ライフサイエンスコーパス: SAS

1 SAS and STATA codes are provided for fitting a linear od

2 SAS are, nevertheless, important because they prompt dos

3 SAS code (SAS Institute, Inc., Cary, North Carolina) for

4 SAS should be considered as an adjuvant treatment to ame

5 SAS-4 deletion led to a progressive loss of centrioles,

6 SAS-6 constitutes the cartwheel, and SAS-6 levels remain

7 SAS-6 from Chlamydomonas reinhardtii and Danio rerio was

8 SAS-6 mutants with alanine substitutions in a previously

9 SAS-6 proteins are thought to impart the ninefold symmet

10 esions using PROC CALIS in SAS, version 9.1 (SAS Institute, Inc., Cary, North Carolina) and Mplus, ve

11 red for centriole duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

12 Sas-6, and DSas-4/CPAP-orthologues of ZYG-1, SAS-6, and SAS-4, respectively-are required for centriol

13 e performed using SAS software (version 9.1; SAS Institute).

14 sponse time on control trials (TMS(CT)), (2) SAS 200 ms prior to the go cue (SAS), (3) suprathreshold

15 cal data and were performed using SAS 9.1.3 (SAS Institute, Cary, NC).

16 centriole duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

17 by the spindle assembly abnormal protein 6 (SAS-6) family.

18 w conserved proteins (ZYG-1/Sak/Plk4, SAS-6, SAS-5/Ana2, and SAS-4), and is often initiated by the fo

19 to the SAS-6 coiled coil recruits the SAS-6-SAS-5 complex to the mother centriole, where a ZYG-1 kin

20 erver ICC 0.95-0.97, interobserver ICC 0.91; SAS intraobserver ICC 0.95-0.99, interobserver ICC 0.93;

21 A SAS macro (SAS Institute Inc., Cary, North Carolina) is

22 d that the release of planned movements by a SAS is mediated by subcortical, possibly brainstem, path

23 The early release of movement by a SAS was significantly delayed (P < 0.001, average delay

24 on, could delay the release of movement by a SAS.

25 Administration of a SAS accelerated ankle dorsiflexion in both groups, but m

26 The presentation of a SAS alone at -200 ms resulted in the release of the plan

27 erty (Web Appendix 1) and a description of a SAS macro (SAS Institute, Inc., Cary, North Carolina) fo

28 In 25% of trials, a SAS was delivered simultaneously with the imperative sti

29 e we define the volume of correlation, Vc, a SAS invariant derived from the scattered intensities tha

30 the imperative stimulus was combined with a SAS, presumably through release of a subcortically store

31 P patients in trials both with and without a SAS would argue in favor of a cortically stored response

32 The histone acetylases, SAS-I and NuA4, functioned in insulation, independently

33 In some cases, alternative SAS would give rise to mRNAs encoding proteins with diff

34 genes have between one and three alternative SAS, but PAS are more dispersed.

35 to 54 degrees S) in southern South America (SAS), to quantify the coupling of SAM and regional wildf

36 -6-depleted embryos, the levels of ZYG-1 and SAS-5 are reduced and the ZYG-1- and SAS-5-dependent rec

37 s centriole assembly by protecting ZYG-1 and SAS-5 from degradation.

38 G-1 and SAS-5 are reduced and the ZYG-1- and SAS-5-dependent recruitment of SAS-6 to the nascent cent

39 ent among standard ABC/2, modified ABC/2 and SAS: (mean) 12.8 (SD 16.3), 8.9 (9.2), 12.8 (13.1) cm(3)

40 We show that SAS-4 and SAS-6 are coordinately recruited to the site of new cent

41 cusing on the conserved components SAS-4 and SAS-6.

42 tion requires notably the proteins SAS-5 and SAS-6, which have functional equivalents across eukaryot

43 Plk4) and its downstream effectors SAS-5 and SAS-6.

44 ner centriole cartwheel components SAS-6 and SAS-5/Ana2/STIL, which then recruit SAS-4/CPAP, which in

45 DSas-4/CPAP-orthologues of ZYG-1, SAS-6, and SAS-4, respectively-are required for centriole duplicati

46 duplication: SPD-2, ZYG-1, SAS-5, SAS-6, and SAS-4.

