ライフサイエンスコーパス: SPL

1 SPL also binds regulators of G protein signaling (RGS) p

2 SPL attributes are compared with field measurements and

3 SPL augmented the activation status of IKKepsilon and en

4 SPL expression in HEK293 cells potentiated apoptosis in

5 SPL genes are posttranscriptionally downregulated by miR

6 SPL is a member of the radical AdoMet superfamily of enz

7 SPL is a radical S-adenosyl-l-methionine (SAM) enzyme, w

8 SPL is a radical S-adenosylmethionine (SAM) enzyme, util

9 SPL utilizes a special [4Fe-4S] cluster to reductively c

10 SPL-KD cells accumulated intracellular and extracellular

11 SPL-KD cells successfully differentiated when treated wi

12 SPL-KD cells transfected with mimics for miR-1 or miR-20

13 SPL-mediated inhibition of virus-induced cell death was

14 SPL-overexpressing cells were partially protected agains

15 the enzyme, sphingosine-1-phosphate lyase 1 (SPL), has neuroprotective effects in Huntington's diseas

16 re was an 8kHz octave band of noise at 105dB SPL for 4h.

17 t it addresses fundamental limitations of 2D SPL by allowing one to compensate for unavoidable imperf

18 was comparable to that obtained with a 50dB SPL acoustic click.

19 sinusoidal acoustical excitation of 40-90dB SPL, in the frequency range from 100Hz to 10kHz.

20 minimal detectable sound level of 31-35dB(A) SPL for humans.

21 Accordingly, SPL-overexpressing cells were much more resistant than c

22 In addition, SPL expression and activity were down-regulated in adeno

23 apoptosis and provide an example of altered SPL expression in a human tumor.

24 Although SPL is robustly expressed in thymic epithelial cells (TE

25 Here, we evaluate data from an SPL instrument - the High Resolution Quantum Lidar Syste

26 thermore, the transcription factors BEL1 and SPL/NZZ, previously described as key regulators of ovule

27 5 mN/m, respectively, whereas PPL:dSP-C and SPL:dSP-C only reached minima of approximately 20 mN/m a

28 Dispersions of PPL:SP-C and SPL:SP-C rapidly adsorbed to high equilibrium surface pr

29 20 cycles/min), dispersions of PPL:SP-C and SPL:SP-C reached low minimum surface tensions of <1 and

30 vivo approaches (mouse insulinoma cells and SPL-deficient mice), that SPL is a potent negative regul

31 whereas parts of the left and right IPL and SPL are specialized for the processing of spatial attrib

32 Finally, memory-related HGP in left IPS and SPL was sufficiently reliable to enable brain-based deco

33 Comparisons of PAC and SPL showed a lack of spatiotemporal correlations.

34 nima of approximately 20 mN/m as did PPL and SPL alone.

35 f short-term synchronization between SCM and SPL are consistent with an abnormal corticoreticular and

36 The activity in the SCM and SPL was in phase in the patients but not in the controls

37 In addition, both MIR156 and SPLs are responsive to auxin signaling suggesting that m

38 ated using a small interfering RNA approach, SPL's anti-influenza viral activity was markedly suppres

39 We postulate an antagonistic effect between SPLs and the heterogeneous MYB-bHLH factors binding to T

40 rthermore, we examined whether the bilateral SPL regions play a causal role in the rate of perceptual

41 Constitutive ablation of SPL in the brain (SPL(fl/fl/Nes)) but not postnatal neuronal forebrain-res

42 This was due to reciprocal modulation by SPL and NRB of the potency of RGS2 to inhibit Ca(2+) sig

43 rnocleidomastoid (SCM) and splenius capitis (SPL) muscles of eight patients with rotational torticoll

44 ipids called sphingadienes increased colonic SPL levels and reduced S1P levels, STAT3 signaling, cyto

45 Under differentiation conditions, SPL-KD cells also demonstrated delayed induction of 3 my

46 By contract, SPL, but not NRB, binds the 3iL of the GPCRs alpha(1B)-a

47 se octave band of noise (8-16 kHz) at 100 dB SPL for 2 hr, either with or without previous whole-body

48 , on the high sound pressure levels (>100 dB SPL) necessary to produce them.

