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1                                              SRF is also dispensable for the KLF3-mediated repression
2                                              SRF is an essential regulator of skeletal muscle differe
3                                              SRF is an essential transcription factor in hematopoiesi
4                                              SRF regulates neutrophil migration, integrin activation,
5                                              SRF split renal function was measured by using the area
6                                              SRF subsequently funded animal research to evaluate sucr
7                                              SRF terminated Project 259 without publishing the result
8                                              SRF thickness >118.25 mum at baseline predicted requirin
9                                              SRF-deficient macrophages fail to spread, transmigrate,
10                                              SRF-VP16iHep mHCC reveal convergent Ras/MAPK and Rho/act
11                                              SRF/MRTF-A-dependent gene transcription is activated whe
12 lators of smooth muscle expression (miR-145, SRF, Nfatc4, and Crip1).
13 The main aim of this study was to compare 2D SRFs between neurons in the noise-selective region (NSR)
14  for retina (ICC = 0.84; 95% CI, 0.83-0.86), SRF (ICC = 0.88; 95% CI, 0.86-0.89), and subretinal tiss
15 2 reduced the nuclear accumulation of MRTF-A.SRF complexes and consequently inhibited alpha-SMA promo
16                  It describes how the MRTF-A/SRF pathway is intricately linked to all the key regulat
17 ription factor/serum response factor (MRTF-A/SRF) transcription pathway as a potential novel therapeu
18 polymerization de-repress MRTFs and activate SRF-dependent genes.
19  conditionally express constitutively active SRF-VP16 in hepatocytes, thereby controlling subsets of
20 ued by expression of a constitutively active SRF.
21 ion resulting in reduced myogenically active SRF, but enhanced SRF activity on target genes involved
22 ng the formation of transcriptionally active SRF/MRTF-A complexes.
23 ically interacted with SRF without affecting SRF-myocardin interaction.
24                                          All SRF-VP16iHep mice develop hyperproliferative liver nodul
25  enrichment of RNase L and TTP targets among SRF-regulated genes suggesting that the RNase L/TTP axis
26 activated smooth muscle gene promoters in an SRF-dependent manner.
27 n proposed that coating the inner wall of an SRF cavity with superconducting thin films increases Hvp
28 ning, funding, and internal evaluation of an SRF-funded research project titled "Project 259: Dietary
29 essive DNA binding complexes and suppress an SRF-dependent transcriptional program that supports surv
30  transmigrate, and phagocytose bacteria, and SRF-deficient neutrophils show defective chemotaxis in v
31 cerebellum, NR3C1 in the cerebral cortex and SRF in the basal forebrain.
32 d similar efficacy in visual improvement and SRF resolution.
33 differentiate, and quantify intraretinal and SRF using area under the receiver operating characterist
34                       All individual IRC and SRF lesions were manually delineated on each of the 128
35 rrelations were computed between the IRC and SRF parameters and the baseline BCVA, final BCVA, and BC
36 s of fit of correlations between the IRC and SRF parameters and the baseline BCVA, final BCVA, and BC
37 rvention, exudative features such as IRC and SRF resolved rapidly in 74% of eyes, whereas PED respond
38 underwent complete quantification of IRC and SRF.
39 ual outcome only in combination with IRC and SRF.
40 acuity, new haemorrhage, presence of IRF and SRF on an optical coherence tomography (OCT) scan.
41 e" treatment (complete resolution of IRF and SRF) or ranibizumab "relaxed" treatment (resolution of I
42 entiates TNF-alpha in inducing NF-kappaB and SRF/SRE activities.
43 us, despite the shared phenotype of KLF3 and SRF-deficient mice, cooperation of these factors appears
44 ting the ability of Pfn to influence MKL and SRF expression.
45 in which RNase L and TTP target SRF mRNA and SRF-induced transcripts.
46 F), resulting in disruption of myocardin and SRF interactions and thereby attenuating expression of s
47  set of genes associated with high Notch and SRF activity.
