ライフサイエンスコーパス: SEP

1 SEP (beta = 0.034, t = 2.4, p = 0.01) and cognitive mili

2 SEP amplitude but not latency effects recovered after 7

3 SEP genes have not been detected in gymnosperms and seem

4 SEP indicators at 3 life stages (childhood, adulthood, a

5 SEP inequalities in CRP emerged in 30s, increased up to

6 SEP inequalities in fibrinogen decreased with age.

7 SEP is associated with T2DM risk factors in children but

8 SEP over the life course is related to the risk of incid

9 SEP values for urea are 7.33 and 0.10 mM for first overt

10 SEP was homogeneous as shown by high-resolution analytic

11 SEP-225289 is a novel compound that, based on in vitro p

12 mine AngII dose (SEE = 26 ng kg(-1) min(-1), SEP = 37 ng kg(-1) min(-1), r2 = 0.99) and collagen/elas

13 perative mJOA (P = .927), T2 HSI (P = .176), SEP amplitude (P = .154), and latency (P = .260) did not

14 nd collagen/elastin (C/E) ratio (SEE = 0.38, SEP = 0.39, r2 = 0.85) in mouse aortas.

15 We validated expression of a SEP from its endogenous RNA, and demonstrated the specif

16 95% CI, 1-13) 72 hours after CPR, and absent SEPs during hypothermia (FPR 3; 95% CI, 1-7) and after r

17 Here, we show that a 69-amino acid SEP, MRI-2, physically interacts with the Ku heterodimer

18 ity with respect to both childhood and adult SEP are urgently required.

19 inal data to examine how childhood and adult SEP relates to BMI across adulthood in three national Br

20 The strength of association between adult SEP and BMI did not vary with age in any consistent patt

21 nces hemostatic markers more than does adult SEP.

22 etween early life, young and later adulthood SEP and the expression of 845 genes involved in human in

23 ly life, young adulthood and later adulthood SEP with each inflammatory markers separately, or by com

24 ustment for young and mid-to-older adulthood SEP and established heart failure risk factors attenuate

25 Of the SEP measures, mid-to-older adulthood SEP showed the strongest association with incident heart

26 However, an SEP of 2.96 mM indicates unsuccessful calibration standa

27 transfer is highly successful, producing an SEP value of 1.07 mM.

28 to 66%), z-severity (-2.4 down to -2.5), and SEP (-195 to -227) (P = 0.0062).

29 easure of cumulative SEP (range, 0 to 8) and SEP mobility.

30 concentrations by LC-MS/MS (rPred > 0.90 and SEP < 100 mug/kg).

31 istics (mother's age and place of birth) and SEP.

32 assessing associations of race/ethnicity and SEP to health outcomes with CIs); illustrate the applica

33 l expression pattern with the C-function and SEP-like genes.

34 plex factor specifically with C-function and SEP-like MBFs.

35 The obtained results (RSQ and SEP, respectively) for the global model of red and white

36 pumping (SEP)-hole filling spectroscopy and SEP-induced population transfer spectroscopy have been u

37 des (SEPs) to validate smORF translation and SEP stability.

38 nd 36 hours and 48 hours after collapse; and SEPs during hypothermia and after rewarming.

39 The decreased concentration of PI/AP3/SEP complexes offers an explanation for the petal defect

40 the anterior hypothalamus (AH), septal area (SEP), and ventromedial nucleus of the posterior division

41 measurements of EEG power spectra as well as SEP-amplitudes and latencies of N10.3 were performed bef

42 Harmonized social class-based SEP data (Registrar General's Social Class) was ascertai

43 Because SEPs are shorter and less abundant than average proteins

44 A better SEP and a more stimulating cognitive milieu in childhood

45 Associations between SEP and BMI were examined using linear regression and mu

46 Conversely, in women, associations between SEP and nonadherence were weak and inconsistent.

47 s evidence for a complex interaction between SEP and other variables, such as ethnicity, sex, and the

48 We investigated the relationship between SEP and statin adherence in a country with universal cov

49 equality to measure the relationship between SEP and use of eye care services across the entire distr

50 We examined the relationships between SEP and T2DM risk factors in UK children of South Asian,

51 There was a linear correlation between SEPs and BOLD fMRI over the full range of frequency cont

52 h we cannot rule out residual confounding by SEP.

