ライフサイエンスコーパス: SPF

1 SPF >/= 15 sunscreen use was associated with significant

2 SPF IL-10-knockout mice had no significant difference in

3 SPF mice colonized with rETBF mimicked WT-ETBF, whereas

4 SPF microbiota did not induce HO-1 in colons of germ-fre

5 SPF optoacoustic imaging was applied to imaging arteries

6 tion between sunscreen use (never, SPF < 15, SPF >/= 15) and melanoma risk by calculating hazard rati

7 neata the proportions were approximately 20% SPF, 5% PMF, and PRF was absent.

8 et ~ UV Index (355 reviews), VisualDx (306), SPF (128), iSore (61), and SpotMole (50).

9 all ESTs) and PRF (15%), with less than 0.5% SPF.

10 C56BL/6J SPF and GF mice were placed on custom diets containing d

11 leading computational prediction algorithms: SPF-Cancer and TransFIC.

12 ng GGQ to reach the PTC while still allowing SPF-stop-codon interaction.

13 in/+) ;Il10(-/-) mice conventionalized by an SPF microbiota had significantly more colon tumors compa

14 The tissue specimens were HPV typed using an SPF(10) line probe assay HPV detection system.

15 004), grade (P =.004), ploidy (P =.006), and SPF (P =.05) were associated with time to recurrence; th

16 tor, progesterone receptor, patient age, and SPF.

17 onserved amino acid motifs (NIKS in eRF1 and SPF in RF2) and by the conserved tripeptide (GGQ) intera

18 sely, colonic macrophages from germ-free and SPF-derived colitis-prone Il10(-/-) mice demonstrated ro

19 is was not significantly different in GF and SPF mice, there was a delay in intestinal epithelial rep

20 We compared the results of HC2 and SPF(10) testing of cervical specimens.

21 valence of carcinogenic HPV types by HC2 and SPF(10)-LiPA among women with normal, atypical squamous

22 observed very good agreement between HC2 and SPF(10)-LiPA for carcinogenic HPV type detection.

23 ong positive correlation between mitosin and SPF (r = 0.57; P = 0.0001), and there were significant n

24 d grade were associated with both ploidy and SPF (P </=.01).

25 the genes of two of these proteins (PRF and SPF) are prone to incessant evolution driven by positive

26 Like PRF and SPF, PMF is produced by a multigene family characterized

27 xtreme in these dimensions than both PRF and SPF.

28 iable with broad-spectrum sunscreens because SPF with primarily UVB sunscreens is dependent on time o

29 For RF2, the separation between SPF and GGQ elongates from 32 angstroms in the crystal t

30 as 35% (n=1,962) by HC2 and 35% (n=2,003) by SPF(10)-LiPA.

31 Samples were tested for HPV by SPF(10) PCR/DEIA/LiPA(25), version 1.

32 Cereal-SPF rats displayed increased gut CD3(+) and CD8alpha(+)

33 T1D was highest in cereal-SPF (65%) and cereal-GF rats (53%) but inhibited and del

34 +) was highest in HC-GF and lowest in cereal-SPF rats.

35 Clean SPF mice had attenuated responses to gluten compared to

36 inst gluten-induced immunopathology in clean SPF mice was reversed after supplementation with a membe

37 nscreen, the UVA filter, and the combination SPF 15 sunscreen and UVA filter, resulting in increasing

38 nd the side chain of serine of the conserved SPF motif of RF2 recognize U1 and A2 of the stop codon,

39 n two separate experiments; however, control SPF kittens housed with highly bacteremic kittens in the

40 ared to 73.6g glucose/100g starch in control SPF and 65.9 g in SPS noodles.

41 thogens and Proteobacteria, and conventional SPF mice that harbor a complex microbiota that includes

42 luten compared to germ-free and conventional SPF mice.

43 uten-induced immunopathology in conventional SPF mice.

44 nation of Sun Protection Factor for DNA (DNA-SPF), using specific DNA repair enzymes and antibodies,

45 -10-deficient mice were maintained in either SPF conditions or germfree conditions or were populated

46 rom mice housed in pathogen free facilities (SPF).

47 actor (PRF), Sodefrin Precursor-Like Factor (SPF), and Plethodontid Modulating Factor (PMF).

48 y use sunscreens with sun protection factor (SPF) 15.

49 en (UVA superior, UVB sun protection factor (SPF) 50) delayed the onset of UVR-driven melanoma, but o

50 benzophenone-3 with a sun protection factor (SPF) of 15, the UVA filter butyl methoxydibenzoylmethane

51 ompare high- with low-sun protection factor (SPF) sunscreens in relation to sunbathing habits in a la

52 n and is expressed as Sun Protection Factor (SPF).

