ライフサイエンスコーパス: SPM

1 SPM and LPM differ in their ability to phagocytose apopt

2 SPM is applicable for analyses of longitudinal data in m

3 SPM risk was quantified by using standardized incidence

4 SPM using a statistical threshold of P less than 0.001 p

5 SPMs and miR-466l regulated transcription factors activa

6 SPMs are also decreased in advanced plaques of fat-fed L

7 llo-beta-lactamases (including NDM-1, GIM-1, SPM-1, IMP-1, -2, -7, -8, -18, and -27, and VIM-1, -2, a

8 s also capable of sequestering an additional SPM(4+) at the cytoplasmic entrance near a cluster of ne

9 Although SPM(4+) remains inside the CD of ROMK, it diffuses aroun

10 few studies have compared survival after an SPM to survival of the same cancer occurring as first pr

11 Overall, 5-year survival after an SPM was 33.1% lower for children, 20.2% lower for AYAs,

12 ing humoral immunity, potentially through an SPM-mediated mechanism.

13 s blocked by 15-lipoxygenase inhibition, and SPM stimulated phagocytosis was diminished by HO-1 inhib

14 re interrogated using region of interest and SPM (statistical parametric mapping) analysis.

15 ittle difference in fatality between MPM and SPM (HR for MPM relative to SPM, 1.24 [95% CI, 0.91-1.69

16 y) as well as the affinity of TiO(2) NPs and SPM, characterized by the attachment efficiency, alpha(h

17 nt decline in brain metabolism, both ROI and SPM-based methods revealed significant changes; SPM, how

18 er than those of Sigmaparabens in sludge and SPM.

19 Meanwhile, the polyamines SPD and SPM were found in all soya milks, being stable to the UH

20 hat impaired biosynthesis of certain SPM and SPM precursors, including 17-HDHA and PD1, contributes t

21 cross-talk involving gammadelta T cells and SPMs that promotes tumor cell growth and thus counteract

22 66l overexpression increased prostanoids and SPMs (e.g., resolvin D1 [RvD1] and RvD5), which enhanced

23 aspirin to n-3 fatty acids did not alter any SPMs in either group.

24 Augmented SPMs were continued.

25 tent with consumption of naturally available SPM as opposed to values in oil (-27 +/- 0.2 per thousan

26 Baseline SPMs did not differ between groups.

27 bridge for the inorganic adsorption between SPM-MWCNTs and sediment.

28 e activating Mps1 and Cdk1 sites lie between SPM and CM1 motifs.

29 Most organisms have both SPM-CM1 (Spc110/Pcp1/PCNT) and CM1-only (Spc72/Mto1/Cnn/

30 at endogenous LPM inhibit differentiation by SPM.

31 an macrophage, which was further enhanced by SPM ( approximately 100%).

32 Survival by SPM status was significantly worse in younger vs older p

33 ine was PUT, followed by SPD, HIS, TYR, CAD, SPM, PHE, and SER, with the total BAs content decreasing

34 We conclude that highly phagocytic, CD138(+) SPM-like cells with an anti-inflammatory phenotype may p

35 dicate that impaired biosynthesis of certain SPM and SPM precursors, including 17-HDHA and PD1, contr

36 -based methods revealed significant changes; SPM, however, was generally more sensitive and region-sp

37 First, characteristic SPM images of nanoparticles are collected in aqueous sus

38 usion (I/R) injury, to increased circulating SPMs and decreased remote organ inflammation.

39 test is a bayesian analogue of the classical SPM-F and allows users to implement model comparison in

40 For the most common SPMs in AYAs, the absolute difference in 5-year survival

41 pro-inflammatory mediators and compromising SPM production that contribute to failed catabasis and h

42 In steady-state conditions, SPM are sustained by circulating precursors, whereas LPM

43 s administration of MaR1, a highly conserved SPM, propagated inflammatory resolution after SCI, as re

44 ntral network, identified in a conventional (SPM) whole-brain analysis, was used to compare different

45 (PMT), the results revealed that the cooled SPM exhibits a wider linear response to inducible substr

46 In ligated carotid arteries at 4 d, SPM treatment was associated with reduced cell prolifera

47 essed obesity-induced changes of n-3-derived SPMs in adipose tissue and the effects of dietary EPA/DH

48 on the biological effects of omega-3-derived SPMs on the humoral immune response.

