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1                                              SCS and SCL were visible in 15 eyes.
2                                              SCS applied with the three frequencies produced similar
3                                              SCS at 500 Hz significantly increased cutaneous blood fl
4                                              SCS at 500 Hz significantly increased SCS-induced vasodi
5                                              SCS at frequencies of 50, 200, or 500 Hz was applied at
6                                              SCS converge to form local gaps that close based on both
7                                              SCS disrupted this pathological circuit behavior in a ma
8                                              SCS injection of low (0.2 mg) and high doses (2.0 mg) of
9                                              SCS is a dominant disorder with mutations reported for a
10                                              SCS is an under-recognized cause of longitudinally exten
11                                              SCS is used to improve peripheral circulation in selecte
12                                              SCS patients had constitutional symptoms, cerebrospinal
13                                              SCS significantly improved cardiac contractile function
14                                              SCS was delivered at one of 3 different frequencies (4 H
15 l and reversed polarities for samples PO-01, SCS-01 and SCS-02, indicating a primary record of the Ea
16 .48 mm/Ma and 11.28 mm/Ma for samples PO-01, SCS-01, SCS-02 and IO-01, respectively.
17 a and 11.28 mm/Ma for samples PO-01, SCS-01, SCS-02 and IO-01, respectively.
18 ific Ocean (PO-01), South China Sea (SCS-01, SCS-02) and Indian Ocean (IO-01).
19           We studied 71 patients (NMOSD, 37; SCS, 34).
20                                Sixteen (47%) SCS cases were initially diagnosed as NMOSD or idiopathi
21 (LarA) of Lactobacillus plantarum harbors a (SCS)Ni(II) pincer complex derived from nicotinic acid.
22 onal theory optimizations, the high-accuracy SCS-MP2/cc-pVinfinityZ energies, and PCM solvation corre
23               However, how infection affects SCS macrophages remains largely unexplored.
24 rs of oxygenated HMP (at 12 degrees C) after SCS and before NMP.
25 mals were perfused either 5 min or 2 h after SCS and c-fos protein examined immunohistochemically.
26 s underwent NMP without oxygenated HMP after SCS.
27  significantly increased 5 min (35 min after SCS began) after 4 Hz SCS in the NRM, but not PAG in ani
28 sed polarities for samples PO-01, SCS-01 and SCS-02, indicating a primary record of the Earth's magne
29  fraction was noted in the SCS (52+/-5%) and SCS+MEDS (46+/-4%) groups compared with the MEDS (38+/-2
30 ere explored by means of B3LYP, B3LYP-D, and SCS-MP2 calculations.
31                                      DFT and SCS-ADC(2) calculations showed that contaminant-contamin
32              We have employed hybrid DFT and SCS-MP2 calculations at the SMD-PCM-6-311++G(2d,2p)//6-3
33 high level quantum chemical methods (DFT and SCS-MP2-calculations).
34  several DFT methods as well as with MP2 and SCS-MP2.
35 were carried out with M06-2X/6-311G(d,p) and SCS-MP2/6-311G(d,p)//M06-2X/6-311G(d,p) methods.
36 ding the neural circuits related to pain and SCS in the dorsal horn, supraspinal structures, and the
37 nts for cell movement between parenchyma and SCS in what we speculate is a program of immune surveill
38  for the lowly heritable traits DPR, PL, and SCS.
39 e authors' hypothesis that a single anterior SCS injection can reach the ocular posterior segment was
40                                 The anterior SCS of adult porcine and canine ex vivo eyes was cannula
41 ection site to the opposite ventral anterior SCS and the posterior SCS.
42                              Calculations at SCS-MP2/CBS-level of theory and experimental data of the
43                              Calculations at SCS-MP2/CBS//TPSS-D3/def2-SVP-level of theory provided p
44 a barrier of 22.7 kcal/mol (22.2 kcal/mol at SCS-MP2/cc-PVTZ).