47 eins (ZYG-1/Sak/Plk4, SAS-6, SAS-5/Ana2, and SAS-4), and is often initiated by the formation of an in

48 SAS-6 constitutes the cartwheel, and SAS-6 levels remain low until centriole assembly is init

49 djacent segment of the SAS-6 coiled coil and SAS-5, prevented SAS-6 recruitment and cartwheel assembl

50 (available as a worksheet, Excel macro, and SAS macro) is a standardized scoring system for making D

51 R, Matlab and SAS/IML code for implementing lossless data reduction is

52 sed on marginal structural models (MSMs) and SAS (SAS Institute, Inc., Cary, North Carolina).

53 Third, PCL formation depends on SAK/PLK4 and SAS-6.

54 Statistical software code in both R and SAS is provided.

55 esponse time (TMS(SAS)), or (4) TMS(SAS) and SAS presented concurrently (TMS+SAS).

56 delay = 35.0 +/- 12.9 ms) when TMS(SAS) and SAS were presented concurrently.

57 available user-friendly programs (Stata and SAS) to implement meta-analysis for dose-response data.

58 examples of such primordial sense-antisense (SAS) coding.

59 Here we show that supercritical antisolvent (SAS) precipitation using carbon dioxide, a process that

60 e to program using standard software such as SAS PROC SURVEYLOGISTIC (SAS Institute Inc., Cary, North

61 ined with high-throughput functional assays, SAS mutational libraries can expedite the functional ass

62 tch used by the widely distributed bacterial SAS enzymes.

63 rs known to prevent ectopic silencing (Bdf1, SAS-I complex, Rpd3L complex, Ku), we identified the Rtt

64 pendent step, whose target is unlikely to be SAS-6, triggers cartwheel assembly.

65 the presence of hyperelasticity in high-BMI SAS patients was also statistically significant (P < 0.0

66 nificant (P < 0.05) when compared to low-BMI SAS patients.

67 Functional orthologues of all but SAS-5 have been found in other species.

68 The dimensions determined by SAS agree well with those obtained by (dried-state) atom

69 ase of targeted ballistic wrist movements by SAS is mediated, in part, by a fast conducting transcort

70 ontractility was unaffected but that cMyBP-C(SAS(t/t)) hearts showed decreased diastolic function at

71 ng wild-type cMyBP-C, the transgenic cMyBP-C(SAS(t/t)), cMyBP-C(ADA(t/t)), and cMyBP-C(DAD(t/t)) mice

72 ning either Ser-273-Ala-282-Ser-302 (cMyBP-C(SAS)), Ala-273-Asp-282-Ala-302 (cMyBP-C(ADA)), or Asp-27

73 A protein called SAS-7 is required for daughter centrioles to become moth

74 apoptosis in human tongue squamous carcinoma SAS cells through mitochondrial pathway.

75 Centriolar SAS-4 remains in dynamic equilibrium with the cytoplasmi

76 Centriolar SAS-6 is subsequently reduced by a mechanism intrinsic t

77 try and electron microscopy, we characterize SAS-6 and show that it self-assembles into stable tetram

78 We provide SAS code (SAS Institute, Inc., Cary, North Carolina) and a simulat

79 SAS code (SAS Institute, Inc., Cary, North Carolina) for implement

80 The centrosomal component SAS-6 localized to basal bodies and the proximal region

81 its the inner centriole cartwheel components SAS-6 and SAS-5/Ana2/STIL, which then recruit SAS-4/CPAP

82 mbryos, focusing on the conserved components SAS-4 and SAS-6.

