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1 SCS and SCL were visible in 15 eyes.
2 SCS applied with the three frequencies produced similar
3 SCS at 500 Hz significantly increased cutaneous blood fl
4 SCS at 500 Hz significantly increased SCS-induced vasodi
5 SCS at frequencies of 50, 200, or 500 Hz was applied at
6 SCS converge to form local gaps that close based on both
7 SCS disrupted this pathological circuit behavior in a ma
8 SCS injection of low (0.2 mg) and high doses (2.0 mg) of
9 SCS is a dominant disorder with mutations reported for a
10 SCS is an under-recognized cause of longitudinally exten
11 SCS is used to improve peripheral circulation in selecte
12 SCS patients had constitutional symptoms, cerebrospinal
13 SCS significantly improved cardiac contractile function
14 SCS was delivered at one of 3 different frequencies (4 H
15 l and reversed polarities for samples PO-01, SCS-01 and SCS-02, indicating a primary record of the Ea
21 (LarA) of Lactobacillus plantarum harbors a (SCS)Ni(II) pincer complex derived from nicotinic acid.
22 onal theory optimizations, the high-accuracy SCS-MP2/cc-pVinfinityZ energies, and PCM solvation corre
25 mals were perfused either 5 min or 2 h after SCS and c-fos protein examined immunohistochemically.
27 significantly increased 5 min (35 min after SCS began) after 4 Hz SCS in the NRM, but not PAG in ani
28 sed polarities for samples PO-01, SCS-01 and SCS-02, indicating a primary record of the Earth's magne
29 fraction was noted in the SCS (52+/-5%) and SCS+MEDS (46+/-4%) groups compared with the MEDS (38+/-2
36 ding the neural circuits related to pain and SCS in the dorsal horn, supraspinal structures, and the
37 nts for cell movement between parenchyma and SCS in what we speculate is a program of immune surveill
39 e authors' hypothesis that a single anterior SCS injection can reach the ocular posterior segment was
47 itochondrial extracts and purified bacterial SCS (BSCS), we showed that this enhanced 32P labeling re
49 rther research into the interactions between SCS and pain pathways in the nervous system using animal
50 e both ATP and GTP fit into the Blastocystis SCS active site, GTP is destabilized by electrostatic di
51 ate the risk of SCS-related complications by SCS exposure and quantify the associated health care cos
52 To elucidate potential sites modulated by SCS we examined distribution of c-fos in Sprague-Dawley
55 mechanisms underlying the pain reduction by SCS and how different frequencies of SCS produce the ana
58 elationship was demonstrated between chronic SCS use and risk of SCS-related complications in patient
62 nce treatment with systemic corticosteroids (SCSs) to control daily symptoms and prevent serious acut
72 and persistent enhancement >2 months favored SCS, and ringlike enhancement favored NMOSD (p < 0.05).
74 points examined in this model, low frequency SCS likely activates supraspinal and spinal mechanisms t
76 was shown that P(i) binds the porcine heart SCS alpha-subunit (SCSalpha) in a noncovalent manner and
79 gher and rib cage movement greater during HF-SCS compared to spontaneous breathing, stimulus amplitud
80 f the external intercostal muscles during HF-SCS is similar to that occurring during spontaneous brea
82 eous breathing, stimulus amplitude during HF-SCS was adjusted such that rib cage movement matched (Pr
83 dorsoventral gradients of activity during HF-SCS were not significantly different compared to spontan
86 , high frequency spinal cord stimulation (HF-SCS) results in more physiological activation of these m
87 tients' follow-up into low, medium, and high SCS exposure (</= 6, >6-12, and >12 mg/d, respectively).