45 ntly higher, 34.6 kcal/mol (41.9 kcal/mol at SCS-MP2/cc-PVTZ).
46       LY294002 at 100 microM also attenuated SCS-induced vasodilation at 60% and 90% of MT (P<0.05).
47 itochondrial extracts and purified bacterial SCS (BSCS), we showed that this enhanced 32P labeling re
48  racemase, which features a pyridinium-based SCS pincer ligand framework bound to nickel.
49 rther research into the interactions between SCS and pain pathways in the nervous system using animal
50 e both ATP and GTP fit into the Blastocystis SCS active site, GTP is destabilized by electrostatic di
51 ate the risk of SCS-related complications by SCS exposure and quantify the associated health care cos
52    To elucidate potential sites modulated by SCS we examined distribution of c-fos in Sprague-Dawley
53 ls that received healthy livers preserved by SCS (n=9) or NELP (n=11) for 6 hr.
54 d from heart-beating donors and preserved by SCS served as controls.
55  mechanisms underlying the pain reduction by SCS and how different frequencies of SCS produce the ana
56              Given that mtGTP synthesized by SCS-GTP is an indicator of TCA flux that is crucial for
57                        Thus, we characterize SCS macrophages as specialized antigen-presenting cells
58 elationship was demonstrated between chronic SCS use and risk of SCS-related complications in patient
59 ent serious acute exacerbations, but chronic SCS use is associated with complications.
60 ses and had more than 6 months of continuous SCS use.
61 tent or continuous systemic corticosteroids (SCS) or high-dose ICS.
62 nce treatment with systemic corticosteroids (SCSs) to control daily symptoms and prevent serious acut
63                                     Defected SCS ADP-forming beta subunit (SCS A-beta) is linked to l
64 l lines established from CHL and RLN-derived SCSs remain cytokine-secretory.
65 ures that, taken together, help discriminate SCS from NMOSD.
66              In this review, we will discuss SCS technologies and applications, as well as translatio
67                                  A disrupted SCS macrophage barrier enables tEVs to enter the lymph n
68 rages of 1.36 mg/dL (HMP) versus 1.40 mg/dL (SCS).
69                                     Low-dose SCS TA was also effective in reducing inflammation; howe
70                                    Effective SCS-sparing strategies might reduce the burden associate
71 medium occupied 39.0% to 52.1% of the entire SCS.
72 and persistent enhancement >2 months favored SCS, and ringlike enhancement favored NMOSD (p < 0.05).
73     Median delay to diagnosis was longer for SCS than NMOSD (5 vs 1.5 months, p < 0.01).
74 points examined in this model, low frequency SCS likely activates supraspinal and spinal mechanisms t
75 one) versus those with low (reference group) SCS exposure.
76  was shown that P(i) binds the porcine heart SCS alpha-subunit (SCSalpha) in a noncovalent manner and
77                                           HF-SCS may provide a more physiological method of inspirato
78 terspace during spontaneous breathing and HF-SCS following C2 spinal section.
79 gher and rib cage movement greater during HF-SCS compared to spontaneous breathing, stimulus amplitud
80 f the external intercostal muscles during HF-SCS is similar to that occurring during spontaneous brea
81                 Stimulus amplitude during HF-SCS was adjusted such that inspired volumes matched spon
82 eous breathing, stimulus amplitude during HF-SCS was adjusted such that rib cage movement matched (Pr
83 dorsoventral gradients of activity during HF-SCS were not significantly different compared to spontan
84                                    During HF-SCS, mean peak SMU firing frequency was highest in the 3
85 ce during either spontaneous breathing or HF-SCS.
86 , high frequency spinal cord stimulation (HF-SCS) results in more physiological activation of these m
87 tients' follow-up into low, medium, and high SCS exposure (</= 6, >6-12, and >12 mg/d, respectively).
