ライフサイエンスコーパス: SBS

1 SBS results were significantly different when AmelxKO an

2 SBS was the major indication for HPN in our cohort.

3 SBSs and insertions/deletions occur predominantly at the

4 SBSs remained relatively stable amongst the UK cohort, w

5 l subjects with intact intestine and from 13 SBS patients dependent on parenteral nutrition because o

6 mportantly, site-directed mutagenesis of 202-SBS or expression of a dominant negative form of STAT3 s

7 AT3 to an oligonucleotide containing the 202-SBS in gel-mobility shift assays and to the 5'-regulator

8 a potential STAT3 DNA-binding site (the 202-SBS) present in the 5'-regulatory region of the Ifi202 g

9 triallyl leucine betaine chloride and 43.5% SBS.

10 ce the half-life of SMD targets that form an SBS by either intramolecular or intermolecular base-pair

11 tely 355 nm) in 10 sec, we then performed an SBS reaction on a chip that contains a self-priming DNA

12 arenteral support volume, bowel anatomy, and SBS features.

13 erodimer (SB) and two heterotrimers (BSB and SBS) by alkyne bridges leads to the formation of coupled

14 ice genes use APA, supported by MPSS-DGE and SBS-DGE tags, respectively.

15 r dideoxynucleotide terminating reaction and SBS.

16 The SeBSe and SBS pincer ligands can be synthesized via two independen

17 s(4,5)P(2) and that three lysine residues at SBS I site, Lys-1420, Lys-1432, and Lys-1434, are respon

18 we reported a single-molecule nanopore-based SBS strategy that accurately distinguishes four bases by

19 e ovary to uncover genes with an ovary-bound SBS that are misregulated upon Su(Hw) loss.

20 e function involves binding to non-clustered SBSs and does not require CP190 or Mod67.2.

21 Compared to the Ugandan cohort, SBSs and RIs in the UK cohort were lower at visit 1, and

22 Maximal transactivation of COL7A1 SBS-driven promoters in either MDA-MB-468 carcinoma cell

23 ed SMAD3, but not SMAD2, can bind the COL7A1 SBS.

24 osophila flies and destroys Escherichia coli SBS 363 cells.

25 life seem to be similar to those who develop SBS as adolescents with regards to long-term outcome, de

26 erences between adult patients who developed SBS during early childhood and those who develop this as

27 ised the pediatric group (PG), 37 developing SBS at age 13 to 25 constituted the adolescent group (AG

28 he two groups, pediatric patients developing SBS early in life seem to be similar to those who develo

29 Thirty patients developing SBS younger than 12 years comprised the pediatric group

30 This ensemble SBS chemistry has been implemented on an existing Illumi

31 Mapping of ovary Su(Hw)(f) SBSs revealed that female fertility requires binding to

32 hort-bowel syndrome with intestinal failure (SBS-IF) to gain insight into its mechanism of action.

33 hort bowel syndrome with intestinal failure (SBS-IF).

34 d underwent a SRSB between 1985 and 2010 for SBS were included.

35 odipy-FL-510, as a reversible terminator for SBS.

36 -s-indacene), as a reversible terminator for SBS.

37 tide analogues as reversible terminators for SBS.

38 d by a single excitonic state but by two for SBS.

39 Human SBSs can form by intermolecular base-pairing between a 3

40 dNTP-azidolinker-fluorophores for the hybrid SBS.

41 ead length of >30 bases by using this hybrid SBS method on a chip and a four-color fluorescence scann

42 In SBS, the remaining SB may dilate as part of intestinal a

43 In SBS-IF patients, feeding fails to decrease proteolysis i

44 Gut adaptation in SBS patients does not appear to involve an increase in g

45 -dNTPs-allyl-fluorophore, for application in SBS.

46 ome parenteral nutrition (HPN) dependence in SBS patients with intestinal failure.

