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1 SAF also significantly lowered fasting glucose (P = 0.03
2 SAF is a potentially valuable clinical screening tool fo
3 SAF partially mediated the association between T2DM and
4 SAF was classified into two types: type 1, which was fil
5 SAF-1 activity was detected in the chondrocytes of OA ca
6 SAF-1 contains several negative and positively functioni
7 SAF-1 DNA-binding activity is increased in both cytokine
8 SAF-1 is a 477-amino acid protein with six zinc fingers.
9 SAF-1, a zinc finger transcription factor, is activated
10 SAF-1-overexpressing mice spontaneously developed AA amy
11 SAF-A oligomerization decompacts large-scale chromatin s
12 SAFs for photons were generated for mildly and severely
13 SAFs of W-CIN cells were remarkably similar to those ind
14 SAFs studied between the obese phantoms and the 50th per
15 role of serum amyloid A-activating factor 1 (SAF-1) in MMP-1 expression was assessed by transient tra
16 factor, serum amyloid A-activating factor 1 (SAF-1), has been shown to regulate several genes, includ
17 factor serum amyloid A-activating factor-1 (SAF-1) has been identified as a regulator of a number of
19 Serum amyloid A (SAA) activating factor-1 (SAF-1) is an inducible transcription factor that plays a
20 amyloid A-activating transcription factor-1 (SAF-1) plays a major role in regulating transcription of
21 factor, serum amyloid A activating factor-1 (SAF-1), leading to markedly higher levels of angiogenesi
25 cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of homology to MAZ/ZF
29 we report that scaffold attachment factor A (SAF-A), originally identified as a structural nuclear pr
39 her levels of matrix metalloproteinase-1 and SAF-1 in the inflamed joints of SAF-1 transgenic mice co
40 on responsive transcription factors AP-1 and SAF-1 synergistically regulate transcriptional induction
42 gy to MAZ/ZF87/Pur-1 protein while SAF-5 and SAF-8 isoforms are unique and are related to SAF-1/MAZ/Z
43 he three cloned SAF cDNAs (SAF-1, SAF-5, and SAF-8), SAF-1 isoform showed a high degree of homology t
47 ctural component of articular cartilage, and SAF-1 in both SAF-1 transgenic and nontransgenic mice.
49 benefits of beam spinning EW excitation and SAF detection and provides the conditions for quantitati
50 P-1 family of proteins, c-Fos and c-Jun, and SAF-1 form a ternary protein complex, which has markedly
53 own to regulate microtubule organization and SAFs known to promote microtubule assembly such as Maski
55 ce of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibited interleukin-1-mediated MMP
56 is regulated by a posttranslational event as SAF DNA-binding and transactivation abilities are increa
59 ests, and noninvasive skin autofluorescence (SAF; a measure of tissue AGE levels) on people aged >55
66 es additional layers of regulation, and both SAFs are only degraded after being released from their i
67 or protein p21 was significantly affected by SAF-1; its expression level was highly induced by cellul
70 rvation, increased DNA binding of the cloned SAF and its hyperphosphorylation, in response to okadaic
75 health care organizations wanting to create SAF teledermatology workflows within the Epic EHR system
76 ion of SAF-1 protein from either end creates SAF-1 isoforms that are highly transcriptionally active.
