ライフサイエンスコーパス: SSR

1 SSR analysis allowed the detection of 157 alleles across

2 SSR dose-related suppression of the nickel CHS response

3 SSR growth requires Ral and the exocyst component Sec5 a

4 SSR had the same effect as cis-UCA, but trans-UCA had no

5 SSR loci also exhibit the highest percentage of 'extende

6 SSR loci are least conserved with 12.71 polymorphic base

7 SSR markers and Real Time qPCR analysis showed that diff

8 SSR processes provide a new alternative to understand th

9 SSR-based cluster analysis revealed that varieties with

10 SSRs (simple sequence repeats) have been shown to have a

11 SSRs are subject to slipped-strand mutations and a commo

12 SSRs can affect neuronal differentiation, brain developm

13 SSRs, on the other hand, are found at low frequency dist

14 d in a dense linkage map of 541 AFLP and 111 SSR markers distributed across 19 linkage groups.

15 The accuracies using 135 SSRs were low, with accuracies of the traits around 0.20

16 agronomic traits relative to 26 RFLP and 15 SSR chromosome 3A-specific markers on 95 single-chromoso

17 A total of 15 SSR amplicons unique to P. philippinensis (one of the po

18 2 molecular markers (538 Bin, 258 AFLPs, 175 SSRs, and an EST).

19 The difference was greatest at 3.8 J/cm(2) SSR [mean difference: 11% (95% CI: 0.5%, 21.4%)].

20 agonists such as nicotine, choline, GTS-21, SSR-180711A and PNU-282987 in the presence of the positi

21 enerated in this study was screened using 22 SSR markers located on linkage group 2 which contains a

22 recting for the multiple tests performed, 25 SSR loci showed evidence of selection.

23 We identified and characterized 2749 SSRs of 963 SSR types in the genome of N. crassa.

24 rent study, we simultaneously genotyped 3105 SSRs in eight rice varieties, which were further validat

25 ingerprints of the rice varieties using 3105 SSRs, which offer much greater discriminative power than

26 s in variation of amino acid repeats, and 4) SSRs are stable molecular markers for genetic studies in

27 A set of 400 SSR primer combinations were tested for amplification an

28 constructed using 2,467 SLAF markers and 43 SSRs, which were distributed on seven linkage groups spa

29 Among 2,431 SSR markers located across the whole peanut genome scree

30 uch greater discriminative power than the 48 SSRs commonly used for rice.

31 WI [CNN(WI3A)], with 142 RFLP probes and 55 SSR markers revealed that the extent of polymorphism is

32 A total of 1,470 markers (580 SSRs and 890 STSs) were anchored by PCR on a subset of a

33 A genetic linkage map composed of 841 SSR, AFLP, and RAPD markers and phenotypic data from 310

34 We identified 12,956 SSRs, including 2,594 repeats for which primers were des

35 dentified and characterized 2749 SSRs of 963 SSR types in the genome of N. crassa.

36 ntial have been reported, we now show that a SSR also impacts small RNA-mediated posttranscriptional

37 haracterized the distribution of 19 abundant SSR types (AST), which account for 71% of total SSRs in

38 netics and forensics, which require accurate SSRs to calculate intra- and inter-species interactions.

39 tinic acetylcholine receptor (nAChR) agonist SSR 180711 (3.0 mg/kg) eliminated the TTX-induced perfor

40 We present Kmer-SSR, which finds all SSRs faster than most heuristic SSR identification algor

41 Comparisons among SSRs of different lengths suggest that LSSR(1-4) are lik

42 AmpSeq-SSR is able to genotype potentially more than a million

43 l method for SSR genotyping, named as AmpSeq-SSR, which combines multiplexing polymerase chain reacti

44 results showed that the accuracies of AmpSeq-SSR were nearly 100 and 94% with a single base resolutio

45 To demonstrate the power of AmpSeq-SSR, we adopted it in two applications.

46 the work we present focused on rice, AmpSeq-SSR can be readily extended to animals and micro-organis

47 Again, 50% had an LLR, but none had an SSR.

48 he method and a case study with 201 AFLP and SSR markers scored on 228 full-sib individuals of autote

49 sing a full-sib population of 238 plants and SSR and STS markers to access the degree of preferential

50 f SNPs (single nucleotide polymorphisms) and SSR (simple sequence repeat) variation as a means of tra

51 h that combines Phenotype MicroArray(TM) and SSR genotyping appeared useful to assess the performance

52 The genes up-regulated by both cis-UCA and SSR were associated with apoptosis, cell growth arrest,

53 pid peroxidation were induced by cis-UCA and SSR, but not trans-UCA, suggesting possible upstream eve

54 netic map comprising over 600 SFPs, GEMs and SSRs.

