ライフサイエンスコーパス: SCR

1 SCR and participants' expectations of UCS presentation w

2 SCR and SCL23 are expressed specifically in the BS cells

3 SCR contributes to increased BVR by interspersed prolong

4 SCR drives the Na(+)/Ca(2+) exchange current inducing a

5 SCR estimates were strongly associated with local malari

6 Human phospholipid scramblase 1 (SCR) catalyzes phospholipid transmembrane (flip-flop) mo

7 l as in subjects aged >64 years (SPR, 91.1%; SCR, 78.2%) 21 days after vaccination.

8 own that the protein product of this is a 24 SCR protein that is secreted with normal fluid-phase act

9 variants on fH to short consensus repeat 6 (SCR 6).

10 issions from diesel vehicles equipped with a SCR.

11 Accurate SCR predictions can benefit homology modelling and seque

12 The low percentage of activity SCR over the local and near-dock cycles contributed to a

13 lockade led to further AP prolongation after SCR, and this strongly correlated with exaggerated BVR.

14 d during Ca(2+)-induced Ca(2+) release after SCR, and this contributes to AP prolongation.

15 In this study, an ammonia SCR system was examined with respect to its impact on bo

16 The action of an SCR catalyst with urea injection was found to increase t

17 The SCR-1/5 and SCR-16/20 fragments were less likely to bind zinc.

18 2013 model years) equipped with both DPF and SCR were 69 +/- 15%, 92 +/- 32%, and 66 +/- 35% lower, r

19 -ray scattering also showed that both FH and SCR-6/8 allotypes strongly aggregated at >10 muM zinc.

20 as detected by parasite prevalence rates and SCR estimates for samples of health care facility attend

21 novel link between zygotic reprogramming and SCR, providing a regulatory mechanism responsible for gl

22 The plant-specific GAI, RGA and SCR (GRAS) family proteins play critical roles in plant

23 tion of FANCJ helicase in regulating SCR and SCR associated gene amplification/duplications and imply

24 -specific transcriptional effects of SHR and SCR combined with data from chromatin immunoprecipitatio

25 et of a specific formative division, SHR and SCR directly activate a D-type cyclin; furthermore, alte

26 Our model reveals the timing of SHR and SCR dynamics and allows us to understand how protein mov

27 chain reaction (PCR), we showed that SHR and SCR regulate a similar but not identical set of stress r

28 Surprisingly, both SHR and SCR regulate expression of SIEL, so that siel/scr and si

29 ameters into a mathematical model of SHR and SCR, which shows that SHR reaches a steady state in minu

30 GA in part through the regulation of SHR and SCR.

31 formation experiments using chimeric SRK and SCR genes constructed with SC- and S36-derived sequences

32 24 accession, in which expression of SRK and SCR had been shown to exhibit a robust SI response.

33 clone was found to contain complete SRK and SCR sequences located close by one another in the derive

34 ctionality of endogenous A. thaliana SRK and SCR sequences.

35 xpectation, however, coexpression of SRK and SCR was found to inhibit SRK-mediated signaling and to d

36 ck between the action potential upstroke and SCR.

37 no direct correlation between REM sleep and SCRs, indicating that REM may only modulate fear acquisi

38 modern pollution reduction measures such as SCR and fuel with low sulfur content.

39 anted to establish whether the below average SCR performance observed in this study is a systemic iss

40 SCR is required for the interaction between SCR and LHP1 as well as with other interacting partners,

41 through SCR 18 to 20; PorB.1A can also bind SCR 6, but only weakly, as evidenced by a low level of b

42 trast, maltreated children exhibited blunted SCR to the CS+ and failed to exhibit differential SCR to

43 enuation of Ca(2+)-induced Ca(2+) release by SCR underlies AP prolongation via increased I(CaL.) Thes

44 configurations including engine-out, DOC+CuZ-SCR+AMOX, V-SCR+AMOX, and DOC+DPF+CuZ-SCR+AMOX.