47 A significant increase in SBS was measured for all samples when the self-etch syst

48 orthy deceleration of growth was observed in SBS children 6 mo after weaning off HPN.

49 Up-regulation of colonic PepT1 in SBS may adaptively improve accrual of malabsorbed di- an

50 longed PN duration and decreased survival in SBS children.

51 Altogether, our data suggest that MHA, SBS, FTNS, EPS, and APSM comprise a phenotypic spectrum

52 For A-tracts, most SBSs occur in the direction of the base-pair flanking th

53 sponding mutants with a single SBS, multiple SBSs probably interact to cause the high affinity bindin

54 Because some Byr4 mutants with multiple SBSs bound Spg1 tighter than the corresponding mutants w

55 ould yield a single-molecule electronic Nano-SBS platform.

56 nanopore-based sequencing by synthesis (Nano-SBS) strategy that can accurately distinguish four bases

57 ucleotide-based polymers to perform nanopore SBS on an alpha-hemolysin nanopore array platform.

58 pore-based sequencing-by-synthesis (Nanopore-SBS) approach, which used a set of nucleotides with poly

59 a nanopore, in combination with the Nanopore-SBS approach, can provide the foundation for a low-cost,

60 body mass index (BMI)-matched controls (non-SBS), for 14 d with the DLW method.

61 n values for age-, BMI-, and sex-matched non-SBS controls.

62 175 kcal/d, P = 0.001) and also for the non-SBS group (2393 +/- 445 compared with 1532 +/- 178 kcal/

63 antly lower in the SBS group than in the non-SBS group (P < 0.01); however, predicted TEE did not dif

64 ort the melt-phase self-assembly behavior of SBS triblock copolymers (S = poly(styrene) and B = poly(

65 The most common cause of SBS in the AG was trauma (n=13), followed by tumors (n=7

66 the abundance of PepT1 mRNA in the colon of SBS patients was more than 5-fold that in control subjec

67 Each cycle of SBS consists of the incorporation of the photocleavable

68 NA template, facilitating the development of SBS as a viable approach for high-throughput DNA sequenc

69 SB diameter of SBS children (n = 61) was measured in contrast SB series

70 of GLP-2 coinfused with PN in a rat model of SBS (60% jejunoileal resection plus cecectomy).

71 study reviews outcomes of a large number of SBS infants and identifies predictors of success.

72 f administration in relation to the onset of SBS, optimal patient selection for use, duration of trea

73 nfants who have liver failure as a result of SBS are frequently referred for consideration for combin

74 permanent sequelae are rare, the symptoms of SBS can be uncomfortable, even disabling, and whole work

75 Pediatric SBS carries extensive morbidity and high mortality, but

76 zards analysis was conducted on 80 pediatric SBS patients.

77 t to calculate the total costs for pediatric SBS patients and to provide an in-depth analysis of thes

78 This is particularly true for pediatric SBS patients who receive some of the most complex medica

79 an (+/- SD) total cost of care for pediatric SBS was US$505 250 +/- US$248 398 (corrected for inflati

80 e major predictors of mortality in pediatric SBS.

81 The comprehensive care of pediatric SBS patients costs significantly more than has previousl

82 t better prediction of outcomes of pediatric SBS, which may help to direct future management of these

83 le fluorescent dideoxynucleotides to perform SBS.

84 R322 using an r-m+ M5a1 derivative and phage SBS for screening.

85 ) </= 0.82, we demonstrate that polydisperse SBS triblock copolymers self-assemble into periodic stru

86 yrene-block-polybutadiene-block-polystyrene (SBS) polymer have been produced.

87 proteins, even though both localize to Rbp9 SBSs.

