ライフサイエンスコーパス: SR

1 SR (serine-arginine)-rich proteins influence multiple st

2 SR proteins function in nuclear pre-mRNA processing, mRN

3 SR-AI expression on liver Mvarphi promotes recovery from

4 SR-B1 also facilitates the efflux of cholesterol from pe

5 SR-B1 has also been identified as a potential marker for

6 SR-BI is the main receptor for high density lipoproteins

7 SRs were recorded and coded according to the Medical Dic

8 resence of the AHR antagonist StemReginin-1 (SR-1) or the AHR agonist 2,3,7,8-tetrachlorodibenzo-p-di

9 The scavenger receptor, class B type 1 (SR-B1), is a multiligand membrane receptor protein that

10 iated by scavenger receptor class B, type 1 (SR-B1).

11 A total of 109 SRs were recorded, and 90 patients (2.1%) presented at l

12 Adult severe asthmatics (SS n = 12; SR n = 23) were assessed for their response to 2 weeks o

13 ramuscular adrenaline was administered in 17 SRs, but only 65% of these were subsequently classified

14 he propagation front elevates local [Ca(2+) ]SR , leading to luminal RyR sensitization and lowering o

15 [Ca(2+) ]SR was smaller than nj-SR [Ca(2+) ]SR .

16 2+) indicator rhod-2) and intra-SR ([Ca(2+) ]SR ; fluo-5N) Ca(2+) in rabbit atrial myocytes revealed

17 er, the degree of depletion of j-SR [Ca(2+) ]SR was smaller than nj-SR [Ca(2+) ]SR .

18 tibiotic and surgical treatment (37 TSSR, 24 SR, 19 OSSR) and 6 with OA.

19 e surface motifs of parent [Ag44(SR)30](4-) (SR = thiolate) nanoparticles (NPs), leading to bimetalli

20 number of paced beats increased from 1 to 5, SR Ca content (assessed with caffeine pulses) increased,

21 Results One hundred one patients (66 SR and 35 HR) were evaluable for outcome.

22 eticulum (SR), or calsequestrin 2 (CASQ2), a SR Ca(2+) binding protein, are linked to CPVT.

23 However, little is known about SR and D-serine function in the amygdala.

24 identify new mechanisms to abrogate acquired SR as well as new potential therapeutic targets in HCC.

25 We identified a mechanism for acquiring SR in HCC that is through the aberrant expression of the

26 Adding SR to the minimum cutoff for minimum diet diversity impr

27 0% of all miRNAs tested significantly affect SR time, revealing pervasive behavioral effects of miRNA

28 Detailed analyses of those miRNAs affecting SR behavior (SR-miRNAs) show that individual miRNAs can

29 4 of 412 [5.8%] vs 120 of 4791 [2.5%]) after SR.

30 Au38(SR)24, and Au102(SR)44 as well as Ag25(SR)18, Ag29(S2R)12, and Ag44(SR)30 (often with a few cou

31 as well as Ag25(SR)18, Ag29(S2R)12, and Ag44(SR)30 (often with a few counterions to compensate charge

32 y replace the surface motifs of parent [Ag44(SR)30](4-) (SR = thiolate) nanoparticles (NPs), leading

33 med to synthesize, appraise, and present all SR evidence on the efficacy and safety of inhaled short-

34 The data imply that an allosteric SR protein-phosphatase platform balances phosphorylation

35 Putative effectors include an SR protein kinase, bilobe proteins, TbSAS4, TbRP2, and B

36 gion for the proper subnuclear storage of an SR protein splicing factor.

37 t facilitates the interaction with SRSF3, an SR protein family member that promotes pri-miRNA process

38 Our data support an SR-B1 nibbling mechanism that is similar to that of stre

39 Furthermore, in vitro studies using an SR-AI-deficient Mvarphi cell line revealed impeded M2 po

40 formation of microdomains between acidic and SR calcium stores, supporting emerging interpretations o

41 By contrast, CD81 and SR-BI fulfil redundant functions during infection by the

42 ble functional similarities between CFIm and SR proteins suggest that interactions between RS-like do

43 ces in Nav subtype expression between CH and SR axon terminals and between CH and SR dendrites and sp

44 CH and SR axon terminals and between CH and SR dendrites and spines.

45 were found between axon terminals of CH and SR or between dendrites and spines of CH and SR.

