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1 Barr virus and the ORF65 gene of herpesvirus saimiri.
2 rcoma-associated herpesvirus and herpesvirus saimiri.
3 om a rhesus monkey infected with herpesvirus saimiri.
4 rved with Epstein-Barr virus and herpesvirus saimiri.
5 tion in T cell transformation by herpesvirus saimiri.
6 rmation by subgroup A strains of herpesvirus saimiri.
7 t, point mutant, and recombinant herpesvirus saimiri.
8 porter gene from the recombinant herpesvirus saimiri.
9 f rhesus monkey rhadinovirus and herpesvirus saimiri.
10 transcripts encoded by KSHV and herpesvirus saimiri.
11 l name, human herpesvirus 8) and herpesvirus saimiri.
12 nscriptional regulating genes in herpesvirus saimiri.
14 er known human herpesviruses and herpesvirus saimiri, a closely related gammaherpesvirus of nonhuman
18 mma herpesvirus with homology to herpesvirus Saimiri and Epstein-Barr virus, both of which can transf
19 to human gammaherpesviruses and herpesvirus saimiri and has been reported to be associated with lymp
20 ee species diverged, L1 has amplified in the Saimiri and Saguinus lineages but L1 activity seems to h
21 the active L1 lineage has evolved rapidly in Saimiri and Saguinus, generating species-specific subfam
23 sviruses, Epstein-Barr virus and Herpesvirus saimiri, and are present in the lesions of more than 95%
28 pvdhfr), we transfected blood specimens from Saimiri boliviensis monkeys infected with the pyrimetham
29 tire blue opsin gene in the squirrel monkey (Saimiri boliviensis) and the five introns of the human b
30 ing KSbcl-2, BHRF1, and ORF16 of herpesvirus saimiri contain poorly conserved Bcl-2 homology 3 (BH3)
31 The Bcl-2 homologs encoded by herpesvirus saimiri, Epstein-Barr virus, and BHV4 were not cleaved b
33 were investigated in four squirrel monkeys (Saimiri) following extracellular injections of the trace
36 al gammaherpesviruses, including herpesvirus saimiri, human herpesvirus 8, and MHV68, encode proteins
37 ase interacting protein (Tip) of Herpesvirus saimiri (HVS) activates the lymphoid-specific member of
38 rus-encoded cyclin (v-cyclin) of herpesvirus saimiri (HVS) and 31% identity and 53% similarity to hum
39 formation by the DNA tumor virus herpesvirus saimiri (HVS) and designated tyrosine kinase interacting
40 ri transforming protein (STP) of herpesvirus saimiri (HVS) and of K1 of KSHV, other members of the ga
43 open reading frame 14 (orf14) of herpesvirus saimiri (HVS) exhibits significant homology with mouse m
44 ently infected marmoset T cells, Herpesvirus saimiri (HVS) expresses six microRNAs (known as miR-HSUR
45 earity with the right end of the herpesvirus saimiri (HVS) genome and more limited homology to the le
52 tein (Tip) of the T lymphotropic Herpesvirus saimiri (HVS) is constitutively present in lipid rafts a
54 ase-interacting protein (Tip) of herpesvirus saimiri (HVS) is required for binding to the cellular Sr
56 f the terminal repeats (TR) from herpesvirus saimiri (HVS) renders it unable to produce infectious vi
57 formation by the DNA tumor virus herpesvirus saimiri (HVS) strain 484, designated tyrosine kinase-int
58 otein oncogene, called STP-A, of herpesvirus saimiri (HVS) subgroup A is not required for viral repli
62 ns encoded by two herpesviruses, herpesvirus saimiri (HVS) which can transform blood lymphocytes and
63 s of the Sm class are encoded by Herpesvirus saimiri (HVS), a gamma Herpesvirus that causes aggressiv
64 marmoset T cells transformed by Herpesvirus saimiri (HVS), a viral U-rich noncoding (nc) RNA, HSUR 1
67 nd of the genome in RRV26-95 and herpesvirus saimiri (HVS), but in KSHV the DHFR gene is displaced 16
68 ssociated herpesvirus (KSHV) and herpesvirus saimiri (HVS), has been shown to encode a latency-associ