49 that systemic administration of DHA-derived SPMs [resolvin D2 (RvD2) and maresin 1 (MaR1)] would inf

50 colony forming units, c.f.u.), the dominant SPMs identified were resolvin (Rv) D5 and protectin D1 (

51 s in SPM differentiation into LPM, yet donor SPM do not generate LPM after transfer into C/EBPbeta-su

52 Each n-3 DPA-SPM displayed protective actions from second organ injur

53 With human leukocytes each n-3 DPA-SPM reduced neutrophil chemotaxis, adhesion and enhanced

54 The n-3 DPA-SPM, including RvD1(n-3 DPA) and MaR1(n-3 DPA), each exe

55 omes observed in longitudinal studies, i.e., SPM relates the stochastic dynamics of variables (e.g.,

56 t not overweight was associated with earlier SPM emergence.

57 a platform for developing more elaborate EC-SPM setups.

58 ray scan patterns, only found on high-end EC-SPM setups such as hopping mode scan, were easily implem

59 lectrochemical-scanning probe microscope (EC-SPM) is presented.

60 f" parts to deliver a simple yet powerful EC-SPM equipment capable of performing simple space-resolve

61 ersus 59 +/- 24 pg/mouse, 6 h), and elevated SPM including resolvin (Rv) D1 (27 +/- 4 versus 16 +/- 5

62 we found a temporal regulation of endogenous SPMs during self-resolving inflammatory arthritis.

63 both ECs and VSMCs, potentially facilitating SPM biosynthesis.

64 sciences to the study of natural flocculated SPM filling both this spatial and dimensional gap.

65 0(5), 4.17 x 10(5) and 1.46 x 10(5)M(-1) for SPM, SPD and PUT, respectively, which correlated well wi

66 ar combination fitting of the EXAFS data for SPM samples, using a large set of EXAFS spectra of Zn mo

67 The present study provides new evidence for SPM activity in the humoral response.

68 laques and offer a mechanistic rationale for SPM therapy to promote plaque stability.

69 These results demonstrate pivotal roles for SPMs and miR-466l in dynamic leukocyte plasticity during

70 between pediatric overweight/obesity and FPM/SPM emergence, adjusting for age, sex, and race/ethnicit

71 t US-representative data and affirm that FPM/SPM emergence varies by race/ethnicity and sex and is po

72 ssociation of body mass index (BMI) with FPM/SPM emergence in a representative sample of US children

73 or paracrine signaling via locally generated SPMs in the vasculature may represent a novel homeostati

74 s suggested a lower incidence of hematologic SPMs in the thalidomide maintenance arm (hazard ratio =

75 we recorded significantly (p < 0.05) higher SPM in the Ajkwa Estuary compared to neighboring estuari

76 dings contribute to our understanding of how SPM synthesis is regulated during the inflammatory respo

77 How SPMs regulate early LPS signaling, including activation

78 However, how SPMs are regulated during the inflammatory response, and

79 ased metabololipidomics was used to identify SPMs from n-3 polyunsaturated fatty acids in human IBD c

80 NDM-1 (New Delhi MBL), IMP-1 (Imipenemase), SPM-1 (Sao Paulo MBL), and VIM-2 (Verona integron-encode

81 ls, we tested whether n-3 fatty acids impact SPM profiles in patients with CAD and promote clot remod

82 Importantly, SPMs lack the immunosuppressive properties of classical

83 Importantly, SPMs were uniquely and directly capable of promoting ova

84 This improved SPM and labeling protocol is an important step toward a

85 contributions to the EXAFS were analyzed in SPM samples, dried and stored under a dry nitrogen atmos