45 OC+CuZ-SCR+AMOX, V-SCR+AMOX, and DOC+DPF+CuZ-SCR+AMOX.

46 NCJ pathological mutants exhibited defective SCR with an increased frequency of LTGC.

47 Defining SCRs requires the comparison of two or more homologous s

48 ive interaction with its pollen coat-derived SCR ligand.

49 nal climates, and planned increase in diesel SCR controls, it is imperative that we understand the ex

50 o the CS+ and failed to exhibit differential SCR to the CS+ vs CS- during early conditioning.

51 Disrupting SCR function abolished periclinal divisions in this late

52 SCI was used to distinguish SCRs from non-SCRs.

53 alytic Reduction after-treatment system (DPF-SCR) equipped heavy-duty diesel engine.

54 of long-tract gene conversion (LTGC) during SCR.

55 een Cu(I) and Cu(II) oxidation states during SCR reaction.

56 ver can be found at http://prodata.swmed.edu/SCR.

57 We also show that stigma-expressed SCR causes entrapment of its SRK receptor in the endopla

58 X-ray curve fits were obtained with extended SCR arrangements, showing that TT30 has a limited degree

59 fH-like protein 1 (FHL-1), which contains fH SCR 6, also bound to fHbp-expressing meningococci.

60 An fH SCRs 6-20 construct enhanced binding of unsialylated Por

61 of fH called fH-like protein-1 (contains fH SCRs 1-7) bound to gonococci but minimally to CHO-CR3.

62 of its five SCRs are highly homologous to fH SCRs 18-20) bound to CHO-CR3 and to unsialylated PorB.1A

63 o complement C3d, followed by the first five SCR domains of complement factor H that bind to compleme

64 gly, fH-related protein 1 (three of its five SCRs are highly homologous to fH SCRs 18-20) bound to CH

65 regulation is probably the common basis for SCR and GA activity in cortex cell proliferation.

66 in ground tissue patterning, we screened for SCR-interacting proteins using the yeast two-hybrid meth

67 nding controversy about the active sites for SCR of NO with NH3 by supported V2O5-WO3/TiO2 catalysts.

68 ith IL-2 was tolerable in the early post-HDC/SCR period.

69 replication fork stalling and chromosomal HR/SCR in mouse cells.

70 n gene products, BRCA1 and BRCA2, control HR/SCR at stalled replication forks.

71 t probably within the functionally important SCR-6/8 domains, and this explains why zinc inhibits FH

72 dentified histidine 337 and histidine 371 in SCR 6 as important for binding to fHbp.

73 llular Ca cycling to show that a decrease in SCR variability leads to a higher DAD amplitude and is d

74 cohesion during mitosis but are defective in SCR and sensitive to ionizing radiation (IR).

75 worst predictions pinpoints difficulties in SCR definitions.

76 does not bypass the requirement for Mms21 in SCR.

77 less coherent fibers and less myelination in SCR and PCR only in male infants, but these abnormalitie

78 of the Fe centers (reduced in NH3, partly in SCR mixture, slight reduction in NO) strongly changed.

79 c binding sites in FH, most of which were in SCR-6.

80 wing that TT30 has a limited degree of inter-SCR flexibility in its solution structure.

81 l, the modelled structures showed that inter-SCR domain contacts are likely, while these contacts are

82 ncludes regions of SCR1, SCR2, and the inter-SCR linker, specifically residues Arg(13), Tyr(16), Arg(

83 2 is primarily focused on SCR1 and the inter-SCR linker, specifically residues Asn(11), Arg(13), Ala(

84 nteractions, suggesting that the intervening SCRs (8-17) may impart a configurational and spatial req

85 conditions including low-temperature (473 K) SCR catalysis and are rationalized through first-princip

86 CFHR4 lacks SCRs homologous to the complement inhibitory domains of

87 kdown of hCtr1 (536 +/- 191 mm(3) for Lenti- SCR-shRNA-PC-3 or 208 +/- 104 mm(3) for Lenti-SCR-shRNA-

88 CR-shRNA-PC-3 or 208 +/- 104 mm(3) for Lenti-SCR-shRNA-DU-145, P < 0.01).

89 -shRNA-PC-3 and 5.57 +/- 1.20 %ID/g in Lenti-SCR-shRNA-DU-145, P < 0.001) by PET quantification.

