ライフサイエンスコーパス: SENV

コーパス検索結果 (left1)

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1 SENV strain prevalence was examined in a race/ethnicity-

2 SENV strains are common among San Francisco Bay area IDU

3 SENV was detected in 54 (22%) of 248 patients with acute

4 SENV-A was more common in black subjects (adjusted odds

5 reaction amplification was used to detect 2 SENV variants (SENV-D and SENV-H) in 1706 patients and c

6 prevalences were as follows: SENV-A, 45.7%; SENV-C/H, 35.6%; and SENV-D, 10.3%.

7 oot of the phylogenetic tree, and compared 8 SENV isolates, 6 prototype TTV isolates, and 7 TTV varia

8 TTV formed group 2, PMV formed group 3, and SENV, SANBAN, and TUS01 isolates formed group 4, the mos

9 follows: SENV-A, 45.7%; SENV-C/H, 35.6%; and SENV-D, 10.3%.

10 action in serum samples from 130 (32.7%) and SENV-H infection in 149 (37.5%).

11 Among Baltimore IDUs, SENV-D and SENV-H infections are common and dynamic, including both

12 oup of DNA viruses whose members (SENV-D and SENV-H) are linked to posttransfusion hepatitis.

13 s used to detect 2 SENV variants (SENV-D and SENV-H) in 1706 patients and control subjects.

14 Two SEN-V variants (SENV-D and SENV-H) were assayed by polymerase chain reaction (PCR)

15 A, B, or C was not influenced by coexisting SENV infection.

16 istence a median of 9.3 years later detected SENV-D in 25 (61.0%), whereas SENV-H was detected on ret

17 Eight different SENV isolates have been found, some with an association

18 Weighted prevalences were as follows: SENV-A, 45.7%; SENV-C/H, 35.6%; and SENV-D, 10.3%.

19 For SENV-D, prevalence was similar in black and white subjec

20 % of IDUs who repeatedly tested positive for SENV-D DNA and in 55.6% of those who repeatedly tested p

21 of those who repeatedly tested positive for SENV-H DNA.

22 No etiological role for SENV in the cause of cryptogenic hepatitis could be demo

23 ic subjects, and the pattern was similar for SENV-C/H.

24 Among Baltimore IDUs, SENV-D and SENV-H infections are common and dynamic, inc

25 We investigated SENV's role in unexplained hepatitis in other settings.

26 on drug users (IDUs) in Baltimore, Maryland, SENV-D infection was detected by polymerase chain reacti

27 scovered group of DNA viruses whose members (SENV-D and SENV-H) are linked to posttransfusion hepatit

28 ing for geographic region, the prevalence of SENV among case and control subjects was not significant

29 The clinical significance of SENV infection in this setting remains unknown.

30 In summary, SENV-D and -H were present in nearly 2% of US donors, an

31 fication was used to detect 2 SENV variants (SENV-D and SENV-H) in 1706 patients and control subjects

32 Two SEN-V variants (SENV-D and SENV-H) were assayed by polymerase chain reac

33 SEN virus (SENV) has been tentatively linked to transfusion-associa

34 SEN virus (SENV) is a recently discovered group of DNA viruses whos

35 nsmissible single-stranded (ss) DNA viruses (SENV) distantly related to the large TT virus (TTV) fami

36 SEN viruses (SENVs) are newly discovered bloodborne viruses that may

37 later detected SENV-D in 25 (61.0%), whereas SENV-H was detected on retesting in only 14 (26.9%) of 5

38 Of 41 IDUs in whom SENV-D DNA was initially detected, retesting for viral p

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