53 fferences in the use of eye care services by SEP persist among US adults with eye diseases.

54 inflammatory marker examined rather than by SEP measures.

55 nt have on neuronal activity, as measured by SEPs, and on the amplitude of the BOLD fMRI signal in ra

56 Lower childhood SEP was associated with higher adult BMI in both genders

57 provides additional evidence that childhood SEP is associated with a sustainable upregulation of the

58 ogen and C-reactive protein, while childhood SEP influences hemostatic markers more than does adult S

59 ucation, interactions of time with childhood SEP (beta = -0.003, t = -1.0, p = 0.32) and childhood co

60 Census tract-level composite SEP index in quintiles was assigned based on residence a

61 on the same side, produced highly correlated SEPs in neighboring cortical columns (r = 0.9) with freq

62 xamined the impact of changes in life-course SEP on incidence of dementia and cognitive impairment bu

63 authors examined the effects of life-course SEP on inflammatory and hemostatic markers: fibrinogen,

64 IND with every 1-unit increase in cumulative SEP disadvantage across the life course (hazard ratio =

65 were used to derive a measure of cumulative SEP (range, 0 to 8) and SEP mobility.

66 short-wavelength region is further degraded (SEP = 2.53 mM).

67 dominantly mediate perisomatic and dendritic SEPs, respectively.

68 We now report that this depressed SEP persists during the subchronic (48 h) period of ICH

69 , worsening cardiac denervation (global diff-SEP > 9) occurred in 14 of 23 (60.9%) patients over 2 y,

70 cline in lateral and inferior segments (diff-SEP -82 to -99) compared with anterior and septal segmen

71 nervation as the 2-y difference in SEP (diff-SEP).

72 isomer (CO-bound or NH-bound) with different SEP excitation wavelengths.

73 -terminal UBA domain and the central domain (SEP) from p47.

74 od having a more prominent role than earlier SEP.

75 ylogenetic and expression analysis for eight SEP-like GERBERA REGULATOR OF CAPITULUM DEVELOPMENT (GRC

76 Neutralization of either SEP-196 or Arg-201 leads to a channel with reduced activ

77 n cell and animal assays for erythropoiesis, SEP displayed potent biological activity and had signifi

78 We expressed SEP-TAC-7.3 in dissociated rat hippocampal neurons to ex

79 The timing of the first SEP duplication approximately coincides with duplication

80 nce interval: -0.04, 0.13) after control for SEP and RPF.

81 logy confirmed selective RPE cell damage for SEP irradiation with 7.5 and 15 micros; slower scan spee

82 takeholder workgroup recognizes the need for SEP-1 but strongly believes that multiple antibiotics li

83 ibiotics listed in the antibiotic tables for SEP-1 are not appropriate and the use of these antibioti

84 ants, indicating the involvement of all four SEP genes in the development of sepals.

85 henotypes were not socially patterned or had SEP patterns that were not related to smoke exposure.

86 odels, participants with a continuously high SEP had lower hazard ratios for dementia/CIND compared w

87 e duration of the localized sleep-like (high SEP amplitude) state was dependent on the duration of pr

88 Such high SEP values for urea with first overtone spectra correspo

89 val: 0.66, 0.98) than did children with high SEP.

90 evidence.In the general population, a higher SEP appears to be associated with an increased 'hypothet

91 h better performance being related to higher SEP and a better cognitive milieu.

92 ce rate ratios for children with the highest SEP were 1.29 (95% confidence interval (CI): 1.15, 1.44)

93 We examined how SEP inequalities in inflammation -assessed using C-react

94 early life to adulthood, we investigate how SEP indicators at different points across the life cours

95 Future research needs to study how SEP impacts the potential donor population from willingn

96 However, SEP amplitudes from single trials fluctuate periodically

97 The discovery of additional human SEPs and their conservation indicate the potential impor

98 ve led to the detection of several new human SEPs (novel human genes), improved confidence in the SEP

99 ICC-SEP form a crucial conduction pathway for spreading exci

100 in cross-section so that the ICC-MY and ICC-SEP networks and the CM could be readily visualized.

101 ker activity in human ICC-MY can entrain ICC-SEP to excite CM bundles.