46 pression in glutamatergic synapses of CH and SR supporting neurotransmitter release and synaptic plas

47 All Nav subtypes in both CH and SR were preferentially associated with asymmetric synaps

48 SR or between dendrites and spines of CH and SR.

49 WCs after implant and autologous IR, DR, and SR breast procedures after mastectomy.

50 r mechanisms leading to RyR2 dysfunction and SR Ca(2+) leak depend on the clinical stage of AF or spe

51 to synthesize the responses of soil EEAs and SR to elevated N.

52 types of scavenger receptors (SRs; MARCO and SR-B1), as blockade of the receptors with antibodies or

53 tiple serine/arginine-rich (SR) proteins and SR related proteins, including U2AF65, all of which are

54 mote CPB expansion into northern regions and SR into the circumpolar latitudes.

55 opathy, central cores, Z-disc streaming, and SR dilation.

56 ed activity, the latency period variance and SR Ca load had the greatest influences.

57 ith the vasopressin V1a receptor antagonist, SR 49059 (-1 +/- 1 mmHg; 1.1 +/- 1.1% total power, respe

58 ugh phosphorylation directs serine-arginine (SR) proteins from nuclear storage speckles to the nucleo

59 scavenger receptor B1 (SCARB1; also known as SR-B1) gene.

60 including the published short-read assembly (SR) constructed for the same individual allowed assessin

61 and FGR (1-4 groups, with SR = 4) to assess SR and FGR effects on ecosystem N cycling and its respon

62 as such as Au25(SR)18, Au38(SR)24, and Au102(SR)44 as well as Ag25(SR)18, Ag29(S2R)12, and Ag44(SR)30

63 ules, giving rise to a core-shell [Ag32@Au12(SR)30](4-).

64 s on thiolate-protected gold clusters (Au130(SR)50, Au144(SR)60, and Au500(SR)120).

65 -protected gold clusters (Au130(SR)50, Au144(SR)60, and Au500(SR)120).

66 rdingly, a new bimetal nanocluster, [Au19Cd2(SR)16](-), is produced.

67 ion induced by cadmium doping into the [Au23(SR)16](-) (R = cyclohexyl) nanocluster, in which two nei

68 strate different doping modes when the [Au23(SR)16](-) nanocluster is doped with different metals (Cu

69 ules of this kind with formulas such as Au25(SR)18, Au38(SR)24, and Au102(SR)44 as well as Ag25(SR)18

70 Electrocrystallization of three known Au25(SR)18(0) clusters is described.

71 kind with formulas such as Au25(SR)18, Au38(SR)24, and Au102(SR)44 as well as Ag25(SR)18, Ag29(S2R)1

72 lusters (Au130(SR)50, Au144(SR)60, and Au500(SR)120).

73 lyses of those miRNAs affecting SR behavior (SR-miRNAs) show that individual miRNAs can affect moveme

74 utlook of the directions in which we believe SR-SIM is headed in the future.

75 Feret diameter and the mean distance between SR and the outer mitochondrial membrane vs. CD hearts.

76 ecies (ROS), intermediate ROS levels between SR-BI(+/+) and nSR-BI(-/-) embryos were detected in SR-B

77 roteins, CD81 and the Scavenger Receptor BI (SR-BI), respectively, to infect hepatocytes.

78 Bimatoprost SR demonstrated favorable efficacy and safety through 6

79 Bimatoprost SR provided rapid, sustained IOP lowering.

80 ients (n = 75) were administered Bimatoprost SR (6 mug, 10 mug, 15 mug, or 20 mug) intracamerally in

81 prost sustained-release implant (Bimatoprost SR).

82 is via SRPK2, a key regulator of RNA-binding SR proteins.

83 ent survival and a low relapse risk for both SR and HR patients with APL.

84 lar EGMs, were obtained and compared in both SR and AF.

