ライフサイエンスコーパス: SLAM-associated protein

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1 n molecule) and its interacting protein SAP (SLAM-associated protein).

2 equires the presence of the adaptor molecule SLAM-associated protein.

3 associates with the recently described human SLAM-associated protein.

4 Cytokines also changed the levels of SLAM-associated protein adaptors, which prevent the inhi

5 The 2 CD84/SLAM adapter proteins, SAP (SLAM-associated protein) and EAT-2 (EWS-activated transc

6 However, compared with XLP caused by SLAM-Associated Protein deficiency (SH2D1A mutation), XI

7 thought to contribute to HLH development in SLAM-Associated Protein deficiency were not observed in

8 a critical role for the adaptor protein SAP (SLAM-associated protein) during NKT cell development.

9 ative disease gene product, SH2D1A/DSHP/SAP (SLAM-associated protein, or SAP) in NK cells.

10 ugh mice that lack the adaptor molecule SAP (SLAM-associated protein) resemble ICOS(-/-) mice (and ca

11 Signaling lymphocytic activation molecule (SLAM)-associated protein (SAP [SH2D1A]) expression in CD

12 Signaling lymphocytic activation molecule (SLAM)-associated protein (SAP) can mediate the function

13 in signaling lymphocyte activation molecule (SLAM)-associated protein (SAP) exhibit a selective impai

14 al signaling lymphocyte activation molecule (SLAM)-associated protein (SAP) in T, invariant natural k

15 Signaling lymphocyte activation molecule (SLAM)-associated protein (SAP)) interactions with SLAM f

16 es signaling lymphocyte activation molecule (SLAM)-associated protein (SAP), is altered in patients w

17 o signaling lymphocytic activation molecule (SLAM)-associated protein (SAP), the X-linked lymphoproli

18 a signaling lymphocytic activation molecule (SLAM)-associated protein (SAP)-deficient mouse model.

19 sed SHIP-1, SHP-1, SHP-2, and CSK but lacked SLAM-associated protein (SAP) and Ewing's sarcoma-activa

20 ) arises from mutations in the gene encoding SLAM-associated protein (SAP) and leads to abnormalities

21 nd the downstream recruitment of the adaptor SLAM-associated protein (SAP) and the Src kinase Fyn, wh

22 Mutations in SH2D1A resulting in lack of SLAM-associated protein (SAP) expression cause the human

23 nked lymphoproliferative disease, which lack SLAM-associated protein (SAP) expression.

24 signals in both cell types, mediated by the SLAM-associated protein (SAP) family of adaptors.

25 Notably, mutations in SLAM-associated protein (SAP) lead to X-linked lymphopro

26 Mutations affecting SLAM-associated protein (SAP) prevent GC formation becau

27 AM) family of receptors that signals through SLAM-associated protein (SAP), an SH2 domain protein tha

28 on T-cell-derived signals through CD40L and SLAM-associated protein (SAP), but not IL-17.

29 ls, T(FR) cell development depends on Bcl-6, SLAM-associated protein (SAP), CD28 and B cells; however

30 ther with their downstream signaling adaptor SLAM-associated protein (SAP), have emerged as key playe

31 aused by mutations in SH2D1A/SAP that encode SLAM-associated protein (SAP), is characterized by an in

32 We now show that a T-cell-specific, SLAM-associated protein (SAP), which contains an SH2 dom

33 SH2D1A encodes the adaptor molecule SLAM-associated protein (SAP), which is expressed in T a

34 were identified by diminished intracellular SLAM-associated protein (SAP), X-linked inhibitor of apo

35 After infection, SLAM-associated protein (SAP)-/- mice show increased T c

36 SLAM-associated protein (SAP)-deficient mice had normal

37 l selection on hematopoietic cells through a SLAM-associated protein (SAP)-dependent mechanism simila

38 defined molecularly by loss of expression of SLAM-associated protein (SAP).

39 ls induced by anti-CD150 is not dependent on SLAM-associated protein (SAP, SH2D1A), because anti-CD15

40 nd if male, Bruton tyrosine kinase (Btk) and SLAM-associated protein (SAP/SH2D1A).

41 he signaling lymphocyte activation molecule (SLAM)-associated protein, SAP, was first identified as t

42 iated in part by the adaptor protein SH2D1A (SLAM-associated protein, SAP).

43 ylation of NTB-A and the association of SAP (SLAM-associated protein), the protein absent in X-linked

44 (signaling lymphocytic activation molecule [SLAM]-associated protein) - was cloned, the crystal stru

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