88                Patients with medium and high SCS exposure had significantly higher risks of SCS-relat
89                              Medium and high SCS exposure were also associated with significantly mor
90 eam spacing of 375 microm and larger hinders SCS advancement, thus providing abilities to engineer cl
91                                       100 Hz SCS had no effect on c-fos expression.
92 ed 5 min (35 min after SCS began) after 4 Hz SCS in the NRM, but not PAG in animals with nerve injury
93                    In uninjured animals 4 Hz SCS increased c-fos expression at the electrode site and
94 d bilaterally 2 h after either 4 Hz or 60 Hz SCS in the spinal cord dorsal horn in the cervical enlar
95     Previously we showed both 4 Hz and 60 Hz SCS reduces hyperalgesia in an animal model of neuropath
96                       These results identify SCS macrophages as crucial gatekeepers to the CNS that p
97                         Our results identify SCS macrophages as primary mediators of NK cell activati
98                                           In SCS, the new index accounts for as high as 75% of the to
99 iologic IOP had no significant difference in SCS distension.
100 s unfavorable for typhoon intensification in SCS, which therefore serves as a natural buffer that shi
101 st that ERK and AKT pathways are involved in SCS-induced vasodilation.
102 ficient LNs VSV replicated preferentially in SCS macrophages but not in adjacent nerves.
103 e and well tolerated therapeutic strategy in SCS and other conditions of excitotoxicity or Ca(2+) ove
104        SCS at 500 Hz significantly increased SCS-induced vasodilation without influencing MT.
105 M1/CHARMM22 level) and high-level ab initio (SCS-MP2) QM/MM calculations to analyze the reactions, an
106     On the other hand, animals from ischemia/SCS control group all died within 12 hr postoperatively
107 [P < .0001] and 1.59 [P < .0001]) versus low SCS exposure.
108 ultiple, single, or zero complex spike (MCS, SCS, ZCS) cells.
109   The zebrafish mutant line, twister, models SCS in terms of a dominant mutation in the alpha subunit
110 blockade of ERK and AKT activation modulated SCS-induced vasodilation.
111  and optB88-vdW), wave function theory (MP2, SCS(MI)-MP2, MP2.5, MP2.X, and CCSD(T)), and empirical c
112 ls (n = 52) with a minimal form of SCS or no SCS.
113   However, on Dongsha Atoll, in the northern SCS, unusually weak winds created low-flow conditions th
114                   Distinguishing FLS from NS/SCS is essential in assessing LSG biopsies, before deter
115 mpared to FLS with a focus score of <1 or NS/SCS, is strongly associated with the ocular and serologi
116 cific or sclerosing chronic sialadenitis (NS/SCS).
117 s also revealed a P(i)-induced activation of SCS activity by more than 2-fold when mitochondrial extr
118 ignificantly with frequent administration of SCS, and patients on such medications should undergo pre
119                           The association of SCS with leukoaraiosis suggests that it is part of a mor
120 srupted organization reduces the capacity of SCS macrophages to retain and present antigen in a subse
121  approach, several application categories of SCS produced materials, such as for energy conversion an
122                       Moreover, depletion of SCS macrophages using clodronate liposomes abolished NK
123                       Further development of SCS injections for treatment of the ocular posterior seg
124                      Furthermore, effects of SCS at 500 Hz are mediated via activation of TRPV1-conta
125           This study investigated effects of SCS at both a normal frequency (as used clinically) and
126                  Cases with a severe form of SCS (n = 27) were randomly selected for comparison with
127 re found between those with a severe form of SCS and controls in rates of parkinsonism (19% vs 17%; o
128 hed controls (n = 52) with a minimal form of SCS or no SCS.
129 atures with the presence of a severe form of SCS.
130 lcium-dependent myopathy in certain forms of SCS.
131 tion by SCS and how different frequencies of SCS produce the analgesic effect are unclear.
