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1 and two gram negative pathogens (E. coli and Salmonella).
2 ling of the tagged amplicon from E. coli and Salmonella.
3 with host adaptation and systemic disease in Salmonella.
4 mples that have been achieved up to diagnose salmonella.
5 ween antimicrobial susceptible and resistant Salmonella.
6 n of zoonotic bacterial pathogens especially Salmonella.
7 d post-translational modification studies in Salmonella.
8 tion and promoting sustained colonization by Salmonella.
9 ion (LOD) down to 10CFUmL(-1) of E. coli and Salmonella.
10 ive technologies for real-time monitoring of salmonella.
11 umber in the infected cells, is derived from Salmonella 5'-leader of the ribosomal RNA transcript and
16 f FlgK differs from that of its orthologs in Salmonella and Burkholderia, whose structures have previ
17 found in only 16 to 29% and 0% of mice with Salmonella and Klebsiella administrations, respectively.
19 ntial pathogens (e.g. Clostridium difficile, Salmonella, and Escherichia coli) that do not produce si
20 an efficient carrier for nasal delivery of a Salmonella antigen that results in protection upon activ
21 hus, this is the first to demonstrate use of Salmonella aptamers for development of the colorimetric
24 stone genes from the entire GH complement of Salmonella are required to degrade glycans to change inf
25 patients infected with an invasive pathogen, Salmonella, are considered, based on recent studies in a
29 epleting cellular Sal-1 strongly renders the Salmonella bacteria less resistant to the host defenses
32 gainst Salmonella colitis and showed reduced Salmonella burdens in viscera, suggesting that adenosine
33 n against systemic infections by E. coli and Salmonella by directly coating bacteria to promote killi
34 oduce this technique into the human pathogen Salmonella by incorporating p-azido-phenylalanine, benzo
36 on of potentially pathogenic Escherichia and Salmonella by reducing the bioavailability of respirator
37 wo-component systems of Escherichia coli and Salmonella can also perceive an osmotic upshift, another
40 Anaerobic bacteria, such as Clostridium and Salmonella, can selectively invade and colonize in tumor
43 espite the increased luminal colonization by Salmonella, CD73(f/f)Villin(Cre) mice were protected aga
45 tigen, which mediated significant killing of Salmonella Choleraesuis and provided full protection aga
48 ombinant Asd(+) plasmid pCZ1 with the cloned Salmonella Choleraesuis O-antigen gene cluster was intro
50 (f/f)Villin(Cre) mice were protected against Salmonella colitis and showed reduced Salmonella burdens
53 -component regulatory system to exist inside Salmonella-containing vacuoles in the macrophage, as wel
55 demonstrated that the 5' leader mRNA of the Salmonella corA gene can adopt two mutually exclusive co
56 approaches in electrochemical biosensors for Salmonella detection are presented and a critical analys
57 t challenges towards a complete solution for Salmonella detection in microbial food control based on
58 the commercially available rapid methods for Salmonella detection is provided along with a critical d
60 it is a major cause of invasive nontyphoidal Salmonella disease, associated with high case fatality.
62 to monitor recruitment of endogenous LC3C to Salmonella during xenophagy, as well as to mitochondria
64 Ag(+) resistance was initially found on the Salmonella enetrica serovar Typhimurium multi-resistance
65 -propanediol utilization microcompartment of Salmonella enterica and use it to analyze the function o
67 at viable Francisella tularensis, as well as Salmonella enterica bacteria transferred from infected c
70 infection with Burkholderia pseudomallei and Salmonella enterica HMBA treatment was also associated w
71 tructures of GusRs from Escherichia coli and Salmonella enterica in complexes with a glucuronide liga
72 d in the EnvZ-OmpR two-component system from Salmonella enterica in vitro and in vivo, which directly
74 Between March 1, 2010, and Jan 31, 2014, 135 Salmonella enterica serotype Typhi (S Typhi) and 94 iNTS
76 ting protein, to influence susceptibility to Salmonella enterica serovar Typhi (S Typhi) infection.
82 ly showed that l-asparaginase II produced by Salmonella enterica serovar Typhimurium (S Typhimurium)
83 BA would be more resistant to infection with Salmonella enterica serovar Typhimurium (S Typhimurium).
88 l compartments and a reduced ability to kill Salmonella enterica serovar Typhimurium compared to that
91 toxified outer membrane vesicles (OMVs) from Salmonella enterica serovar Typhimurium displaying the v
94 n simultaneously in pathogen and host during Salmonella enterica serovar Typhimurium infection and re
95 alis, Escherichia coli K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococ
96 ed a metabolically competent, but avirulent, Salmonella enterica serovar Typhimurium mutant for its a
97 we present the structure of the prototypical Salmonella enterica serovar Typhimurium pathogenicity is
98 antigen from Mycobacterium tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.