48 tile melanoma cells had increased Notch- and SRF-dependent transcription.
49 scription factors (MRTFs), which function as SRF coactivators, serve as sensors of actin polymerizati
50 rrative case study method was used to assess SRF Project 259 from 1967 to 1971 based on sugar industr
51  CREB/ATF family, heat-shock factor 1, ATF6, SRF, and E2F1 transcription factors.
52                                     Baseline SRF had no effect on visual recovery; however, recurrenc
53 rs for BCVA change at month 12 were baseline SRF (P = 0.05), PVD (P = 0.03), IRC (P = 0.05), treatmen
54    No robust associations were found between SRF and baseline BCVA (R2 = 0.06; P = .14) or BCVA chang
55  resolution of all retinal fluid (i.e., both SRF and intra-retinal fluid [IRF]) in patients with nAMD
56 via distinct mechanisms, the actions of both SRF and MRTF-A.
57                         The presence of both SRF and PVD at baseline was associated with similar BCVA
58                        In patients with both SRF and PVD at baseline, similar BCVA outcomes were obse
59 inal cysts resolved most rapidly followed by SRF, whereas PED decreased at a slower rate and intensit
60 duced by TGF-beta1 and MYOCD, and reduced by SRF deficiency in VSMCs.
61  follow-up; all recurrent cases had complete SRF resolution after another PDT treatment.
62 ific role of the transcription factors CREB, SRF, and MEF2 in the depression and potentiation compone
63   In this study, we tested the role of CREB, SRF, and MEF2 in ocular dominance plasticity (ODP), a pa
64 to block the transcription function of CREB, SRF, and MEF2 in the visual cortex, and measured visuall
65                          We found that CREB, SRF, and MEF2 are all required for ODP, but have differe
66 novel MRTF/SRF inhibitor, markedly decreased SRF reporter gene activity and showed a greater inhibito
67 gether, these results suggest that decreased SRF expression induces replicative stress and chromosoma
68 act as general antagonists of MRTF-dependent SRF target gene expression, competing directly with the
69 C data, we identified over 700 TCF-dependent SRF direct target genes involved in signaling, transcrip
70 1 patients; 16 of these eyes (84%) developed SRF on OCT.
71 Forty-six (90%) study participants developed SRF during the study period, with 9 (20%) experiencing s
72 kness of 39 study participants who developed SRF at the first visit increased from 280 (26) microm at
73 upon postnatal and adult depletion of either SRF or its cofactors Myocardin Related Transcription Fac
74 tiple cancer-related pathways including Elk1/SRF, AP1, NFkappaB and STAT, and reduces EGFR expression
75  observation), when exudative signs emerged (SRF in 3/6 eyes and retinal cystoid spaces in 5/6 eyes).
76 -induced SM gene transcription by empowering SRF binding to CArG box in SM gene promoters.
77 educed myogenically active SRF, but enhanced SRF activity on target genes involved in proliferation.
78 xpression of Olfm2 dose dependently enhanced SRF binding.
79  of the CCTepsilon subunit by siRNA enhances SRF signaling in cultured mammalian cells by an actin as
80                              On examination, SRF appeared as elevated, yellow-orange pockets in the f
81 genes controlled by the transcription factor SRF, and overexpression of SRF rescues impaired chromoso
82 s through their partner transcription factor SRF.
83  transcription factor serum-response factor (SRF) along with its co-activator, myocardin-related tran
84 ial role of myocardin/serum response factor (SRF) and Notch signaling in the transcriptional regulati
85  transcription factor serum response factor (SRF) and set the chromatin state of SRF-targeted genes e
86 factor A (MRTF-A) and serum response factor (SRF) and the other using the transcriptional coactivator
87                   The serum response factor (SRF) binds to coactivators, such as myocardin-related tr
88 al remodelling on the serum response factor (SRF) co-factors Megakaryoblastic Leukemia-1 and -2 (MKL1
89                       Serum response factor (SRF) controls multiple genes governing adhesion and migr
90 tic leukemia-1 (MKL1)/serum response factor (SRF) during myofibroblast differentiation resulted in de
91 (MRTFs) co-activating serum response factor (SRF) in this process is largely unknown.