132 , non-ischemic livers preserved with 6 hr of SCS or NELP and unpreserved ischemic livers were transpl
133  of warm ischemia and preserved with 5 hr of SCS or NELP, and transplanted into syngeneic recipients.
134                      However, the impacts of SCS A-beta deficiency on mitochondria specifically in ne
135 derlying chronic pain and the interaction of SCS with these circuits may underlie this plateau in cli
136  by down-regulating the expression levels of SCS A-beta in cultured mouse neurons, we have found that
137  extent, validity, and historical meaning of SCS findings, as well as their influence and the influen
138 7)) was also used to elucidate mechanisms of SCS vasodilation at these higher frequencies.
139 this study suggest that 0.2 mg and 2.0 mg of SCS TA was as effective in reducing inflammation as 2.0
140 composition, structure, and nanostructure of SCS products, and routes to regulate the size and morpho
141 and inflammation disrupt the organization of SCS macrophages in a manner that involves the migration
142 rnished our knowledge on the pathogenesis of SCS A-beta deficiency-related mitochondrial diseases and
143     Given the negative emission potential of SCS and biochar and their potential advantages compared
144                   However, the properties of SCS macrophages are poorly defined.
145                                   Removal of SCS macrophages did not compromise adaptive immune respo
146            We sought to evaluate the risk of SCS-related complications by SCS exposure and quantify t
147 s were used to estimate the adjusted risk of SCS-related complications for patients with medium and h
148 nstrated between chronic SCS use and risk of SCS-related complications in patients with severe asthma
149 S exposure had significantly higher risks of SCS-related complications, including infections and card
150 rial diseases and revealed the vital role of SCS A-beta in maintaining neuronal mitochondrial quality
151 edical records and MRI reports suggestive of SCS.
152 slowly decaying synaptic currents typical of SCS that transit to a much faster decay upon the appeara
153 ital-optimized spin-component scaled MP2 [OO-SCS-MP2]) methods.
154 onal models used to investigate and optimize SCS.
155 P) has been suggested as an improvement over SCS.
156 s were carried out with M06-2X/6-311+G(d,p), SCS-MP2/6-311+G(d,p)//M06-2X/6-311+G(d,p), and B97D/6-31
157     Lithospheric storage of OC in peripheral SCS basins potentially exceeded 4,000 Gt (equivalent to
158 ntrast arrived at the opposite and posterior SCS 7.8 (4.6) and 7.7 (4.6) seconds after injection, res
159 osite ventral anterior SCS and the posterior SCS.
160                   In the relatively pristine SCS sediments, the most prevalent and abundant ARGs are
161 EDS (carvedilol plus ramipril 2.5 mg PO QD), SCS plus MEDS (concurrent therapy), or CTRL group.
162 anted with the electrode who did not receive SCS.
163 37) (2.37 mg/kg, i.v.) significantly reduced SCS-induced vasodilation at 500 Hz (P<0.05, n=6) as comp
164 f currents by salicylidine salycylhydrazide (SCS) indicate expression of the beta2 or beta3 subunit,
165 ional theory (DFT) and spin component scaled SCS-MP2 methods.
166 al currents (significant current scattering, SCS) could reliably discriminate between consciousness a
167  fat, and protein yield; somatic cell score (SCS); productive life (PL); and daughter pregnancy rate
168 the Zhongsha Islands in the South China Sea (SCS) deep basin during the period from August 2012 to Ja
169 re observed in the southern South China Sea (SCS) from 5 years of direct measurements.
170 ce temperature (SST) of the South China Sea (SCS) increased by 2 degrees C in response to the develop
171   The deep ocean bed of the South China Sea (SCS) is considered to be largely devoid of anthropogenic
172 in the Oligo-Miocene of the South China Sea (SCS) is evaluated herein.
173 River Estuary (PRE) and the South China Sea (SCS) were analyzed.
174 etected in the northwestern South China Sea (SCS) with its intensity, size, polarity and structure be
175 rough the Luzon Strait into South China Sea (SCS).