83 in acute exacerbations of COPD, SGRQ-C, CRQ-SAS, and FEV1 were greater in benralizumab-treated patie

84 e self-administered standardised format (CRQ-SAS), pre-bronchodilator forced expiratory volume in 1 s

85 MS(CT)), (2) SAS 200 ms prior to the go cue (SAS), (3) suprathreshold TMS 70 ms prior to the mean SAS

86 To date, SAS-6 is one of the two clearly conserved components of

87 efold symmetry, suggesting that two distinct SAS-6 oligomerization architectures can direct the same

88 ZYG-1/Sak/Plk4) and its downstream effectors SAS-5 and SAS-6.

89 showing that, instead, Caenorhabotis elegans SAS-6 self-assembles into a spiral arrangement.

90 nally, in the presence of embryonic extract, SAS-6 tetramers assembled into high density complexes, p

91 Before these tests, subjects received five SAS while standing to verify normal function of the reti

92 SAMS is the most frequent SAS, and mild myalgia may affect 5% to 10% of statin use

93 A new user-friendly SAS macro (SAS Institute, Inc., Cary, North Carolina) is

94 For 488 genes, SAS were identified downstream of the originally assigne

95 cal low-gradient (PLG) or high-gradient (HG) SAS.

96 onsistent high-gradient (MG >/=40 mm Hg) (HG-SAS group) and with that of patients with a moderate AS

97 showed better survival in PLG-SAS than in HG-SAS, both in the overall population (48% versus 31%; P<0

98 Patients were categorized into HG-SAS (n=144) and PLG-SAS (n=205) according to mean transv

99 because the majority of them evolve into HG-SAS over time.

100 G-SAS are en route toward the more severe HG-SAS form, because the majority of them evolve into HG-SA

101 associated with improved survival in the HG-SAS group (hazard ratio: 0.18; p = 0.001) and in the PLG

102 r: 64 +/- 4%) compared with patients with HG-SAS (1-year: 96 +/- 1%; 5-year: 82 +/- 3%) or MAS (1-yea

103 a less malignant form of AS compared with HG-SAS, because their spontaneous outcome is better.

104 ording to the AVA, with 187 patients with HG-SAS.

105 ment or the removal of luminal SAS-6 hinders SAS-6 (or centriole) assembly at the outside wall of mot

106 sults provide mechanistic insights in to how SAS responses are rapidly established by light condition

107 ons conducive to widespread fire activity in SAS will continue throughout the 21st century.

108 We used Cox proportional hazards analysis in SAS 9.2 survey procedures to estimate associations after

109 gram the previous risk-averaging approach in SAS.

110 the odds of skin lesions using PROC CALIS in SAS, version 9.1 (SAS Institute, Inc., Cary, North Carol

111 A varimax-rotation was conducted in SAS 9.1 using the PROC FACTOR CORR to extract factors fr

112 Web site, contain implementation examples in SAS software (SAS Institute, Inc., Cary, North Carolina)

113 Hyperelastic floppy eyelid in SAS patients was statistically significant (P < 0.05) wh

114 rovide helpful hints for creating figures in SAS/GRAPH that meet the requirements of the Journal.

115 Floppy eyelid syndrome was more frequent in SAS patients than in normal subjects (P < 0.05), but no

116 its kinase activity-independent function in SAS-6 recruitment.

117 The MCMC procedure in SAS (called PROC MCMC) is particularly designed for Baye

118 tions have shown how the GENMOD procedure in SAS (SAS Institute Inc., Cary, North Carolina) can be us

119 wise elimination (Proc Logistic procedure in SAS).

120 ASF1 and CAF-1-dependent pathways, including SAS-I- and Rtt109p-dependent acetylation events at H4-K1

121 ilure in worm embryos, indicating that large SAS-5 assemblies are necessary for function in vivo.

122 monstrate that oligomerization of Leishmania SAS-6 can be inhibited by a small molecule in vitro and

123 ation to centrioles requires SAS-4 and, like SAS-4, HYLS-1 is stably incorporated into the outer cent

124 fit by using standard software packages like SAS and R).

125 esent in a human tongue carcinoma cell line, SAS.