49 focused on high-intensity exposures (>100 dB SPL).

50 pigs were exposed to broadband noise (102 dB SPL, 3 h/day, 5 days) while receiving daily injections o

51 ense sound exposure (broadband noise, 105 dB SPL, 5 h) on GSH and cysteine levels in the guinea pig c

52 ol and tone-exposed hamsters (10 kHz, 115 dB SPL, 4h) before and after application of carbachol to th

53 he noise exposure (4-kHz octave band, 115 dB SPL, 5 h) created permanent threshold shifts at frequenc

54 exposed to noise (4 kHz octave band, 115 dB SPL, 5 h).

55 cleus neurons are typically above 100-120 dB SPL (sound pressure level re 20 microPa).

56 d creatine plus tempol and exposed to 120 dB SPL one-octave band noise centered at 4 kHz for 5 h.

57 exposure (narrow band noise, 12 kHz, 120 dB SPL, 1 hour) on the physiological response of the inferi

58 ea pig following noise exposure (5 h, 120 dB SPL, 1 OCB).

59 nds centered on 2, 8, or 32 kHz at 80-120 dB SPL.

60 l distortion generated by the 110- to 120-dB SPL produced at the open ear with fortissimo playing; (c

61 Results indicate that a precursor (>20 dB SPL) induced efferent activation, resulting in a decreas

62 meteors can generate audible sound at 25 dB SPL.

63 tinuous noise with an overall level of 33 dB SPL.

64 nd 10-kHz tone bursts (dexamethasone: 8.4-dB SPL vs. placebo: 53.4-dB SPL, P=.0003).

65 xamethasone: 8.4-dB SPL vs. placebo: 53.4-dB SPL, P=.0003).

66 nd pressure level [SPL] vs. placebo: 33.4-dB SPL, P=.0078) and 10-kHz tone bursts (dexamethasone: 8.4

67 n increased gap detection threshold at 50 dB SPL, but not at 60 or 80 dB SPL.

68 75th percentile was associated with a 1.6-dB SPL (sound pressure level) decrease in DPOAE amplitude (

69 ental frequency (f0) were presented at 60 dB SPL to the ear contralateral to the hemisphere from whic

70 e LA making it hyperactive to sounds>/=60 dB SPL.

71 evel coding by the AN population above 60 dB SPL.

72 espond to low-frequency sounds (80 Hz; 65 dB SPL) by freezing-a common anti-predatory behavior charac

73 requency tones (80 Hz at approximately 65 dB SPL)-recordings that also represent the first record of

74 SRTs were measured for 65-dB SPL sentences presented in speech-weighted noise or four

75 Hz with a fixed probe level of either 70 dB SPL or 8 dB SL (whichever was greater) and probe duratio

76 ion (PPI) to tone prepulses (4-24 kHz, 70 dB SPL) was stronger, and baseline acoustic startle respons

77 t elevated sound-evoked thresholds (60-70 dB SPL).

78 for input sound levels between 50 and 70 dB SPL.

79 s exhibiting hearing loss in excess of 70 dB SPL.

80 SSwap had WBN sound levels from 40 to 78 dB SPL, and separations of 22.5, 45, 90, and 180 degrees :

81 rains; 10-50 Hz in increments of 5 Hz; 80 dB SPL) in carefully screened cannabis users and controls.

82 Tone bursts (80 dB SPL) were binaurally presented via insert earphones in t

83 For a high-input level (80 dB SPL), slow compression was preferred over fast compressi

84 otal OHC power output is about 2 pW at 80 dB SPL, with a maximum of about 10 fW per OHC.

85 reshold at 50 dB SPL, but not at 60 or 80 dB SPL.