55 suggest a diverse array of interspersed and SSRs in the common ancestor of amniotes and a genomic co

56 etween asymptomatic sensitization, LLRs, and SSRs.

57 , the development of traditional 'anonymous' SSRs from genomic DNA is costly and time-consuming.

58 Because SSRs are commonly located in promoters, untranslated reg

59 From 40 BES-SSRs based on long motifs SSRs (>/= trinucleotides) anal

60 In total, 3,789 BES-SSRs were identified, with a distribution of one SSR (si

61 This study identified highly polymorphic BES-SSRs containing tri- to hexanucleotides motifs and bring

62 cally between chromosomes as well as between SSR types, 2) the size variation of tri-nt SSRs in exons

63 atistically significant associations between SSRs and gene functional classifications were detected,

64 fic SNP genotyping primers, and Brachypodium SSR flanking primers in several genome projects have bee

65 hereditary neurodegenerative disorders, but SSR alleles can also contribute to normal variation in b

66 h and the percentage of immunosuppression by SSR was calculated.

67 thousands of transgenic insertions carrying SSR recognition sites have been distributed throughout t

68 We thoroughly characterize SSR density and variability as a function of N-->C posit

69 of tri-nucleotide (nt) SSRs, the most common SSRs in N. crassa, was significantly biased in exons.

70 In this study, we compared SSRs in housekeeping and tissue-specific genes in human

71 ile being the most comprehensive and correct SSR detection software available.

72 Public cotton SSR libraries (17,343 markers) were curated for sequence

73 rocess that generalises the recently defined SSR processes and is able to produce power laws with arb

74 high-throughput software tool for designing SSR flanking primers and SNP genotyping primers is incre

75 mmon prefix arrays for efficiently detecting SSRs in large sets of sequences.

76 is the first attempt to extensively develop SSR markers from Peronosclerospora genomic DNA.

77 The newly developed SSR markers can be readily used to distinguish isolates

78 between GC content and the span of (AG)n-Di-SSR variation.

79 tology (GO) terms with abundance of (AG)3-Di-SSRs shared by the VP strains were associated with purin

80 he numbers of triplexes formed from (AG)n-Di-SSRs between VP and N_VP in Mycoplasma suggested the pot

81 sma suggested the potential role of (AG)n-Di-SSRs in gene regulation.

82 Di-amino acids coded by (AG)n-Di-SSRs included all three six-fold code amino acids (Arg,

83 of (AG)n dimeric simple sequence repeats (Di-SSRs) in the genomes, coding and non-coding regions than

84 d Syndapin colocalize with Past1 in distinct SSR subdomains and collapse into Amphiphysin-dependent m

85 ast, the sheep isolates representing diverse SSR genotypes closely resembled the global gene expressi

86 number of different bands detected for each SSR primer pair using a LI-COR- DNA Analyzer ranged from

87 and polymorphism information content of each SSR locus was calculated with an adapted formula taking

88 ng fruiting body development and develop EST-SSR markers assessing the genetic value of breeding mate

89 developed 26 polymorphic and informative EST-SSR markers to assess the genetic diversity in 82 strain

90 variation (no more than two variants per EST-SSR genotype) and recently published chromosomal evidenc

91 sed-sequence tag simple-sequence repeat (EST-SSR)) primers to survey genetic variation in populations

92 at is known about the transferability of EST-SSRs from one taxon to another with particular reference

93 cuss the advantages and disadvantages of EST-SSRs in the context of population genetic applications.

94 These EST-SSRs exhibited high transferability in closely related s

95 100% recall and accurately identifies every SSR of any specified length.

96 Existing SSR detection applications are hampered by one or more l

97 steomyelitis and cystic fibrosis carry fewer SSRs than do isolates from patients with acute infection

98 . trichocarpa genome assembly using flanking SSR markers with known physical positions.

99 c situations and it should be considered for SSR computation.