86 ate of ASWs, including the fraction found in SPM and in sludge, in addition to the aqueous portion of

87 The development of hidden image in SPM is achieved by referencing of images obtained in the

88 We report the archaeal lipidome in SPM from diverse oceanic regimes.

89 nsfer of SPM into Cebpb(-/-) mice results in SPM differentiation into LPM, yet donor SPM do not gener

90 rivers, we have determined Zn speciation in SPM as a function of both the seasonal water flow variat

91 ic acid accelerated resolution via increased SPMs and promoted human monocyte reprogramming.

92 In 10(7) c.f.u. E. coli infections, SPMs (RvD1, RvD5, PD1) together with ciprofloxacin also

93 In skin infections, SPMs enhanced vancomycin clearance of Staphylococcus aur

94 Aspartame was found in 92% of influent SPM samples at a mean concentration of 444 ng/g dw, foll

95 he groundwork for further investigation into SPMs as alternatives to immunosuppressive therapies like

96 I, 0.23-3.66), respectively; IRs of invasive SPMs were 1.71 (95% CI, 0.86-3.43) and 0.91 (95% CI, 0.2

97 SCML2A binds to PRC1 via its SPM domain and interacts with ncRNAs through a novel RNA

98 vD5, an action reversed by blockage of a key SPM biosynthesis enzyme 15-lipoxygenase type 1.

99 se in the cytoplasmic:nuclear ratio of a key SPM biosynthetic enzyme, 5-lipoxygenase.

100 sion and subcellular localization of the key SPM biosynthetic enzyme 5-lipoxygenase (LOX) in vascular

101 s, MerTK cleavage during inflammation limits SPM biosynthesis and the resolution response.

102 soft tissues may result from dysregulated LM-SPM production, and that inhibition of 15-PGDH activity

103 increased AT-RvD3, RvD6, AT-PD1, and AT-LXB4 SPMs identified in Lovaza-treated patients with CAD enha

104 ice: small and large peritoneal macrophages (SPM and LPM, respectively).

105 unconventional small peritoneal macrophages (SPMs) that, in comparison with large peritoneal macropha

106 With human macrophages, SPM (10 pM-10 nM) elevated heme oxygenase (HO)-1 ( appro

107 Second primary malignancies (SPM) were observed in 8 patients (16%).

108 tient-years) of second primary malignancies (SPMs) was 3.62.

109 development of second primary malignancies (SPMs) was conducted, and no increase in incidence was no

110 er incidence of second primary malignancies (SPMs), including both hematologic and solid malignancies

111 We used statistical parametric mapping (SPM V.12) software to compare groups and investigate vox

112 (VBA) using statistical parametric mapping (SPM) for comparison of USPIO-PEG-Abeta1-42 injected AD t

113 Statistical Parametric Mapping (SPM) is the dominant paradigm for mass-univariate analys

114 formed using statistical parametric mapping (SPM).

115 thing during statistical parametric mapping (SPM).

116 l to ratios in suspended particulate matter (SPM) and in fresh and weathered oil.

117 aeal lipids in suspended particulate matter (SPM) and sediments mainly focused on a small class of fu

118 d primarily to suspended particulate matter (SPM) at sites in areas with a high land-cover fraction o

119 tration in the suspended particulate matter (SPM) can reach 2000 mg/kg in the most urbanized areas.

120 s in estuarial suspended particulate matter (SPM) concentration.

121 speciation in suspended particulate matter (SPM) in the oxic water column of the Seine River from up

122 Suspended particulate matter (SPM) is present in the natural aquatic environment as lo

123 TMPP sorbed to suspended particulate matter (SPM) was 56.4% of the total mass in wastewater, which wa

124 2-month period suspended particulate matter (SPM) was collected from the lake and analyzed using a co

125 inal effluent, suspended particulate matter (SPM), and sludge collected from two WWTPs (denoted as WW

126 ent, effluent, suspended particulate matter (SPM), and sludge collected from two WWTPs in the Albany

127 in wastewater, suspended particulate matter (SPM), and sludge from two WWTPs in the Albany area in Ne

128 TiO(2) NPs and suspended particulate matter (SPM), is analyzed in more detail.

129 ssolved phase, suspended particulate matter (SPM), wetland sediments, and vegetation.