92                       Serum response factor (SRF) is a ubiquitously expressed transcription factor an
93 scriptional regulator serum response factor (SRF) is controlled by both Ras/MAPK (mitogen-activated p
94   We demonstrate that serum response factor (SRF) is induced by both platelet-derived growth factor (
95 oding proteins of the serum response factor (SRF) pathway are located on 5q.
96 ion factor-A (MRTF-A)/serum response factor (SRF) pathway.
97 es, especially in the serum response factor (SRF) pathway.
98 tion of Rho-dependent serum-response factor (SRF) signaling.
99 lators and to promote serum response factor (SRF) signalling has raised the question of whether MRL p
100  transcription factor serum response factor (SRF) to activate mitogen-induced transcription.
101 -kappaB and myocardin/serum response factor (SRF) to convey hypertrophy signaling in cardiac myoblast
102 teraction between the serum response factor (SRF) transcription factor and one of its principal co-ac
103 levated expression of serum-response factor (SRF), a master regulator of mitogen-induced transcriptio
104 s where they activate serum response factor (SRF), a regulator of actin and other cytoskeletal protei
105 cardin for binding to serum response factor (SRF), resulting in disruption of myocardin and SRF inter
106  transcription factor Serum Response Factor (SRF), suffer from loss of BBB integrity and intracerebra
107 scriptional regulator serum response factor (SRF), whereas another is calcineurin Abeta.
108 ator that activates a serum response factor (SRF)-dependent gene program required for cardiogenesis a
109 gnificantly inhibited serum response factor (SRF)-dependent reporter gene (SRE-LUC) activity and mRNA
110 tes the expression of serum-response factor (SRF)-dependent target genes in response to the Rho-actin
111 ) are coactivators of serum response factor (SRF)-mediated gene expression.
112 lastic leukemia (MKL)/serum-response factor (SRF)-mediated gene transcription is a highly conserved m
113 merization results in serum response factor (SRF)-mediated transcription through nuclear retention of
114 r TGF-beta1 and MYOCD/serum response factor (SRF)-regulated TSPANs in VSMC by using RNA-seq analyses
115  binding sites of the serum-response factor (SRF).
116 tional coactivator of serum response factor (SRF).
117 romoter activity in a serum response factor (SRF)/CArG box-dependent manner.
118 transcription factor, serum response factor (SRF); however, the mechanisms dynamically regulating SMC
119 CREB (cAMP response element binding factor), SRF (serum response factor), and MEF2 (myocyte enhancer
120  was controlled by the transcription factors SRF and MEF2.
121 sue by quantifying spatial receptive fields (SRFs) in two functionally distinct cortical regions in t
122  incomplete resolution of sub-retinal fluid (SRF) </=200 mum at the foveal centre relative to a T&E p
123 as intra-retinal (IRF) or sub-retinal fluid (SRF) were evident on SD-OCT, followed by a gradual exten
124 sed hole showed persistent subretinal fluid (SRF) after gas absorption.
125 uity (BCVA), resolution of subretinal fluid (SRF) demonstrated by optical coherence tomography (OCT),
126 F corresponded to areas of subretinal fluid (SRF) on spectral-domain OCT and was found to persist in
127                    On OCT, subretinal fluid (SRF) was detected in 77% of CM patients and 60% of UM pa
128 al cystoid fluid (IRC) and subretinal fluid (SRF) was developed.
129 intraretinal cysts (IRCs), subretinal fluid (SRF), and pigment epithelial detachment (PED), presentin
130 tinal cystoid fluid (IRC), subretinal fluid (SRF), and pigment epithelial detachment (PED).