90 eam spacing of 375 microm and larger hinders SCS advancement, thus providing abilities to engineer cl
92 ed 5 min (35 min after SCS began) after 4 Hz SCS in the NRM, but not PAG in animals with nerve injury
94 d bilaterally 2 h after either 4 Hz or 60 Hz SCS in the spinal cord dorsal horn in the cervical enlar
95 Previously we showed both 4 Hz and 60 Hz SCS reduces hyperalgesia in an animal model of neuropath
100 s unfavorable for typhoon intensification in SCS, which therefore serves as a natural buffer that shi
103 e and well tolerated therapeutic strategy in SCS and other conditions of excitotoxicity or Ca(2+) ove
105 M1/CHARMM22 level) and high-level ab initio (SCS-MP2) QM/MM calculations to analyze the reactions, an
106 On the other hand, animals from ischemia/SCS control group all died within 12 hr postoperatively
109 The zebrafish mutant line, twister, models SCS in terms of a dominant mutation in the alpha subunit
111 and optB88-vdW), wave function theory (MP2, SCS(MI)-MP2, MP2.5, MP2.X, and CCSD(T)), and empirical c
113 However, on Dongsha Atoll, in the northern SCS, unusually weak winds created low-flow conditions th
115 mpared to FLS with a focus score of <1 or NS/SCS, is strongly associated with the ocular and serologi
117 s also revealed a P(i)-induced activation of SCS activity by more than 2-fold when mitochondrial extr
118 ignificantly with frequent administration of SCS, and patients on such medications should undergo pre
120 srupted organization reduces the capacity of SCS macrophages to retain and present antigen in a subse
121 approach, several application categories of SCS produced materials, such as for energy conversion an
127 re found between those with a severe form of SCS and controls in rates of parkinsonism (19% vs 17%; o
132 , non-ischemic livers preserved with 6 hr of SCS or NELP and unpreserved ischemic livers were transpl
133 of warm ischemia and preserved with 5 hr of SCS or NELP, and transplanted into syngeneic recipients.
135 derlying chronic pain and the interaction of SCS with these circuits may underlie this plateau in cli
136 by down-regulating the expression levels of SCS A-beta in cultured mouse neurons, we have found that
137 extent, validity, and historical meaning of SCS findings, as well as their influence and the influen
139 this study suggest that 0.2 mg and 2.0 mg of SCS TA was as effective in reducing inflammation as 2.0
140 composition, structure, and nanostructure of SCS products, and routes to regulate the size and morpho
141 and inflammation disrupt the organization of SCS macrophages in a manner that involves the migration
142 rnished our knowledge on the pathogenesis of SCS A-beta deficiency-related mitochondrial diseases and
143 Given the negative emission potential of SCS and biochar and their potential advantages compared
147 s were used to estimate the adjusted risk of SCS-related complications for patients with medium and h
148 nstrated between chronic SCS use and risk of SCS-related complications in patients with severe asthma
149 S exposure had significantly higher risks of SCS-related complications, including infections and card
150 rial diseases and revealed the vital role of SCS A-beta in maintaining neuronal mitochondrial quality
152 slowly decaying synaptic currents typical of SCS that transit to a much faster decay upon the appeara
156 s were carried out with M06-2X/6-311+G(d,p), SCS-MP2/6-311+G(d,p)//M06-2X/6-311+G(d,p), and B97D/6-31
157 Lithospheric storage of OC in peripheral SCS basins potentially exceeded 4,000 Gt (equivalent to
158 ntrast arrived at the opposite and posterior SCS 7.8 (4.6) and 7.7 (4.6) seconds after injection, res
163 37) (2.37 mg/kg, i.v.) significantly reduced SCS-induced vasodilation at 500 Hz (P<0.05, n=6) as comp
164 f currents by salicylidine salycylhydrazide (SCS) indicate expression of the beta2 or beta3 subunit,
166 al currents (significant current scattering, SCS) could reliably discriminate between consciousness a
167 fat, and protein yield; somatic cell score (SCS); productive life (PL); and daughter pregnancy rate
168 the Zhongsha Islands in the South China Sea (SCS) deep basin during the period from August 2012 to Ja
170 ce temperature (SST) of the South China Sea (SCS) increased by 2 degrees C in response to the develop
171 The deep ocean bed of the South China Sea (SCS) is considered to be largely devoid of anthropogenic
174 etected in the northwestern South China Sea (SCS) with its intensity, size, polarity and structure be
182 mphocytes that survey the subcapsular sinus (SCS) and associated macrophages for pathogen entry.