53 ology [AAD] criteria, sun protection factor [SPF], or vehicle) could be used to predict price per oun

54 eens with comparable sun protection factors (SPFs), but with different levels of UVA protection, espe

55 rats remained diabetes-free, whereas HC-fed SPF rats were less protected (7 vs. 29%).

56 cattery cats transmitted B. henselae to five SPF kittens in two separate experiments; however, contro

57 6 at 10%, 15% and 20% in sweet potato flour (SPF) and 5% and 10% in sweet potato starch (SPS) in redu

58 nd HPV73 (which is not targeted by HC2), for SPF(10)-LiPA, we defined the carcinogenic HPV types as t

59 54.96% for NUTRIOSE (15%)+GG (1%) fortified SPF noodles and 53.3% for NUTRIOSE (5%)+GG (0.5%) fortif

60 Four SPF H. felis-uninfected cats served as controls.

61 om the SSF into the soymilk pellet fraction (SPF) following incubation with 0.9% PGA for 1h.

62 ctor in breast cancer, and S-phase fraction (SPF), as measured by flow cytometry, is the most clinica

63 low cytometric (ploidy and S-phase fraction [SPF]) and histopathologic analyses (Nottingham Combined

64 They applied a single-pulse-frame (SPF) sequence, which enabled motion insensitive optoacou

65 abnormally hygienic specific pathogen free (SPF) barrier facilities.

66 ricted flora (RF) vs specific pathogen free (SPF).

67 nd the microbiota in specific pathogen-free (SPF) and germ-free (GF) mice given more than 40 unique d

68 Specific pathogen-free (SPF) C57BL/6J or germfree 129S6/SvEv mice were orally in

69 feces collected from specific-pathogen-free (SPF) chickens experimentally infected with avian HEV wer

70 characterization in specific-pathogen-free (SPF) chickens.

71 s animals died under specific pathogen-free (SPF) conditions between 6 and 7 months of age.

72 o, WT mice raised in specific pathogen-free (SPF) conditions fared better against I/R-induced injury

73 C/Tnd mice housed in specific pathogen-free (SPF) conditions induced KLK5 and activated the protease-

74 se when reared under specific-pathogen-free (SPF) conditions, suggesting the involvement of a microbi

75 causes colitis under specific pathogen-free (SPF) conditions.

76 colitis when kept in specific-pathogen-free (SPF) environments.

77 ce with conventional specific pathogen-free (SPF) gut microbiota increases both bone formation and re

78 Five specific-pathogen-free (SPF) Helicobacter-free cats were studied before and for

79 otentiate colitis in specific-pathogen-free (SPF) IL-10(-/-) mice.

80 lar to those seen in specific pathogen-free (SPF) IL-2(-/-) mice.

81 biota composition of specific pathogen-free (SPF) INS-GAS mice was quantified by pyrosequencing.

82 rring these fleas to specific-pathogen-free (SPF) kittens housed in a controlled, arthropod-free Univ

83 Specific Pathogen-Free (SPF) layer chickens were infected with ALV-J or maintain

84 erm-free mice, clean specific-pathogen-free (SPF) mice colonized with a microbiota devoid of opportun

85 free (GF) and normal specific-pathogen-free (SPF) mice have revealed the impact of host immunosurveil

86 onventionally raised specific pathogen-free (SPF) mice treated with azoxymethane (AOM) and dextran su

87 ops spontaneously in specific pathogen-free (SPF) mice with a targeted disruption in the IL-10 gene (

88 in sharp contrast to specific pathogen-free (SPF) mice, germ-free (GF) mice are resistant to Concanav

89 ared with disease in specific pathogen-free (SPF) mice, ileitis in GF mice is significantly attenuate

90 were colonized with specific pathogen-free (SPF) microbiota.

91 H pylori-specific pathogen-free (SPF) monkeys were experimentally challenged with wild-ty

92 and exposed them to specific-pathogen-free (SPF) or colorectal cancer-associated bacteria.

93 rats were housed in specific pathogen-free (SPF) or germ-free (GF) conditions and weaned onto diabet

94 , the development of specific-pathogen-free (SPF) oysters has enabled assessment of the infection pro

95 nts, we utilized the specific-pathogen-free (SPF) pig model for HEV and a unique inoculation procedur

96 tious DNA clones, 40 specific-pathogen-free (SPF) pigs were randomly assigned into five groups of eig

97 ely transferred into specific pathogen-free (SPF) RAG-/- mice, but not in germfree RAG-/- mice.

