ライフサイエンスコーパス: ST segment depression

1 r systemic inflammation, blood pressure, and ST segment depression.

2 ring which the patient developed > or = 1-mm ST-segment depression.

3 e, smoking status, diabetes, heart rate, and ST-segment depression.

4 elevation or bundle branch block, and 7% had ST-segment depression.

5 ischemia, as shown by a decrease in maximal ST-segment depression (1.84+/-0.14 versus 1.63+/-0.13 mm

6 diographic abnormalities (13 of 15 rabbits), ST-segment depression (10 of 15 rabbits), and preventric

7 hest in the patients achieving <10 METs with ST-segment depression (14 of 70, 19.4%).

8 d self-reported symptoms and reduced maximal ST segment depression (-142 vs. -156 muV, p = 0.046) ove

9 0.83 mm, P=0.027), and time to resolution of ST-segment depression (229+/-30 versus 207+/-28 s, P=0.0

10 529 +/- 190 s, p = 0.002) and longer time to ST segment depression (530 +/- 215 vs. 419 +/- 205 s, p

11 ssion compared with patients without resting ST segment depression (71 +/- 6% vs. 42 +/- 4%) and spec

12 lysis compared with patients without resting ST segment depression (77 +/- 7% vs. 45 +/- 4%) and spec

13 rdiovascular death risk for exercise-induced ST-segment depression (age-adjusted hazard ratio, 1.02;

14 rval, a Q wave, abnormal QRS axis deviation, ST segment depression and a pathologic T wave more frequ

15 ng, left ventricular cavity dilation, marked ST segment depression and blood pressure decrease indica

16 t in a large cohort of patients with resting ST segment depression and no prior myocardial infarction

17 increased significantly before the onset of ST segment depression and persisted throughout the ische

18 tes, hypertension, smoking history, baseline ST segment depression and previous coronary artery bypas

19 he subgroup (n = 29) with dobutamine-induced ST-segment depression and abnormal SPECT imaging, the pr

20 were independently predictive of mortality (ST-segment depression and age > or = 65 years).

21 croV of additional horizontal or downsloping ST-segment depression and by an ST-segment/heart rate (S

22 nts decreases heart rate variability, causes ST-segment depression and endothelial dysfunction, incre

23 prolongation of the time to exercise-induced ST-segment depression and resolution of myocardial perfu

24 axis, Sokolow-Lyon and Cornell voltages, and ST-segment depression and slope.

25 was to investigate the relationship between ST-segment depression and the rate-pressure product (RPP

26 ficacy criteria were changes in time to 1-mm ST-segment depression and time to limiting angina during

27 Time to onset of > or = 1-mm ST-segment depression and time to onset of angina were s

28 iables--diabetes, hypertension, magnitude of ST segment depression, and exercise rate-pressure produc

29 eline characteristics, number of episodes of ST-segment depression, and baseline serum cholesterol le

30 ST-segment depression, appearing in unfiltered bipolar e

31 Chest pain and ST segment depression are extremely common findings in p

32 ulated for patients with and without resting ST segment depression as determined by visual or compute

33 When QTD of > 60 ms was added to ST-segment depression as a condition for positive test,

34 Familial history of sudden cardiac death and ST-segment depression associated with PTWI were predicti

35 mg/h) significantly reduced the magnitude of ST segment depression at angina onset during ETT compare

36 was used to group patients as follows: 1) no ST segment depression at any time (control group); 2) ST

37 ro V of additional horizontal or downsloping ST segment depression at peak exercise.

38 was 61 +/- 10 years; 97% were male; 38% had ST segment depression at study entry; 30% had an anterio

39 ercise duration at peak and in times to 1 mm ST-segment depression at trough and peak and to angina a

40 ere exercise duration and time to > or =1-mm ST-segment depression, average daily anginal attack coun

41 There were two patients with ST-segment depression before and one after caffeine inta

42 ted risk > or =20%, failure to reach THR and ST-segment depression both more than doubled the risk of

43 ents with CAD with profound exercise-induced ST segment depression but good ETT duration is an approp

44 The classification of resting ST segment depression by method of analysis (visual vs.

45 dicators of ischemia during the speech test (ST-segment depression, chest pain) did not predict death

46 Duke score, including ST-segment depression, chest pain, and exercise capacity

47 on results from the exercise test, including ST-segment depression, chest pain, and exercise duration

48 test increased in 349 patients with resting ST segment depression compared with patients without res

49 At 12-min exercise, the amount of ST-segment depression compared with placebo and controll

50 icant reduction in the number of episodes of ST-segment depression compared with the placebo group.

51 ographic criteria, including left precordial ST segment depression, complete atrioventricular heart b

52 es performance of the simple and HR-adjusted ST-segment depression criteria for the identification of

53 levation criteria and those only meeting the ST-segment depression criteria.