126 er the recruitment or the removal of luminal SAS-6 hinders SAS-6 (or centriole) assembly at the outsi

127 The removal or release of luminal SAS-6 requires Plk4 and the cartwheel protein STIL.

128 A SAS macro (SAS Institute Inc., Cary, North Carolina) is given in an

129 ppendix 1) and a description of a SAS macro (SAS Institute, Inc., Cary, North Carolina) for doubly ro

130 A new user-friendly SAS macro (SAS Institute, Inc., Cary, North Carolina) is provided t

131 Here we show that Leishmania major SAS-6 crystallizes as a 9-fold symmetric cartwheel and p

132 ) suprathreshold TMS 70 ms prior to the mean SAS-evoked response time (TMS(SAS)), or (4) TMS(SAS) and

133 gnosis codes in the Veterans Affairs Medical SAS data sets.

134 lyzed data from the Veterans Affairs Medical SAS Datasets and Decision Support System for entire coho

135 ated-measures regression models (PROC MIXED; SAS Institute), adjusted for all other dietary water sou

136 st and estimate effect-measure modification; SAS code is provided.

137 Recombinant and native SAS-6 self-oligomerizes into tetramers with approximatel

138 on and ciliation can arise in the absence of SAS-6 self-oligomerization.

139 We found that acute administration of SAS at concentrations equivalent to those used to treat

140 l analysis demonstrates the applicability of SAS to monitor complex solution-based self-assembly.

141 ural analysis to uncover the architecture of SAS-5 and examine its functional implications in vivo.

142 Whereas the molecular architecture of SAS-6 and its role in initiating centriole formation are

143 Assembly of SAS-6 dimers to form the centriolar cartwheel requires t

144 he centriole assembly initiation capacity of SAS-6 is separate from or directly related to its struct

145 ion of the ciliary axoneme, and depletion of SAS-6 prevented centriole assembly.

146 ay structure of the amino-terminal domain of SAS-6 from zebrafish, and we show that recombinant SAS-6

147 vide a proof-of-principle that inhibition of SAS-6 oligomerization by small molecules is feasible.

148 In this report, we address the issue of SAS coding in a wider scope.

149 d by the observation that elevated levels of SAS-6 in Drosophila cells resulted in higher order struc

150 Management of SAS requires making the possible diagnosis, altering or

151 necessary to form higher-order oligomers of SAS-5: a trimeric coiled coil and a novel globular dimer

152 Ana2 is the likely functional orthologue of SAS-5 and that it is also related to the vertebrate STIL

153 In some organisms, overexpression of SAS-6 causes the formation of supernumerary centrioles.

154 o structural errors, even in the presence of SAS-6 self-oligomerization.

155 The FREQ, LOGISTIC, and GENMOD procedures of SAS (SAS Institute, Cary, NC) were used to evaluate poss

156 that PpIX-SDT suppress the proliferation of SAS cells via arresting cell cycle at G2/M phase and act

157 he ZYG-1- and SAS-5-dependent recruitment of SAS-6 to the nascent centriole fails.

158 urogenesis in mice by conditional removal of SAS-4, a protein that is required for centriole biogenes

159 al centriole duplication factors upstream of SAS-6 recruitment and procentriole formation.

160 riole biogenesis does not strictly depend on SAS-6 self-assembly, and may require preexisting centrio

161 Correlation analyses were run on SAS 9.2.

162 exo- Protoporphyrin based SDT (PpIX-SDT) on SAS cells in vitro and in vivo.

163 show that overexpression of either Cad99C or SAS causes a dramatic increase in apical membrane at the

164 Overexpression of Cad99C or SAS results in similar, but distinct effects, suggesting

165 to DM, but evidence linking statins to other SAS is largely anecdotal.

166 pression and apoptosis rate in wild-type p53 SAS cells were found in the SDT group, while p53-mutated

167 designed for use with the statistics package SAS.

168 However, when the SAS software package (SAS Institute Inc., Cary, North Carolina) is used for an

169 XED in the SAS statistical software package (SAS Institute Inc., Cary, North Carolina).

170 within the SAS statistical software package (SAS Institute, Inc., Cary, North Carolina).

171 egression using the SAS statistical package (SAS Institute Inc., Cary, North Carolina).