130 to study the dynamics of the Sao Paulo MBL (SPM-1) from beta-lactam-resistant Pseudomonas aeruginosa

131 Augmented standard preventive measures (SPMs) were not able to control this outbreak.

132 mmon PBS pulse media or sucrose pulse media (SPM).

133 conserved specialized proresolving mediator (SPM) hosting potent anti-inflammatory and proresolving p

134 al of the specialized proresolving mediator (SPM) resolvin D1 (RvD1) to actively promote the resoluti

135 ncreased specialized pro-resolving mediator (SPM) biosynthesis, including Resolvin D1 (RvD1), RvD2, a

136 lly regulates local levels of lipid mediator/SPM.

137 y specialized pro-resolving lipid mediators (SPM) in histologically-defined stable and vulnerable reg

138 by specialized proresolving lipid mediators (SPM) that limit the host response within the affected ti

139 These specialized proresolving mediators (SPM) constitute separate families that include lipoxins,

140 ecently, specialized proresolving mediators (SPM) have been found to exert profound beneficial effect

141 genus of specialized proresolving mediators (SPM) includes essential fatty acid-derived lipoxins, res

142 hesis of specialized proresolving mediators (SPM).

143 termed specialized pro-resolving mediators (SPM).

144 am specialized proresolving lipid mediators (SPMs) 14-hydroxydocosahexaenoic acid, 17-hydroxydocosahe

145 Specialized proresolving lipid mediators (SPMs) are endogenous autacoids that actively promote res

146 of specialized proresolving lipid mediators (SPMs) derived from eicosapentaenoic acid (EPA) and docos

147 es specialized proresolving lipid mediators (SPMs) derived from n-3 (omega-3) fatty acids.

148 Specialized proresolving lipid mediators (SPMs) derived from omega-3 polyunsaturated fatty acids [

149 at specialized proresolving lipid mediators (SPMs) generated from docosahexaenoic acid (DHA) can modu

150 of specialized proresolving lipid mediators (SPMs), generation of specific growth factors contributin

151 al specialized proresolving lipid mediators (SPMs), such as resolvins and protectins, which derive fr

152 by specialized proresolving lipid mediators (SPMs).

153 se specialized proresolving lipid mediators (SPMs; lipoxins, resolvins, and protectins) stimulate res

154 -derived specialized proresolving mediators (SPMs) and discuss their place within what is currently u

155 Specialized proresolving mediators (SPMs) are powerful endogenous bioactive regulators of in

156 Special proresolving mediators (SPMs) limit the intensity of inflammation; however, thei

157 -derived specialized proresolving mediators (SPMs) play a critical role in the active resolution of i

158 dogenous specialized proresolving mediators (SPMs) that actively stimulate resolution of inflammation

159 Specialized proresolving mediators (SPMs), identified in recent years, are endogenous mediat

160 Specialized proresolving mediators (SPMs), including resolvin D2 (RvD2), promote the active

161 s called specialized proresolving mediators (SPMs).

162 nvolving specialized proresolving mediators (SPMs; resolvins and maresins) and microRNAs (miRNAs).

163 es) and specialized pro-resolving mediators (SPMs).

164 uded patients with single primary melanomas (SPMs) and multiple primary melanomas (MPMs) of any stage

165 ecifically in the synaptic plasma membranes (SPMs) of the ipsilateral dorsal horn.

166 ecular layers in surface plasmon microscopy (SPM) is suggested.

167 n the wide-field surface plasmon microscopy (SPM).

