.
160 mostly increases the DNA binding activity of SAF-1.
161                         Increased binding of SAF-1 to the MMP-14 promoter correlated with the transcr
162  overexpression of SAF-1 and coexpression of SAF-1 and MMP-14 in the macrophages present in the ather
163 specific subcellular compartmentalization of SAF plays an important role in mediating the specificity
164    The data also demonstrate that control of SAF-1 activity can suppress induced expression of MMP-1.
165                                  Deletion of SAF-1 binding elements from the VEGF promoter as well as
166 thermore, we found that terminal deletion of SAF-1 protein from either end creates SAF-1 isoforms tha
167 oth phosphorylation and dephosphorylation of SAF-A serine 59 by PLK1 and PP2A, respectively, are requ
168 ed 293 patients with a lifetime diagnosis of SAF disorder, bipolar disorder and major depressive diso
169 n constructs, the core DNA-binding domain of SAF-1 is mapped between amino acids 282 and 361, which c
170                          In vivo evidence of SAF-1 and PKA-Calpha interaction was further revealed by
171           Prolonged high level expression of SAF-1 in cultured cells failed to produce any stable cel
172 ys a major role in controlling expression of SAF-regulated genes by increasing the interaction betwee
173                          To test the fate of SAF-1-overexpressing cells, the cells were monitored for
174                        The high frequency of SAF on MR arthrograms (10.5%), the absence of subchondra
175 e cells is primarily due to the induction of SAF activity.
176 PKA) and presented evidence for induction of SAF-1-regulated genes by a PKA signaling pathway.
177 Previously, we showed in vivo interaction of SAF-1 and protein kinase A (PKA) and presented evidence
178 einase-1 and SAF-1 in the inflamed joints of SAF-1 transgenic mice compared with their levels in nont
179        We present evidence that knockdown of SAF-1 by small interfering RNAs inhibits AP-1-mediated i
180 on assay, in vivo, markedly higher levels of SAF-1 interaction with the VEGF promoter was detected in
181 processing activity during overexpression of SAF-1 and coexpression of SAF-1 and MMP-14 in the macrop
182                   Although overexpression of SAF-1 could increase expression of the MMP-9 promoter an
183                            Overexpression of SAF-1 in OA chondrocytes increased the expression of the
184       Our study shows that overexpression of SAF-1 predisposes animals to arthritis.
185 ical conditions that favor overexpression of SAF-1, such as an acute inflammatory condition, can trig
186 on of MMP-9 gene concurrent participation of SAF-1 and AP-1 is required.
187  markedly induces in vivo phosphorylation of SAF-1 and transcription of SAF-regulated reporter genes.
188                  In vitro phosphorylation of SAF-1 by PKC-beta markedly increased its DNA binding abi
189 ably lowers the transactivation potential of SAF-1.
190 ng activity and transactivation potential of SAF-1.
191 ptional repression and active recruitment of SAF-1-mediated transcriptional activation.
192 chanism of synergy and the essential role of SAF-1 and AP-1 in up-regulating human MMP-1 expression u
193     Together these results suggest a role of SAF-1 in the pathogenesis of inflammation-induced arthri
194            To assess the pleiotropic role of SAF-1 in vivo, we generated SAF-1 transgenic mice, in wh
195 t under native condition, N and C termini of SAF-1 are engaged in an inhibitory intramolecular intera
196 hosphorylation of SAF-1 and transcription of SAF-regulated reporter genes.
197 gated linoleic acid (CLA) and safflower oil (SAF), on body weight and composition in obese postmenopa
198 g/d of ethyl esters of either safflower oil (SAF; control), eicosapentaenoic acid (EPA), or docosahex
199        Mechanistically, this is dependent on SAF-A's AAA(+) ATPase domain, which mediates cycles of p
200 bony fossa in the roof of the acetabulum, or SAF.
201 IR and steatosis, activity, and fibrosis (or SAF) scoring systems.
202  fluorescently labeled streptavidin (SAF) or SAF conjugated to biotinylated Cy3 adenoviral-vector (BC
203 duce any stable cell line that overexpresses SAF-1.
204 he cells that were programmed to overproduce SAF-1 were found to undergo growth arrest and reduce DNA
205 dy we provide evidence that, similar to p53, SAF-1 is able to activate p21 gene expression by promoti
206 were made to the reference (50th) percentile SAF values.