131 intraretinal cysts (IRCs), subretinal fluid (SRF), and pigment epithelial detachments (PEDs), decreas
132  intraretinal fluid (IRF), subretinal fluid (SRF), and sub-retinal pigment epithelium (RPE) fluid and
133 id material and persistent subretinal fluid (SRF), but also a RPE-independent visual cycle for cone p
134 tinal cystoid fluid (IRC), subretinal fluid (SRF), pigment epithelial detachment, and vitreomacular i
135  intraretinal fluid (IRF), subretinal fluid (SRF), sub-retinal pigment epithelium (RPE) fluid, and su
136 ccult CNV, and presence of subretinal fluid (SRF).
137  coefficient of 0.90 for IRC and of 0.96 for SRF.
138 ivation of SRF by GSK-3 that is critical for SRF-dependent axon growth in mammalian central neurons.
139 asis, underscoring an essential function for SRF and its pathway in health and disease.
140 Thus, competition between TCFs and MRTFs for SRF determines the balance between antagonistic prolifer
141 This serine phosphorylation is necessary for SRF activity and for its interaction with MKL-family cof
142 ollectively, our results identify a role for SRF in proliferation and migration during craniofacial d
143 the most accurate CT volumetry technique for SRF and the prediction of postdonation kidney function (
144      In 1965, the Sugar Research Foundation (SRF) secretly funded a review in the New England Journal
145 ters commonly evaluate split renal function (SRF) with Tc-99m-mercapto-acetyltriglycin (MAG3) scintig
146                                 Furthermore, SRF downregulation in diploid hPSCs induces replication
147                                 We generated SRF-VP16iHep mice, which conditionally express constitut
148  Only 2 participants (4%) were found to have SRF at the last study visit after discontinuation of tre
149 transcription factor depletion in the heart (SRF(HKO)) or of cardiac hypertrophy triggered by transve
150                                     However, SRF alone is not sufficient for regulating smooth muscle
151                       These results identify SRF and its MRTF cofactors as major transcriptional regu
152 inding supports previous reports implicating SRF and MEF2 in long-term depression (required for Dc-OD
153 ds achieving higher acceleration gradient in SRF cavity accelerator beyond the theoretical limit of b
154           We hypothesize that impairments in SRF/MRTF activity contribute to human SVD pathology.
155 pression of a wide range of genes, including SRF itself and many important structural and regulatory
156 h insulin alone, insulin+TNF-alpha increased SRF/SRE binding and beta-MHC expression, which was rever
157 ein 70-interacting protein (CHIP), increased SRF activity, as well as beta-myosin heavy chain (MHC) a
158  At the molecular level, direct and indirect SRF/MRTF target genes, encoding structural components of
159  Olfm2 expression inhibited TGF-beta-induced SRF binding to SM gene promoters in a chromatin setting,
160 P axis represents a viable target to inhibit SRF-driven proliferation in neoplastic diseases.
161   It did so both by independently inhibiting SRF gene expression and nuclear import of MRTF-A.
162                             Using integrated SRF ChIP-seq and Hi-C data, we identified over 700 TCF-d
163 utilized Nb ellipsoid to simulate an inverse SRF cavity and investigate the effect of coating it with
164           Specific alterations, such as IRC, SRF, and PED, as baseline or follow-up features are sign
165  baseline, the proportions of eyes with IRC, SRF, and PED were balanced between the aflibercept and r
166 the action of MKL that is independent of its SRF-related activity.
167  agreement between CT volumetry SRF and MAG3-SRF (bias, 95% limits of agreement: ROI vs MAG3 0.4%, -7
168  at day 3 was r = 0.85 to 0.88, between MAG3-SRF and PDKF (r = 0.84).
169 ic SRF was determined and compared with MAG3-SRF, postoperation donor kidney function, and graft func
170 wever, in severely dilated kidneys, the mean SRF split renal function measurement was underestimated
171  atypical actin-regulatory protein, mediates SRF/MRTF-A-dependent gene transcription elicited by nerv
172 r actin as a regulatory switch that mediates SRF/MRTF-A-dependent gene transcription.