98 ere transferred from specific pathogen-free (SPF) to conventional (non-SPF) animal rooms, and evaluat

99 espiratory tracts of specific-pathogen-free (SPF) young turkeys.

100 uces stool output in specific pathogen-free (SPF), but not GF mice.

101 s from germ-free and specific pathogen-free (SPF)-derived mice produce IL-10, but not IL-12 p40, when

102 hat stimulation with purified fecal Ags from SPF, but not GF mice leads to the generation of IL-4-sec

103 -10(-/-) mice were colonized with stool from SPF mice that harbored or did not harbor endogenous H. h

104 PF conditions from birth or transferred from SPF conditions at weaning have predominantly ileal tumor

105 is study, these structure-property-function (SPF) and structure-property-hazard (SPH) relationships a

106 of establishing structure-property-function (SPF) and structure-property-hazard (SPH) relationships t

107 rse and richer gut microbiota than did group SPF.

108 e abundant in groups XZ and JD than in group SPF, whereas Firmicutes showed the inverse pattern.

109 ntly lower in groups XZ and JD than in group SPF.

110 Furthermore, transfer of MLN cells from BM-->SPF Tg(epsilon26) mice into SPF Tg(epsilon26) recipients

111 beit in lower frequency than in control BM-->SPF Tg(epsilon26) mice.

112 In SPF and germfree mice, ETBF caused colitis but was letha

113 estored by anti-CD8beta Ab, and augmented in SPF mice bearing the TAP(-/-) genotype.

114 TBF induced acute then persistent colitis in SPF mice and rapidly lethal colitis in WT germfree mice.

115 al microbiota and the severity of colitis in SPF mice, but not in GF mice or mice given antibiotics.

116 ose fiber reduced the severity of colitis in SPF mice, whereas methylcellulose increased severity.

117 inant microbiota antigen, induced colitis in SPF RAG-/- mice.

118 bacter hepaticus, induces chronic colitis in SPF-reared IL-10(-/-) mice and that the disease is accom

119 e the SI-Ep under steady-state conditions in SPF mice.

120 ined by rapid and selective pDC depletion in SPF mice transferred with RF CD8(+) T cells.

121 Bacterial communities differed in SPF rats fed cereal compared with HC.

122 itis was induced in wild-type mice housed in SPF conditions by infection with Salmonella typhimurium.

123 ignificantly correlated with inflammation in SPF-housed Apc(Min/+) ;Il10(-/-) , but not in Apc(Min/+)

124 IL-10-deficient mice kept in SPF conditions developed colitis in all segments of the

125 osteoclastogenesis and alveolar bone loss in SPF mice compared with GF mice.

126 %) significantly reduced the cooking loss in SPF noodles, this was enhanced in SPS noodles and guar g

127 ut microflora compared to mice maintained in SPF facilities.

128 as against 33.8% and 40.68%, respectively in SPF and SPS controls.

129 indings demonstrate that the colitis seen in SPF IL-2(-/-) mice depends upon the presence of intestin

130 ic cells lining the alveolar bone surface in SPF compared with GF mice.

131 more and larger tumors compared with that in SPF mice after AOM and DSS treatment despite the lack of

132 aralleling detection of IAV in H9N2-infected SPF chickens and chickens from LBM showed that pan-IAV F

133 Stomachs of H pylori-infected SPF male mice had significant reductions in Bacteroidete

134 is was inoculated by gastric intubation into SPF C57BL/6 wild-type and p53 hemizygous mice that were

135 cells from BM-->SPF Tg(epsilon26) mice into SPF Tg(epsilon26) recipients induced active colitis, but

136 s from these mice were then transferred into SPF or GF recipients.

137 IELs are reduced, and in lethally irradiated SPF IL-2(+/+) mice, reconstituted with IL-2(-/-) bone ma

138 mulative sunburn with two sunscreens labeled SPF 6, but with different UVR-absorbing properties, one

139 They also show that labeled SPF is much more reliable with broad-spectrum sunscreens

140 Sun Protection Factor for Lethal Damage (LD-SPF), by measuring cell viability and apoptosis induced

141 o develop in germfree INS-GAS mice than male SPF INS-GAS mice.

142 In colons of germ-free, wild-type mice, SPF microbiota induced production of HO-1 via activation

143 Moreover, SPF WT mice i.p. administered 10 mg/kg MDP were protecte

144 he association between sunscreen use (never, SPF < 15, SPF >/= 15) and melanoma risk by calculating h

145 up of mdx mice and controls (housed in a non-SPF facility) using MRI at 1, 3, 6, 9 and 12 months afte

146 fic pathogen-free (SPF) to conventional (non-SPF) animal rooms, and evaluated clinically and histolog

147 ter species [non-specific-pathogen-free (non-SPF) conditions].