100 etic marker, we developed a novel method for SSR genotyping, named as AmpSeq-SSR, which combines mult

101 However, the existing methods for SSR genotyping suffer from low sensitivity, low accuracy

102 ysis demonstrates that miR-8 is required for SSR morphogenesis.

103 lated, positively for TEs and negatively for SSRs, with relative nucleosome binding affinities along

104 a 9.5-fold higher risk for LLRs but not for SSRs.

105 erved between species than TFBS derived from SSRs and non-repetitive DNA.

106 We have used the FLP/FRT SSR system to provide a proof of concept, though any of

107 These findings provide new genes, SSR markers, and germplasm to enhance the breeding of co

108 Genic-SSR's coupled with the functional annotations provided b

109 Of the 80 genic-SSR primer sets, 62 were amplified in C. ensifolium succ

110 The 7,936 genic-SSR markers were identified.

111 were analyzed based on 55 polymorphic genic-SSR data among 59 accessions.

112 nce repeats derived from gene regions (genic-SSR).

113 The genic-SSR identified in this study constitute a set of markers

114 Unigenes containing the 62 genic-SSRs were searched against Non-redundant (Nr), Gene Onto

115 A total of 80 genic-SSRs were selected, and primers were designed according

116 Only 5 (5.3%) patients had SSRs, but 41 (43.6%) had large local reactions (LLRs) af

117 l homozygosity underlying fixed heterozygote SSR genotypes, which in turn reflect a selfing mating sy

118 ch finds all SSRs faster than most heuristic SSR identification algorithms in a parallelized, easy-to

119 We discuss several applications showing how SSR processes can be used to understand Zipf's law in wo

120 tions, accurately and intuitively identifies SSRs quickly and in a more user-friendly manner than any

121 Here we review studies implicating SSRs not just in disease but also in circadian rhythmici

122 we show that imposing energy conservation in SSR cascades allows us to recover Fermi's classic result

123 olerance were performed with 267 informative SSR markers in this study.

124 Kmer-SSR, coupled with the filter options, accurately and int

125 The exhaustive Kmer-SSR option has 100% precision and 100% recall and accura

126 We present Kmer-SSR, which finds all SSRs faster than most heuristic SSR

127 eta11 (imp-beta11) and dfz2 mutants had less SSR, and some boutons lacked the postsynaptic marker Dis

128 Long SSRs (LSSRs) are common in eukaryotes but rare in most p

129 Long SSRs in Mycobacterium leprae are mostly associated with

130 erally rare in prokaryotic genomes, and long SSRs composed of iterations of mono-, di-, tri-, and tet

131 results concerning associations between long SSRs and genes related to different cellular functions i

132 ons were detected, suggesting that most long SSRs are not related to a particular cellular function o

133 However, we also found a number of long SSRs associated with housekeeping genes, including rRNA

134 Significantly long SSRs are generally rare in prokaryotic genomes, and long

135 A lower SSR using CE was defined as the best scenario for using

136 th greater effect than the RE leads to lower SSR using the CE regardless of the AE proportion and the

137 or their placement with independently mapped SSRs, demonstrating the previously un-utilized power tha

138 Segregation of 193 microsatellite (SSR) loci was evaluated in the F(1) progeny of 169 full-

139 genotypes and an established microsatellite (SSR) framework map, we produced a high-density genetic m

140 to genotype potentially more than a million SSRs at once using the current sequencing techniques.

141 SSR is 100% accurate and detected >1000 more SSRs than the second best algorithm, while offering grea

142 From 40 BES-SSRs based on long motifs SSRs (>/= trinucleotides) analyzed in high-resolution ge

143 sporter 1 (GlyT1) inhibitors including NFPS, SSR 504734, Lu AA21279, Org 25935, SB-710622, GSK931145,

144 Noisy SSR processes further allow us to explain a wide range o

145 n SSR types, 2) the size variation of tri-nt SSRs in exons might be an important mechanism in generat

146 The distribution of tri-nucleotide (nt) SSRs, the most common SSRs in N. crassa, was significant

147 her Populus linkage studies, and addition of SSR loci from the poplar genome project provided coarse

148 Moreover, comparison of SSR-based data with phenylpropanoid molecules exhibited

149 The density of SSR in the walnut genome analyzed was also slightly high

150 Given the high density of SSR recognition-site insertions in Drosophila, our metho

151 nt immunity, revealing the complex nature of SSR resistance.