102 were therefore to determine the role of ICC-SEP in activating the thick CM in the human jejunum.

103 ion, leading to a sporadic activation of ICC-SEP.

104 In large mammals, ICC also line septa (ICC-SEP) between circular muscle (CM) bundles, suggesting th

105 Importantly, SEPs recorded after InsP3 infusion into distal astrocyte

106 eps in the SEP discovery workflow to improve SEP isolation and identification.

107 eduction in CMRO2 and there was no change in SEP cortical amplitude (N20-P27).

108 athetic innervation as the 2-y difference in SEP (diff-SEP).

109 ter understand the observed heterogeneity in SEP differences in health across the lifespan.

110 power spectrum (by maximally 62%), increased SEP-latencies (by approximately 6-10%) and amplitudes (b

111 ked potential (SEP) amplitudes and increased SEP latencies in control genotypes.

112 At 8 months, posture influenced mid-latency SEP components, but by 10 months effects were observed a

113 tive assessments revealed that short-latency SEPs and somatosensory-induced gamma-oscillations predom

114 ive decline and examine how individual-level SEP factors (educational level, annual income, and occup

115 ors examined the relation between early-life SEP and incident, hospitalized heart failure among middl

116 Early-life SEP indicators assessed included parental education, occ

117 proportional hazards regression, early-life SEP was inversely related to incident heart failure afte

118 rradiation patterns forming separated lines (SEP) or interlaced lines (INT), different dwell times (2

119 Local SEP fluctuations also followed local differences in the

120 Low SEP in childhood is associated with modest increase in a

121 Low SEP was associated with overall and rapidly increasing s

122 Nearly 24% of the sample maintained a low SEP throughout life.

123 odds of asthma alone in children with a low SEP.

124 compared with those with a continuously low SEP at all 3 life stages (hazard ratio = 0.49, 95% confi

125 clinical characteristics, and copayment, low SEP was associated with statin nonadherence (proportion

126 dherence trajectories showed that men in low SEP were likely to belong to trajectories presenting a f

127 dent on the duration of prior wake-like (low SEP amplitude) state (r = 0.5), suggesting a use depende

128 Lower SEP was associated with a reduced likelihood of receivin

129 In a commercial setting, lower SEP motivates people to donate.

130 As compared with the lowest SEP, the adjusted incidence rate ratios for children wit

131 ; mean severity z score, -2.47 to -2.0; mean SEP, -167 to -139), with lateral and proximal inferior s

132 ; mean severity z score, -2.8 to -2.62; mean SEP, -271 to -230).

133 everity z score was -2.7+/-1.2, and the mean SEP was -202+/-131 (range, -358 to 0).

134 Here, we measured SEP gating and perceptual sensory attenuation in a singl

135 Neighborhood SEP was derived by linking the participant's individual

136 vided information distinct from neighborhood SEP.

137 graphic characteristics, higher neighborhood SEP was significantly associated with cognitive function

138 r individual educational level, neighborhood SEP remained associated with baseline cognition but not

139 ults suggest that the effect of neighborhood SEP on cognitive decline among Latinos is primarily acco

140 s were strongly associated with neighborhood SEP but also provided information distinct from neighbor

141 netics, and compartmentalization of neuronal SEPs.

142 nced the amplitude and frequency of neuronal SEPs.

143 faster detection and characterization of new SEPs and smORFs.

144 At all ages we observed SEPs over central regions contralateral to the stimulate

145 Co-administration of SEP and L-Cit decreased infarct size and improved corona

146 Co-administration of SEP and L-Cit limits diabetic cardiomyopathy and ischemi

147 litus were abrogated by co-administration of SEP and L-Cit.

148 roximately 24 h after oral administration of SEP-225289, to assess occupancy at trough levels.

149 Phylogenetic analyses of SEP sequences show that several gene duplications occurr

150 Assessment of SEP was based on parental occupation defined using NS-SE

151 estimate direct and indirect associations of SEP and RPF, and we estimated average marginal controlle

152 Plasma concentration of SEP-225289 was assessed before ligand injection, and sub

153 biological embedding and the consequences of SEP experiences from early life to adulthood, we investi

154 ferent ethnic groups and the contribution of SEP to ethnic differences in T2DM risk in young people h

155 tudy was conducted during the development of SEP-225289 to investigate its dopamine and serotonin tra

156 may differ according to other dimensions of SEP such as education or income.