86 long exposure to moderate-level noise (84 dB SPL) in mice with varying degrees of cochlear de-efferen

87 imuli at low sound pressure levels (</=84 dB SPL), revealing a previously unrecognised consequence of

88 tave density, 4-Hz rate) applied to an 85-dB SPL, 2-kHz lowpass-filtered pink-noise carrier.

89 lt mice were exposed (8-16 kHz, 100 or 91 dB SPL for 2 h) and then evaluated from 1 h to approximatel

90 ignificant (tuned to 1.5 and 4 kHz; 60-98 dB SPL), and capable of mediating behavioral responses of p

91 a 3 hour 4 kHz octave band noise at 117 dB (SPL).

92 ity blocks of 65, 72.5, 80, 87.5, and 95 dB (SPL).

93 (TECs), in this study, we show that deleting SPL in CD11c(+) dendritic cells (DCs), rather than TECs

94 neuronal forebrain-restricted SPL deletion (SPL(fl/fl/CaMK)) caused marked accumulation of S1P.

95 Dense SPL-IR areas included the periaqueductal grey, trigemina

96 sing the expression of functionally distinct SPL transcription factors.

97 d genes in the enhancer of split complex (E-(SPL)-C).

98 Endogenous SPL expression was induced by DNA damage in WT cells, wh

99 ther, these results indicate that endogenous SPL may play a physiological role in stress-induced apop

100 the potential roles of 2 putative endogenous SPLs, an ouabain-like compound (OLC) and an alpha(1) Na(

101 that of NH listeners when compared at equal SPL (sound pressure level).

102 Exogenous SPL expression inhibited influenza A virus replication,

103 his indicates that IKKepsilon is crucial for SPL-mediated inhibition of influenza virus replication.

104 ed in effects opposite to those observed for SPL overexpression.

105 In this study, we show a crucial role for SPL in mediating cellular responses to stress.

106 hat 5R-SP, but not 5S-SP, is a substrate for SPL is consistent with the expectation that 5R is the SP

107 Here, we report a cantilever-free SPL architecture that can generate 100 nanometer-scale m

108 that canopy and ground characteristics from SPL are similar to discrete return lidar despite differe

109 t canopy structure and ground elevation from SPL point clouds.

110 ty were observed in hippocampal neurons from SPL(fl/fl/Nes) mice.

111 Further, SPL expression led to constitutive activation of p38.

112 hrough activation of the SPOROCYTELESS gene (SPL, also known as NOZZLE,NZZ), a regulator of sporogene

113 Spleens of hu-SPL-SCID mice with Ag-specific titers < or = 1:1 x 10(6)

114 tumors removed from spleens of five such hu-SPL-SCID mice, produced Abs that were specific for the i

115 Specific human IgG titers in the hu-SPL-SCID mice reached approximately 1:4 x 10(5) when the

116 d V6/V6A as functional counterparts of human SPL because they contained the most widespread shift sig

117 the functional characteristics of the human SPL shifting area.

118 ate whether a potential homolog of the human SPL shifting region exists in monkeys (Macaca mulatta),

119 ation of LpSpl and its comparison with human SPL reveals high structural conservation, thus supportin

120 result questions the currently hypothesized SPL mechanism which excludes the involvement of protein

121 Moreover, the identified SPL activity defines a distinct pathway in control of th

122 Substance P-like immunoreactivity (SPL-IR) was semiquantitatively mapped, and correlated ve

123 Importantly, SPL expression was significantly down-regulated in human

124 ective movement coincided with activation in SPL.

125 nd functional assays in model systems and in SPL(-/-) and NRB(-/-) mice to show that SPL and NRB reci

126 Protein radicals are known to be involved in SPL catalysis; however, how these radicals are quenched

127 platelet function we previously observed in SPL(-/-) mice, these data show that (1) regulated seques

128 Here we review the current progress in SPL mechanistic studies.

129 des the involvement of protein residue(s) in SPL reaction, suggesting that some protein residue(s), w

130 ranscription factor (TF) families, including SPLs, MYBs, ERFs and bZIPs, might regulate corresponding

131 , we show that one consequence of inhibiting SPL is intracellular inhibition of histone deacetylases,

132 ith control animals, mice lacking intestinal SPL exhibited greater disease activity, colon shortening

133 To investigate SPL's role in myogenesis at the cellular level, we gener

134 hat a member of miR156 family and one of its SPL target genes have inverse expression levels, which i

135 aracterized a murine myoblast SPL-knockdown (SPL-KD) cell line lacking SPL.