152 The peak of SSR density is centered around the -35 bp position where

153 to account for miR-8-mediated regulation of SSR architecture, consistent with its localization in th

154 ective of this study was to develop a set of SSR markers useful for genetic studies in sainfoin and t

155 Specificity of SSR markers was verified using controls consisting of: i

156 Differences among genomes in terms of SSR representations and their possible interpretations a

157 ecific genes in human and mouse, in terms of SSR types and distributions in different regions includi

158 ence repeats (SSRs) constitutes the basis of SSRs as an effective genetic marker with various applica

159 d 2,000 were suitable for the development of SSRs, of which 194 were evaluated in low-resolution scre

160 tive variability and density distribution of SSRs in coding regions.

161 enic bacteria, we observe over enrichment of SSRs near protein N-termini significantly beyond expecta

162 Although several examples of SSRs that affect transcription or coding potential have

163 which is expected if the primary function of SSRs is their contribution to antigenic variation.

164 o study evolution and biological function of SSRs.

165 gnaling in eradication of S. aureus, loss of SSRs may represent an advantageous mechanism to adapt to

166 an inverse correlation between the number of SSRs and the length of disease course.

167 and immune cells, depending on the number of SSRs, which leads to differences in neutrophil recruitme

168 In order to fully exploit the potential of SSRs as genetic marker, we developed a novel method for

169 We analyze representations of SSRs in >300 prokaryotic genomes and report significant

170 o Hymenoptera venoms was common, the risk of SSRs in sensitized subjects was low in our study.

171 d that ESTs are a potentially rich source of SSRs that reveal polymorphisms not only within the sourc

172 that allow users to easily view a subset of SSRs based on user input.

173 karyotes as well as among different types of SSRs.

174 We also characterized the size variation of SSRs among natural accessions using Polymorphic Index Co

175 was to adapt a genotyping approach based on SSR (Simple Sequence Repeat) marker to a discriminating

176 bly amplify the aerosol extinction effect on SSR.

177 n and wind stilling amplification effects on SSR variations at a decadal scale.

178 equence, we assess evolutionary pressures on SSRs.

179 were identified, with a distribution of one SSR (simple sequence repeat) per 8.36 kbp and 2,000 were

180 n a more user-friendly manner than any other SSR identification algorithm.

181 roof of concept, though any of several other SSR systems could be used in the same way.

182 In pathogens, SSRs can enhance antigenic variance of the pathogen popu

183 th an average of 4.8 polymorphic markers per SSR across 34 Peronosclerospora, Peronospora and Scleros

184 To identify more biologically pertinent SSRs, we also developed several filters that allow users

185 Using polymorphic SSRs, we have built linkage maps from three line-cross p

186 of primer designs including generic primers, SSR primers together with SSR detection, and SNP genotyp

187 In prokaryotes, SSR loci can cause phase variation, a microbial survival

188 also a module available for finding putative SSRs in the analysed datasets.

189 tive SNPs and 184 unigenes with high quality SSRs, of which 42% were novel to Prunus.

190 or 7.6 J/cm(2) of solar-simulated radiation (SSR) and to 3 unexposed control sites.

191 red the effect of solar-simulated radiation (SSR) on the elicitation phase of contact hypersensitivit

192 s were induced by solar-simulated radiation (SSR), 16 of which were also up-regulated by cis-UCA.

193 It was found that surface solar radiation (SSR) decreased considerably with wind stilling in heavil

194 The survival rate (SSR) and success rate (SR) were calculated using life-ta

195 tera venom without systemic sting reactions (SSRs) is commonly observed in the general population.

196 15-crown-5 ether, a selective shift reagent (SSR), was added after the last chromatographic dimension

197 Site-specific recombinases (SSRs) are valuable tools for manipulating genomes.

198 scribe a simple site-specific recombination (SSR) strategy that simultaneously removes pre-defined DN

199 Sample Space Reducing (SSR) processes are simple stochastic processes that offe

200 emonstrate that these sample-space-reducing (SSR) processes necessarily lead to Zipf's law in the ran

201 (referred as to single surface referencing (SSR)) uses a single functionalized surface split into th

202 sin gene (RH1) and 9 microsatellite regions (SSRs) were amplified and analysed.

