ライフサイエンスコーパス: SCU

1 edematous (n = 11) and nonedematous (n = 11) SCU when they were infected and malnourished (clinical p

2 edematous (n = 11) and nonedematous (n = 11) SCU when they were infected and malnourished (clinical p

3 h edematous (n = 14) or nonedematous (n = 7) SCU when they were infected and malnourished (postadmiss

4 Although SCUs may have provided unmeasured benefits to families a

5 e observe a pattern of better outcomes among SCU residents.

6 embryonal and undifferentiated small cells (SCU) progressively lose EGFR and ASAP1 expression.

7 r in the group that previously had edematous SCU than in the nonedematous group.

8 d in the group that previously had edematous SCU, but it did not change in the other group.

9 in the children who previously had edematous SCU.

10 r than the rate at recovery in the edematous SCU group, and there were no significant differences bet

11 e as when recovered, children with edematous SCU cannot.

12 At clinical phase 1, children with edematous SCU had rates of total methionine flux, flux from protei

13 and infected state, children with edematous SCU have slower methionine production than do children w

14 wer, however, in the children with edematous SCU than in those with nonedematous SCU.

15 , is much greater in children with edematous SCU than in those with nonedematous SCU.

16 sm will be slower in children with edematous SCU than in those with nonedematous SCU.

17 Differences in SCU among functional categories reflect differences in l

18 s reveal that PFOS anions are immobilized in SCU-8 by driving forces including electrostatic interact

19 e gBGC model explains 70% of the variance in SCU among genes.

20 ved in the speed of decline for residents in SCUs and traditional units in any of the 9 outcomes.

21 ent a mesoporous cationic thorium-based MOF (SCU-8) containing channels with a large inner diameter o

22 een children with edematous and nonedematous SCU and inherent differences in protein metabolism when

23 in children with edematous and nonedematous SCU.

24 ized that, in edematous but not nonedematous SCU, impaired protein breakdown leading to inadequate am

25 s of children with edematous or nonedematous SCU in the malnourished and recovered states.

26 roduction than do children with nonedematous SCU because of a slower rate of release from protein bre

27 Whereas children with nonedematous SCU can maintain arginine flux at the same rate as when

28 dematous SCU than in those with nonedematous SCU.

29 dematous SCU than in those with nonedematous SCU.

30 dematous SCU than in those with nonedematous SCU.

31 We argue that the strong heterogeneity of SCU induced by gBGC in mammalian genomes precludes any o

32 The anion-exchange properties of SCU-8 were explored with many anions including small oxo

33 ratory, and physiologic variables at time of SCU admission, at 24 hrs, as well as vital status at the

34 sage, which was interpreted as evidence that SCU is adaptively constrained to optimize translation ef

35 We demonstrate here that SCU is not driven by constraints on tRNA abundance, but

36 tal status at the time of discharge from the SCU and hospital.

37 The median length of stay in the SCU was 2 days, and the median hospitalization for patie

38 The causes of admission to the SCU were pulmonary (15/43), cardiac (14/43), wound relat

39 urgical procedures required admission to the SCU.

40 ck malignant disease were transferred to the SCU.

41 h edematous severe childhood undernutrition (SCU) can maintain production rates of glycine and serine

42 ous form of severe childhood undernutrition (SCU) during food deprivation are not clear.

43 onedematous severe childhood undernutrition (SCU) have lower plasma and erythrocyte-free concentratio

44 onedematous severe childhood undernutrition (SCU) have lower plasma and erythrocyte-free concentratio

45 patients admitted to the special care unit (SCU) of Memorial Sloan-Kettering Cancer Center between J

46 Alzheimer disease special care units (SCUs) in nursing homes are increasingly prevalent, but l

47 Synonymous codon usage (SCU) varies widely among human genes.

48 bryonal tumor cells are EGFR-negative, while SCU cells are EGFR-negative and ASAP1-negative.

49 e production is reduced in all children with SCU because of a decreased contribution from protein bre

50 ular nitric oxide synthesis in children with SCU.

51 ease from protein breakdown in children with SCU.

52 homocysteine remethylation in children with SCU.

53 cluding 1228 residents in 48 facilities with SCUs.