136 ast SPL-knockdown (SPL-KD) cell line lacking SPL.

137 By contrast, HGP in left SPL increased for CRs early after stimulus onset (200-30

138 eriod between the more abstract role of left SPL in activating the appropriate S-R associations and t

139 g response to a 100-dB sound pressure level (SPL) acoustic stimulus and maintains its response to sub

140 ed as functions of the sound pressure level (SPL) and duration of 2-kHz tone bursts.

141 thresholds of 70-80 dB sound pressure level (SPL).

142 c input power at 10 dB sound pressure level (SPL).

143 of approximately 65 dB sound pressure level (SPL).

144 (dexamethasone: 6.7-dB sound pressure level [SPL] vs. placebo: 33.4-dB SPL, P=.0078) and 10-kHz tone

145 ver a range of masker sound pressure levels (SPLs) and frequencies.

146 By regulating S1P levels, SPL also controls SC recruitment and muscle regeneration

147 Single photon lidar (SPL) is an innovative technology for rapid forest struct

148 Digitalis-like sodium pump ligands (SPLs) effect natriuresis via inhibition of renal tubular

149 xpression of SQUAMOSA PROMOTER BINDING-LIKE (SPL) family members, SPL3, SPL5, and SPL9, is upregulate

150 ssion of the SQUAMOSA promoter-binding-like (SPL) transcription factors.

151 s of SQUAMOSA PROMOTER BINDING PROTEIN LIKE (SPL) genes showed that wall ingrowth deposition was incr

152 n of SQUAMOSA PROMOTER BINDING PROTEIN LIKE (SPL), NAC, YUCCA and AGAMOUS-LIKE genes associated with

153 the SQUAMOSA PROMOTER BINDING PROTEIN-LIKE (SPL) gene family appear universally conserved in land pl

154 the SQUAMOSA PROMOTER BINDING PROTEIN-LIKE (SPL) gene family.

155 eted SQUAMOSA PROMOTER BINDING PROTEIN-LIKE (SPL) genes, which are deeply conserved and known to have

156 gets SQUAMOSA PROMOTER BINDING PROTEIN-LIKE (SPL) genes: SPL3, SPL9 and SPL10 are involved in the rep

157 n of SQUAMOSA PROMOTER-BINDING PROTEIN-LIKE (SPL) transcription factors renders Arabidopsis plants in

158 even SQUAMOSA-PROMOTER BINDING PROTEIN-LIKE (SPL) transcription factors, including SPL13, are targete

159 Scanning probe lithography (SPL) is a promising candidate approach for desktop nanof

160 Activity in the superior parietal lobe (SPL) increased proportionally to viewpoint disparity dur

161 tivation in the left superior parietal lobe (SPL).

162 ucture of bilateral superior parietal lobes (SPL) could account for interindividual variability in pe

163 Superior and inferior parietal lobule (SPL and IPL) possessed both types of structure.

164 eye fields (FEF), superior parietal lobule (SPL) and intraparietal sulcus (IPS).

165 es (IPL), the left superior parietal lobule (SPL) and the right precuneus-SPL, which were all more ac

166 cus (IPS) and left superior parietal lobule (SPL) differing in time and sign for recognized old items

167 l sulcus (aIPS) or superior parietal lobule (SPL) disrupts on-line adaptive adjustments of grasp when

168 itical role of the superior parietal lobule (SPL) in shifting spatial attention, a finding not predic

169 eye fields (FEF), superior parietal lobule (SPL), and right supramarginal gyri (SMG).

170 e field (FEF), the superior parietal lobule (SPL), and the right ventrolateral prefrontal cortex (VLP

171 e spatial topography of spike-phase locking (SPL) was similar to that of PAC.