203 Among all these regions, SSRs in the 5'-UTR show the most distinction between hou

204 ors, with 50 nuclear simple sequence repeat (SSR) (also known as microsatellite) primer pairs and a p

205 ted QTL, we analyzed simple sequence repeat (SSR) diversity from 102 markers dispersed throughout the

206 n many forest trees (simple sequence repeat (SSR) FST=0.21), with major genetic groups corresponding

207 , regardless of their short sequence repeat (SSR) genotype, induced similar global gene expression pa

208 DNA, genomic DNA and simple sequence repeat (SSR) loci were re-sequenced in Gossypium herbaceum (A1 g

209 were genotyped at 25 simple-sequence repeat (SSR) loci.

210 andraces assessed by simple sequence repeat (SSR) markers and allelic variation at the GmTfl1 locus s

211 er analysis with 151 simple sequence repeat (SSR) markers in 90 individuals of the population indicat

212 iously characterised Simple Sequence Repeat (SSR) markers to estimate genetic differentiation across

213 639033]), developing simple sequence repeat (SSR) markers, and identifying single nucleotide polymorp

214 ed on a whole-genome simple sequence repeat (SSR)-based genetic map and on mapping sex determination

215 M in the preexisting simple sequence repeat (SSR)-based map, while 111 genes mapped in 19 of the 26 g

216 surface antigens by simple sequence repeat (SSR)-mediated phase variation.

217 Microsatellite (simple sequence repeat - SSR) and single nucleotide polymorphism (SNP) markers ar

218 Simple sequence repeats (SSR) from BESs were analyzed and found to be more abunda

219 ait loci (QTLs) and simple sequence repeats (SSR) markers linked to three QTLs for PM resistance.

220 orphisms (AFLP) and simple sequence repeats (SSR)] we have generated genetic linkage maps for Physcom

221 stribution of SNPs, simple sequence repeats (SSRs) and isozyme alleles and a graphical view of the pl

222 tems evaluated were simple sequence repeats (SSRs) and single nucleotide polymorphisms (SNPs).

223 sequences including simple sequence repeats (SSRs) and TEs.

224 Simple sequence repeats (SSRs) are indel mutational hotspots in genomes.

225 Simple Sequence Repeats (SSRs) are used to address a variety of research question

226 cient genotyping of simple sequence repeats (SSRs) constitutes the basis of SSRs as an effective gene

227 Simple sequence repeats (SSRs) have been successfully used for various genetic an

228 Simple-sequence repeats (SSRs) have increasingly become the marker of choice for

229 Simple sequence repeats (SSRs) in DNA sequences are composed of tandem iterations

230 Simple sequence repeats (SSRs) in DNA sequences are tandem iterations of a single

231 ion of hypermutable simple sequence repeats (SSRs) is a widespread and stochastic mechanism to genera

232 ct, by carrying out simple sequence repeats (SSRs) markers analysis, we characterise the genetic prof

233 f retroelements and simple sequence repeats (SSRs) not found in the chicken.

234 lyzed the multilocus short sequence repeats (SSRs) of 211 and 56 isolates of Mycobacterium avium subs

235 hether the number of short sequence repeats (SSRs) present in the polymorphic region modulates the in

236 n instances such as Simple Sequence Repeats (SSRs), a 'good enough' solution may not accurately portr

237 informative marker simple sequence repeats (SSRs), as long as a sufficient number of loci are employ

238 .5% of BESs contain simple sequence repeats (SSRs), most of which are AT/TA dimer repeats.

239 Simple sequence repeats (SSRs), sometimes described as genetic 'stutters,' are DN

240 ymerase slippage of simple sequence repeats (SSRs), to generate phenotypic variation and enhanced fit

241 etrotransposons and simple sequence repeats (SSRs).

242 elements (TEs) and simple sequence repeats (SSRs).

243 I) were scanned for Simple Sequence Repeats (SSRs); unique SSRs were then tested for amplification an

244 a binary CE on the sample size requirement (SSR) has not been addressed.

245 h is sometimes possible with high-resolution SSR markers.

246 f the subsynaptic muscle membrane reticulum (SSR) at the Drosophila melanogaster larval neuromuscular

247 functions within the subsynaptic reticulum (SSR) surrounding presynaptic terminals.

248 ic development of the subsynaptic reticulum (SSR), an elaboration of the postsynaptic plasma membrane

249 plasma membrane (the subsynaptic reticulum, SSR).

