ライフサイエンスコーパス: SD rat

1 liver and kidney after an acute exposure in SD rats.

2 Cl were significantly higher in F344 than in SD rats.

3 imulated GEPR-9s, seizure-naive GEPR-9s, and SD rats.

4 ture was greater in BHE/cdb rats than in the SD rats.

5 e lower in both SHR and WKY[LJ] rats than in SD rats.

6 ffects on the other urinary parameters in Nx SD rats.

7 cated in vivo in neonatally infected LEW and SD rats.

8 efore the outset of extinction training than SD rats.

9 detection and compared between WKY, WIS and SD rats.

10 WKY rats consistently had lower levels than SD rats.

11 5-HT turnover when compared to both WKY and SD rats.

12 IC neurons of SN-GEPR-3s compared to control SD rats.

13 lality and urea concentration (P < 0.001) in SD rats.

14 -fold (P = 0.0001) higher in BN rats than in SD rats.

15 Dawley (SD) rats and 5/6 nephrectomized (Nx) SD rats.

16 studies on ageing (F344) by comparison with SD rats.

17 o a dorsal spinal cord transection lesion of SD rats.

18 nephrine (NE) release in BSTL of WKY but not SD rats.

19 and increased nephron number, compared with SD rats.

20 WKY response was not different from that of SD rats.

21 chemia and 0 to 24 hours reperfusion in male SD rats.

22 iographically in seven urethane anesthetized SD rats.

23 oyang (GMFS) in healthy male Sprague Dawley (SD) rats.

24 tment in Long-Evans (LE) and Sprague-Dawley (SD) rats.

25 ne rats (GEPR-9s) and normal Sprague-Dawley (SD) rats.

26 red normotensive rat strain, Sprague-Dawley (SD) rats.

27 and behavioral responses in Sprague-Dawley (SD) rats.

28 inhibit gastric emptying in Sprague-Dawley (SD) rats.

29 GEPR-3s) compared to control Sprague-Dawley (SD) rats.

30 b data from a prior study in Sprague-Dawley (SD) rats.

31 ollowing gonadectomy in male Sprague-Dawley (SD) rats.

32 HR), Wistar-Kyoto (WKY), and Sprague-Dawley (SD) rats.

33 ective assessment of baseline craving (mean [SD] rating: 2 days, 26.05 [9.85]; 1 week, 18.70 [11.01];

34 , 3.00 [3.77]) and cue-induced liking (mean [SD] rating: 2 days, 3.06 [2.34]; 1 week, 2.33 [2.87]; 1

35 and 1 year, 1.00 [1.26]) and wanting (mean [SD] rating: 2 days, 3.44 [2.62]; 1 week, 2.72 [2.87]; 1

36 MicroPET images of SD rats after [(18)F]10 administration allowed easy asse

37 tion of T-2 toxin and DON in Sprague-Dawley (SD) rats after a single dose exposure.

38 coincided with the inflammatory response in SD rats and may constitute a neuroprotective mechanism.

39 MicroPET images of SD rats and nonhuman primates after [18F]27 administrati

40 resistance locus on chromosome (chr) 6q16 in SD rats and, surprisingly, a locus on chr3q21-23 that wa

41 in anesthetized non-diabetic Sprague Dawley (SD) rats and 5/6 nephrectomized (Nx) SD rats.

42 ferred to the right atrium of Spague-Dawley (SD) rats and acetylcholine (ACh) or noradrenaline (NA) r

43 d streptozocin diabetic male Sprague-Dawley (SD) rats and of control and diabetic female Lewis rats w

44 h intravenous AUC (64.9 muM.h, 2.0 mg/kg) in SD rats, and significant in vivo antitumor activity (T/C

45 ial isolation in non-anxious Sprague Dawley (SD) rats, and a depression model, Wistar-Kyoto (WKY) rat

46 y (BN), Long-Evans (LE), and Sprague-Dawley (SD) rats, and protein expression in the retina was measu

47 y of AMPA receptors in the IC of GEPR-9s and SD rats are similar, our results indicate that altered A

48 d with Long-Evans (LE) rats, Sprague-Dawley (SD) rats are more sensitive to the PPI-disruptive effect

49 We previously reported that Sprague-Dawley (SD) rats are significantly more sensitive than Long Evan

50 nal neovascularization in BN rats but not in SD rats, as demonstrated by fluorescein retinal angiogra

51 molecules was attenuated in LEW compared to SD rats at all times examined.

52 In a subgroup of male SD rats, blood-retinal barrier (BRB) integrity was also

53 Compared with Sprague-Dawley (SD) rats, Brown-Norway (BN) rats exhibit impaired caroti

54 e shown that CCG was maximal at d 9 of RI in SD rats but did not occur in JCR rats.

55 transient ( approximately 4-fold, d 3 RI) in SD rats but greater and sustained in JCR rats ( approxim

56 it decreased gastric emptying in S5B/P1 and SD rats but not in OM rats.

57 a high-fate diet in Osborne-Mendel (OM) and SD rats but not in S5B/Pl rats, whereas it decreased gas

58 henotype in drug-dependent and singly housed SD rats, characterized by slowed norepinephrine clearanc

59 n (r = 0.99) with the observed values in the SD rat corneas.