207 were made to the reference (50th) percentile SAF values.
208 tions of radiation transport were performed; SAFs for photons were generated for 10th, 25th, 75th, an
209 ted that unphosphorylated and phosphorylated SAF-1 proteins are structurally distinct.
210 tion between promoter DNA and phosphorylated SAF.
211 is, we asked whether DNA-PKcs phosphorylates SAF-A in mitosis.
212 alone or pHrodo complexed to E. coli, pHrodo-SAFs report pH in both the cytoplasm and phagosomes, as
213                       In the MMP-9 promoter, SAF-1 and AP-1 DNA-binding elements are present in close
214                  In the human VEGF promoter, SAF-1- and KLF-4-binding elements are overlapping, where
215 ctor-1/c-Myc-associated zinc finger protein (SAF-1/MAZ), and mutation of the SAF-1RE had little effec
216 nopausal women with type 2 diabetes received SAF or CLA (8 g oil/d) during two 16-wk diet periods sep
217                        We assessed reference SAF and skin reflectance (SR) values in a Saudi populati
218  microtubule assembly caused by the released SAFs would lead to excessive microtubule sliding that re
219                          Myocardin is a SAP (SAF-A/B, Acinus, PIAS) domain transcription factor that
220 c Leukemia-1 (MKL1), a gene encoding an SAP (SAF-A/B, Acinus and PIAS) DNA-binding domain.
221 isorders (that is, bipolar, schizoaffective (SAF), major depression) based on contemporary diagnostic
222 or four known SR protein-binding motifs: SF2/SAF, SC35, SRp40, and SRp55.
223 e with Xist, three of these proteins--SHARP, SAF-A and LBR--are required for Xist-mediated transcript
224                       MR arthrograms showing SAF were evaluated for subchondral reactions.
225                                        Since SAF-A, DNA-PK catalytic subunit (DNA-PKcs), and protein
226 ation of fluorescently labeled streptavidin (SAF) or SAF conjugated to biotinylated Cy3 adenoviral-ve
227 ited States, and development of a successful SAF workflow within it is needed.
228  used to study the relationships among T2DM, SAF, and gray matter volume (GMV).
229 olishing reentry with chloroquine terminates SAF more effectively than traditional Na+-channel blocka
230 ore effective than flecainide in terminating SAF in isolated sheep hearts by significantly increasing
231  increased in SAF-1-transgenic mice and that SAF-1 induces VEGF transcription by directly binding to
232                          We demonstrate that SAF improves the surface selectivity of TIRF, even at sh
233 cription factor genes have demonstrated that SAF-Sp1 heteromer is a highly potent transactivator of S
234 transfection assay, we provide evidence that SAF potentiates SAA gene expression through SAS element.
235 , these results provide direct evidence that SAF-1 plays a key role in the development of AA amyloido
236 nsgenic mice, supporting the hypothesis that SAF is important in mediating silencer function.
237                    Our studies indicate that SAF belongs to a family of transcription factors charact
238 es, immunofluorescence studies indicate that SAF is present primarily in the cytoplasm in T cells in
239            Together these data indicate that SAF-1 controls cell cycle progression via p21 induction,
240                    The results indicate that SAF-1 is involved in the regulation of MMP1 gene express
241 e kinase 1 (PLK1) rather than DNA-PKcs, that SAF-A interacts with PLK1 in nocodazole-treated cells, a
242 green fluorescent protein reporter show that SAF-1 contains two independent nuclear localization sign
243                         We further show that SAF-1 is phosphorylated in vitro by PKA-Calpha and that
244                           Here, we show that SAF-1-mediated induction of VEGF is repressed by KLF-4 t
245                        Our results show that SAF-A and caRNAs form a dynamic, transcriptionally respo
246                                 We show that SAF-A is phosphorylated on serine 59 in mitosis, that ph
247 ivation of SRp20 by SRrp86, we now show that SAF-B, hnRNP G, and 9G8 all antagonize the activity of S
248             In this paper we have shown that SAF-1, a major member of the SAF family, is abundantly p
249  distinct DNA-binding elements suggests that SAF-1 and AP-1 function in a mutually beneficial manner
250                                          The SAF results were compared with values from similar studi
251                                          The SAF-1.c-Fos.c-Jun ternary complex efficiently promotes t
252 No subchondral changes were found around the SAF.
253                           For each case, the SAF score was created including the semiquantitative sco
254 near in the number of genomes to compute the SAF likelihood.