172 Spore photoproduct lyase (SPL) repairs 5-thyminyl-5,6-dihydrothymine, a thymine di

173 Spore photoproduct lyase (SPL) repairs a covalent UV-induced thymine dimer, spore

174 onine) enzyme, the spore photoproduct lyase (SPL), at the bacterial early germination phase.

175 adical SAM enzyme, spore photoproduct lyase (SPL), at the early germination phase.

176 mage by the enzyme spore photoproduct lyase (SPL).

177 S1P lyase (SPL) catalyzes the irreversible degradation of sphingosi

178 Overexpression of S1P lyase (SPL), which induces the degradation of S1P, interfered w

179 l S1P levels are kept low through S1P lyase (SPL)-mediated metabolism.

180 reversibly degraded by the enzyme S1P lyase (SPL).

181 ating the S1P-metabolizing enzyme S1P lyase (SPL).

182 Sphingosine-1-phosphate (S1P) lyase (SPL) irreversibly degrades the bioactive sphingolipid S1

183 Sphingosine 1-phosphate (S1P) lyase (SPL) is an intracellular enzyme that mediates the irreve

184 ic knockout mouse models in which S1P-lyase (SPL), the enzyme responsible for irreversible S1P cleava

185 Altogether, the miR156-SPL module mediates the response to recurring HS in Arab

186 lowers, and we found that miR172-AP2, miR156-SPLs were critical regulatory nodes contributing to the

187 ve to auxin signaling suggesting that miR156/SPL modules might be involved in the proper timing of th

188 Despite the important roles of the miR156/SPL network, our understanding of its downstream genes t

189 ely, these results unravel a role for miR156/SPLs modules in lateral root development in Arabidopsis.

190 enerated and characterized a murine myoblast SPL-knockdown (SPL-KD) cell line lacking SPL.

191 C141 can be readily alkylated in the native SPL by an iodoacetamide treatment, suggesting that it is

192 l organ-building" gene SPOROCYTELESS/NOZZLE (SPL/NZZ) plays a central role in regulating anther cell

193 Additional grafting of NP SPL with NP THY improves the efficiency of tolerance ind

194 leen (NP SPL), but not in those receiving NP SPL alone, indicating that pig thymus grafting was neces

195 THY with or without neonatal pig spleen (NP SPL), but not in those receiving NP SPL alone, indicatin

196 In this study, we examined the ability of SPL to modulate the activity of beta-cell M(3) muscarini

197 Constitutive ablation of SPL in the brain (SPL(fl/fl/Nes)) but not postnatal neur

198 romote S1P metabolism through the actions of SPL.

199 rom that of PPL alone, and the adsorption of SPL:dSP-C was improved only slightly over SPL alone.

200 Coexpression of SPL or NRB with the alpha(1B)AR in Xenopus oocytes revea

201 Accordingly, deletion of SPL in mice enhanced binding of RGS2 to NRB and Ca(2+) s

202 ivates SHP-1 and causes dephosphorylation of SPL tyrosine residues, PGI2 and forskolin cause phosphor

203 , this suggests that DYT1 acts downstream of SPL/NZZ and EMS1/EXS.

204 t the concept that the inhibitory effects of SPL on M3R activity are mediated by RGS4.

205 hb2 partially reversed beneficial effects of SPL overexpression.

206 Arabidopsis, and decreased the expression of SPL genes regulated by miR156.

207 study, we report that ectopic expression of SPL/NZZ not only affects flower development in the wild-

208 ves and flowers, while ectopic expression of SPL/NZZ resulted in ectopic expression of AG and SEP3 in

209 ver, the S1P degradation-incompetent form of SPL also enhanced IFN responses, suggesting that SPL's p

210 aled that SPL, as well as the mutant form of SPL, interacts with IKKepsilon.