77 he most influential variables in determining SAF values in men, whilst in female participants, SR was
82 sentative plots were made of photon electron SAFs, evaluating the traditional assumption that all ele
84 MP1 promoter, and interference of endogenous SAF-1 activity by antisense SAF-1 messenger RNA inhibite
86 promoter as well as knockdown of endogenous SAF-1 markedly inhibited IL-1beta- and TGF-beta-mediated
88 , in addition to tissue-specific expression, SAFs, a family of zinc finger transcription factors, und
89 These data show that transcription factor SAF-1 is involved in the regulation of IL-6-mediated ind
90 n that the inflammatory transcription factor SAF-1 is, at least in part, responsible for the marked i
93 tor serum amyloid A (SAA)-activating factor (SAF), a family of zinc finger proteins, plays a signific
94 show that serum amyloid A-activating factor (SAF)-1, a novel transcription factor, and the AP-1 famil
96 , referred to as silencer-associated factor (SAF), is a member of the helix-turn-helix factor family
97 n, and a nuclear factor, SAS-binding factor (SAF), that interacts with the SAS element has been ident
99 nflammation-responsive transcription factor, SAF (for SAA activating factor), has been implicated in
100 le matrix contains spindle assembly factors (SAFs) such as Eg5 and dynein which are known to regulate
101 of importin-bound spindle assembly factors (SAFs), which stimulate microtubule (MT) nucleation and o
104 rization of a self-assembling peptide fiber (SAF) system based on alpha-helical coiled-coil building
105 m fillets and the Sparus aurata fibroblasts (SAF-1) cell-line during an 8day storage period at +4 deg
106 oring system (steatosis, activity, fibrosis [SAF]) allowing the use of an algorithm (fatty liver inhi
107 ation with supercritical-angle fluorescence (SAF) detection efficiently rejects the fluorescence orig
109 To identify possible activation partners for SAF-1, we used a yeast two-hybrid system that detected i
111 sis suggests that L(pro) contains a SAP (for SAF-A/B, Acinus, and PIAS) domain, a protein structure a
115 ed to study how specific absorbed fractions (SAFs) vary with changes in adult body size, for persons
117 ta by first computing site allele frequency (SAF) likelihood for each site (i.e. the likelihood a sit
118 the task's speed-accuracy tradeoff function (SAF), which prevented us from falsely interpreting varia
120 iotropic role of SAF-1 in vivo, we generated SAF-1 transgenic mice, in which CMV immediate-early prom
126 sent in the atherosclerotic plaque implicate SAF-1 as a key regulator of MMP-14 gene induction in mac
127 sly unreported and significant difference in SAF values between men and women, with median (range) va
129 vidence that VEGF expression is increased in SAF-1-transgenic mice and that SAF-1 induces VEGF transc
141 Serial administration of the English MPN-SAF among 53 patients showed that most MPN-SAF items are
142 rative Neoplasm Symptom Assessment Form [MPN-SAF], coadministered with the Brief Fatigue Inventory) t
143 N-SAF among 53 patients showed that most MPN-SAF items are well correlated (r > 0.5, P < .001) and hi
144 tomatic elements represented on both the MPN-SAF and the European Organisation for Research and Treat
153 ether, these results show that activation of SAF-1 in response to IL-1 and -6 is mediated via MAP kin
154 tribution of MAP kinase in the activation of SAF-1, we prepared two independent mutant proteins in wh
162 overexpression of SAF-1 and coexpression of SAF-1 and MMP-14 in the macrophages present in the ather
163 specific subcellular compartmentalization of SAF plays an important role in mediating the specificity
164 The data also demonstrate that control of SAF-1 activity can suppress induced expression of MMP-1.
166 thermore, we found that terminal deletion of SAF-1 protein from either end creates SAF-1 isoforms tha
167 oth phosphorylation and dephosphorylation of SAF-A serine 59 by PLK1 and PP2A, respectively, are requ
168 ed 293 patients with a lifetime diagnosis of SAF disorder, bipolar disorder and major depressive diso
169 n constructs, the core DNA-binding domain of SAF-1 is mapped between amino acids 282 and 361, which c
172 ys a major role in controlling expression of SAF-regulated genes by increasing the interaction betwee
177 Previously, we showed in vivo interaction of SAF-1 and protein kinase A (PKA) and presented evidence
178 einase-1 and SAF-1 in the inflamed joints of SAF-1 transgenic mice compared with their levels in nont
180 on assay, in vivo, markedly higher levels of SAF-1 interaction with the VEGF promoter was detected in
181 processing activity during overexpression of SAF-1 and coexpression of SAF-1 and MMP-14 in the macrop
185 ical conditions that favor overexpression of SAF-1, such as an acute inflammatory condition, can trig
187 markedly induces in vivo phosphorylation of SAF-1 and transcription of SAF-regulated reporter genes.