176  the Pacific Ocean (PO-01), South China Sea (SCS-01, SCS-02) and Indian Ocean (IO-01).
177                      Single-cell sequencing (SCS) has emerged as a powerful new set of technologies f
178                      Single-cell sequencing (SCS) is a powerful new tool for investigating evolution
179 treams, which support suspended cell sheets (SCS) of various sizes and curvatures.
180                                 Here we show SCS macrophage development depended on lymphotoxin-alpha
181 and that we called 'Sertoli Cell Signature' (SCS).
182 mphocytes that survey the subcapsular sinus (SCS) and associated macrophages for pathogen entry.
183 yer of macrophages at the subcapsular sinus (SCS) captures pathogens entering the lymph node, prevent
184 s and preferentially bind subcapsular sinus (SCS) CD169(+) macrophages in tumor-draining lymph nodes
185 h maintained a protective subcapsular sinus (SCS) macrophage phenotype within virus draining lymph no
186 ed with CD169(+)-positive subcapsular sinus (SCS) macrophages and collagen fibers.
187                           Subcapsular sinus (SCS) macrophages capture antigens from lymph and present
188 s, relocalized toward the subcapsular sinus (SCS) near invaded macrophages, and engaged in prolonged
189 hages, which populate the subcapsular sinus (SCS) of LNs, are critical for the clearance of viruses f
190 structures with a single formal charge site (SCS), whereas ArgArg x H(+) has a salt-bridge (SB) struc
191 histidine-tryptophan-ketoglutarate solution (SCS), followed by kidney autotransplantation.
192 rces for the persistent TRWs in the southern SCS.
193 ward thickening of the suprachoroidal space (SCS) compared with monotherapy alone (13.4 mum vs 5.3 mu
194 roidal layer (SCL) and suprachoroidal space (SCS).
195  is unclear whether spinal cord stimulation (SCS) at higher frequencies induces further increases in
196                     Spinal cord stimulation (SCS) is an alternative approach for treatment of neuropa
197                     Spinal cord stimulation (SCS) is an established and cost-effective therapy for tr
198                     Spinal cord stimulation (SCS) reduces the incidence of ventricular tachyarrhythmi
199 ve method--epidural spinal cord stimulation (SCS)--has been suggested as an alternative approach for
200 fiber (ZCS) input during visual stimulation; SCS cells fired complex spikes associated with learned s
201 We investigated whether static cold storage (SCS) can be safely replaced with a novel technique of pr
202  perfusion (NEVKP) with static cold storage (SCS) in a porcine model of DCD autotransplantation.
203  livers by conventional static cold storage (SCS) is associated with an unacceptable risk of primary
204 se preserved using only static cold storage (SCS).
205  compared with standard static cold storage (SCS).
206 of oxygenated HMP after static cold storage (SCS).
207 ith HMP were compared to static cold stored (SCS) transplant controls.
208 iatum is known as the Swiss cheese striatum (SCS); however, its clinical impact is unknown.
209 rom 15 cohorts of the Seven Countries Study (SCS), the first systematic study of diet, risk character
210 ieved using a structured chimeric substrate (SCS) that releases a downstream activator after cleavage
211       Defected SCS ADP-forming beta subunit (SCS A-beta) is linked to lethal infantile Leigh or leigh
212 from 18 CHL-derived single-cell suspensions (SCSs) compared to reactive lymph nodes (RLNs).
213 e ACh receptors cause slow-channel syndrome (SCS) and Escobar syndrome, two forms of congenital myast
214                       Slow-channel syndrome (SCS) is an autosomal-dominant disease resulting from mut
215 ty and the slow-channel myasthenic syndrome (SCS), IP(3)R(1) knockdown eliminated NMJ Ca(2+) overload
216  the field of solution combustion synthesis (SCS) obtained during recent years.