85 ient adequacy were mostly obtained when both SR and DDS were maximal.

86 ssential to prevent internalization of FX by SR-AI, and the presence of FX is needed to interfere wit

87 r cells were also significantly increased by SR-1 as quantified by standard hematopoietic colony-form

88 imulated LCRs and examined LCR regulation by SR Ca pumping rate (Pup) that provides a major contribut

89 partners, a mechanism that may be shared by SR-BI and CD36, scavenger receptor proteins highly homol

90 -fire' (or FDUF) mechanism: Ca(2+) uptake by SR Ca(2+) pumps at the propagation front elevates Ca(2+)

91 ocytoplasmic shuttling among seven canonical SR protein family members.

92 ian genomes encode many poorly characterized SR-like proteins, including subunits of the mRNA 3'-proc

93 K1 and CHO-ldlA7 cells (LDLR(-/-)) with (CHO-SR-B1) and without SR-B1 overexpression and in human Huh

94 tostability compared to that of conventional SR probes, it can provide images of nuclear DNA at unpre

95 wild type levels, suggesting that decreased SR Ca(2+) release is not the major driver of the myopath

96 activity in response to increasing diastolic SR [Ca(2+)] are influenced by CSQ2 and are disrupted in

97 Dietary SR is recommended as the most appropriate measure of foo

98 -refractory acute graft-versus-host disease (SR-aGVHD) remains suboptimal.

99 During SR, EBW are present in over a third of patients, particu

100 ccur is already present in some areas during SR.

101 pact on bipolar EGM voltages obtained during SR and AF.

102 sized that increased plant diversity (either SR or FGR) and elevated CO2 would enhance plant N pools

103 nfected mice deficient for the gene encoding SR-AI (msr1).

104 For SR and HR patients with APL, the overall survival was 98

105 1 of 369) for DR, and 3.2% (167 of 5150) for SR.

106 The EFS for SR patients in AAML0631 was noninferior to that of patie

107 ed oxidative stress but not apoptosis in FRD-SR-AIP mice, in which a CaMKII inhibitor is targeted to

108 Binding studies showed that FX, SR-AI, and PTX2 independently bind to each other (KD,app

109 Here, we demonstrate that the FX/SR-AI-complex comprises a third protein, pentraxin-2 (PT

110 th two mutated modules participated in GroEL(SR)-mediated protein folding in vitro.

111 GroES, and a single-ring GroEL variant GroEL(SR) forms a stable complex with GroES, arresting the cha

112 To weaken GroEL(SR)-GroES interaction in a controlled manner, we used gr

113 hree mutated modules collaborated with GroEL(SR) to perform chaperone function in vivo: three GroES(7

114 hree GroES(7) variants functioned with GroEL(SR) under both normal and heat-shock conditions.

115 interaction, <0.05), with men showing higher SRs and SRa, although lower SRe (all P<0.001).

116 ntified scavenger receptor class A member I (SR-AI) as a receptor for coagulation factor X (FX), medi

117 by the scavenger receptor, class B, type I (SR-BI), the so-called HDL receptor.

118 atomically precise displacement of SR-Ag(I)-SR-protecting modules of Ag NPs by the incoming SR-Au(I)

119 g modules of Ag NPs by the incoming SR-Au(I)-SR modules, giving rise to a core-shell [Ag32@Au12(SR)30

120 n of RSNO and a copper(II) thiolate [Cu(II)]-SR intermediate formed upon reaction of an additional eq

121 ly contributes significantly to the impaired SR Ca(2+) -release observed in skeletal muscle from Tric

122 [5.8%] vs 129 of 5286 [2.4%]) after implant SR was higher in women who had received adjuvant radioth

123 In SR, vertical values were on average 66+/-119% larger tha

124 st completely (from 54% to 2%, p < 0.001) in SR-BI(-/-) embryos and normalized ROS and gene expressio

125 BV is tolerable and has activity in SR-aGVHD and merits further investigation.

126 to CH axon terminals compared to SR, and in SR dendrites and spines compared to CH.