250 d elements (LINEs) and a paucity of A-T rich SSRs, characterize turtles and archosaur genomes, wherea

251 issect the genetics of Sclerotinia stem rot (SSR) [caused by Sclerotinia sclerotiorum (Lib.) de Bary]

252 SA-SSR is 100% accurate and detected >1000 more SSRs than t

253 Here, we present an innovative algorithm, SA-SSR, based on suffix and longest common prefix arrays fo

254 rich repeats near termini, yielding U-shaped SSR density curves.

255 We show that a single SSR reaction can simultaneously carry out double excisio

256 he integrated linkage map with gene-specific SSR markers coupled with computational prediction of AFL

257 One hundred and seventy-nine gene-specific SSR markers were mapped in 46 linkage groups, and 1574 p

258 Specifically, SSR densities in 5'-UTRs in housekeeping genes are about

259 Within each stratum, SSR was computed for multiple scenarios by varying the e

260 In the case of the studied SSRs, the results allowed the initial characterisation a

261 ped to linkage group (LG) 3 of the sunflower SSR map.

262 recting the choice of the most appropriate T-SSR system for genetic engineering include that, wheneve

263 Tyrosine-type site-specific recombinases (T-SSRs) have opened new avenues for the predictable modifi

264 in our mechanistic understanding of simple T-SSRs and their application in developmental and syntheti

265 This finding supports a hypothesis that SSR polymorphism has functional implications.

266 Taken together, our results suggest that SSRs may have an effect on gene expression and may play

267 The SSR markers characterized and tested in this study provi

268 the relative overlap, can help to define the SSR in specific situations and it should be considered f

269 hods were successfully applied to design the SSR and to support the experimental data.

270 mphiphysin-dependent membrane nodules in the SSR of past1 mutants.

271 Integrating the SSR- and AFLP-based maps generated 31 linkage groups com

272 the elaboration of increasing layers of the SSR and uncover new roles for an EHD protein at synapses

273 ectly corresponds to the mixing ratio of the SSR process and noise.

274 during persistent carriage by analyzing the SSRs of eight loci in multiple isolates from 21 carriers

275 we conclude that 1) the distributions of the SSRs in the sequenced N. crassa genome differ systematic

276 f LSSR(5-11) in some genomes may reflect the SSRs' general tendency to expand rather than their speci

277 These SSRs are located within the reading frame or in the prom

278 These SSRs are stable and suitable for studying the molecular

279 From this SSR dependence on wind speed, we further derived proxies

280 to determine their genetic diversity through SSR markers.

281 We show that by adding noise to SSR processes the corresponding rank distributions remai

282 types (AST), which account for 71% of total SSRs in the N. crassa genome, using a Poisson log-linear

283 ific genes and CGG, the dominant type of tri-SSR in 5'-UTR, accounts for 74-79% of the tri-SSRs in ho

284 Tri-SSRs in 5'-UTRs of housekeeping genes are more GC rich t

285 SR in 5'-UTR, accounts for 74-79% of the tri-SSRs in housekeeping genes, as compared to 42-57% in tis

286 75% of the tri-SSRs in the 5'-UTR of housekeeping genes have 4-5 repeat

287 -mediated site-specific recombination" (tRNA-SSR) for this mechanism.

288 g that these species may evolutionarily tune SSR positions in coding regions to facilitate phase vari

289 large genomic segment that lies between two SSR recognition-site insertions can be "captured" as a t

290 d to the distal part of chr.3 flanked by two SSR markers within an interval of 1.8 cM corresponding t

291 arated by approximately 6 cM, along with two SSR markers.

292 d for Simple Sequence Repeats (SSRs); unique SSRs were then tested for amplification and polymorphism

293 Second, using SSRs and putatively neutral sequenced loci, coalescent s

294 upstream of the TSS at the same point where SSR frequency is at its highest.

295 icant epistatic interactions associated with SSR resistance, with candidate genes involved in a wide

296 g generic primers, SSR primers together with SSR detection, and SNP genotyping primers (including sin

297 , at least some of the genes associated with SSRs encode potential antigens, which is expected if the

298 ulation might also apply to other genes with SSRs, which could be targeting sites of cis- or trans-en

299 rresponding SR of 93.9%, whereas the 16-year SSR and SR for the R implants were 97.2% and 95.2%, resp

300 The M implants had a 16-year SSR of 95.7% and a corresponding SR of 93.9%, whereas th