168 brief overview of scanning probe microscopy (SPM) methods addressing nanoscale electrochemical functi

169 overview of using Scanning Probe Microscopy (SPM), in particular Scanning Tunneling Microscopy (STM),

170 labeled with superparamagnetic microspheres (SPMs).

171 subset, referred to as small peritoneal MO (SPM), expresses substantially lower levels of CD11b and

172 erformance relative to a single-phase model (SPM) similar to LandGEM.

173 The Stochastic Process Model (SPM) represents a general framework for modeling the joi

174 t molars (FPMs) and second permanent molars (SPMs) is an important developmental milestone influencin

175 econd motif that we named Spc110/Pcp1 motif (SPM) is also important for MT nucleation.

176 cytic MPhi resembling small peritoneal MPhi (SPM) that expressed CD138(+) and the scavenger receptor

177 microfluidic sample preparation multiplexer (SPM) and assay procedure is developed to improve amplifi

178 microfluidic sample preparation multiplexer (SPM) has been developed and evaluated for Ebola virus de

179 erparamagnetic multiwalled carbon nanotubes (SPM-MWCNTs) in an aqueous system containing Lake Tai sed

180 penemases such as KPC, NMC-A, IMI, SME, NDM, SPM, IMP, VIM, and OXA-23, 40, 48, 58, 72, 181, and 232

181 dence of second primary malignant neoplasms (SPMs) is a well-known late effect after cancer, few stud

182 The new SPM can run up to 80 assays in parallel using a pneumati

183 Our study shows that the novel SPM 17-hydroxydosahexaenoic acid (17-HDHA), resolvin D1,

184 The actions of SPM on B lymphocytes remain unknown.

185 Therefore, the adsorption of SPM-MWCNT on the sediment should proceed through a multi

186 However, when normalized to the amount of SPM, concentrations were on average >20 times higher at

187 Bulk analysis of SPM clearly shows an increase of Ti-containing particles

188 Application of SPM's voxel-based morphometry on the modulated images of

189 ings highlight the potential applications of SPM as endogenous and nontoxic adjuvants, and as anti-in

190 nduced inflammation, providing a new area of SPM activity to investigate in this major area of therap

191 The temporally initiated biosynthesis of SPM and their direct impact on leukocyte trafficking and

192 dence that the inappropriate biosynthesis of SPM in the lesioned spinal cord hampers the resolution o

193 ry in adult female mice, the biosynthesis of SPM is not induced in the lesion site up to 2 weeks afte

194 clinical and pathological characteristics of SPM, MPM, and both in Cox proportional hazards regressio

195 suggesting that the greater driving force of SPM allowed achievement of steady state.

196 n we investigate expression and functions of SPM in intestinal inflammation.

197 The impact of SPM status on cancer-specific death was examined using m

198 WT, suggesting a more stable interaction of SPM in the deep pore cavity.

199 nd Ab production supports the involvement of SPM during the late stages of inflammation and pathogen

200 Cebpb(-/-) mice exhibit elevated numbers of SPM-like cells but lack functional LPM.

201 Accurate prediction of SPM hydrodynamics requires the quantification of 3D floc

202 The ratio of SPM images allows one to visualize this redistribution a

203 Adoptive transfer of SPM into Cebpb(-/-) mice results in SPM differentiation

204 The package estimates several versions of SPM currently available in the literature including disc

205 stimulating local endogenous biosynthesis of SPMs and the generation of specific growth factors.

206 We compared plasma concentrations of SPMs in men and women with features of the MetS and in h

207 To assess the survival impact of SPMs in adolescents and young adults (AYAs) (15-39 years

208 clusion and Relevance: The adverse impact of SPMs on survival is substantial for AYAs and may partial

209 The cumulative incidence of SPMs did not differ significantly among the TT trial com

210 omotion of inflammation and the induction of SPMs.

211 r refractory MM (N = 703), the overall IR of SPMs was 3.98 (95% confidence interval [CI], 2.51-6.31)

212 ce also have increased circulating levels of SPMs and less lung injury after I/R.