127 in CD34(+)CD31(+) hematoendothelial cells in SR-1-treated hESCs, as well as a twofold expansion of CD

128 ce, in the absence of significant changes in SR Ca(2+) load.

129 The FRD-induced decrease in SR-mitochondrial distance is likely to additionally favo

130 /+) and nSR-BI(-/-) embryos were detected in SR-BI(-/-) with NTD (NTD SR-BI(-/-)).

131 sponsible for the age-associated increase in SR Ca content but not the decrease in Ca(2+) transient a

132 n frequency of mDCs in SS, but reduced it in SR asthmatics.

133 nd FX levels were correspondingly reduced in SR-AI-deficient mice.

134 y accounts for the steady-state reduction in SR calcium content and its more pronounced decline after

135 lr1-6SA-GFP protein, in which six serines in SR/RS clusters are substituted with alanines, primarily

136 protecting modules of Ag NPs by the incoming SR-Au(I)-SR modules, giving rise to a core-shell [Ag32@A

137 tead, decreased peak ICa-L offsets increased SR load such that Ca(2+) release from the SR was maintai

138 calcium kinetics, associated with increased SR calcium load.

139 l stores, which is consistent with increased SR luminal Ca(2+) These findings define critical roles f

140 In contrast, increasing SR (holding FGR constant and despite increasing total pl

141 tial support of these hypotheses, increasing SR or FGR (holding the other constant) enhanced total pl

142 clude that dysferlin prevents injury-induced SR Ca(2+) leak.

143 Ca(2+) -free medium and S107, which inhibits SR Ca(2+) leak, suggest the SR as the primary source of

144 uorescent Ca(2+) indicator rhod-2) and intra-SR ([Ca(2+) ]SR ; fluo-5N) Ca(2+) in rabbit atrial myocy

145 eous confocal imaging of cytosolic and intra-SR calcium revealed a transient elevation of store Ca(2+

146 This cycling initially involves SR release of Ca via the ryanodine receptor, which is re

147 myocytes revealed that Ca(2+) release from j-SR resulted in a cytosolic Ca(2+) transient of higher am

148 Ca(2+) -induced Ca(2+) release (CICR) from j-SR ryanodine receptor (RyR) Ca(2+) release channels.

149 the peripheral subsarcolemmnal junctional (j-SR) and the much more abundant central non-junctional (n

150 nj-SR; however, the degree of depletion of j-SR [Ca(2+) ]SR was smaller than nj-SR [Ca(2+) ]SR .

151 lore this possibility, studying early larval SR behavior in a collection of 81 Drosophila miRNA mutan

152 due to UNC-68 leakiness and/or malfunctional SR Ca(2+) homeostasis.

153 chondrial area, mean Feret diameter and mean SR-mitochondrial distance vs. CD-WT hearts.

154 resolved structured illumination microscopy (SR-SIM) is among the most rapidly growing fluorescence m

155 Our data suggest that dysferlin modulates SR Ca(2+) release in skeletal muscle, and that in its ab

156 oavailability, we selected a small molecule, SR-18292, that reduces blood glucose, strongly increases

157 uestrin accompanied by drastic morphological SR changes in fully depleted cells.

158 Most SRs occurred during the up-dosing phase (75.8%) and were

159 We propose that the new SR-TT junctions formed during exercise, and that contain

160 We also demonstrate that these new SR-TT junctions contain the molecular machinery that med

161 evation of [Ca(2+) ]i initiates CICR from nj-SR and sustains propagation of CICR to the cell centre.

162 We propose that Ca(2+) release from nj-SR is activated by cytosolic and luminal Ca(2+) (tandem

163 higher amplitude compared to release from nj-SR; however, the degree of depletion of j-SR [Ca(2+) ]SR

164 uch more abundant central non-junctional (nj-SR) SR.

165 tion of j-SR [Ca(2+) ]SR was smaller than nj-SR [Ca(2+) ]SR .

166 f Pax3, Alx1 and Alx3 genes was found in NTD SR-BI(-/-) embryos.

167 os were detected in SR-BI(-/-) with NTD (NTD SR-BI(-/-)).

