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1 SGLT2 inhibitors protected against the risk of major adv
2 ucose with a sodium-glucose cotransporter 2 (SGLT2) inhibitor could improve insulin-mediated tissue g
3 hypoglycemic sodium-glucose cotransporter 2 (SGLT2) inhibitor dapagliflozin and the insulin sensitize
4 iflozin, the sodium-glucose cotransporter 2 (SGLT2) inhibitor, on renal hemodynamics and tubular func
6 nt with the sodium-glucose co-transporter-2 (SGLT2) inhibitor empagliflozin reduced albuminuria in pa
8 g effects of sodium-glucose cotransporter 2 (SGLT2) inhibitors are already established, guidance is n
9 Carbasugar sodium-glucose cotransporter 2 (SGLT2) inhibitors are highly promising drug candidates f
11 gon release, sodium-glucose cotransporter 2 (SGLT2) inhibitors induce stimulation of endogenous gluco
13 ering agents-sodium-glucose cotransporter 2 (SGLT2) inhibitors-has been reported to decrease the risk
15 2 diabetes, sodium-glucose cotransporter-2 (SGLT2) inhibitors are known to reduce glucose concentrat
17 rofile, the sodium-glucose co-transporter-2 (SGLT2) inhibitors empagliflozin and canagliflozin have b
18 gonists and sodium-glucose co-transporter-2 (SGLT2) inhibitors reduce glycaemia and weight, and impro
20 the efficacy and safety of canagliflozin, an SGLT2 inhibitor, with glimepiride in patients with type
21 el glucose lowering agent, empagliflozin, an SGLT2 inhibitor which targets the kidney to block glucos
22 ynthetic route towards some novel carbasugar SGLT2 inhibitors, featuring an underexploited, regiosele
23 he way towards the development of carbasugar SGLT2 inhibitors as potential antidiabetic/antitumor age
24 However, the clinical usage of carbasugar SGLT2 inhibitors has been underexplored, due to the leng
25 ealed the unexpected SAR of these carbasugar SGLT2 inhibitors, and enabled the discovery of a highly
30 ure and diabetic glomerular hyperfiltration, SGLT2 inhibitors may induce protective effects on the ki
33 synthetic route towards some small-molecule SGLT2 inhibitors by a chemo- and diastereospecific palla
35 fraction may be mitigated by the actions of SGLT2 inhibitors to reduce blood pressure, body weight,
40 way, sodium-related physiological effects of SGLT2 inhibitors and clinical correlates of natriuresis,
41 summarize the key pharmacodynamic effects of SGLT2 inhibitors and the clinical evidence that support
45 rmacokinetic and pharmacodynamic profiles of SGLT2 inhibitors in clinical trials and examine possible
48 ntify significant differences between use of SGLT2 inhibitors and use of other glucose-lowering drugs
50 rong rationale to expect benefit from use of SGLT2 inhibitors in patients with type 2 diabetes at hig
51 ed with other glucose-lowering drugs, use of SGLT2 inhibitors was associated with a decreased risk of
52 ed with other glucose-lowering drugs, use of SGLT2 inhibitors was associated with decreased risk of c
54 ular mortality and morbidity in new users of SGLT2 inhibitors versus new users of other glucose-lower
55 onsistent with data from clinical studies on SGLT2 inhibitors and provide a rationale for the mode of
56 iven by findings for empagliflozin (the only SGLT2 inhibitor for which data from a dedicated long-ter
59 discovery of two highly selective and potent SGLT2 inhibitors, thereby paving the way towards the dev
60 tes and a broad cardiovascular risk profile, SGLT2 inhibitor use was associated with reduced cardiova
62 ecursors was synthesized and tested as SGLT1/SGLT2 inhibitors using a cell-based fluorescence assay o
64 ge pancreatic and prostate cancers, and that SGLT2 inhibitors, currently in use for treating diabetes
65 e adenocarcinomas, and provide evidence that SGLT2 inhibitors block glucose uptake and reduce tumor g
66 nical practice guidelines now recommend that SGLT2 inhibitors with proven cardiovascular benefit be p
67 the GLP-1 receptor agonist exenatide and the SGLT2 inhibitor dapagliflozin with exenatide or dapaglif
68 blood pressure and glycaemic effects of the SGLT2 inhibitor dapagliflozin with placebo in patients w
69 thod is demonstrated by the synthesis of the SGLT2 inhibitor, canagliflozin (1a), from commercially a
73 produced, it is important to understand why SGLT2 inhibitors inhibit <50% of the filtered glucose lo
74 nital tract infections were more common with SGLT2 inhibitors (odds ratios, 1.42 [CI, 1.06 to 1.90] a
75 clinical trials, large clinical trials with SGLT2 inhibitors are now investigating the potential use
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