54 test increased in 206 patients with resting ST segment depression determined by visual ECG analysis

55 ST-segment depression did not predict mortality.

56 However, ST-segment depression did occur in two patients requirin

57 419 +/- 205 s, p = 0.0001) and less frequent ST segment depression during ambulatory monitoring (9% v

58 Patients with stable symptoms but profound ST segment depression during ETT are often referred for

59 comes in patients with profound (> or =2 mm) ST segment depression during exercise treadmill testing

60 utine daily activities was best predicted by ST segment depression during mental or bicycle exercise

61 tal or exercise stress is more predictive of ST segment depression during routine daily activities th

62 The presence of ST segment depression during routine daily activities wa

63 and systolic blood pressure changes at 1-mm ST segment depression during treadmill exercise testing

64 In fact, asymptomatic (or silent) ST-segment depression during ambulatory electrocardiogra

65 ics have a higher prevalence of asymptomatic ST-segment depression during exercise treadmill testing

66 ients with coronary artery disease (CAD) and ST-segment depression during exercise were studied befor

67 he duration of exercise at the onset of 1-mm ST-segment depression during exercise, also remained unc

68 Eleven (15%) of 71 patients had ischemic ST-segment depression during exercise, whereas 27 patien

69 d 63% (P<.01), respectively, for > or = 1 mm ST-segment depression during stress.

70 rcentages of patients with only asymptomatic ST-segment depression during the 48-hour AECG were simil

71 Among asymptomatic men, ST-segment depression, failure to reach THR, and exercis

72 Adjustment of ST-segment depression for the change in HR with exercise

73 dex was calculated by dividing the change in ST segment depression from rest to peak exercise by the

74 rogen levels and an increase in time to 1-mm ST-segment depression from (mean+/-SEM) 309+/-27 seconds

75 ess imaging procedure for patients with rest ST-segment depression greater than 1 mm, complete left b

76 exercise electrocardiography was defined by ST segment depression > 0.1 mV, ischemia by exercise ech

77 ST segment depression > or = 1 mm was more common with e

78 (reversible left ventricular dysfunction or ST segment depression > or = 1 mm) developed in 106 of 1

79 ssociated with a 1.50-fold increased risk of ST-segment depression > or =0.1 mm (95% CI, 1.19 to 1.89

80 s of angina, and time to ischemic threshold (ST-segment depression > or =1 mm from baseline) during e

81 d relative to 3 exercise test variables: (1) ST-segment depression > or =1 mm, (2) failure to achieve

82 >/= 8%; and/or 3) horizontal or downsloping ST-segment depression >/= 1 mm in 2 or more leads lastin

83 h exercise-induced horizontal or downsloping ST-segment depression >/=1 mm, and 38 subjects (age, 77.

84 ST-segment depression >/=50 microV (n = 127) and CRP >7.

85 The presence of either major ST segment depression (>2 mm) or elevation on the admiss

86 Importantly, the time to ST-segment depression (>/=1 mm) also increased from 327+

87 Heart rate adjustment of the magnitude of ST segment depression has been proposed as an alternativ

88 fixed magnitude of horizontal or downsloping ST segment depression has demonstrated only limited diag

89 ge and Framingham CHD risk score, among men, ST-segment depression (hazard ratio [HR], 1.88; 95% CI,

90 dered as a continuous variable and age, sex, ST-segment depression, heart failure, previous revascula

91 ncluding younger age of onset, more frequent ST-segment depression, higher prevalence of neurologic d

92 le heart rate adjustment of the magnitude of ST segment depression improves the prediction of death f

93 nd heart rate changes related to episodes of ST segment depression in 17 patients with stable coronar

94 ST segment depression in leads other than those showing

95 ST segment depression in response to mental or exercise

96 Consideration of ST-segment depression in addition to elevation increased

97 effects of sub-endocardial ischaemia on the ST-segment depression in ECG to patterns of transmural a

98 onitoring occurs more often than symptomatic ST-segment depression in patients with coronary artery d

99 0.14 versus 1.63+/-0.13 mm, P=0.011), sum of ST-segment depressions in 12 leads (7.64+/-1.01 versus 6

100 er-protocol population (n=257), time to 1-mm ST-segment depression increased in the 5 and 10 mg BID g

101 Time to > or =1-mm ST-segment depression increased significantly from basel

102 ST-segment depression is associated with increased expos

103 Whether exercise-induced ST-segment depression <1 mm is an independent predictor

104 The risk of pollution-associated ST-segment depression may be greatest in those with myoc

105 ents reaching > or =10 METs without exercise ST-segment depression, none had > or =10% LV ischemia.

106 left ventricular cavity dilation and marked ST segment depression, occur more often with exercise th

107 At a matched specificity of 96%, ST-segment depression of > 160 microV identified CAD wit

108 ved the sensitivity of each method: adjusted ST-segment depression of > 176 had a sensitivity of 87%

109 Neither the number of Q waves, residual ST-segment depression of >or=0.5 mm or elevation of >or=

110 ing was defined as horizontal or downsloping ST-segment depression of 1 mm (0.1 mV) or more for 1 min

111 % or more; and/or horizontal or down-sloping ST-segment depression of 1 mm or more in 2 or more leads

112 (in the same direction as) the QRS complex; ST-segment depression of 1 mm or more in lead V1, V2, or

113 or = 4 ischemic episodes or > or = 20 min of ST segment depression on 72-h electrocardiogram were ran

114 total exercise time, frequency of angina and ST segment depression on exercise.