217              Succinyl Coenzyme A synthetase (SCS) is a key mitochondrial enzyme.
218                     Succinyl-CoA synthetase (SCS) is the only mitochondrial enzyme capable of ATP pro
219 ically performed by succinyl-CoA synthetase (SCS), has arisen in diverse bacterial groups, including
220 is of mtGTP made by succinyl-CoA synthetase (SCS-GTP) to an anaplerotic pathway producing phosphoenol
221  study investigated the effects of long-term SCS on ventricular function in a postinfarction canine h
222 on (average: 6.1 vs 7.4 days, P = .003) than SCS organs.
223 nd had systemic autoimmunity more often than SCS (p < 0.05).
224 bies virus transmitted by insect bites, that SCS macrophages perform a third vital function: they pre
225 concentration is increased, we conclude that SCS activation by P(i) binding represents another mitoch
226              Furthermore, we have found that SCS A-beta deficiency has detrimental influence on neuro
227 n cultured mouse neurons, we have found that SCS A-beta deficiency induces severe mitochondrial dysfu
228                           Here, we show that SCS reproducibly alleviates motor deficits in a primate
229                   These results suggest that SCS should be considered as an additional treatment opti
230                                          The SCS can be activated by various upstream DNAzymes, can b
231                                          The SCS findings included 30-fold cohort differences in rate
232                                          The SCS is characterized by a precise organization of bindin
233                                          The SCS was accessed using microneedles in a minimally invas
234  demonstrated a visible SCS at baseline, the SCS expanded significantly after suprachoroidal CLS-TA i
235 /6-31+G(d,p) structures and energies for the SCS and SB forms of these protonated dipeptides indicate
236 251 mug kg(-1)) and those collected from the SCS (12-83 mug kg(-1)).
237 f fluid and that it is possible to image the SCS with ultrasound contrast.
238 e 3 also were significantly decreased in the SCS (27+/-17 and 27%, respectively; P<0.03) and MED (58+
239 covery in ejection fraction was noted in the SCS (52+/-5%) and SCS+MEDS (46+/-4%) groups compared wit
240 g isotopic compositions were observed in the SCS (delta(202)Hg, from -2.82 to -2.10 per thousand; Del
241 n fraction was significantly improved in the SCS compared with the MED and CTRL groups (P<0.001 for b
242                 In the mean, the DWBC in the SCS flows southwestward with core velocity of 2.0 cm/s a
243  RT-PCR of reperfusion biopsy samples in the SCS group showed high expression of inflammatory cytokin
244  also revealed ultrastructural damage in the SCS group that was not seen in the HMP group.
245  numbers of CD68 positive macrophages in the SCS group when compared to the HMP group.
246                                       In the SCS group, left ventricular ejection fraction was 65+/-5
247       The large positive Delta(199)Hg in the SCS indicated that a fraction of Hg has undergone Hg(2+)
248 nalysis in the PRE sediments, but not in the SCS sediments.
249 pe fractionation by natural processes in the SCS.
250 e and resistance mechanism than those in the SCS.
251  toxicity following injection of TA into the SCS in porcine eyes.
252                           Migration into the SCS intrinsically requires S1pr1, whereas movement from
253 injection of 0.2 mg or 2.0 mg of TA into the SCS or IVT.
254 n of microbubble contrast injection into the SCS were assessed by 2D and 3D ultrasound.
255  is required for their accumulation near the SCS and for efficient IL17 induction.
256 eys, near the end of his long career, of the SCS design, conduct, and findings, with his discussion a
257                  Mean (SD) distension of the SCS with PBS increased from 1.57 (0.48) mm after injecti
258                 In the deep ocean bed of the SCS, source-related signatures of Hg isotopes may have b
259 ed to image the effect and distension of the SCS.
260 e to RVO, but may result in expansion of the SCS.