148 one-fourth of GPX-DKO mice raised under non-SPF conditions from birth or transferred from SPF condit

149 bone height is significantly less in normal SPF mice compared with their age- and strain-matched GF

150 nsfer of CD4-positive cells from GF, but not SPF mice induces severe colitis in SCID recipients.

151 Application of SPF-15 sunscreens to mouse skin before each UV irradiati

152 helper cells in the junctional epithelium of SPF mice compared with GF mice suggest that the adaptive

153 cells and IL17+ cells in the periodontium of SPF mice demonstrate possible molecular mechanisms media

154 ight promote GIN in achlorhydric stomachs of SPF mice.

155 Conclusion Use of SPF >/= 15 rather than SPF < 15 sunscreens reduces melan

156 Moreover, use of SPF >/= 15 sunscreen by all women age 40 to 75 years cou

157 n attributable fraction) with general use of SPF >/= 15 sunscreens by women age 40 to 75 years was 18

158 ock was determined by inoculating 1-week-old SPF chickens intravenously with 200 microl of each of se

159 In 4-week-old SPF chickens, NDV-Belize-3/08 behaved as a typical velog

160 Germ-free or SPF-raised wild-type and Il10(-/-) mice were given intra

161 mice was transplanted into germfree (GF) or SPF Tg(epsilon26) mice.

162 nsal microflora without any known pathogens (SPF), <9% of GPX-DKO mice develop tumors in the ileum or

163 determine the fraction of cells in S phase (SPF).

164 ctions with multiple residues beyond the PXT/SPF motifs.

165 However, in the X-ray structure of RF2, SPF and GGQ are only 23 A apart, indicating that they ca

166 Another conserved motif of RFs (SPF in RF2) has been proposed to interact directly with

167 tions: a specific pathogen-free animal room (SPF), a general animal room (XZ) and a farmhouse (JD).

168 246 and 246X lines had similar SPFs that were approximately twofold greater than contro

169 ot adhere to AAD guidelines (broad spectrum, SPF >/=30, and water resistant) for sunscreens.

170 fared better against I/R-induced injury than SPF Nod2(-/-) mice.

171 y, HC2 was more likely to test positive than SPF(10)-LiPA for the carcinogenic HPV types (87% and 79%

172 Conclusion Use of SPF >/= 15 rather than SPF < 15 sunscreens reduces melanoma risk.

173 was assessed by eye and objectively, and the SPF of each sunscreen was modeled with changes in solar

174 ys, the Hybrid Capture 2 assay (HC2) and the SPF(10) assay, for the detection of carcinogenic HPV.

175 TSLP also induced scratching behavior in the SPF NC/Tnd mice.

176 itated with the 7S and 11S proteins into the SPF by 0.9% PGA.

177 VA filter butyl methoxydibenzoylmethane, the SPF 15 sunscreen and the UVA filter together, and the lo

178 ripeptide anticodon PVT motif instead of the SPF motif of RF2, which confers the specificity towards

179 henicol acetyl transferase activity with the SPF 15 sunscreen, the UVA filter, and the combination SP

180 he factor-binding site of the ribosome, the 'SPF' loop of the protein is situated close to the mRNA,

181 d development of eczematous lesions in these SPF NC/Tnd mice, which normally do not suffer from AD.

182 In striking contrast to SPF IL-2(-/-) mice, germfree IL-2(-/-) mice do not devel

183 mice without colitis transferred disease to SPF Tg(epsilon26) recipients within 2-4 weeks.

184 /-) mice before transition from germ-free to SPF conditions reduced their development of colitis.

185 itosin as assessed by IHC may be superior to SPF as a prognostic factor in node-negative breast cance

186 Genotyping for 25 HPVs was performed using SPF(10)/LiPA(25).

187 In addition, we show that GF versus SPF mice have reduced intestinal absorption and increase

188 After 1 week, SPF and GF mice were given dextran sulfate sodium (DSS)

189 han under the laboratory conditions in which SPF is tested and assigned.

190 cantly decreased melanoma risk compared with SPF < 15 use (hazard ratio, 0.67; 95% CI, 0.53 to 0.83).

191 tigens in ConA-treated GF mice compared with SPF counterparts.

192 enteric lymph nodes of GF mice compared with SPF mice, as well as lower relative gene expression of F