168 postattachment receptors CD81, CLDN1, OCLN, SR-BI, and LDLR greatly impaired both HCV cell-free and

169 previously showed that approximately 50% of SR-BI(-/-) embryos fail to close the anterior neural tub

170 of HTS permitted identification of 19.9% of SR patients without MRD at any detectable level who had

171 Absence of SR-like RNA-binding protein Slr1 slows hyphal formation

172 and is markedly elevated with acquisition of SR.

173 erebroventricular (i.c.v.) administration of SR-3306, a brain-penetrant and selective pan-JNK (JNK1/2

174 toid DCs (pDCs) was observed in the blood of SR as compared to SS asthmatics (P = .03).

175 ease by conformationally inducing closure of SR channels.

176 Although the voltage dependence of SR calcium release was not statistically different betwe

177 a historical overview of the development of SR-SIM, we review how this method can be implemented in

178 ion is an atomically precise displacement of SR-Ag(I)-SR-protecting modules of Ag NPs by the incoming

179 Features of SR EBW indicate that muscular connections between endo-

180 The versatile functions of SR (serine/arginine-rich) proteins in pre-mRNA splicing

181 These collective functions of SR-B1 ultimately affect programmed cell death, female fe

182 bitory activity and a critical gatekeeper of SR calcium cycling and contractility in the heart.

183 Thus, the local interplay of SR Ca pump and release channels regulates LCRs and Ca tr

184 sum, our results suggest the involvement of SR-BI in the maternal provision of embryonic vitamin E t

185 to characterize the cellular localization of SR and D-serine in the mouse and human amygdala.

186 icate that posttranslational modification of SR proteins underlies the regulation of their mRNA expor

187 gesting a unique mechanism for regulation of SR Ca homeostasis.

188 RIC-A for normal physiological regulation of SR Ca(2+) -release in skeletal muscle.

189 Here, we evaluated the role of SR-BI in embryonic vitamin E uptake during murine neural

190 he regulation and functional significance of SR-B1 in mediating cholesterol movement into and out of

191 The strength of SR-SIM is that it can be readily applied to samples prep

192 cin in the NAc and CARTpt in the striatum of SR rats.

193 nd 90 patients (2.1%) presented at least one SR.

194 olangiocytes with knocked down either SCT or SR by short hairpin RNAs show reduced EV secretion durin

195 PS stimulation, and EVs isolated from SCT or SR knocked down cholangiocytes fail to induce inflammato

196 Mechanistically, SRPK2 promotes SR protein binding to U1-70K to induce splicing of lipog

197 Depressive Symptomatology-Self Report [QIDS-SR] or the Beck Depression Inventory [BDI]), obtained fo

198 Inventory of Depressive Symptomatology (QIDS-SR) scale and 14 items from the clinician-rated Hamilton

199 items of the MADRS, the HAM-D, and the QIDS-SR but not the BDI.

200 Three symptom clusters in the QIDS-SR scale were identified at baseline in STAR*D.

201 rine by the neuronal enzyme serine racemase (SR).

202 In contrast, in the stratum radiatum (SR) of CA1, Nav 1.1, Nav 1.2, and Nav 1.6 were selective

203 s, stratum oriens (SO) and stratum radiatum (SR).

204 o investigate effects of strontium ranelate (SR) on alveolar bone loss (ABL) in rats with experimenta

205 ongitudinal systolic strain and strain rate (SR) at systolic (SRs), early diastolic (SRe), and late d

206 e Vegetation Index (NDVI), the Simple Ratio (SR), and the Normalized Pigment Chlorophyll Index (NPCI)

207 retin (SCT) that binds to secretin receptor (SR), is a key mediator in cholangiocyte pathophysiology.

208 sequence to deliver it to the SRP receptor (SR) on the membrane, where the signal peptide is transfe

209 b) through two types of scavenger receptors (SRs; MARCO and SR-B1), as blockade of the receptors with

210 r delayed (DR) and secondary reconstruction (SR) compared with immediate reconstruction (IR) procedur

211 eticulum Ca ATPase (SERCA) pump then refills SR Ca stores.