60 dia from uninfected cultures of both LEW and SD rats could prevent BDV-induced DG damage in infected

61 In control SD rats, DeltaPO2(t1-ra) > DeltaPO2(t2-t1) (P <0.05) was

62 DSP-4-treated SD rats demonstrated a dependence-like phenotype, wherea

63 [(18)F]10 administration to Sprague-Dawley (SD) rats demonstrated high uptake of the radiotracer fro

64 Eleven weeks after A/I, SD rats developed proteinuria, severe kidney damage, dec

65 rat retina are highly strain dependent, and SD rats exhibit low-level inflammation similar to that o

66 In addition, SD rats expressed more Fos-LI in the area postrema and m

67 ry bulbs of adult transgenic Sprague Dawley (SD) rats expressing green fluorescent protein (GFP) were

68 excretion rate were increased compared with SD rats fed the 25% protein diet (GFR, 2.66 +/- 0.16 ver

69 In SD rats fed the 50% protein diet, GFR, RPF, and kallikre

70 o be significantly greater in LE compared to SD rats for both drugs, with F1 rats intermediate.

71 We found that SD rats have significantly more duodenal myenteric neuro

72 In OIR-SD rats, however, hyperpermeability was observed from P1

73 In age-matched normal BN and SD rats, however, there was no detectable difference in

74 essure (MAP) in young female Sprague-Dawley (SD) rats, however, the underlying mechanisms are unclear

75 f analogues ([18F] 22-28) in Sprague-Dawley (SD) rats identified [18F]27 as the most promising radiot

76 ry (IHC) across a large bank of PhIP-induced SD rat mammary gland carcinomas.

77 Therefore, SD rats may be a good model for the study of early infla

78 In conclusion, SD rats may express more myenteric Fos-LI in response to

79 In SD rats, melanin was not detected in all tissues assesse

80 e and miR-224 antagonist using an orthotopic SD rat model.

81 Similarly, in Nx SD rats (n = 5-6), luseogliflozin significantly increase

82 , HR, RBF, or creatinine clearance (CrCl) in SD rats (n = 7).

83 nically catheterized, awake, Sprague Dawley (SD) rats (n = 78).

84 R, WKY and standard, outbred Sprague-Dawley (SD) rats on a delay discounting task where the primary m

85 of diabetes), while diabetic Sprague Dawley (SD) rats only showed retinal hyperpermeability from 3 to

86 Postnatal day 4 (P4) SD rat pups were subjected to bilateral common carotid a

87 Wistar and Sprague-Dawley (SD) rats received 1 of 3 diets: control (CON); Western (

88 IC neurons of SN-GEPR-3s compared to control SD rats, respectively.

89 Hyperoxia-treated SD rats showed changes in PEDF and VEGF levels that were

90 protective factors in the disease-resistant SD rat strain.

91 ay respiratory output is similar in F344 and SD rat strains.

92 d eyes were approximately 1.5-fold higher in SD rats than in BN rats.

93 In addition to Sprague-Dawley (SD) rats that have CT responses typical of most rat stra

94 sly, using skin chambers mounted on backs of SD rats, that neutralizing antibodies directed against V

95 In diabetic SD rats, the superior, but not the inferior, hemiretina

96 In 3-month-old diabetic male SD rats, total and postreceptor retinal thickness increa

97 f the NO system in WF versus Sprague Dawley (SD) rats was examined with the 5/6 renal ablation/infarc

98 rone BHE/cdb rats and normal Sprague Dawley (SD) rats was studied.

99 sing a phenol red gavage technique in fasted SD rats, we showed that insulin administration accelerat

100 On gestation day 22 SD rats were anesthetized and the uterine horns were ext

101 Forty male SD rats were divided into four surgery groups: bilateral

102 To test this, young female SD rats were either sham implanted or implanted s.c. wit

103 Two days after the injections, the SD rats were exposed to light of 2000 lux for 48 hours.

104 Normal male SD rats were fed regular powdered diet with or without t

105 HCR, LCR, and SD rats were grouped by strain and randomly assigned to

106 Male SD rats were infused with glucosamine at 0.1 mg x kg(-1)

107 Male SD rats were pretreated with alpha-lipoic acid (ALA) and

108 SD rats were randomized to AAN and control groups.

109 SD rats were randomly divided into 6 groups: Sham, R/I,

110 te matter damage (WMD), postnatal day 4 (P4) SD rats were subjected to bilateral common carotid arter

111 Male Spragu-Dawley (SD) rats were subjected to left descending artery occlus

112 120 male Sprague Dawley (SD) rats were treated with lithium chloride-pilocarpine

113 aturally higher CO and lower H2S levels than SD rat, whereas SH carotid bodies have reduced CO and gr

114 primary rat hepatocytes (PRH) or intact male SD rats with replication-defective recombinant adenoviru

115 ers, LCR), and 3) unselected Sprague-Dawley (SD) rats with or without free access to running wheels f

116 as decreased in the BHE/cdb rats compared to SD rats, with the exception of the comparison made at 37