90 kdown of hCtr1 (7.21 +/- 1.48 %ID/g in Lenti-SCR-shRNA-PC-3 and 5.57 +/- 1.20 %ID/g in Lenti-SCR-shRN

91 The most distally located SCRs (SCR1-2) mediate the interaction of CR2 with its fo

92 The most distally located SCRs, SCR1-2, mediate the interaction of CR2 with its fo

93 naling, we coexpressed an Arabidopsis lyrata SCR variant with its cognate SRK receptor in the stigma

94 understanding this microRNA (miRNA)-mediated SCR mechanism may shed light on the improvements of iPS

95 e cells in the presence of LPS, MM-2 and Mel-SCR were negligibly active.

96 e third analogue is a scrambled peptide (Mel-SCR) that contains the amino acid composition of melitti

97 ear-dock driving cycles, resulted in minimal SCR activity.

98 reshold, which are used in some of the newer SCRs, have the potential to control NOx emissions during

99 tor the Cu-CHA catalyst in action during NH3-SCR in the 150-400 degrees C range, targeting Cu oxidati

100 ective catalytic reduction of NO by NH3 (NH3-SCR) over a Fe-ZSM-5 zeolite catalyst.

101 ion kinetics on low-temperature standard NH3-SCR, supplemented by DFT calculations, as strong evidenc

102 and the isostructural Cu-SAPO-34) in the NH3-SCR of NOx.

103 SCI was used to distinguish SCRs from non-SCRs.

104 ntaneous activation of SRK in the absence of SCR ligand, these thioredoxins are thought to be essenti

105 ated to the previously described affinity of SCR for cholesterol-rich domains in membranes.

106 n of randomness by a statistical analysis of SCR events, which do not follow a Poisson process observ

107 otential risk factors for the development of SCR.

108 ard deviation of the latency distribution of SCR within a large population of myocytes in intact tiss

109 ovide evidence that the N-terminal domain of SCR is required for the interaction between SCR and LHP1

110 ed in terms of associating dimers of each of SCR-16/20 and C3d.

111 sting controversies on the interpretation of SCR geochemistry and the involvement of the putative Yel

112 Size distribution analyses of mixtures of SCR-16/20 or FH with C3d by analytical ultracentrifugati

113 In this study, the performance of SCR was studied by utilizing NOx, NH3, and particle meas

114 were used to predict how these properties of SCR determine DAD magnitude.

115 to measure the spatiotemporal properties of SCR within large myocyte populations in intact rat heart

116 elucidate the molecular basis of the role of SCR in ground tissue patterning, we screened for SCR-int

117 ing the sugar response and that this role of SCR is important for root growth.

118 regulating the initiation and termination of SCR in mammalian cells are poorly understood.

119 action potential upstroke, waiting times of SCR events after the upstroke are narrowly distributed,

120 strate that the variability of the timing of SCR in a population of cells in tissue decreases with SR

121 This result demonstrates that the timing of SCR occurs with less variability as the sarcoplasmic ret

122 ovide a better understanding of the usage of SCR in combination with a higher sulfur level fuel and a

123 A database of SCRs was compiled from 386 SCOP superfamilies containing

124 ened arousal and increased generalization of SCRs and explicit measures of shock expectancy.

125 ings indicate that gradual implementation of SCRs at power plants will result in an industry-wide inc

126 ial conditioning to the CS+ vs CS-, based on SCR and self-reported fear.

127 hus, the proposed state of the art review on SCR will create a renewed interest at all levels includi

128 n in conjunction with inactivation of SRK or SCR.