157 Different single doses of SEP-225289 were administered to healthy volunteers in 3

158 cupancies increased with increasing doses of SEP-225289.

159 The present study examined the effects of SEP and L-Cit on diabetic cardiomyopathy and ischemia/re

160 To gain insights into the evolution of SEP genes, we isolated nine genes from plants that occup

161 use of social class as the sole indicator of SEP-while it was available in each cohort in both childh

162 However, the influence of SEP on emerging T2DM risks in different ethnic groups an

163 ld income) and distal (education) markers of SEP were used.

164 ere the outcome was reported by a measure of SEP.

165 n future studies using different measures of SEP.

166 Virginia Orange that match the properties of SEP.

167 A cumulative indicator score of SEP was calculated that ranged from 0 (always in the hig

168 ith a pH sensitivity that approaches that of SEP, making it amenable for detection of single exocytos

169 ectra in absorbance units produced values of SEP that ranged from 0.41 to 2.02 mM and an average of 1

170 In vivo animation of SEPs and somatosensory-induced gamma oscillations both m

171 We substantiated the astrocytic origin of SEPs through paired neuron-astrocyte recordings, where w

172 DNA repair and underscores the potential of SEPs to serve important biological functions in mammalia

173 P assignments, and enabled quantification of SEPs under different cellular conditions.

174 With repetitive recordings of SEPs, this somatotopic pattern was reliably recorded for

175 meostasis, trauma had exaggerated effects on SEP amplitude and mitochondrial morphology, suggesting a

176 The dipole on SEPs is consistent with the previously accepted notion t

177 verlapping expression pattern with the other SEP genes.

178 cortex does not significantly reduce the P1 SEP component but strongly reduces the N1 and P2 compone

179 The p47 SEP domain adopts a novel fold with a betabetabetaalphaa

180 The peak-to-peak SEP amplitudes increased as the power in the high freque

181 d but still provides acceptable performance (SEP = 1.20 mM).

182 m reflexes 72 hours after CPR and performing SEP.

183 rane chimera, named super ecliptic phluorin (SEP)-TAC-7.3, functions and traffics as a wild-type (WT)

184 The GFP-based superecliptic pHluorin (SEP) enables detection of exocytosis and endocytosis, bu

185 scent protein mutant superecliptic pHluorin (SEP) is particularly well suited to these applications.

186 To demonstrate the utility of the pHuji plus SEP pair, we perform simultaneous two-color imaging of c

187 n reading frame (sORF)-encoded polypeptides (SEPs) has raised important questions about the functiona

188 ause they detect smORF-encoded polypeptides (SEPs) to validate smORF translation and SEP stability.

189 gether the results suggest a role for poplar SEP-class genes in reproductive viability.

190 Socio-economic position (SEP) and ethnicity influence type 2 diabetes mellitus (T

191 cal process linking socio-economic position (SEP) to health.

192 haracteristics, and socio-economic position (SEP).

193 anges in life-course socioeconomic position (SEP) and cognitive decline or incidence of dementia.

194 the relation between socioeconomic position (SEP) and cognitive milieu in childhood and change in cog

195 preeclampsia, higher socioeconomic position (SEP) and maternal birth in Hong Kong.

196 lating to link lower socioeconomic position (SEP) and poorer health, and the inflammatory system stan

197 associations between socioeconomic position (SEP) and risk of childhood acute lymphoblastic leukemia

198 Children with a low socioeconomic position (SEP) had more asthma alone (adjusted multinomial odds ra

199 Lower socioeconomic position (SEP) has consistently been associated with poorer health

200 and persons of lower socioeconomic position (SEP) have a greater prevalence of caries.

201 Lower socioeconomic position (SEP) in both childhood and adulthood is associated with

202 eview focuses on how socioeconomic position (SEP) may influence hypothetical and actual living kidney

203 financial impacts by socioeconomic position (SEP) of a 20% SSB tax for Australia.