88 luenced by cellular factors, on a fine scale SBSs are influenced by the local DNA sequence-context, a

89 Whereas on a genome-wide scale SBSs are influenced by cellular factors, on a fine scale

90 Each participant's sexual behavior score (SBS) was estimated based on the number and type of unpro

91 aly (MHA) and Fechtner (FTNS) and Sebastian (SBS) syndromes are autosomal dominant platelet disorders

92 In well selected SBS patients, outcomes as diverse as survival, macronutr

93 demonstrate that the SMAD binding sequence (SBS) representing the TGF-beta response element in the r

94 P), we identified 24 SlmA-binding sequences (SBSs) on the chromosome.

95 on electron microscopy analyses of seventeen SBS triblock copolymers with poly(1,4-butadiene) volume

96 Although mutants with a single SBS bound Spg1 and inhibited GTP dissociation, the equil

97 than the corresponding mutants with a single SBS, multiple SBSs probably interact to cause the high a

98 to a region containing a Smad binding site (SBS) and a medea box.

99 s mRNAs that harbor a Staufen1-binding site (SBS) in their 3' untranslated regions (UTRs).

100 grades mRNAs harboring a STAU1-binding site (SBS) in their 3'-untranslated regions (3' UTRs).

101 ate association testing or the site-by-site (SBS) testing may underutilize the longitudinal feature o

102 16-binding site and four Spg1-binding sites (SBS), designated SBS1-SBS4.

103 ) binding to clustered Su(Hw) binding sites (SBSs) and recruitment of the insulator proteins Centroso

104 enome-wide analysis of Su(Hw)-binding sites (SBSs) in the ovary, showing that tissue-specific binding

105 separation which creates a slightly smoother SBS.

106 ng was compared through shear bond strength (SBS) studies with 2 different systems (etch-and-rinse an

107 Single base substitutions (SBSs) and insertions/deletions are critical for generati

108 ) laser on the corneal stromal bed surfaces (SBS).

109 dications for HPN were short bowel syndrome (SBS) (59%), PNDD (14%), congenital enteropathies (10%),

110 vances in the areas of short bowel syndrome (SBS) and small bowel transplantation (SBT).

111 evelopment program for short bowel syndrome (SBS) are reviewed.

112 posed in patients with short-bowel syndrome (SBS) as a rehabilitative therapy, but its effects on abs

113 Short bowel syndrome (SBS) can lead to intestinal failure and require total or

114 Short bowel syndrome (SBS) is a serious malabsorption disorder, and dietetic m

115 ediatric patients with short bowel syndrome (SBS) is now possible because of parenteral nutrition and

116 Short bowel syndrome (SBS) may induce a plethora of clinical symptoms ranging

117 tcome of neonates with short bowel syndrome (SBS) who require parenteral nutrition (PN).

118 SRSB) in patients with short bowel syndrome (SBS) who were "permanently" dependent on parenteral nutr

119 olume in patients with short bowel syndrome (SBS) with intestinal failure, increasing intestinal wet

120 ation in children with short bowel syndrome (SBS).

121 urred by patients with short-bowel syndrome (SBS).

122 s exist in humans with short-bowel syndrome (SBS).

123 managing patients with short bowel syndrome (SBS).

124 ation in children with short bowel syndrome (SBS).

125 Sick-building syndrome (SBS) is an increasingly common problem.

126 ms (often denoted as sick building syndrome (SBS)), chronic respiratory symptoms, and respiratory inf

127 te a version of the Sequencing by Synthesis (SBS) chemistry that potentially can become a preferred t

128 DNA sequencing by synthesis (SBS) offers a robust platform to decipher nucleic acid s

129 DNA sequencing by synthesis (SBS) offers an approach for potential high-throughput se

130 port four-color DNA sequencing by synthesis (SBS) on a chip, using four photocleavable fluorescent nu

131 DNA sequencing by synthesis (SBS) on a solid surface during polymerase reaction can d

132 DNA sequencing by synthesis (SBS) on a solid surface during polymerase reaction offer

133 DNA sequencing by synthesis (SBS) using reversible fluorescent nucleotide terminators

134 DNA sequencing-by-synthesis (SBS) technology, using a polymerase or ligase enzyme as

135 by using Illumina's sequencing-by-synthesis (SBS) technology, which allowed us to characterize the co