173 in a possible feedback loop of the actin/MKL/SRF signaling circuit.
174  study, we examined the possible role of MKL/SRF in the context of regulation of profilin (Pfn), a ma
175          Furthermore, disruption of the MKL1/SRF target gene, smooth muscle alpha-actin (alpha-SMA) v
176 tify the transcription factor binding motifs SRF and PRDM1 as important regulators of PIP3-sensitive
177                                Thus, the MRF-SRF and YAP-TEAD pathways interact indirectly through th
178 yocardin-related transcription factor (MRTF) SRF cofactor family.
179                                         MRTF-SRF signaling is thus critical for expression of genes r
180                             We compared MRTF-SRF and YAP-TEAD target gene sets and identified genes d
181 lore the impact of the actin-controlled MRTF-SRF (myocardin-related transcription factor-serum respon
182           In CAFs, expression of direct MRTF-SRF genomic targets is also dependent on YAP-TEAD activi
183 ts stabilizing partner KLHL40, enhances MRTF-SRF activity.
184          We used vav-iCre to inactivate MRTF-SRF signaling early during hematopoietic development.
185 et gene expression is also dependent on MRTF-SRF signaling.
186                                Both the MRTF-SRF and the YAP-TEAD transcriptional regulatory networks
187                        We show that the MRTF-SRF pathway is activated in cancer-associated fibroblast
188  that YAP-TEAD activity is sensitive to MRTF-SRF-induced contractility, while MRTF-SRF signaling resp
189 o MRTF-SRF-induced contractility, while MRTF-SRF signaling responds to YAP-TEAD-dependent TGFbeta sig
190 y different WH2 domains correlates with MRTF-SRF activation.
191 scription factor/Serum response factor (MRTF/SRF) pathway plays a key role in fibroblast activation a
192 scription factor/serum response factor (MRTF/SRF) pathway represents a promising therapeutic target t
193                     CCG-222740, a novel MRTF/SRF inhibitor, markedly decreased SRF reporter gene acti
194 ts of new pharmacological inhibitors of MRTF/SRF signalling in a preclinical model of fibrosis.
195          We conclude that inhibitors of MRTF/SRF-regulated gene transcription such as CCG-222740, pot
196 d showed a greater inhibitory effect on MRTF/SRF target genes than the previously described MRTF-A in
197 ed" treatment (resolution of IRF or >200 mum SRF only at foveal centre).
198 ckness of the retina was Delta = 5+/-67 mum, SRF was Delta = 1.5+/-35 mum, and subretinal tissue comp
199 ls of HRT2 concomitantly disrupted myocardin/SRF and Notch transcription complex formation at respect
200 agged1 ligand- and Notch1-enhanced myocardin/SRF complex formation at the promoter CArG element.
201  promyogenic transcription factors myocardin/SRF in a CHIP-dependent manner.
202 identified the first VSMC-enriched and MYOCD/SRF and TGF-beta1/SMAD-dependent TSPAN family member, wh
203 2 is regulated by 2 parallel pathways, MYOCD/SRF and TGF-beta1/SMAD, via distinct binding elements wi
204  two regions: (1) compared with NSR neurons, SRF properties of FMSR neurons were more strongly depend
205  on OCT had better mean VA than eyes with no SRF (72.8 vs. 66.6 letters; P = 0.006).
206 , induces MRTF translocation to the nucleus, SRF-activation and CCN1/2 transcription.
207 al a novel phosphorylation and activation of SRF by GSK-3 that is critical for SRF-dependent axon gro
208 o DeltaNT in a distinct assay (activation of SRF luciferase).
209 ndingly, TRIM32 attenuated the activation of SRF signaling and hypertrophy due to dysbindin, whereas
210 gnal-responsive transcriptional activator of SRF.
211 nce, and the presence and characteristics of SRF noted on optical coherence tomography.