129 the three N-terminal domains (called CCPs or SCRs) of the important complement regulator, human facto

130 H4(+),ads intermediates dominate the overall SCR reaction, especially for hydrothermally aged catalys

131 it is still possible to effectively predict SCRs for a protein.

132 ogous structures, it is necessary to predict SCRs of a protein using information from only a set of h

133 neural networks were then trained to predict SCRs with various features deduced from a single structu

134 Predicted SCR-6/8 dimer structures showed that zinc binding sites

135 ) current (I(CaL)) with and without previous SCR indicated that I(CaL) was increased during Ca(2+)-in

136 pose that Scc1 sumoylation by Mms21 promotes SCR by antagonizing Wapl at a step after cohesin loading

137 ust temperature being high enough for proper SCR function.

138 oat-localized S-locus cysteine-rich protein (SCR).

139 e pollen S-locus Cys-Rich/S-locus Protein11 (SCR/SP11) ligand and the pistil S Receptor Kinase (SRK).

140 A number of putative SCR-interacting proteins were identified, among them LIK

141 tion rate [SPR], 97.2%; seroconversion rate [SCR], 90.1%) as well as in subjects aged >64 years (SPR,

142 rates and age-specific seroconversion rates (SCRs) of antibodies against Plasmodium falciparum antige

143 For AMA-1, the seroconversion rates (SCRs) ranged from 0.121 (Ngodhe) to 0.202 (Ungoye), and

144 ous allotypes of FH and both the recombinant SCR-6/8 allotypes with Tyr/His402.

145 Sister chromatid recombination (SCR) is a potentially error-free pathway for the repair

146 processed by sister chromatid recombination (SCR), generating error-free or error-prone homologous re

147 hesis during sister chromatid recombination (SCR).

148 h sister chromatid homologous recombination (SCR).

149 troduction of selective catalytic reduction (SCR) aftertreatment to meet stringent diesel NOx emissio

150 e presence of selective catalytic reduction (SCR) and selective noncatalytic reduction (SNCR) technol

151 in Cu/SSZ-13 selective catalytic reduction (SCR) catalysts have been recently identified as isolated

152 anadium-based selective catalytic reduction (SCR) catalysts, and ammonia oxidation (AMOX) catalysts a

153 st supporting selective catalytic reduction (SCR) chemistry.

154 ith EGR and a selective catalytic reduction (SCR) device were measured on two different routes with t

155 s compared to selective catalytic reduction (SCR) equipped diesel vehicles.

156 ssion limits, selective catalytic reduction (SCR) is increasingly utilized in ships, likely also in c

157 NH3-assisted selective catalytic reduction (SCR) of harmful nitrogen oxides and to unveil the SCR me

158 ctive for the selective catalytic reduction (SCR) of nitrogen oxides (NO x ) with ammonia (NH3), but

159 mistry in the selective catalytic reduction (SCR) of NO over Cu-exchanged SSZ-13.

160 technologies, selective catalytic reduction (SCR) of NOx by NH3 over Cu- and Fe-ion exchanged zeolite

161 ivity for the selective catalytic reduction (SCR) of NOx with NH3 are established through experimenta

162 , and ammonia selective catalytic reduction (SCR) of NOx.

163 ers (DPF) and selective catalytic reduction (SCR) on heavy-duty diesel truck emissions were studied a

164 el and have a Selective Catalytic Reduction (SCR) system for NO(X) abatement.

165 d with a urea selective catalytic reduction (SCR) system.

166 oduct of urea-selective catalytic reduction (SCR) systems that are being phased-in to control on-road

167 er (DPF), and selective catalytic reduction (SCR) were tested on a chassis dynamometer.

168 equipped with selective catalytic reduction (SCR), two LNG's equipped with three-way catalyst (TWC),

169 dentification of a stability control region (SCR), residues 97-118, in the Tm sequence that controls

170 revalence of structurally conserved regions (SCRs) even in highly divergent protein families.