255 thod produces an accurate SFS, computing the SAF likelihood is quadratic in the number of samples seq
256 ity score but to report it separately in the SAF score.
257 orylation analyses revealed two sites in the SAF-1 protein, serine 187 and threonine 386, as the targ
258 Introduction of a specific mutation into the SAF binding site in the CD4 silencer abrogates silencer
259 cartilage defect was seen at the site of the SAF at arthroscopy.
260 have shown that SAF-1, a major member of the SAF family, is abundantly present in human AA amyloidosi
261  algorithm, all non-negligible values of the SAF likelihood are concentrated on a few cells around th
262 gh fluorescence collection efficiency of the SAF microlens array, the SP-PCR assay on the LOC platfor
263 ed multiplexed SP-PCR directly on top of the SAF microlens array.
264 tudy was to determine whether the use of the SAF score and FLIP algorithm can decrease interobserver
265 m 42% to 75% in Group 2 after the use of the SAF score and FLIP algorithm.
266                             The width of the SAF was measured on coronal and sagittal MR images.
267 romoter was used to direct expression of the SAF-1 transgene in multiple organs.
268 reduced ability to promote expression of the SAF-1-regulated promoter.
269 ger protein (SAF-1/MAZ), and mutation of the SAF-1RE had little effect on IL-6 induction of gammaFBG
270              The FLIP algorithm based on the SAF score should decrease interobserver variations among
271 of the terminal negative modules renders the SAF-1 protein functionally very active.
272 age defects at arthroscopy indicate that the SAF of the acetabulum likely represents a variant.
273                           With regard to the SAF score, concordance was substantial in Group 1 for st
274                     Myocardin belongs to the SAF-A/B, Acinus, PIAS (SAP) domain family of transcripti
275 e suggest describing liver lesions using the SAF score.
276                                    Also, the SAFs were used with standardized decay data to develop d
277                  With preceding designs, the SAFs are thickened, highly ordered structures in which m
278 f self-assembly and self-organization in the SAFs is unprecedented for a designed peptide-based mater
279  with C-terminal thioester moieties into the SAFs.
280       Here, we describe the structure of the SAFs determined to approximately 8 A resolution using cr
281 designed a self-assembling fiber system, the SAFs, in which two small alpha-helical peptides are prog
282 SAF-8 isoforms are unique and are related to SAF-1/MAZ/ZF87/Pur-1 at the zinc finger domains but diff
283                                  Relative to SAF, EPA and DHA supplementation resulted in higher MVs
284     Furthermore, overexpression of wild-type SAF-1 in transfected liver cells can increase transcript
285  While the DNA-binding activity of wild-type SAF-1 protein was markedly increased upon phosphorylatio
286 ng variations in position along an unchanged SAF as a change in skill.
287           We surmised that reentry underlies SAF, and that abolishing reentry with chloroquine termin
288  highly induced by cellular conditions where SAF-1 is abundant.
289 -4-binding elements are overlapping, whereas SAF-1 induces and KLF-4 suppresses VEGF expression.
290 ansient acute phase induction of SAA whereas SAF and NF-kappa B activities are necessary for persiste
291                            To assess whether SAF-1 is directly linked to the pathogenesis of AA amylo
292  of homology to MAZ/ZF87/Pur-1 protein while SAF-5 and SAF-8 isoforms are unique and are related to S
293 mpacts large-scale chromatin structure while SAF-A loss or monomerization promotes aberrant chromosom
294  four hip joints with SAF type 1 and 13 with SAF type 2.
295 ts with the SAA promoter in association with SAF as an SAF.Sp1 heteromeric complex.
296 d obesity all showed strong association with SAF, particularly when gender differences were taken int
297 SAA expression by acting in conjunction with SAF.
298 icipants, SR was also highly correlated with SAF.
299 ose from arthroscopy in four hip joints with SAF type 1 and 13 with SAF type 2.
300 AP kinase phosphorylation site, PPTP, within SAF-1 could be phosphorylated by MAP kinase in vitro.

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