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1                                              SUDEP excludes other forms of seizure-related sudden dea
2                                              SUDEP usually occurs when the seizures are not witnessed
3                                              SUDEP-prone Kcna1-/- mice with partial genetic ablation
4   Demographic and clinical information of 61 SUDEP cases were collected.
5                                       The 61 SUDEP cases were categorized as definite SUDEP (n = 54),
6 s of BECTS are present in the North American SUDEP Registry (NASR).
7 ocampus, culminating in seizure activity and SUDEP.
8 duced the incidence of ictal bradycardia and SUDEP in DS mice.
9 ot cardiac, KO of Scn1a produced cardiac and SUDEP phenotypes similar to those found in DS mice.
10 profound postictal cerebral dysfunction, and SUDEP.
11 to mechanisms and prevention of epilepsy and SUDEP.
12 us Scn8a(N1768D/+) mice exhibit seizures and SUDEP, confirming the causality of the de novo mutation
13 disorder, including spontaneous seizures and SUDEP.
14 iorespiratory findings related to SUDEP, and SUDEP animal models.
15 the ryanodine receptor-2 (RyR2) gene in both SUDEP and sudden cardiac death cases linked to catechola
16  = 54), probable SUDEP (n = 5), and definite SUDEP plus (n = 2).
17  61 SUDEP cases were categorized as definite SUDEP (n = 54), probable SUDEP (n = 5), and definite SUD
18                     Two deaths were definite SUDEP, and 1 was probable SUDEP.
19                   In this review, we discuss SUDEP population studies, case-control studies, witnesse
20         Sudden unexpected death in epilepsy (SUDEP) accounts for approximately 1 in 5 deaths in patie
21         Sudden unexpected death in epilepsy (SUDEP) can affect individuals of any age, but is most co
22 taneous sudden unexpected death in epilepsy (SUDEP) in two of our strain combinations.
23         Sudden unexpected death in epilepsy (SUDEP) is increasingly recognized as a common and devast
24         Sudden unexpected death in epilepsy (SUDEP) is the leading cause of death in patients with re
25         Sudden unexpected death in epilepsy (SUDEP) is the most common cause of death in intractable
26         Sudden unexpected death in epilepsy (SUDEP) is the most common cause of death in patients wit
27         Sudden unexpected death in epilepsy (SUDEP) is the most common cause of death in patients wit
28        Sudden unexplained death in epilepsy (SUDEP) is the most common cause of premature mortality i
29         Sudden unexpected death in epilepsy (SUDEP) refers to the sudden death of a seemingly healthy
30 dels of sudden unexpected death in epilepsy (SUDEP) that exhibit audiogenic generalized convulsive se
31 d with sudden unexplained death in epilepsy (SUDEP), we propose that dysfunctions of KCNQ channels ma
32 ice to sudden unexplained death in epilepsy (SUDEP), which we recorded fortuitously in one animal.
33 bute to sudden unexpected death in epilepsy (SUDEP).
34 isk for sudden unexpected death in epilepsy (SUDEP).
35  due to sudden unexpected death in epilepsy (SUDEP).
36 lity is sudden unexpected death in epilepsy (SUDEP).
37 risk of sudden unexpected death in epilepsy (SUDEP).
38 isms of Sudden Unexpected Death in Epilepsy (SUDEP).
39 use of sudden unexplained death in epilepsy (SUDEP).
40 ial for sudden unexpected death in epilepsy (SUDEP).
41 isk for sudden unexpected death in epilepsy (SUDEP).
42 ia, and sudden unexpected death in epilepsy (SUDEP).
43 ause of sudden unexpected death in epilepsy (SUDEP).
44 isk for sudden unexpected death in epilepsy (SUDEP).
45 nation, sudden unexpected death in epilepsy (SUDEP, 18% of all deaths) did not fully explain the earl
46 redictor of early death even after excluding SUDEP (e.g. reduction in years of life for those who had
47 ltiple seizures was a strong risk factor for SUDEP.
48 ic seizures are the greatest risk factor for SUDEP; most often, SUDEP occurs after this type of seizu
49 l apnea, may be a pathogenetic mechanism for SUDEP.
50 ous system and establish an animal model for SUDEP.
51                                         Four SUDEP cases (7%) had mutations in common genes responsib
52 king Kcna1 a strong candidate gene for human SUDEP.
53        To search for genetic risk factors in SUDEP cases, we performed an exome-based analysis of rar
54 ppression (PGES) and apnea are implicated in SUDEP.
55 es is common and suspected to play a role in SUDEP.