69 ri transforming protein (STP) of herpesvirus saimiri (HVS), Kaposi's sarcoma-associated herpesvirus (
70 normal donors by infection with Herpesvirus saimiri (HVS), to evaluate functional properties of thes
71 RNAs encoded by the lymphotropic Herpesvirus saimiri (HVS), we determined the specific sequence and s
73 es have demonstrated that Tip of herpesvirus saimiri (HVS), which is a T-lymphotropic tumor virus, is
74 -defensin-1 and CAF derived from herpesvirus saimiri (HVS)-transformed CD8(+) cells inhibited HIV-1 i
81 ects on TCR signal transduction, herpesvirus saimiri-immortalized CD4 Th cells from XLP patients and
82 ystander apoptosis was tested in herpesvirus saimiri-immortalized primary CD4(+) T cells (CD4/HVS), w
85 he role of StpC in cell transformation by H. saimiri may be mediated by signaling that results in NF-
87 hat, unlike the ORF57 homolog in herpesvirus saimiri, nucleolar trafficking is not required for ORF57
88 s, including 66 with homology to herpesvirus saimiri ORFs, and 5 internal repeat regions are present
89 rame 16 (ORF16) of the oncogenic herpesvirus saimiri protects cells from heterologous virus-induced a
90 at the X-linked locus among three species of Saimiri representing a wide range of geographical and be
93 World monkeys - the diurnal squirrel monkey (Saimiri sciureus) and the nocturnal owl monkey (Aotus tr
97 nomolgus (Macaca fascicularis) and squirrel (Saimiri sciureus) monkey eyes (n = 20), matrix placement
100 oon (Papio hamadryas) and 1 squirrel monkey (Saimiri sciureus) to respond to stimuli representing the
101 ys (Cebus apella) and four squirrel monkeys (Saimiri sciureus) were given demonstration trials in whi
102 characterised (humans and squirrel monkeys (Saimiri sciureus)), the aVOR response to roll head rotat
103 nkeys (Aotus trivirgatus), squirrel monkeys (Saimiri sciureus), and macaque monkeys (Macaca fascicula
104 s in three NWM species, the squirrel monkey (Saimiri sciureus), the tamarin (Saguinus oedipus), and t
106 ysiological study employed squirrel monkeys (Saimiri sciureus), which moved freely on the walls and f
109 mitochondrial genomes from Aotus lemurinus, Saimiri sciureus, Saguinus oedipus, Ateles belzebuth, an
110 red reporter genes cloned within herpesvirus saimiri sequences, recombinant viruses were readily iden
111 quencies were approximately the same for all Saimiri species, with a slight but significant differenc
114 orming protein (STP) oncogene of Herpesvirus saimiri subgroup A strain 11 (STP-A11) is not required f
116 d by transformation of PBMC with herpesvirus saimiri, suggesting that this phenomenon is an intrinsic
119 11 of subgroup A contains a gene called the saimiri transformation-associated protein, STP, which is
120 n the cell culture medium of the herpesvirus saimiri-transformed CD8(+) T-cell line, K#1 50K, has pot
121 2-kDa viral protein expressed by herpesvirus saimiri-transformed lymphocytes, is essential for transf
123 of the R1 gene is equivalent to that of the saimiri transforming protein (STP) of herpesvirus saimir
124 position equivalent to the gene encoding the saimiri transforming protein (STP) of herpesvirus saimir
128 viral small nuclear RNA (snRNA) Herpesvirus saimiri U RNA 1 (HSUR 1) also contains an AUUUA-rich seq
130 nuclear RNAs, called HSURs (for Herpesvirus saimiri U RNAs), that are abundantly expressed in HVS-tr
132 lex of six New World primates (Cebus apella, Saimiri ustius, Saguinus midas niger, Alouatta caraya, A
133 ve cells were cotransfected with herpesvirus saimiri virion DNA and one of the engineered reporter ge
136 sm modulating gene expression in herpesvirus saimiri, whereby ORF 50a transcription is downregulated
137 uses, the Epstein-Barr virus and herpesvirus saimiri, which are known to be associated with lymphomas
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