171 to the BS cells, where it directly regulates SCR and SCL23 expression.

172 vel function of FANCJ helicase in regulating SCR and SCR associated gene amplification/duplications a

173 mprises 15 or 16 short complement regulator (SCR) domains in a partially folded-back but flexible str

174 r H (FH) with 20 short complement regulator (SCR) domains is associated with age-related macular dege

175 m the first four short complement regulator (SCR) domains of complement receptor type 2 (CR2) that bi

176 s composed of 20 short complement regulator (SCR) domains.

177 The C-terminal short complement regulator (SCR)-20 domain of factor H (FH), the major serum regulat

178 We believe that subclinical rejection (SCR) may be 1 of the factors that contribute to graft lo

179 ittle is known about the mechanisms relating SCR and triggered activity on the tissue scale.

180 due to spontaneous calcium (Ca(2+)) release (SCR) from intracellular stores after the end of a preced

181 ctivity because spontaneous calcium release (SCR) activates sufficient Ca-sensitive inward currents t

182 that contained only the apparently relevant SCRs (6, 7, and 18-20) bound to CHO-CR3 and to gonococci

183 ene encodes a unique short consensus repeat (SCR) domain protein.

184 that are composed of short consensus repeat (SCR) domains.

185 ains (fH contains 20 short consensus repeat [SCR] domains) fused to murine Fc, we observed strong bin

186 n of the N-terminal short consensus repeats (SCRs) that are conserved in FHR2 and FHR5.

187 or 16 extracellular short consensus repeats (SCRs), that promotes B lymphocyte responses and bridges

188 or 16 extracellular short consensus repeats (SCRs), that promotes B lymphocyte responses and bridges

189 thylation during somatic cell reprogramming (SCR) of hHFCs.

190 gh-dose chemotherapy (HDC)/stem-cell rescue (SCR).

191 Wapl, a negative cohesin regulator, rescues SCR defects of Mms21-deficient or Scc1 15KR-expressing c

192 4-fold) in a schedule-controlled responding (SCR) behavioral assay.

193 the unconditioned skin conductance response (SCR).

194 Skin conductance responses (SCR) and self-reported fear were acquired.

195 Additionally, skin conductance responses (SCRs) were weakly correlated to the activity in the amyg

196 c arousal [i.e., skin conductance responses (SCRs)] and explicit measures of shock expectancy served

197 kinase (SRK) and its S-locus cysteine rich (SCR) ligand.

198 kinase (SRK) and the S-locus cysteine-rich (SCR) genes, as well as unlinked modifier loci required f

199 pollen coat-localized S-locus cysteine-rich (SCR) ligand [2-5] and the resulting rejection of pollen

200 oding its ligand, the S-locus cysteine-rich (SCR) protein, which is localized in the pollen coat.

201 localized ligand, the S-locus cysteine-rich (SCR) protein.

202 localized ligand, the S-locus cysteine-rich (SCR) protein.

203 The origin of the Steens-Columbia River (SCR) flood basalts, which is presumed to be the onset of

204 se reporter construct revealed that in root, SCR, but not SHR, repressed ABI4 and ABI5 directly and s

205 ct with the AtSHR binding protein, Scarecow (SCR).

206 tion factors SHORT-ROOT (SHR) and SCARECROW (SCR) are key regulators of root growth and of the asymme

207 SHORT-ROOT (SHR) and SCARECROW (SCR) are key regulators of stem cell renewal and radial

208 ow that embryos that lack SCZ and SCARECROW (SCR) functions do not form a ground tissue because they

209 AS proteins, SHORT-ROOT (SHR) and SCARECROW (SCR), cooperatively direct asymmetric cell division and

210 controlled by SHORTROOT (SHR) and SCARECROW (SCR).