213 The levels of SPMs, particularly resolvin D1 (RvD1), and the ratio of

214 infections and an unexpected high number of SPMs, which may limit its use.

215 ysis, we identified unbalanced production of SPMs (i.e., D- and E-series resolvins, protectin D1, mar

216 cularly resolvin D1 (RvD1), and the ratio of SPMs to pro-inflammatory leukotriene B4 (LTB4), are sign

217 The risk of SPMs must be taken into account before initiating lenali

218 with MM without apparent increase in risk of SPMs.

219 s research highlights the beneficial role of SPMs in pulmonary bacterial infections and provides the

220 acute inflammation in aging and the roles of SPMs.

221 ssolved organic matter (DOM) and sediment on SPM-MWCNTs under various conditions and the interaction

222 all and for each cancer occurring as a PM or SPM by age at diagnosis.

223 ed as having 14 cancers occurring as a PM or SPM were included.

224 e error rates for the fMRI software packages SPM, FSL, and AFNI, as well as a nonparametric permutati

225 The impact of a particular SPM diagnosis on survival may inform age-specific preven

226 t properties of the silicon photomultiplier (SPM) with a thermoelectric cooler and the possibilities

227 Plasma SPMs were measured with the use of liquid chromatography

228 plementation did not alter any of the plasma SPMs in MetS and control subjects.

229 lated vascular cells and tissues can produce SPMs and assessed expression and subcellular localizatio

230 M2 macrophages produced SPM including maresin-1 (299 +/- 8 vs 45 +/- 6 pg/2.5 x

231 and in sterile inflammation in vivo promotes SPM biosynthesis by a mechanism involving an increase in

232 In coffee brews, the order was PUT, SPM, TYR, CAD, SPD, PHE, HIS, and SER, but at a very low

233 e form of amorphous zinc sulfide) in the raw SPM samples stored under dry nitrogen vs an oxygen first

234 mice had both delayed resolution and reduced SPMs.

235 Results SPM exhibited excellent ICCs (0.97, 0.85, and 0.83 for T

236 r adult (>/=40 years) patients with the same SPMs.

237 have lower levels and/or absence of specific SPMs that were restored with Lovaza; these SPMs promote

238 thways, producing epimeric forms of specific SPMs, whereas other drugs can disrupt timely resolution.

239 These results demonstrate that specific SPMs are temporally and differentially regulated during

240 Finally, we determined spermine (SPM) sensitivity of these uncharacterized SNPs and found

241 lations carried out with Mg(2+) or spermine (SPM(4+)) show that these ions interact with pore-lining

242 ost important biogenic polyamines; spermine (SPM), spermidine (SPD) and putrescine (PUT), or electroc

243 The results showed that DOM can stabilize SPM-MWCNTs by providing sterically and electrostatically

244 omain theory behavior for a superparamagnet (SPM) but can be accounted for in a carrier-mediated RKKY

245 were ascertained during a longer period than SPM.

246 New results demonstrate that SPM regulate aspects of the immune response, including r

247 We show here that SPM biosynthesis is increased and inflammation resolutio

248 These results indicate that SPM may provide novel therapeutic tools for the treatmen

249 Transcription factor analysis revealed that SPM and LPM express abundant CCAAT/enhancer binding prot

250 The results also show that SPM(4+) behaves differently inside these two channels.

251 animal studies is reviewed, indicating that SPMs are physiologic mediators and pharmacologic agonist

252 The SPM is shown to give lower error in estimating methane c

253 The SPM precursor docosahexaenoic acid accelerated resolutio

254 representing 20 million children), and the SPM analysis included ages 9 to 13 y ( n = 2,774 represe

255 sults demonstrate the importance of both the SPM properties (concentration, size, density) as well as

256 We developed an R package stpm for the SPM-methodology.

257 ing an on-chip concentration protocol in the SPM and the single molecule detection capability of the