211 Importantly, influenza virus infection of SPL-overexpressing cells induced rapid activation of ext

212 In contrast, the lack of SPL expression led to an elevated cellular susceptibilit

213 Overexpression of SPL protected against cytokine toxicity.

214 re, ectopic expression and overexpression of SPL/NZZ altered expression of AG, SEP3, and AP2 in roset

215 Clinically, the pattern of SPL EMG autospectra and of SCM-SPL coherence may provide

216 PGI2 and forskolin cause phosphorylation of SPL Ser94 without reducing tyrosine phosphorylation.

217 ets, and (2) differential phosphorylation of SPL tyrosine and serine residues provides a key to under

218 telets where constitutive phosphorylation of SPL(Y398) creates an atypical binding site for SHP-1.

219 uthors show that age dependent regulation of SPL transcription factors by miR156 influence flowering

220 However, little is known about the role of SPL expressed in peripheral cell types including pancrea

221 Here, we investigated the role of SPL in colitis-associated cancer (CAC).

222 e a direct relationship between structure of SPL and individuals' perceptual switch rate.

223 In contrast to that of SPL, the overexpression of S1P-producing sphingosine kin

224 ms of SP-C versus dSP-C combined with PPL or SPL.

225 of SPL:dSP-C was improved only slightly over SPL alone.

226 have previously reported that overexpressed SPL displays anti-influenza viral activity; however, the

227 e exposed to an impulse noise at 155 dB peak SPL.

228 arietal lobule (SPL) and the right precuneus-SPL, which were all more activated during localization c

229 Fifteen genes in the E. pusilla SPL (EpSPL) family were identified, nine of which contai

230 acts together with other microRNA-regulated SPL transcription factors to control the timing of flowe

231 In contrast with related SPL techniques, this approach affords a direct-write lit

232 ependent of ceramide generation but required SPL enzymatic activity and the actions of p38 MAP kinase

233 not postnatal neuronal forebrain-restricted SPL deletion (SPL(fl/fl/CaMK)) caused marked accumulatio

234 hus, we show for the first time that the S1P/SPL/S1P-receptor axis regulates the expression of a numb

235 he pattern of SPL EMG autospectra and of SCM-SPL coherence may provide a sensitive and specific featu

236 In fibroblasts, silencing SPL promoted tumorigenic transformation through a pathwa

237 rescued the egress phenotype of DC-specific SPL knockout mice.

238 Spinophilin (SPL) and neurabin (NRB) are structurally similar scaffol

239 Spinophilin (SPL), a multidomain scaffolding protein known to modulat

240 omprising the scaffold protein, spinophilin (SPL), and the tyrosine phosphatase, SHP-1, and show that

241 s formed by a scaffold protein, spinophilin (SPL), the tyrosine phosphatase, Src homology region 2 do

242 ernal forces exerted by the splanchnopleure (SPL) and the omphalomesenteric veins (OVs) drive rotatio

243 nzyme catalysis (employing Bacillus subtilis SPL) revealed that it is the 6-H(proR) atom of SP that i

244 One [Y99(Bs) in Bacillus subtilis SPL] is downstream of the cysteine, suggesting that SPL

245 Thus, association of each target SPL gene to a trait or set of traits is essential for de

246 miR156 targets SPL transcription factor genes that are master regulator

247 Furthermore, we demonstrate that SPL can induce microsporogenesis in the absence of AG fu

248 In this study, we demonstrate that SPL functions as a positive regulator of IKKepsilon to p

249 lly defined SP substrates demonstrating that SPL specifically repairs only the 5R isomer of SP.

250 We found that SPL is induced during myoblast differentiation.

251 Our results indicate that SPL is the first member of the radical SAM superfamily (

252 e levels, and tumorigenesis, indicating that SPL prevents transformation and carcinogenesis.