211 gulates the transcription factors SCARECROW (SCR) [2] and JACKDAW (JKD), which in turn inhibit moveme

212 required for proper expression of SCARECROW (SCR), a key regulator of root patterning, and for stem-c

213 In the Arabidopsis root, SCARECROW (SCR) is required for the first cell division, but repres

214 iption factors, SHORT-ROOT (SHR), SCARECROW (SCR) and SCARECROW-LIKE 23 (SCL23), affect BS cell fate

215 action with its downstream target SCARECROW (SCR) control root patterning and cell fate specification

216 root primordia, within which the SCARECROW (SCR) transcription factor was specifically expressed.

217 SHR, SCR and SCL23 homologs are present in many plant species

218 a pathway that partially overlaps with SHR, SCR, PLETHORA1 and PLETHORA2 (PLT1 and PLT2).

219 io-temporal dynamics of SHR movement and SHR-SCR interaction is currently unavailable.

220 e SHR-SCR binary and JACKDAW (JKD)/IDD10-SHR-SCR ternary complexes.

221 we present the crystal structures of the SHR-SCR binary and JACKDAW (JKD)/IDD10-SHR-SCR ternary compl

222 eady state in minutes, while SCR and the SHR-SCR complex reach a steady-state between 18 and 24 hr.

223 ,ads intermediates exhibit a higher specific SCR activity (TOF).

224 ype alleles, the rad57 defect in spontaneous SCR was not strongly suppressed by these same factors.

225 e canonical Brassicaceae S locus genes (SRK, SCR), and is situated in a genomic position that differs

226 ompatibility upon transformation with an SRK-SCR gene pair isolated from its self-incompatible close

227 sult of transformation with a functional SRK-SCR gene pair, we identify Auxin Response Factor 3 (ARF3

228 her abolish nor weaken SI in A. thaliana SRK-SCR plants.

229 ecules in the SI response of A. thaliana SRK-SCR plants.

230 are not essential for specificity in the SRK-SCR interaction.

231 t several additional and highly diverged SRK/SCR genes from A. lyrata and another crucifer plant, Cap

232 ocation or recombination event involving SRK/SCR and Lal2/SCRL likely occurred, together with neofunc

233 us to test the functional equivalence of SRK/SCR gene pairs from different taxa and to assay the func

234 ctively, and crossing data show that the SRK/SCR haplotype is functional in self-incompatibility.

235 Significantly, the low-temperature standard SCR mechanism proposed here provides full consistency wi

236 ce both Cu(II) and Cu(I) ions under standard SCR conditions at 473 K.

237 = .02) and higher diffusivities in superior (SCR) and posterior corona radiatae (PCR) (group x age x

238 Suppressing SCR via inhibition of ryanodine receptors, Ca(2+)/calmod

239 enting the active sites for high-temperature SCR.

240 ovides full consistency with low-temperature SCR kinetics.

241 Low-temperature SCR, up to approximately 200 degrees C, is characterized

242 s C-reactive protein via its most N-terminal SCR, which leads to classical complement pathway activat

243 The study further suggests the fact that SCR-equipped engines operating within the Not-To-Exceed

244 Our findings indicate that SCR contains an amino acid segment at the C-terminal reg

245 p experiments and sugar assays, we show that SCR is primarily involved in sugar transport whereas SCL

246 These results together suggest that SCR has a SHR-independent role in mitigating the sugar r

247 believe that there are data to suggest that SCR leads to progressive fibrosis and loss of graft func

248 e prediction web servers also suggested that SCR-6 and other domains bind zinc.

249 The SCR database and the prediction server can be found at h

250 The SCR revaluation effect was not dependent on explicit mem

251 The SCR-1/5 and SCR-16/20 fragments were less likely to bind

252 The particles were measured after the SCR with an engine exhaust particle sizer spectrometer (

253 energies and apparent reaction orders at the SCR conditions, even on zeolite frameworks other than SS

254 for typical highway driving conditions, the SCR technology is proving to be effective in controlling

255 ccurrence of major volcanic dikes during the SCR-Northern Nevada Rift flood basalt event both in spac

256 g information along the coiled-coil from the SCR.