172 During S phase, SAS-6 molecules are first recruited to the proximal lume

173 were categorized into HG-SAS (n=144) and PLG-SAS (n=205) according to mean transvalvular gradient (me

174 AVA] </=1.0 cm(2)) aortic stenosis (AS) (PLG-SAS group) with that of patients with a severe AS (AVA <

175 urvival curves showed better survival in PLG-SAS than in HG-SAS, both in the overall population (48%

176 Our study indicates that PLG-SAS is a less malignant form of AS compared with HG-SAS,

177 azard ratio: 0.18; p = 0.001) and in the PLG-SAS group (hazard ratio: 0.50; p = 0.04) but not in the

178 /=50%, we identified 187 patients in the PLG-SAS group.

179 ly, at last echocardiographic follow-up, PLG-SAS demonstrated significant increases in mean gradient

180 further demonstrated that patients with PLG-SAS are en route toward the more severe HG-SAS form, bec

181 nt for other risk factors, patients with PLG-SAS had a 1.71-fold increase in overall mortality and a

182 Patients with PLG-SAS had reduced overall survival (1-year: 89 +/- 2%; 5-y

183 on a few conserved proteins (ZYG-1/Sak/Plk4, SAS-6, SAS-5/Ana2, and SAS-4), and is often initiated by

184 st understory and are shade tolerant prevent SAS when exposed to shade.

185 f the SAS-6 coiled coil and SAS-5, prevented SAS-6 recruitment and cartwheel assembly.

186 A simple SAS software program (SAS Institute, Inc., Cary, North Carolina) was developed

187 arily conserved centriole/basal body protein SAS-4 regulates centriole duplication in metazoa and bas

188 interaction with the core centriolar protein SAS-4.

189 lf-oligomerization of the centriolar protein SAS-6, but how the 9-fold symmetry is invariantly establ

190 mo-oligomerization of the centriolar protein SAS-6, but whether SAS-6 self-assembly can dictate cartw

191 lf-oligomerization of the centriolar protein SAS-6.

192 centriole component, the coiled-coil protein SAS-7, as a regulator of centriole duplication, assembly

193 The highly conserved protein SAS-6 constitutes the center of the cartwheel assembly t

194 The conserved centriole protein, SAS-6, is a cartwheel component that functions early in

195 Second, the centriolar proteins SAS-6, Ana1, and Bld10p/Cep135 are in the PCL.

196 iole formation requires notably the proteins SAS-5 and SAS-6, which have functional equivalents acros

197 calization of CEP135(full) binding proteins (SAS-6 and CPAP) and the pericentriolar localization of g

198 We provide SAS code (SAS Institute, Inc., Cary, North Carolina) and

199 from zebrafish, and we show that recombinant SAS-6 self-associates in vitro into assemblies that rese

200 AS-6 and SAS-5/Ana2/STIL, which then recruit SAS-4/CPAP, which in turn helps assemble the outer centr

201 Here, we show that ZYG-1 recruits SAS-6 to the mother centriole independently of its kinas

202 that the protein phosphatase PP2A regulates SAS-5 to control centriole duplication.

203 are presented to explain how PP2A regulates SAS-5.

204 HYLS-1 localization to centrioles requires SAS-4 and, like SAS-4, HYLS-1 is stably incorporated int

205 o and that inhibiting proteolysis can rescue SAS-5 levels and the centriole duplication defect of PP2

206 n marginal structural models (MSMs) and SAS (SAS Institute, Inc., Cary, North Carolina).