212                             A combination of SRF, GATA6 and CRP2 required CSRP2BP for robust smooth m
213 ption factor/coactivator complex composed of SRF/Mkl1.
214 nt to overcome the axonal growth deficits of SRF-deficient and GSK-3-inhibited neurons.
215                         The determination of SRF is conducted at a reading centre while the assessmen
216 SRF interaction promoted the dissociation of SRF from HERP1, a transcriptional repressor.
217 PTEN interacts with the N-terminal domain of SRF and PTEN-SRF interaction promotes SRF binding to ess
218 K-regulated ternary complex factor family of SRF partner proteins.
219              A constitutively active form of SRF/Mkl1 was not sufficient to induce focal adhesion ass
220 t of CCG-1423, a small molecule inhibitor of SRF/MRTF-A-dependent transcription that exhibits efficac
221 s in the nuclear subcellular localization of SRF and MAL.
222  37 of 46 (80%) individuals; the location of SRF accumulation varied.
223                                      Loss of SRF leads to defects in B-cell and T-cell development.
224 xpression levels are maintained with loss of SRF, integrin activation and trafficking are disrupted.
225 and RS renal scintigraphy for measurement of SRF split renal function was shown in patients with mode
226             The detection and measurement of SRF were also highly accurate with an AUC of 0.92 (range
227 nscription factor SRF, and overexpression of SRF rescues impaired chromosome condensation and segrega
228                              The presence of SRF did not lead to permanent ocular sequelae.
229                     Agreement on presence of SRF was 87% and sub-RPE fluid was 80%, with more SD OCT
230                              The presence of SRF was common in study participants undergoing treatmen
231 ller total CNV leakage area, and presence of SRF.
232 t on visual recovery; however, recurrence of SRF during follow-up showed a tendency for an additional
233 g SRF activity antagonizes Myc repression of SRF target genes, attenuates Myc-induced apoptosis, and
234  half-time PDT showed complete resolution of SRF within 6 months after PDT, but 3 eyes that received
235 eyes for up to 8 years despite resolution of SRF.
236 ssociated with improved VA and resolution of SRF.
237 genomics approach to investigate the role of SRF (serum response factor) in the serum response of fib
238 eview focuses, in particular, on the role of SRF in myeloid maturation and neutrophil function.
239                Here we elucidate the role of SRF in neutrophils, the primary defense against infectio
240            The well-established dual role of SRF with alternative cofactors and responsiveness to two
241  factor (SRF) and set the chromatin state of SRF-targeted genes early growth response 1 (egr1) and c-
242 y to ECM stiffness, and compare with that of SRF/MAL, which is another important regulator of differe
243                                        Olfm2-SRF interaction promoted the dissociation of SRF from HE
244                  A protocol to adjudicate on SRF has been established by the central reading centre a
245                   Similar effects of PGE2 on SRF gene expression were observed in fibroblasts from th
246  revealing essential requirements of ongoing SRF/MRTF activity for maintenance of cerebral small vess
247 ying peripheral areas of previous or ongoing SRF and choroidal hyperpermeability that can assist in t
248 A via their respective DNA-binding partners (SRF and TEAD) and is therefore indirect, arising as a co
249 t Q8wks), and 52% (ranibizumab) of patients; SRF resolved in 75% (both aflibercept Q4wks/Q8wks) and 6
250 ow-up period, but 1 eye exhibited persistent SRF, which was resolved progressively during the 12 mont
251 s that received half-dose PDT had persistent SRF before loss to follow-up at months 5, 7, and 8 (P =
252   GSK-3 binds to and directly phosphorylates SRF on a highly conserved serine residue.
253  technique for the evaluation of predonation SRF and allows a reliable prediction of donor's PDKF.