257 In the case of the LSD and ULSD fuels, the SCR system also significantly reduced emissions of compo

258 However, for all tested fuels, the SCR system produced significantly (p < 0.05) higher emis

259 Greater FA in the SCR and precentral gyrus white matter were associated wi

260 the hydrophobic core is destabilized in the SCR by Ala residues at three consecutive d positions.

261 rtly in the first amino acid residues in the SCR C-terminal extracellular coil.

262 rity Fe sites was synthesized, tested in the SCR reaction and characterized by UV-vis, X-ray absorpti

263 oth species were found to participate in the SCR reaction, their relative population depends on the c

264 ammonia level was induced downstream of the SCR catalyst.

265 We demonstrate the importance of the SCR in controlling and transmitting the stability signal

266 nts expressing ABI4 under the control of the SCR promoter manifested a short-root phenotype.

267 For all studied fuels, the use of the SCR system yielded statistically significant (p < 0.05)

268 , we explore the possible interaction of the SCR TMD with cholesterol by using a variety of experimen

269 loading, the low temperature behavior of the SCR was enhanced.

270 The single mutation A109L prevents the SCR from transmitting stabilizing information and separa

271 of harmful nitrogen oxides and to unveil the SCR mechanism.

272 However, under operations where the SCR's do not reach minimum operating temperature, like c

273 While the SCR/SP11-SRK recognition system has been identified in s

274 d) PorB.1A-bearing gonococci bind fH through SCR 18 to 20; PorB.1A can also bind SCR 6, but only weak

275 urine Fc, we observed strong binding through SCRs 18-20, whereas weaker binding occurred through SCRs

276 -20, whereas weaker binding occurred through SCRs 6-10.

277 diated Cu(I)-->Cu(II) redox step integral to SCR.

278 ense beta-adrenergic stimulation, leading to SCR.

279 catalyzing the hydrolysis reaction prior to SCR.

280 hasize the need for model inputs relative to SCR performance as a function of driving cycle and engin

281 was not much longer than that of the unbound SCR-16/20 dimer.

282 conditioned diminution of the unconditioned SCR.

283 cies are solvated and mobilized by NH3 under SCR conditions.

284 ns including engine-out, DOC+CuZ-SCR+AMOX, V-SCR+AMOX, and DOC+DPF+CuZ-SCR+AMOX.

285 adium-based selective catalytic reductors (V-SCR) retrofit.

286 e emission rate of V(V) determined for the V-SCR equipped vehicle (103 ng/mile) was 40-fold greater t

287 adium-containing fine-particle PM from the V-SCR identified V(2)O(5) as the dominant vanadium species

288 The V(V) content of the V-SCR PM (6.6 mug/g) was 400-fold greater than that in PM

289 hment in lateral root primordia operates via SCR-mediated formative cell division and coincides with

290 e upstroke are narrowly distributed, whereas SCR amplitudes follow a broad normal distribution with a

291 s (in 25-30 nm size), the formation of which SCR either increased or decreased.

292 SHR reaches a steady state in minutes, while SCR and the SHR-SCR complex reach a steady-state between

293 exposure and were negatively associated with SCR to the CS+ during early conditioning in the total sa

294 lity, oligomeric state, and association with SCR using a combination of Fluorescent Correlation Spect

295 maximum dimension of the C3d complexes with SCR-16/20 at 29 nm was not much longer than that of the

296 nd one hydraulic hybrid diesel equipped with SCR, were measured using a portable emissions measuremen

297 es in the interactions of fHbp variants with SCR 6 were evident.

298 omposition of NO(x) emitted with and without SCRs and SNCRs; further the isotopic composition of powe

299 allow active and inactive species in deNO(x)-SCR to be identified.

300 ic reduction of NO(x) using ammonia (deNO(x)-SCR) and characterizing the underlying distribution of c