115 final multivariate model included older age, ST segment depression on presentation, history of compli

116 ssess the effect of heart rate adjustment of ST segment depression on risk stratification for the pre

117 tudy is to demonstrate the effect of resting ST segment depression on the diagnostic characteristics

118 ous studies evaluating the effect of resting ST segment depression on the diagnostic characteristics

119 or of AECG abnormalities was the presence of ST segment depression on the initial exercise treadmill

120 1 and 327 mg/dL, and at least one episode of ST-segment depression on ambulatory ECG monitoring.

121 Of the ARCA patients, 2 had inferior ST-segment depression on EST; subsequently, 1 had normal

122 ated initial serum cardiac biomarker levels, ST-segment depression on presenting electrocardiogram, a

123 Time to 1-mm ST-segment depression on treadmill exercise testing and

124 uggestive of myocardial ischemia, defined as ST segment depression or elevation of >/=1 mm in two con

125 positive exercise stress tests (> or = 1 mm ST-segment depression or reversible perfusion defects) a

126 1.02 per 1 mg, P = 0.0001), and presence of ST-segment depression (OR = 2.59, P = 0.007 and OR = 2.3

127 Such patients represent a high-risk (ST-segment depression) or low-risk (normal or nonspecifi

128 greater than the 87% sensitivity of adjusted ST-segment depression (P < .0001).

129 after nitroglycerin administration with less ST-segment depression (P=0.003) and therefore myocardial

130 gment elevation (simultaneous group); and 3) ST segment depression persisting after ST segment elevat

131 a positive troponin at baseline, diabetes or ST-segment depression, recurrent angina, prior aspirin u

132 Blinded analysis of ST segment depression resolution patterns was used to gr

133 We hypothesized that the timing of ST segment depression resolution relative to ST segment

134 t depression at any time (control group); 2) ST segment depression resolving simultaneously with ST s

135 Mean exercise duration to 1-mm ST-segment depression significantly increased in each gr

136 most useful electrocardiogram findings were ST-segment depression (specificity, 95%; LR, 5.3 [95% CI

137 c relationship between baseline quantitative ST-segment depression (ST) and cardiac troponin T (cTnT)

138 he treating provider to identify significant ST-segment depressions, ST-segment elevations, or T-wave

139 ST segment depression (STD) >/=50 micro V and rate-corre

140 protein (CRP) and electrocardiographic (ECG) ST-segment depression (STD) have additive utility for pr

141 nges in T-wave polarity, > or =1 mm STE, and ST-segment depression (STD) were 22, 24, 29, and 35 s, r

142 arction (AMI) and the application of similar ST-segment depression (STEMI-equivalent) criteria with c

143 ECG changes were defined as Q waves, ST-segment depression, T-wave changes, ventricular condu

144 assigned at admission-ST-segment elevation, ST-segment depression, T-wave inversion, or the presence

145 minutes, P < .01), and the maximum depth of ST-segment depression tended to be less in the diabetic

146 imultaneous versus independent resolution of ST segment depression that occurs concomitant with ST se

147 azine produced a dose-dependent reduction in ST-segment depression that became more marked as exercis

148 agnostic value of adjusting the magnitude of ST-segment depression, the ST-segment (ST)/HR index, and

149 of Ad5FGF-4 on total ETT time, time to 1 mm ST-segment depression, time to angina, and Canadian Card

150 rt the use of heart rate-adjusted indexes of ST segment depression to improve the predictive value of

151 nse and intensification of minor preexercise ST-segment depression to levels > or =1 mm independently

152 coronary artery disease and exercise-induced ST-segment depression underwent assessment of forearm en

153 mental or bicycle exercise stress, although ST segment depression was rare during mental stress.

154 se, achieving > or =10 METs with no ischemic ST-segment depression was associated with a 0% prevalenc

155 Exercise-induced ST-segment depression was associated with increased IMT

156 ST-segment depression was completely resolved in 13 of 2

157 Time to > or =1 mm ST-segment depression was increased with fasudil at both

158 ST-segment depression was more prevalent in patients wit

159 Exercise-induced ST-segment depression was present in all patients, but t

160 The RPP and ST-segment depression were assessed before starting exer

161 ymptoms, but abnormal lactate metabolism and ST-segment depression were unaffected after 10 min.

162 antly decreased for 20 min after recovery of ST-segment depression when events were triggered by high

163 reases exercise duration and reduces maximal ST-segment depression while exerting minimal effects on

164 years) with known stable CAD and > or =2 mm ST segment depression who are performing ETT according t

165 by heart rate adjustment of exercise-induced ST-segment depression will benefit from therapy aimed at

166 ck, or left-axis deviation) or exercise ECG (ST-segment depression with exercise, chronotropic incomp