261 dently associated with the visibility of the SCS: disarrangement of plexiform layers, CRT, and multip
262 contrast was visible in 24.0%to 27.2% of the SCS; in 10 of 12 eyes, contrast reached the posterior po
263 ermore, quantum chemical calculations on the SCS-MP2 and DFT levels gave insights into the reaction m
264               These results suggest that the SCS can expand, in a dose-dependent manner, to accommoda
265                                    Thus, the SCS macrophage layer may act as a sensor or valve during
266 2, 28 surviving animals were assigned to the SCS (delivered at the T4/T5 spinal region for 2 hours 3
267 roducing plasmacytoid dendritic cells to the SCS and in addition were a major source of IFN-I themsel
268 hat stability of the SB form relative to the SCS form generally increases with increasing gas-phase b
269                        Delivery of TA to the SCS using microneedles was simple, effective, and not as
270 e 1 survivors were equally randomized to the SCS, MEDS (carvedilol plus ramipril 2.5 mg PO QD), SCS p
271                       Macrophages within the SCS of B cell-deficient LNs, or of mice that lack LTalph
272 ctions to form the organic component of the (SCS)Ni(II) pincer cofactor of LarA.
273                     We report a theoretical (SCS-MP2//B3LYP) and experimental study of the uncatalyze
274 ed only with the highest level of QM theory (SCS-MP2) used.
275 anisms underlying motor improvements through SCS are unknown.
276 neutrophils and macrophages when compared to SCS controls.
277 rial DNA pol gamma and Twinkle contribute to SCS A-beta deficiency-mediated mtDNA instability.
278 ing enzyme (18%, p = 0.30) were exclusive to SCS.
279 SNI) and those without injury in response to SCS.
280 C) machine perfusion (NMP) after traditional SCS (0 degrees C-4 degrees C) for 7 to 9 hours.
281 wo hours of oxygenated HMP after traditional SCS restores hepatic ATP levels and improves hepatobilia
282 MP grafts were matched 1:3 with transplanted SCS livers.
283 tion, animals transplanted with NEVKP versus SCS grafts demonstrated similar serum creatinine peak le
284   In the 15 eyes that demonstrated a visible SCS at baseline, the SCS expanded significantly after su
285 EVKP-preserved kidneys (NEVKP, 39 +/- 6.4 vs SCS, 18 +/- 10.6 mL/min; P = 0.012).
286 reatinine peak levels (NEVKP, 2.0 +/- 0.5 vs SCS 2.7 +/- 0.7 mg/dL; P = 0.11) and creatinine clearanc
287 wer peak values (NEVKP, 5.5 +/- 1.7 mg/dL vs SCS, 11.1 +/- 2.1 mg/dL, P = 0.002).
288 ce on day 10 (NEVKP, 65.9 +/- 18.8 mL/min vs SCS 61.2 +/- 15.6 mL/min; P = 0.74).
289                         All recipients in WI-SCS group died within 6 hours after transplantation.
290  preserved either by simple cold storage (WI-SCS group) or HMP (WI-HMP group) using Belzer-MPS soluti
291 ndrome was observed in all animals of the WI-SCS group but none of the control or WI-HMP groups.
292 ter-preserved liver histology relative to WI-SCS group.
293 atients evaluated between 1996 and 2015 with SCS or NMOSD whose first myelitis episode was accompanie
294 gies might reduce the burden associated with SCS-related complications in patients with severe asthma
295 e were less elevated after HMP compared with SCS preservation alone.
296 rved with HMP than for organs preserved with SCS (34.2% vs 42.0%, P < .001), despite a slightly longe
297 ar factors that bind to genomic regions with SCS could functionally interact with SOX9, we identified
298 P = 0.59), whereas animals transplanted with SCS grafts had persistently elevated serum creatinine an
299                       Over the past 5 years, SCS methods for DNA and RNA have had a broad impact on m
300                         In the coming years, SCS will greatly improve our understanding of invasion,

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