207 have shown how the GENMOD procedure in SAS (SAS Institute Inc., Cary, North Carolina) can be used to

208 REQ, LOGISTIC, and GENMOD procedures of SAS (SAS Institute, Cary, NC) were used to evaluate possible

209 (CES-D), the Zung Self-Rating Anxiety Scale (SAS), and the Child Attitude Toward Illness Scale (CATIS

210 Modern small-angle scattering (SAS) experiments with X-rays or neutrons provide a compr

211 Small-angle scattering (SAS) has a proven ability to detect and characterize sol

212 agments called substrate activity screening (SAS) has been applied to the development of low molecula

213 Substrate activity screening (SAS) is a fragment-based method for the rapid developmen

214 f cruzain, the substrate activity screening (SAS) method was used to screen a library of protease sub

215 adherin 99C (Cad99C) and Stranded at Second (SAS) - in conferring apical character in Drosophila tubu

216 modified ABC/2, semiautomated segmentation (SAS), fully automatic measurement methods; shape, densit

217 construction of a systematic allelic series (SAS) using massively parallel single-nucleotide mutagene

218 ies; but such "sluggish attention shifting" (SAS) appeared only when dyslexics shifted their attentio

219 epidemiologic cohort data by using a simple SAS program.

220 A simple SAS software program (SAS Institute, Inc., Cary, North C

221 An SIS/SAS model of gonorrhea transmission in a population of h

222 We identified 5' splice-acceptor sites (SAS) and polyadenylation sites (PAS) for 6959 and 5948 g

223 ain implementation examples in SAS software (SAS Institute, Inc., Cary, North Carolina) and R languag

224 icles, and Penta-BDE-spiked artificial soil (SAS).

225 ampled into a Statistical Association Space (SAS) to evaluate their dynamic correlation.

226 portant molecular events underlying specific SAS responses have been identified.

227 30% of patients with severe aortic stenosis (SAS; indexed aortic valve area <0.6 cm(2)/m(2)) present

228 Startling acoustic stimuli (SAS) can accelerate reaction times ("StartReact" effect)

229 released when a startling acoustic stimulus (SAS) is presented immediately prior to, or coincident wi

230 nic material, the surface active substances (SAS), reduced sulphur species (RS) and catalytically act

231 ensuing hyperexcitability by sulfasalazine (SAS), a US Food and Drug Administration-approved drug th

232 rd software such as SAS PROC SURVEYLOGISTIC (SAS Institute Inc., Cary, North Carolina).

233 ted with various statin-associated symptoms (SAS), including statin-associated muscle symptoms (SAMS)

234 dy mass index (BMI) in sleep apnea syndrome (SAS) patients compared to normal subjects.

235 ponse known as the shade avoidance syndrome (SAS).

236 ocess known as the shade avoidance syndrome (SAS).

237 ons known as the "shade-avoidance syndrome" (SAS).

238 at the tetrameric small alarmone synthetase (SAS) RelQ from the Gram-positive pathogen Enterococcus f

239 We conclude that SAS-7 functions at the earliest step in centriole duplic

240 Our results firmly establish that SAS-6 can impose cartwheel symmetry on its own and indic

241 Here, we found that SAS-6 assembly can be shaped by preexisting (or mother)

242 We show that SAS-4 and SAS-6 are coordinately recruited to the site o

243 ubules with repeating subunits and show that SAS-6 concentrates at the core of the cartwheel.

244 We also show that SAS-7 binds SPD-2 and regulates SPD-2 centriolar recruit

245 These results suggest that SAS-6 self-assembly may be an initial step in the format

246 Intriguingly, our genetic data suggest that SAS-7 is required for daughter centrioles to become comp

247 Sequence analysis suggests that SAS-6 spirals are restricted to specific nematodes.

248 The SAS Macro for our empirical Bayes test for periodicity i

249 y a direct interaction between ZYG-1 and the SAS-6 coiled coil that explains its kinase activity-inde

250 tone" gene (eroded somewhat, though) for the SAS origin of the two aaRS classes.

251 found that the probability densities in the SAS increase as the peaks in two cues are approached.

252 epidemiologic data using PROC NLMIXED in the SAS statistical software package (SAS Institute Inc., Ca

253 f the high probability density region in the SAS suggests a nonlinear correlation between two cues.

254 This study introduces the SAS/MCMC procedure and demonstrates the application of t

255 Our approach prevents over-fitting of the SAS data and can be used with a newly defined metric, RS

256 We propose that members of the SAS-5/Ana2/STIL family of proteins are key conserved com

257 teraction between an adjacent segment of the SAS-6 coiled coil and SAS-5, prevented SAS-6 recruitment

258 ic activity are abolished by deletion of the SAS-specific C-terminal helix 5alpha.