136 hnologies, based on sequencing-by-synthesis (SBS), are starting to deliver large amounts of DNA seque

137 E) and the Illumina sequencing-by-synthesis (SBS-DGE) sequencing platforms.

138 atory arthritis (chondrocalcinosis) and that SBS patients may be prone to develop extreme hypomagnesa

139 The side cuts were straight, and the SBS appeared smoother in cuts obtained using 150 kHz.

140 Comparisons between PGS and the SBS testing revealed that PGS provided smaller phenotype

141 Both the SBS and medea box are necessary for complex formation an

142 nificantly higher than predicted TEE for the SBS group (1875 +/- 276 compared with 1517 +/- 175 kcal/

143 Measured TEE was significantly lower in the SBS group than in the non-SBS group (P < 0.01); however,

144 of the polymerase reaction and increases the SBS efficiency.

145 The quality and smoothness of the SBS post-FS laser dissection were graded for statistics.

146 The average values of the SBS quality of the anterior lamellar cut were found to b

147 veral surgical options for management of the SBS, including construction of intestinal valves or reve

148 te estimates and higher sensitivity than the SBS testing with comparable specificities.

149 enotype-related biological pathways than the SBS testing.

150 mcision, pregnancy) were integrated with the SBS, generating a RI for each HESN.

151 -act sex in the UK, had major impacts on the SBSs and RIs of each cohort.

152 ithstanding the substantial success of these SBS platforms, challenges continue to limit the ability

153 Optimization of the steps involved in this SBS approach will further increase the read-length.

154 ore UPF1 and ~two- to fivefold more of those SBS-containing mRNAs that were tested, and it comparably

155 Patients were matched to SBS controls with the same digestive characteristics.

156 Remarkably, SlmA binding to SBSs dramatically enhanced its ability to interfere with

157 TEE was measured in 22 participants, 11 with SBS and 11 sex-, age-, and body mass index (BMI)-matched

158 e total charges incurred by 41 children with SBS over the past decade, encompassing both inpatient an

159 organ transplantation rates in children with SBS.

160 roviders and families who have children with SBS.

161 rom a contemporary cohort of 49 infants with SBS and cholestasis whose PN course included soybean ILE

162 l of a fish oil-based ILE in 42 infants with SBS who developed cholestasis (serum direct bilirubin >2

163 the management of a 60 year old patient with SBS and recurrent joint attacks was for different medica

164 The patient with SBS presented with a body mass index of 16.5 kg/m2 after

165 A minority of patients with SBS and inflammatory bowel diseases had colon-in-continu

166 0.5% [n = 2/19]), whereas most patients with SBS and vascular or other diseases had colon-in-continui

167 al energy expenditure (TEE) in patients with SBS by using the doubly labeled water (DLW) method to in

168 In assessment of patients with SBS complaints, specific building-related illnesses sugg

169 er, and dietetic management of patients with SBS is extremely challenging.

170 small bowel transplantation in patients with SBS permanently dependent on PN.

171 ndomized, controlled trials in patients with SBS support the safety and efficacy of teduglutide as an

172 Measured TEE in patients with SBS was significantly higher than predicted by using sta

173 transplant surgical option for patients with SBS who require long-term PN.

174 We collected data from 85 patients with SBS with intestinal failure, according to the European S

175 stimate energy requirements of patients with SBS, and revision is needed to prevent underfeeding and

176 the effects of teduglutide on patients with SBS, we associated reduced parenteral support volume wit

177 e performed a 24-week study of patients with SBS-IF who were given subcutaneous teduglutide (0.05 mg/

178 as generally well tolerated in patients with SBS-IF.

179 reduces parenteral support in patients with SBS-IF.

180 stive capacity in parenterally fed rats with SBS.

181 transporter PepT1 in adults with or without SBS.