204 umulative effects of socioeconomic position (SEP) on cardiovascular disease have been described, but

205 ence of neighborhood socioeconomic position (SEP) on cognitive decline and examine how individual-lev

206 luence of early-life socioeconomic position (SEP) on incident heart failure in blacks and whites is u

207 g cancer varies with socioeconomic position (SEP), it is not known whether socioeconomic inequalities

208 ures of neighborhood socioeconomic position (SEP).

209 a single measure of socioeconomic position (SEP).

210 earch shows that low socioeconomic position (SEP; especially low income) is associated with statin no

211 tween high and low socio-economic positions (SEP) were compared.

212 ignificantly reduced sound-evoked potential (SEP) amplitudes and increased SEP latencies in control g

213 nents of the somatosensory evoked potential (SEP) before and during movement.

214 d by measuring the somatic evoked potential (SEP) elicited by electrical stimulation of the swine sno

215 re regarding somatosensory evoked potential (SEP) gating is commonly cited as a potential mechanism u

216 e amplitude of the somatic evoked potential (SEP) in the swine is diminished at the infarct site and

217 y (HSI), and somatosensory evoked potential (SEP) were analyzed by using a logistic regression model

218 omponent of somatosensory evoked potentials (SEP) and the area of high-frequency oscillations (HFO).

219 decomposed somatosensory evoked potentials (SEP) into three major components: P1, N1, and P2, where

220 n ratio and somatosensory evoked potentials (SEP) were obtained and CPP was increased from 68 +/- 4 t

221 ys recorded somatosensory evoked potentials (SEP) with an almost twice SNR (signal-to-noise ratio) wh

222 Measuring somatosensory evoked potentials (SEP), electroencephalogram (EEG), direct current (DC) po

223 dent spontaneous slow excitatory potentials (SEP) originate at dendritic locations and are significan

224 edian-nerve somatosensory-evoked potentials (SEPs) and somatosensory-induced gamma-oscillations were

225 Surface evoked potentials (SEPs) during auditory clicks and whisker twitches are us

226 we recorded somatosensory evoked potentials (SEPs) from 6.5-, 8-, and 10-month-old infants while pres

227 g the extracellular sound-evoked potentials (SEPs) from the antennal nerve while monitoring the likel

228 ecording of somatosensory evoked potentials (SEPs) in an ovine model.

229 edian nerve somatosensory-evoked potentials (SEPs) to predict poor outcome in patients treated with m

230 signal and somatosensory evoked potentials (SEPs) using short, typically 300 micros, square stimulat

231 tion 0.982 and standard error of prediction (SEP) between 0.03% and 0.06%.

232 values of the standard error of prediction (SEP) for glucose concentration are maintained to a range

233 blished as the standard error of prediction (SEP) for internal calibration models built with the spec

234 mug kg(-1) and standard error of prediction (SEP) of 14.8 mug kg(-1).

235 Pred)>0.91 and standard error of prediction (SEP) of 24mg/100g phenolics, 7mg/100g monomeric anthocya

236 a, providing a standard error of prediction (SEP) of less than 1 mM over the concentration range of 3

237 Standard error of prediction (SEP) values are 1.12 and 0.45 mM for glucose models gene

238 (PLSR) showed standard error of prediction (SEP) ~2.33, 0.06, 0.41, 0.30 and 0.014 g/L for estimatio

239 tion (RSQ) and standard error of prediction (SEP), respectively) for the developed models were: 0.82

240 bination of sequential extraction procedure (SEP) and chemical composition analysis of impoundment sl

241 elivered median nerve stimulation to produce SEPs during a force-matching paradigm (used to quantify

242 Bilateral stimuli produced SEP amplitude fluctuations in each hemisphere that usual

243 score severity and severity-extent product (SEP) on 9-segment bullseye maps and decline in cardiac s

244 score severity, and severity-extent product (SEP).

245 diatoms, are the main cause of the prominent SEP.

246 hesis of "synthetic erythropoiesis protein" (SEP), a 51-kilodalton protein-polymer construct consisti

247 ter and less abundant than average proteins, SEP detection using proteomics faces unique challenges.

248 ulate matter during the summer export pulse (SEP) average 408, 283, 24.1, 1.1, and 67.5 mumol.m(-2).d

249 ation scheme of stimulated emission pumping (SEP) induces shifts of a single water molecule between t

250 Stimulated emission pumping (SEP)-hole filling spectroscopy and SEP-induced populatio

251 The recurrent SEP is enigmatic because it is focused in time despite t

252 ng health promotion policies may help reduce SEP inequalities in health.