253 us replication in the cells, indicating that SPL protects cells from influenza virus via the activati

254 sulinoma cells and SPL-deficient mice), that SPL is a potent negative regulator of M3R-mediated signa

255 Our previous studies proved that SPL utilizes the 5'-dA* generated by the SAM cleavage re

256 alpha(1B)AR in Xenopus oocytes revealed that SPL reduces, whereas NRB increases, the intensity of Ca(

257 Biochemical analyses revealed that SPL, as well as the mutant form of SPL, interacts with I

258 d in SPL(-/-) and NRB(-/-) mice to show that SPL and NRB reciprocally regulate Ca(2+) signaling by GP

259 We also show that SPL/RGS/SHP1 complexes are present in resting platelets

260 These data suggest that SPL or other G-protein-coupled receptor-associated prote

261 e energy transfer technology, suggested that SPL is able to recruit regulator of G-protein signaling

262 downstream of the cysteine, suggesting that SPL uses a novel hydrogen atom transfer (HAT) pathway wi

263 also enhanced IFN responses, suggesting that SPL's pro-IFN function is independent of S1P.

264 r large areas quickly strongly suggests that SPL should be considered as an efficient and potentially

265 The SPL and right VLPFC showed heightened activity only duri

266 SP repair by the SPL C141A mutant yields TpTSO(2)(-) and TpT simultaneous

267 Interestingly, the SPL overexpression did not suppress the cytokine-induced

268 Furthermore, the right VLPFC but not the SPL showed the greatest activation during the nogo decis

269 periods but the higher point density of the SPL data provides more structural detail.

270 significant peak in the autospectrum of the SPL EMG at 10-12 Hz, which was absent in all patients wi

271 xpression of miR156-regulated members of the SPL family of transcription factors and provide evidence

272 dy and analyzed in part with the help of the SPL functions obtained in the present study.

273 llustrate the functional significance of the SPL-IKKepsilon-IFN axis during host innate immunity agai

274 er94 blocks cAMP-induced dissociation of the SPL/RGS/SHP-1 complex.

275 Thus, we propose that the role of the SPL/RGS/SHP1 complex in platelets is time and context de

276 riggering dephosphorylation and decay of the SPL/RGS/SHP1 complex.

277 tionally closer to motor processing than the SPL and the right VLPFC.

278 Our results indicate that the SPL/NZZ gene is engaged in controlling stamen identity v

279 ized sources, first in the aIPS and then the SPL.

280 collis had evidence of a 4-7 Hz drive to the SPL and SCM that was absent in coherence spectra from co

281 Hence, the SPLs potentially rearrange the complex, attenuating its

282 ncreased potency of forward maskers as their SPL was increased, despite the fact that the excitatory

283 In support of this, SPL-deficient cells were defective in mounting an effect

284 Thus, SPL and NRB bind all members of the R4 subfamily of RGS

285 Seven of nine NP THY/SPL-grafted ATX mice and two of six NP THY-grafted ATX m

286 se in reaction time when rTMS was applied to SPL.

287 deletion of NRB enhanced binding of RGS2 to SPL and reduced Ca(2+) signaling by alphaAR.

288 Moreover, cytokine-treated SPL-overexpressing cells exhibited increased expression

289 es supporting the conserved function of TTG1-SPL complex.

290 hereby illuminating the significance of TTG1-SPLs interactions in trichome formation control.

291 s induced by DNA damage in WT cells, whereas SPL knockdown diminished apoptotic responses.

292 Here, we asked whether SPL and other RGS18 binding proteins such as 14-3-3gamma

293 n of GPCR-mediated Ca(2+) signaling in which SPL/NRB forms a functional pair of opposing regulators t

294 arkedly by dSP-C in corresponding films with SPL or DPPC and not at all in films with PPL.

295 ory with a time constant that increases with SPL from approximately 200 to 370 msec.

296 HC power output increases and saturates with SPL.

297 tively mapped, and correlated very well with SPL-IR observed in other species.

298 PS and subsequent on-line adjustments within SPL.

299 rast to the >5 turnovers exhibited by the WT SPL reaction, suggesting that the enzyme catalytic cycle

300 ax) KIE of 2.8 +/- 0.3 determined for the WT SPL reaction.

301 tom transfer mechanism in the wild-type (WT) SPL reaction.

302 stage, miR156-mediated repression of zygotic SPL transcripts prevents premature accumulation of trans