183 yer of macrophages at the subcapsular sinus (SCS) captures pathogens entering the lymph node, prevent
184 s and preferentially bind subcapsular sinus (SCS) CD169(+) macrophages in tumor-draining lymph nodes
185 h maintained a protective subcapsular sinus (SCS) macrophage phenotype within virus draining lymph no
188 s, relocalized toward the subcapsular sinus (SCS) near invaded macrophages, and engaged in prolonged
189 hages, which populate the subcapsular sinus (SCS) of LNs, are critical for the clearance of viruses f
190 structures with a single formal charge site (SCS), whereas ArgArg x H(+) has a salt-bridge (SB) struc
193 ward thickening of the suprachoroidal space (SCS) compared with monotherapy alone (13.4 mum vs 5.3 mu
195 is unclear whether spinal cord stimulation (SCS) at higher frequencies induces further increases in
199 ve method--epidural spinal cord stimulation (SCS)--has been suggested as an alternative approach for
200 fiber (ZCS) input during visual stimulation; SCS cells fired complex spikes associated with learned s
201 We investigated whether static cold storage (SCS) can be safely replaced with a novel technique of pr
203 livers by conventional static cold storage (SCS) is associated with an unacceptable risk of primary
209 rom 15 cohorts of the Seven Countries Study (SCS), the first systematic study of diet, risk character
210 ieved using a structured chimeric substrate (SCS) that releases a downstream activator after cleavage
213 e ACh receptors cause slow-channel syndrome (SCS) and Escobar syndrome, two forms of congenital myast
215 ty and the slow-channel myasthenic syndrome (SCS), IP(3)R(1) knockdown eliminated NMJ Ca(2+) overload
219 ically performed by succinyl-CoA synthetase (SCS), has arisen in diverse bacterial groups, including
220 is of mtGTP made by succinyl-CoA synthetase (SCS-GTP) to an anaplerotic pathway producing phosphoenol
221 study investigated the effects of long-term SCS on ventricular function in a postinfarction canine h
224 bies virus transmitted by insect bites, that SCS macrophages perform a third vital function: they pre
225 concentration is increased, we conclude that SCS activation by P(i) binding represents another mitoch
227 n cultured mouse neurons, we have found that SCS A-beta deficiency induces severe mitochondrial dysfu
234 demonstrated a visible SCS at baseline, the SCS expanded significantly after suprachoroidal CLS-TA i
235 /6-31+G(d,p) structures and energies for the SCS and SB forms of these protonated dipeptides indicate
238 e 3 also were significantly decreased in the SCS (27+/-17 and 27%, respectively; P<0.03) and MED (58+
239 covery in ejection fraction was noted in the SCS (52+/-5%) and SCS+MEDS (46+/-4%) groups compared wit
240 g isotopic compositions were observed in the SCS (delta(202)Hg, from -2.82 to -2.10 per thousand; Del
241 n fraction was significantly improved in the SCS compared with the MED and CTRL groups (P<0.001 for b
243 RT-PCR of reperfusion biopsy samples in the SCS group showed high expression of inflammatory cytokin
256 eys, near the end of his long career, of the SCS design, conduct, and findings, with his discussion a
261 dently associated with the visibility of the SCS: disarrangement of plexiform layers, CRT, and multip
262 contrast was visible in 24.0%to 27.2% of the SCS; in 10 of 12 eyes, contrast reached the posterior po
263 ermore, quantum chemical calculations on the SCS-MP2 and DFT levels gave insights into the reaction m
266 2, 28 surviving animals were assigned to the SCS (delivered at the T4/T5 spinal region for 2 hours 3
267 roducing plasmacytoid dendritic cells to the SCS and in addition were a major source of IFN-I themsel
268 hat stability of the SB form relative to the SCS form generally increases with increasing gas-phase b
270 e 1 survivors were equally randomized to the SCS, MEDS (carvedilol plus ramipril 2.5 mg PO QD), SCS p
281 wo hours of oxygenated HMP after traditional SCS restores hepatic ATP levels and improves hepatobilia
283 tion, animals transplanted with NEVKP versus SCS grafts demonstrated similar serum creatinine peak le
284 In the 15 eyes that demonstrated a visible SCS at baseline, the SCS expanded significantly after su
286 reatinine peak levels (NEVKP, 2.0 +/- 0.5 vs SCS 2.7 +/- 0.7 mg/dL; P = 0.11) and creatinine clearanc
290 preserved either by simple cold storage (WI-SCS group) or HMP (WI-HMP group) using Belzer-MPS soluti
291 ndrome was observed in all animals of the WI-SCS group but none of the control or WI-HMP groups.
293 atients evaluated between 1996 and 2015 with SCS or NMOSD whose first myelitis episode was accompanie
294 gies might reduce the burden associated with SCS-related complications in patients with severe asthma
296 rved with HMP than for organs preserved with SCS (34.2% vs 42.0%, P < .001), despite a slightly longe
297 ar factors that bind to genomic regions with SCS could functionally interact with SOX9, we identified
298 P = 0.59), whereas animals transplanted with SCS grafts had persistently elevated serum creatinine an
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