253 of En and Inv in postmitotic neurons reduces SEP amplitude but also reduces synaptic strength at the

254 Mutants in five rice (Oryza sativa) SEP genes suggest both redundant and unique functions in

255 SEPALLATA (SEP) genes exhibit distinct patterns of expression and f

256 SEPALLATA (SEP) MADS box transcription factors mediate floral devel

257 protein, MIP1, and C-function and SEPALLATA (SEP)-like MBFs.

258 te functional diversification for SEPALLATA (SEP)-like MADS box genes that often function redundantly

259 OF OVEREXPRESSION OF CO 1 (SOC1), SEPALLATA (SEP) and SHORT VEGETATIVE PHASE (SVP) Arabidopsis thalia

260 es, such as AN and members of the SEPALLATA (SEP) family, to activate precociously, while the express

261 Members of the SEPALLATA (SEP) MADS-box subfamily are required for specifying the

262 Sepiapterin (SEP) is a tetrahydrobiopterin precursor, and L-citrullin

263 ndent on an interaction between phospho-Ser (SEP)-196 and Arg-201, reducing Arg-201 interaction with

264 Use of eye care services; SEP was measured by the PIR and educational attainment.

265 nagement Bundle, Severe Sepsis/Septic Shock (SEP-1) performance measure to the Hospital Inpatient Qua

266 ng acute and chronic recordings, significant SEP responses in the triangle between the ansate, diagon

267 Since SEP indicators fluctuated independently from whole anima

268 ity of our labeling approach using synthetic SEP.

269 We found that SEP inequalities in inflammation followed heterogeneous

270 These observations lead us to speculate that SEP-1 has two separable yet coordinated functions: to re

271 tional and evolutionary studies suggest that SEP genes may have been critical for the origin of the f

272 The SEP is approximately threefold greater than mean wintert

273 The SEP was measured on the SI cortex ipsilateral to the sit

274 The SEP-1 performance measure requires, among other critical

275 Interestingly, the ICH also attenuated the SEP on the contralateral SI cortex.

276 The ICH rapidly attenuated the SEP on the ipsilateral cortex as we reported earlier.

277 vel human genes), improved confidence in the SEP assignments, and enabled quantification of SEPs unde

278 Here, we optimize several steps in the SEP discovery workflow to improve SEP isolation and iden

279 , there is no evidence of discordance in the SEP of donors and recipients that would suggest undisclo

280 e of these antibiotics, as called for in the SEP-1 measure, is not in alignment with prudent antimicr

281 ing that similar mechanisms may modulate the SEP.

282 thy diet explained part, but not all, of the SEP inequalities in inflammation; in general, BMI exerte

283 Of the SEP measures, mid-to-older adulthood SEP showed the stro

284 Here, we examine expression of the SEP-like gene LEAFY HULL STERILE1 (LHS1) in phylogenetic

285 lthough it reduced the N120 component of the SEP.

286 These and other data demonstrate that the SEP genes play central roles in flower meristem identity

287 Additional studies indicate that the SEP proteins form multimeric complexes with the products

288 ator of PKC inhibition and suggests that the SEP-196/Arg-201 interaction is critical for maintaining

289 These SEP values are 1.51 and 1.14 mM for spectrometers B and

290 characterization of MRI-2 suggests that this SEP may participate in DNA repair and underscores the po

291 Risk exposure related to SEP accumulates across the life course and contributes t

292 te children and was significantly related to SEP.

293 l cortical columns, we examined single trial SEP amplitudes from multiple cortical locations across s

294 ice at 6 to 8 weeks of age received vehicle, SEP, or L-Cit orally alone or in combination for 8 weeks

295 Whether SEP and L-Cit are effective at reducing diabetic heart d

296 healthy human subjects to determine whether SEP gating correlated with the behavior.

297 childhood ALL was negatively associated with SEP among Hispanics but was positively associated among

298 and 95% confidence intervals associated with SEP and race/ethnicity, adjusted for sex, age, and year

299 of statin adherence and its association with SEP.

300 to the risk of incident heart failure, with SEP later in adulthood having a more prominent role than