212 ially overlaps with that of splicing-related SR proteins and in tex1 plants the ratio of certain alte

213 nts invariably develop sorafenib resistance (SR).

214 teroid-sensitive (SS) and steroid-resistant (SR) asthmatics.

215 ive, SS) but not in others (shock-resistant, SR).

216 ts on microbially mediated soil respiration (SR).

217 proves muscle function, but does not restore SR Ca(2+) transients in I4895T fibres to wild type level

218 nhibitor of the sarco/endoplasmic reticulum (SR) Ca(2+) ATPase (SERCA) and is abnormally elevated in

219 2+) release from the sarcoplasmic reticulum (SR) - a process termed Ca(2+) -induced Ca(2+) release (C

220 elative roles of the sarcoplasmic reticulum (SR) and surface membrane, are unclear.

221 cycling between the sarcoplasmic reticulum (SR) and the cytosol via the sarco-/endoplasmic reticulum

222 unctions between the sarcoplasmic reticulum (SR) and transverse-tubules (TTs).

223 ease events from the sarcoplasmic reticulum (SR) as a potential cause of proarrhythmic cellular ectop

224 mulation to regulate sarcoplasmic reticulum (SR) Ca(2+) -release.

225 phosphorylation and sarcoplasmic reticulum (SR) Ca(2+) leak.

226 POINTS: Spontaneous sarcoplasmic reticulum (SR) Ca(2+) release events increased in fructose-rich die

227 system, dividing the sarcoplasmic reticulum (SR) Ca(2+) store into the peripheral subsarcolemmnal jun

228 2+) release from the sarcoplasmic reticulum (SR) in response to plasma membrane (PM) excitation.

229 in X-1) localizes to sarcoplasmic reticulum (SR) in the heart and interacts with the small membrane p

230 associated with the sarcoplasmic reticulum (SR) in ventricular myocytes; a median separation of 20 n

231 hannel (RyR2) in the sarcoplasmic reticulum (SR) membrane and the SR Ca(2+) binding protein calseques

232 ule apply inside the sarcoplasmic reticulum (SR) of a working cell?

233 releases (LCRs) from sarcoplasmic reticulum (SR) regulate cardiac pacemaker cell function by activati

234 Ca(2+) leak from the sarcoplasmic reticulum (SR) through the RyR1.

235 cycling through the sarcoplasmic reticulum (SR), a Ca storage organelle, is critical for proper card

236 annel located in the sarcoplasmic reticulum (SR), or calsequestrin 2 (CASQ2), a SR Ca(2+) binding pro

237 ptor 1 (RyR1) in the sarcoplasmic reticulum (SR).

238 This overview of systematic reviews (SRs) aimed to synthesize, appraise, and present all SR e

239 eak-to-peak voltages (Vmax) in sinus rhythm (SR) and AF.

240 o occurs to some degree during sinus rhythm (SR).

241 hose obtained in patients with sinus rhythm (SR).

242 hydroxylates multiple serine/arginine-rich (SR) proteins and SR related proteins, including U2AF65,

243 Species richness (SR) and functional group richness (FGR) are often confou

244 and functional diversity, species richness (SR) showed stronger associations and better diagnostic p

245 tation if turned upside down (self-righting, SR), suggesting that other miRNAs might also be involved

246 S stimulation, and demonstrates that the SCT/SR axis may be important for this event.

247 reduced in the latter, indicating a smaller SR content.

248 more abundant central non-junctional (nj-SR) SR.

249 o allow repositioning of the 'activated' SRP-SR complex on the ribosome.

250 anslating Escherichia coli ribosome, the SRP-SR in the 'activated' state and the translocon.

251 lic strain and strain rate (SR) at systolic (SRs), early diastolic (SRe), and late diastolic atrial c

252 It can be concluded that SR can reduce RANKL activity and osteoclast numbers, as

253 Our results showed that SR-BI(-/-) embryos had a very low vitamin E content in c

254 more, for the first time, we have shown that SR Ca content is increased in old atrial myocytes.