259 inding to the SAS-6 coiled coil recruits the SAS-6-SAS-5 complex to the mother centriole, where a ZYG

260 Here, we report that the SAS-4 homolog in the flagellated protozoan Trypanosoma b

261 We propose that ZYG-1 binding to the SAS-6 coiled coil recruits the SAS-6-SAS-5 complex to th

262 the Yale Open Data Access Project using the SAS Clinical Trials Data Transparency platform.

263 dependent, unblinded, statistician using the SAS procedure Proc Plan.

264 tion of N-glycosylation efficiency using the SAS software, employing the 120 sequences studied as a t

265 ng, and full cohort Cox regression using the SAS statistical package (SAS Institute Inc., Cary, North

266 However, when the SAS software package (SAS Institute Inc., Cary, North Ca

267 complex survey designs available within the SAS statistical software package (SAS Institute, Inc., C

268 -evoked response time (TMS(SAS)), or (4) TMS(SAS) and SAS presented concurrently (TMS+SAS).

269 or to the mean SAS-evoked response time (TMS(SAS)), or (4) TMS(SAS) and SAS presented concurrently (T

270 , average delay = 35.0 +/- 12.9 ms) when TMS(SAS) and SAS were presented concurrently.

271 TMS(SAS) and SAS presented concurrently (TMS+SAS).

272 ar association of hyperelasticity and FES to SAS patients but no association between obesity and FES.

273 ed mother centrioles becomes inaccessible to SAS-6, correlating with a block for reduplication.

274 owth-promoting molecular pathways leading to SAS However, it is unknown how plants that complete thei

275 CEP135 physically links to SAS-6 near the site of microtubule nucleation and binds

276 presence of FES was estimated in relation to SAS and BMI.

277 Tetrahymena thermophila, which expresses two SAS-6 homologues, TtSAS6a and TtSAS6b.

278 Unlike SAS-4, HYLS-1 is dispensable for centriole assembly and

279 We used SAS survey procedures to plot distributions of TBI and p

280 Data were analysed using SAS 9.1.3.

281 was performed as the primary analysis using SAS v9.

282 Data were analyzed using SAS statistical software.

283 om 1 310 727 examinations (analyzed by using SAS 9.3) provided median values, as well as means and 25

284 out by fitting linear mixed models by using SAS PROC MIXED.

285 Armitage trend analyses were conducted using SAS 9.2.

286 Analyses were conducted using SAS-callable SUDAAN to correct for complex sampling desi

287 on historical data and were performed using SAS 9.1.3 (SAS Institute, Cary, NC).

288 Our analysis was performed using SAS 9.3 and Stata 12.

289 nparametric comparisons were performed using SAS software (version 9.1; SAS Institute).

290 , methods for evaluating mass and validating SAS-based models and resolution have been inadequate.

291 of the centriolar protein SAS-6, but whether SAS-6 self-assembly can dictate cartwheel and thereby ce

292 are well understood, the mechanisms by which SAS-5 and its relatives function is unclear.

293 egulates SPD-2 centriolar recruitment, while SAS-7 centriolar localization is SPD-2-independent.

294 Data were analysed with SAS using linear regression models.

295 Participants were randomly assigned with SAS (version 9.2, block size 2-9) in a 1:1 ratio, strati

296 We show that PP2A physically associates with SAS-5 in vivo and that inhibiting proteolysis can rescue

297 IM-FRET revealed that Pol30p interacted with SAS-I and Rtt109p in the nuclei of living cells.

298 total of 135 participants (81 patients with SAS and 54 normal subjects) had a full ophthalmologic ex

299 We prospectively studied 349 patients with SAS and preserved left ventricular ejection fraction.

300 Analyses were performed with SAS PROC MIXED.

301 ng to ordinary least squares regression with SAS procedures for multiple imputation and analysis of c