192 chanism of synergy and the essential role of SAF-1 and AP-1 in up-regulating human MMP-1 expression u
193 Together these results suggest a role of SAF-1 in the pathogenesis of inflammation-induced arthri
195 t under native condition, N and C termini of SAF-1 are engaged in an inhibitory intramolecular intera
197 gated linoleic acid (CLA) and safflower oil (SAF), on body weight and composition in obese postmenopa
198 g/d of ethyl esters of either safflower oil (SAF; control), eicosapentaenoic acid (EPA), or docosahex
202 fluorescently labeled streptavidin (SAF) or SAF conjugated to biotinylated Cy3 adenoviral-vector (BC
204 he cells that were programmed to overproduce SAF-1 were found to undergo growth arrest and reduce DNA
205 dy we provide evidence that, similar to p53, SAF-1 is able to activate p21 gene expression by promoti
208 tions of radiation transport were performed; SAFs for photons were generated for 10th, 25th, 75th, an
212 alone or pHrodo complexed to E. coli, pHrodo-SAFs report pH in both the cytoplasm and phagosomes, as
215 ctor-1/c-Myc-associated zinc finger protein (SAF-1/MAZ), and mutation of the SAF-1RE had little effec
216 nopausal women with type 2 diabetes received SAF or CLA (8 g oil/d) during two 16-wk diet periods sep
218 microtubule assembly caused by the released SAFs would lead to excessive microtubule sliding that re
221 isorders (that is, bipolar, schizoaffective (SAF), major depression) based on contemporary diagnostic
223 e with Xist, three of these proteins--SHARP, SAF-A and LBR--are required for Xist-mediated transcript
226 ation of fluorescently labeled streptavidin (SAF) or SAF conjugated to biotinylated Cy3 adenoviral-ve
229 olishing reentry with chloroquine terminates SAF more effectively than traditional Na+-channel blocka
230 ore effective than flecainide in terminating SAF in isolated sheep hearts by significantly increasing
231 increased in SAF-1-transgenic mice and that SAF-1 induces VEGF transcription by directly binding to
233 cription factor genes have demonstrated that SAF-Sp1 heteromer is a highly potent transactivator of S
234 transfection assay, we provide evidence that SAF potentiates SAA gene expression through SAS element.
235 , these results provide direct evidence that SAF-1 plays a key role in the development of AA amyloido
238 es, immunofluorescence studies indicate that SAF is present primarily in the cytoplasm in T cells in
241 e kinase 1 (PLK1) rather than DNA-PKcs, that SAF-A interacts with PLK1 in nocodazole-treated cells, a
242 green fluorescent protein reporter show that SAF-1 contains two independent nuclear localization sign
247 ivation of SRp20 by SRrp86, we now show that SAF-B, hnRNP G, and 9G8 all antagonize the activity of S
249 distinct DNA-binding elements suggests that SAF-1 and AP-1 function in a mutually beneficial manner
255 thod produces an accurate SFS, computing the SAF likelihood is quadratic in the number of samples seq
257 orylation analyses revealed two sites in the SAF-1 protein, serine 187 and threonine 386, as the targ
258 Introduction of a specific mutation into the SAF binding site in the CD4 silencer abrogates silencer
260 have shown that SAF-1, a major member of the SAF family, is abundantly present in human AA amyloidosi
261 algorithm, all non-negligible values of the SAF likelihood are concentrated on a few cells around th
262 gh fluorescence collection efficiency of the SAF microlens array, the SP-PCR assay on the LOC platfor
264 tudy was to determine whether the use of the SAF score and FLIP algorithm can decrease interobserver
269 ger protein (SAF-1/MAZ), and mutation of the SAF-1RE had little effect on IL-6 induction of gammaFBG
278 f self-assembly and self-organization in the SAFs is unprecedented for a designed peptide-based mater
281 designed a self-assembling fiber system, the SAFs, in which two small alpha-helical peptides are prog
282 SAF-8 isoforms are unique and are related to SAF-1/MAZ/ZF87/Pur-1 at the zinc finger domains but diff
284 Furthermore, overexpression of wild-type SAF-1 in transfected liver cells can increase transcript
285 While the DNA-binding activity of wild-type SAF-1 protein was markedly increased upon phosphorylatio
289 -4-binding elements are overlapping, whereas SAF-1 induces and KLF-4 suppresses VEGF expression.
290 ansient acute phase induction of SAA whereas SAF and NF-kappa B activities are necessary for persiste
292 of homology to MAZ/ZF87/Pur-1 protein while SAF-5 and SAF-8 isoforms are unique and are related to S
293 mpacts large-scale chromatin structure while SAF-A loss or monomerization promotes aberrant chromosom
296 d obesity all showed strong association with SAF, particularly when gender differences were taken int
300 AP kinase phosphorylation site, PPTP, within SAF-1 could be phosphorylated by MAP kinase in vitro.
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