255 The SR family proteins play important roles in splicing regu

256 arise as a result of K(+) fluxes across the SR via TRIC-A.

257 sarcoplasmic reticulum (SR) membrane and the SR Ca(2+) binding protein calsequestrin 2 (CSQ2).

258 n molecule-1 (STIM1), which functions as the SR Ca(2+) sensor, and Orai1, the Ca(2+)-permeable channe

259 fear conditioning and extinction engages the SR/D-serine system in the brain.

260 Finally, the SR-B1-linked selective HDL-cholesteryl ester uptake path

261 increased RyR1-mediated Ca(2+) leak from the SR into the cytoplasm.

262 ed SR load such that Ca(2+) release from the SR was maintained during ageing.

263 tly acts to enhance calcium release from the SR.

264 group and 63 beats per minute +/- 14 in the SR group (P < .01).

265 8) and 95.6% and 98.1%, respectively, in the SR group (P = .32).

266 y was 98.5% in the AF group and 98.4% in the SR group (P = .96).

267 ower-magnitude LTP typically observed in the SR layer, demonstrating that cadherins-6, -9, and -10 ar

268 on activation of the RyR1 Ca(2+) pore in the SR, under control of conformational changes of CaV1.1, l

269 mobility of calsequestrin as [Ca(2+)] in the SR--measured with a calsequestrin-fused biosensor--was l

270 k and catastrophic structural changes in the SR.

271 propagation front elevates Ca(2+) inside the SR locally, leading to luminal RyR sensitization and low

272 lowed by re-sequestration of Ca(2+) into the SR.

273 ggesting a compromised ability to refill the SR.

274 e immediate role of SOCE in replenishing the SR calcium store, the evolution of intracellular calcium

275 , which inhibits SR Ca(2+) leak, suggest the SR as the primary source of Ca(2+) responsible for the l

276 Then the SR generates an earlier, more synchronized, and stronger

277 which a CaMKII inhibitor is targeted to the SR.

278 precipitation-stressed dry mosses, while the SR and NPCI were highly effective.

279 con positioned through interactions with the SR in the vicinity of the ribosome exit tunnel where the

280 tion of protein phosphatase 1 (PP1) with the SR protein splicing factor (SRSF1) to understand the fou

281 [Ca(2+) ]i produced by interfering with the SR was accompanied by a decrease of the amplitude of the

282 tatory modulator of RyR1 channels within the SR terminal cisternae.

283 Most of the SRs occurred in subcutaneous allergen immunotherapy (SCI

284 Thirteen SRs containing 56 relevant trials and 5526 patients were

285 y localized to CH axon terminals compared to SR, and in SR dendrites and spines compared to CH.

286 very low vitamin E content in comparison to SR-BI(+/+) embryos.

287 avioral responses in the same individuals to SR and to TSD over long-time intervals.

288 thy adults, neurobehavioral vulnerability to SR exposures, separated by 78-3,058 days (mean: 935 days

289 rds this end, we sought to determine how two SR proteins-SRSF3 and SRSF7, regulators of pre-mRNA spli

290 y during consolidation 1, an additional two (SR) or three (HR) consolidation courses that included hi

291 factorial experiments to independently vary SR (one or four species, with FGR = 1) and FGR (1-4 grou

292 This increase of [Ca(2+) ]i was larger when SR function was impaired either by making the ryanodine

293 Whereas SR-BI(-/-) embryos with closed neural tubes (nSR-BI(-/-)

294 ion of the SCF repertoire, including whether SRs compete for Cul1 and, if so, how this competition is

295 prospectively enrolled (83 with AF, 83 with SR).

296 ies, with FGR = 1) and FGR (1-4 groups, with SR = 4) to assess SR and FGR effects on ecosystem N cycl

297 performance similar to that in patients with SR.

298 es (MedDRA) and risk factors associated with SRs were identified.

299 lls (LDLR(-/-)) with (CHO-SR-B1) and without SR-B1 overexpression and in human Huh7 hepatocytes.

300 roscopy (XPS) and synchrotron radiation-XPS (SR-XPS) analysis of 10-100 nm thick PEDOT(PSS) films.