254                The difference of predonation SRF between preserved and donated kidney was the lowest
255 ain of SRF and PTEN-SRF interaction promotes SRF binding to essential promoter elements in SM-specifi
256 s with the N-terminal domain of SRF and PTEN-SRF interaction promotes SRF binding to essential promot
257       Finally, we demonstrate that regulated SRF expression, in turn, is critical for the effects of
258  a mechanism by which RNase L down-regulates SRF-induced genes.
259 cofactor usage, leading to a PDGF-responsive SRF-driven transcriptional program in the midface.
260                                    Restoring SRF activity antagonizes Myc repression of SRF target ge
261 nd performed manual measurements of retinal, SRF, and subretinal tissue complex thicknesses at the fo
262 tical coherence tomographic imaging revealed SRF beneath the interdigitation zone.
263  absence of either RNase L or TTP stabilized SRF mRNA, and a subset of established TTP targets was al
264 k regulation in which RNase L and TTP target SRF mRNA and SRF-induced transcripts.
265                       Analysis of direct TCF-SRF target genes and chromatin modifiers confirmed this
266                          Here we report that SRF is phosphorylated and activated by GSK-3 to promote
267                We have previously shown that SRF is essential for megakaryocyte maturation and platel
268                                          The SRF pathway registers changes in G-actin levels, leading
269                                          The SRF target gene and actin-binding protein, vinculin, is
270 ly with increasing sound levels; and (3) the SRF size and centroid elevation were correlated with the
271                     RNase L destabilized the SRF transcript and formed a complex with SRF mRNA in cel
272                            Disruption of the SRF pathway results in myelodysplasia and immune dysfunc
273     These data support a central role of the SRF/MRTF pathway in the pathobiology of lung fibrosis an
274 led to MRTF nuclear shuttling to promote the SRF transcriptional activity required for entosis.
275                     We hypothesized that the SRF/MRTF pathway inhibitor CCG-203971 would modulate myo
276 peting directly with the MRTFs for access to SRF.
277 hrough interfering with myocardin binding to SRF.
278  of Ras homolog family member A signaling to SRF, results in aberrant myeloid differentiation and hyp
279                     The results suggested to SRF that gut microbiota have a causal role in carbohydra
280           Furthermore, PM blebbing triggered SRF-mediated up-regulation of the metastasis-associated
281                                     In turn, SRF cooperated with MEF2 to sustain the expression of LM
282 However, the molecular mechanisms underlying SRF-dependent axon growth remains unknown.
283 rane (Collagen IV), were down-regulated upon SRF depletion.
284 orrelation between predonation CT volumetric SRF of the preserved kidney and PDKF at day 3 was r = 0.
285                   Preoperation CT volumetric SRF was determined and compared with MAG3-SRF, postopera
286 alysis showed agreement between CT volumetry SRF and MAG3-SRF (bias, 95% limits of agreement: ROI vs
287 lone overcomes the MZ B cell deficiency when SRF is absent.
288 nt with MEK inhibitors is not indicated when SRF is present.
289 bleb dynamics for cell-in-cell invasion when SRF is suppressed.
290 ecessary for both Dc-ODP and Pc-ODP, whereas SRF and MEF2 are only needed for Dc-ODP.
291 the SRF transcript and formed a complex with SRF mRNA in cells providing a mechanism by which RNase L
292                                    Eyes with SRF in the foveal center on OCT had better mean VA than
293             Olfm2 physically interacted with SRF without affecting SRF-myocardin interaction.
294             CSRP2BP directly interacted with SRF, CRP2 and myocardin.
295 oters show enriched genome-wide overlap with SRF ChIP-seq peaks, PDGF selectively activates a network
296  transient, occurring in 9 participants with SRF (20%; 95% CI, 10%-33%).
297                      In the 26 patients with SRF, the fovea was affected in 85%.
298 nctioning as an indispensible regulator with SRF to maintain the differentiated SM phenotype.
299 tor for CRP2 that works synergistically with SRF and myocardin to regulate smooth muscle gene express
300 nt treatments compared with patients without SRF, without PVD, or without either who may require more

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