258 epresenting about half of the Zn pool in the SPM consist of Zn-sorbed on iron oxyhydroxides (ferrihyd

259 There was an increase in the SPM precursors 18-HEPE, 17-HDHA, and 14-HDHA after n-3 f

260 DPA supplementation increased the SPM resolvin D5n-3DPA (RvD5n-3DPA) and maresin (MaR)-1,

261 is study, we investigated the ability of the SPM 17-HDHA to enhance the adaptive immune response usin

262 Although cooling of the SPM lowered dark count rates and improved the minimum de

263 ned >68% of the dissolved Cu and >92% of the SPM-bound Cu, which represented 84.4% of the total Cu in

264 mechanisms for HO-1/CO, which is part of the SPM-initiated resolution circuit.

265 ent the first software implementation of the SPM-methodology by providing an R package stpm, which wa

266 nding, and interaction with PRC1 through the SPM domain.

267 The SPMs present in self-resolving arthritic joints include

268 ulting in increased CaV2.2 expression in the SPMs of the dorsal horn.

269 These SPM-like cells are not restricted to the peritoneum and

270 c SPMs that were restored with Lovaza; these SPMs promote macrophage phagocytosis of blood clots.

271 Thicker SPM, however, had higher fatality (HR for >4 mm, 13.56 [

272 were similar, relative fatality for thicker SPM was greater than that for thicker MPM.

273 g actions of MaR1 in SCI, we found that this SPM facilitated several hallmarks of resolution of infla

274 All three SPMs were found to be effective in elevating murine Ab l

275 ptotic PMN and microparticles contributed to SPM biosynthesis during efferocytosis.

276 ng minerals dominates the Zn contribution to SPM under such conditions.

277 Although overall fatalities due to SPM and MPM were similar, relative fatality for thicker

278 itors and hydrolyzed beta-lactam products to SPM-1.

279 between MPM and SPM (HR for MPM relative to SPM, 1.24 [95% CI, 0.91-1.69; P = .18]).

280 The fabricated biosensor responded to SPM, SPD and PUT over an extended dynamic range of 0.04-

281 idine (EDDP), amphetamine, and MDA sorbed to SPM ranged from 34.3% to 41.1% of the total mass in the

282 fraction of the total mass of ASWs sorbed to SPM was in the rank order: aspartame (50.4%) > acesulfam

283 The use of the probiotic in addition to SPMs was associated with a marked reduction of C. diffic

284 th muscle cells (VSMCs) converted 17-HDHA to SPMs, including resolvin D1 (RvD1) and other D-series re

285 using three widely used automated toolboxes: SPM ( www.fil.ion.ucl.ac.uk/spm/ ), FreeSurfer ( surfer.

286 IR of invasive (hematologic and solid tumor) SPMs was 2.08, consistent with the background incidence

287 n metrics such as TBV, GMV, and WMV by using SPM.

288 Analysis was performed using SPM or predefined regions of interest (ROIs).

289 ity together with promoting resolution using SPM represents a novel therapeutic strategy to resolve c

290 ET images were normalized and smoothed using SPM.

291 Together, they suggest that low vascular SPMs may enable progression of chronic vascular inflamma

292 In vitro, SPM treatment inhibited mouse aortic smooth muscle cell

293 Voxelwise SPM analyses revealed the first signs of increased corti

294 - 39 pg/2.5 x 10(5) cells; P < .01), whereas SPM including lipoxin A(4) (977 +/- 173 vs 675 +/- 167 p

295 8% of 8-y-olds had all FPMs emerged, whereas SPM emergence varied more.

296 n the MetS are required to determine whether SPMs affect the ability to mount an appropriate response

297 Moreover, we treated obese mice with SPM precursors and investigated the effects on inflammat

298 difference in outcome between patients with SPM and MPM related to factors other than closer surveil

299 association of pathogens and pollutants with SPM and their impact on aquatic life.

300 respectively, but they were lower than with SPM.