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1 ol x min(-1) x [20 microl packed cells](-1), SHRSP vs. WKY, respectively, P = 0.01) and inhibition of
3 derived from SHRSP(HD)/WKY-0(HD) (n=115) and SHRSP(HD)/WKY-1(HD) (n=139) crosses (WKY-0(HD) and WKY-1
6 is were performed on F2 hybrids derived from SHRSP(HD)/WKY-0(HD) (n=115) and SHRSP(HD)/WKY-1(HD) (n=1
10 The stroke-prone spontaneously hypertensive (SHRSP) rat is a model of human insulin resistance and is
12 ease in response to insulin: 1.4 +/- 0.15 in SHRSP, 2.29 +/- 0.22 in WKY; n = 4, P = 0.02), but the s
16 ence and expression are apparently normal in SHRSP, it is likely that the molecular mechanism for def
17 cerebral ischemia via vascular protection in SHRSP rats and neural protection in both the SHRSP and W
19 map was constructed in two F2 intercrosses (SHRSP x BN and FHH x ACI), containing a total of 4736 si
20 ts for defects in insulin action in the male SHRSP rat model compared with the normotensive, insulin-
21 ide screen in an F2 cross obtained by mating SHRSP and SHR, in which latency to stroke on Japanese ra
22 pathogenesis of thrombotic microangiopathy, SHRSP were adrenalectomized and infused with vehicle, An
23 ive effect against stroke in the presence of SHRSP alleles and STR-2 co-localized with the genes enco
24 in spontaneously hypertensive stroke-prone (SHRSP) rats protects against cerebral ischemia induced b
25 stroke-prone spontaneously hypertensive rat (SHRSP(HD)), is a primary, genetically determined trait a
26 stroke-prone spontaneously hypertensive rat (SHRSP) as a model organism, mated it with the stroke-res
27 stroke-prone spontaneously hypertensive rat (SHRSP) is a genetically determined model of "salt-sensit
28 spontaneously hypertensive stroke-prone rat (SHRSP) is an experimental model of stroke characterized
32 SHRSP rats and neural protection in both the SHRSP and WKY rats, indicating that SEH inhibition has b
33 chromosome 3: in animals homozygous for the SHRSP(HD) allele, HR was 414+/-49 compared with 383+/-44
34 genome scan in an F2 cross derived from the SHRSP and the normotensive reference strain, WKY rat.
36 responsible for large infarct volumes in the SHRSP in response to a focal ischaemic insult by perform
37 that the insulin resistance observed in the SHRSP is manifest at the level of skeletal muscle, that
39 y of stroke the phenotypic expression of the SHRSP is (i) either increased or decreased, depending on
41 sulin action on 2-deoxy-D-glucose transport (SHRSP 3.3 +/- 1.5 vs. 21.0 +/- 7.4 pmol x min(-1) x [20
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