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1 SJS is an autosomal recessive skeletal dysplasia charact
2 SJS, SJS/TEN, and TEN pose a substantial health care bur
3 SJS/TEN mostly manifest as a reaction to new drug use, b
4 SJS/TEN was associated with nonwhite race, particularly
8 ity (15 drug-induced liver injury [DILI], 33 SJS/TEN, 20 hypersensitivity syndrome, and 46 nevirapine
9 nts, we calculated an incidence rate of 5.76 SJS/TEN cases per million person-years between 1995 and
10 re (SJS: 9.8 +/- 0.3 days, $21,437 +/- $807; SJS/TEN: 16.5 +/- 1.0 days, $58,954 +/- $5,238; TEN: 16.
13 apine-induced rash plus 3 with both DILI and SJS phenotype) and 155 age-, sex- and ethnicity-matched
16 atistically significant associations between SJS/TEN and pre-existing depression, lupus erythematosus
21 ased cytotoxicity in in vitro models of both SJS/TEN (elicited by drug-specific antigen) and GVHD (el
22 ged length of stay and higher costs of care (SJS: 9.8 +/- 0.3 days, $21,437 +/- $807; SJS/TEN: 16.5 +
24 g ophthalmic records of patients with either SJS (<30% body surface area involvement) or TEN (> = 30%
30 nternational experts, a case report form for SJS/TEN has been created to help standardize the collect
33 ents with epilepsy and gout, odds ratios for SJS/TEN were significantly increased only in the presenc
34 was found to markedly increase the risk for SJS (odds ratio [OR] = 17.52; 95% confidence interval, 3
36 on possible immunomodulating treatments for SJS/TEN and estimate their effects on mortality compared
37 sed systemic immunomodulating treatments for SJS/TEN, which is of great relevance for treating physic
38 those who have already suffered damage from SJS, but emphasis on the prevention of damage in the acu
39 ion, Clinical Modification codes to identify SJS, SJS/TEN, and TEN (n = 2,591, n = 502, and n = 564,
41 0.05) comparing between the moderate (15% in SJS, 0% in TEN) and severe groups (20% in SJS, 33% in TE
43 number of cases with mild involvement (5% in SJS, 42% in TEN, p = 0.01), while no statistically signi
46 9-to-TIMP-1 ratios were markedly elevated in SJS and OCP tears (SJS>OCP) when compared with those of
50 wann cell physiology and suggest that PNH in SJS originates distally from synergistic actions of peri
52 nd MPO levels were elevated significantly in SJS and OCP tears (SJS>OCP) when compared with controls.
54 between HLA-B*1502 and carbamazepine-induced SJS and TEN and that reported sufficient data for calcul
55 HLA-B*1502 allele and carbamazepine-induced SJS and TEN in Han-Chinese, Thai, and Malaysian populati
58 ocytes (CTLs) from patients with CBZ-induced SJS/TEN and analyzed the interaction between HLA-B and C
61 son syndrome and toxic epidermal necrolysis (SJS/TEN) are rare but severe adverse reactions with high
62 son syndrome and toxic epidermal necrolysis (SJS/TEN), although a detailed description is lacking in
63 ty-five patients (50 eyes) with acute ocular SJS who presented within 4 weeks of onset of symptoms we
66 opriate and useful" for documenting cases of SJS/TEN, making it more reliable and valuable for future
69 l observational study on the epidemiology of SJS/TEN contributes to the understanding of this still u
70 e observational study on the epidemiology of SJS/TEN using data from the UK-based Clinical Practice R
71 32Y mutation as the sole causative factor of SJS in the human family harboring this alteration and im
74 luded 86 patients (167 eyes) with history of SJS/TEN who underwent PROSE treatment from January 1, 20
80 a quality based on an international panel of SJS/TEN experts who performed a Delphi consensus-buildin
81 n suggestive of resorption among patients of SJS was 36.7 months and among patients of chemical injur
82 o the eyes and eyelids in the acute phase of SJS can prevent the devastating scarring and visual prob
84 ging, the severe, chronic ocular problems of SJS can be at least partially alleviated with autologous
90 ong signal for an increased risk of DRESS or SJS requires particular vigilance in telaprevir-treated
93 Clinical Modification codes to identify SJS, SJS/TEN, and TEN (n = 2,591, n = 502, and n = 564, respe
95 he relatively mild Schwartz-Jampel syndrome (SJS) and severe neonatal lethal dyssegmental dysplasia,
97 developed to model Schwartz-Jampel syndrome (SJS), a skeletal disease resulting from decreased perlec
98 een HLA-B*1502 and Stevens-Johnson syndrome (SJS) and toxic epidermal necrolysis (TEN) among carbamaz
103 crolysis (TEN) and Stevens-Johnson Syndrome (SJS) are rare, acute, life-threatening dermatologic diso
104 eatments for acute Stevens-Johnson syndrome (SJS) as well as the emerging treatment options for patie
106 By comparison, Stevens-Johnson syndrome (SJS) is a life-threatening severe cutaneous adverse reac
107 injury (DILI) and Stevens-Johnson syndrome (SJS) or toxic epidermal necrosis (TEN) is even rarer and
108 Three cases of Stevens-Johnson Syndrome (SJS), and 11 cases of Drug Reaction with Eosinophilia an
110 f SCARs, including Stevens-Johnson syndrome (SJS), toxic epidermal necrolysis (TEN), drug rash with e
111 ed by PPI, such as Stevens-Johnson syndrome (SJS), toxic epidermal necrolysis (TEN), or drug rash wit
112 pine (CBZ)-induced Stevens-Johnson syndrome (SJS)/toxic epidermal necrolysis (TEN) as a model to stud
119 th chronic ocular surface disease related to SJS/TEN, PROSE treatment offers sustained and significan
122 harboring only the mutation associated with SJS, displayed a mild phenotype, inconsistent with SJS.
124 ients who developed DILI in association with SJS/TEN from a registry of DILI patients from a single c
125 rience of DILI occurring in association with SJS/TEN including the etiologic agents, clinical and bio
126 h mild ocular involvement when compared with SJS, but no significant difference between the number of
129 Thus, it is crucial that all patients with SJS be evaluated by an ophthalmologist familiar with the
130 Heterozygous mutations in two patients with SJS either produced truncated perlecan that lacked domai
133 s study included 30 eyes of 30 patients with SJS-induced dry keratinized ocular surfaces; the patient
136 Retrospective study of 49 patients with SJS/TEN hospitalized in a referral care center from 2005
139 sitivity, such as that seen in patients with SJS/TEN, as well as the alloreactivity seen in patients
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