56 euptake inhibitor (SSRI), blocks GCS-induced SUDEP in both DBA/2 and DBA/1 mice.
57 luding epilepsy, sudden death syndromes like SUDEP and SIDS, and cardiac arrhythmia.
58         In cases with an LQTS gene mutation, SUDEP may occur as a result of a predictable and prevent
59 ve seizures and often associated with (near) SUDEP.
60           Understanding the genetic basis of SUDEP may inform cascade testing of at-risk family membe
61  of SUDEP, while searching for biomarkers of SUDEP risk embedded in electroencephalography (EEG) and
62                                     Cases of SUDEP among children who received a diagnosis of BECTS a
63  tonic-clonic seizures precede most cases of SUDEP, patients must be better educated about prevention
64                                 The cause of SUDEP remains unknown.
65 ng video-EEG recording and who later died of SUDEP.
66                         A crucial element of SUDEP is brainstem dysfunction, for which postictal gene
67                             The frequency of SUDEP varies depending on the severity of the epilepsy,
68 in-heart association as potential indices of SUDEP susceptibility.
69 the Kcna1 knockout mouse (Kcna1-/-) model of SUDEP, while searching for biomarkers of SUDEP risk embe
70 etion acts as protective genetic modifier of SUDEP and suggest measures of brain-heart association as
71 nd increase in duration of PGES, the odds of SUDEP increased by a factor of 1.7%(p < 0.005).
72 res indicated significantly elevated odds of SUDEP with PGES durations of >50 seconds (p < 0.05).
73 rtant contributors to the pathophysiology of SUDEP, and serotonin (5-HT) system dysfunction may be in
74 greater importance to the pathophysiology of SUDEP.
75 ave important implications for prevention of SUDEP in DS patients.
76                     A sizeable proportion of SUDEP cases have clinically relevant mutations in cardia
77             The substantial lifetime risk of SUDEP and the lack of a clear pathophysiological connect
78 gher the risk of SUDEP; furthermore, risk of SUDEP is also elevated in male patients, patients with l
79 entifying which patients are most at risk of SUDEP is hindered by a complex genetic etiology, incompl
80                                      Risk of SUDEP may be increased in direct proportion to duration
81 o clinicians on ways to minimise the risk of SUDEP, information to pass on to patients, and medicoleg
82 ractory epilepsy patients who are at risk of SUDEP.
83 ery might contribute to reducing the risk of SUDEP.
84 IEE13 (OMIM 614558), which has a 10% risk of SUDEP.
85 onic-clonic seizures, the higher the risk of SUDEP; furthermore, risk of SUDEP is also elevated in ma
86  were found dead in circumstances typical of SUDEP.
87  greatest risk factor for SUDEP; most often, SUDEP occurs after this type of seizure in bed during sl
88 n of sudden unexpected death in epilepsy, or SUDEP.
89 udden unexpected death in epilepsy patients (SUDEP).
90 tropine-sensitive ictal bradycardia preceded SUDEP.
91 ath, which may lead to novel ways to prevent SUDEP.
92 could be a promising strategy for preventing SUDEP.
93 gorized as definite SUDEP (n = 54), probable SUDEP (n = 5), and definite SUDEP plus (n = 2).
94 aths were definite SUDEP, and 1 was probable SUDEP.
95 en repository established in 2011 to promote SUDEP research.
96 , exposure to toxic substances, or suicide); SUDEP classification was determined by the consensus of
97           Video monitoring demonstrated that SUDEP occurred immediately following generalized tonic-c
98                                   Given that SUDEP often occurs in association with generalised seizu
99                  These findings suggest that SUDEP is caused by apparent parasympathetic hyperactivit
100 ger in the generalized motor seizures of the SUDEP group (p < 0.001).
101 t tested that blocks RA selectively in these SUDEP models, which may be clinically relevant.
102  effects of the 5-HT(2B/2C) agonist in these SUDEP models.
103 sfunction in these systems may contribute to SUDEP associated with mutations of Scn8a.
104  hypoxemia and hypercapnia may contribute to SUDEP.
105 are, and thus has potential to contribute to SUDEP.
106 s, but physiological mechanisms that lead to SUDEP are unknown.
107  model might be shared with those leading to SUDEP.
108 eizure cardiorespiratory findings related to SUDEP, and SUDEP animal models.
109      Understanding the mechanisms underlying SUDEP is paramount to developing preventative strategies
110 gest the most probable mechanisms underlying SUDEP.
111 psy and respiratory problems associated with SUDEP.

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