99 sine harbors bacteriostatic activity against Salmonella enterica serovar Typhimurium that is not shar
100 egulatory system coordinates the response of Salmonella enterica serovar Typhimurium to diverse envir
101 he current study, we examined the ability of Salmonella enterica serovar Typhimurium to infect the ce
102 bound in vivo by the CspA family members of Salmonella enterica serovar Typhimurium to link the cons
104 ic bacteria, either Klebsiella pneumoniae or Salmonella enterica serovar Typhimurium, enhanced transl
105 nterohemorrhagic Escherichia coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogat
106 create FLIM-phasor maps of Escherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aer
107 To examine individual functions, strains of Salmonella enterica serovar Typhimurium, the murine mode
108 that TcpB protein can efficiently attenuate Salmonella enterica serovar Typhimurium-induced pyroptos
112 hlortetracycline on the temporal dynamics of Salmonella enterica spp. enterica in feedlot cattle.
113 (EPEC), and Citrobacter rodentium Moreover, Salmonella enterica strains encode up to three NleB orth
114 S. bongori, S. enterica subsp. salamae, and Salmonella enterica subsp. arizonae The beta-lactamase T
115 aced this domain with a nuclease domain from Salmonella enterica subsp. arizonae This modified V. cho
119 onlethal gastric infections of Gram-negative Salmonella enterica Typhimurium (ST), a major source of
121 bserved PPI, including Bacillus subtilis and Salmonella enterica which are predicted to have up to 18
123 aMN):DMB phosphoribosyltransferases (CobT in Salmonella enterica), in a reaction that is considered t
125 the foodborne pathogens E. coli O157:H7 and Salmonella enterica, in detail a nucleic acid lateral fl
126 ccus pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, Klebsiella pneumoniae, and Escheric
127 ted microorganisms such as Escherichia coli, Salmonella enterica, Listeria innocua, Mycobacterium par
129 leic acid, a major lipid in E. coli Last, in Salmonella enterica, ubiK was required for proliferation
134 ons of people each year and among pathogens, Salmonella Enteritidis is most widely found bacteria cau
135 domonas aeruginosa, Pseudomonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escheric
140 this study provides an explanation as to how Salmonella evades activation of autophagy mechanisms as
141 clusion, we demonstrate a novel strategy for Salmonella evading the host immune clearance, in which S
142 Taken together, these data indicate that Salmonella flagellin has unique adjuvant properties that
143 g of human TLR5 and a secreted derivative of Salmonella flagellin structurally analogous to a clinica
146 the proportion of multi-drug resistant (MDR) Salmonella from day 4 through day 26, which was the last
147 results reveal a means whereby intracellular Salmonella gain access to the host cell cytosol from wit
151 antimicrobials reduced overall prevalence of Salmonella; however, these treatments increased the prop
152 ated to determine the periodic prevalence of Salmonella in a population of dogs and cats in the Unite
154 s defective transepithelial translocation by Salmonella In conclusion, we define a novel antimicrobia
156 xpression promotes intracellular survival of Salmonella in macrophages, and contributes to the resist
160 helminth infection increased colonization by Salmonella independently of T regulatory or Th2 cells.
164 stinct microbiota members prevent intestinal Salmonella infection by enhancing antibacterial IFNgamma
165 n of specific gut bacteria that protect from Salmonella infection by priming host IFN-gamma responses
166 espond to bacterial and helminth infections: Salmonella infection caused an increase in the abundance
167 e show that a chronic systemic non-typhoidal Salmonella infection in an immunocompromised human patie
168 We found that T cell clonotypes in a mouse Salmonella infection model span early activated CD4(+) T
169 regulate a maximal innate immune response to Salmonella infection, allowing a sustained inflammatory
172 lular proliferation within host cells during Salmonella infections, although none have been found to
175 of typhoid fever and invasive non-typhoidal salmonella (iNTS) disease in sub-Saharan Africa, and the
176 stic dissection revealed how VAC14 regulates Salmonella invasion and typhoid fever susceptibility and
181 l mechanism used by macrophages to eradicate Salmonella is production of reactive oxygen species.
182 he Escherichia coli genome, and reveals that Salmonella is remarkably amenable to genome-scale modifi
184 By screening 73 clinical and environmental Salmonella isolates, we identified EspJ homologues in S.
185 ite its potential importance in Escherichia, Salmonella, Klebsiella, Shigella, and Yersinia opportuni
190 Furthermore, by visualizing this machine in Salmonella mutants we obtained major insights into the m
193 s employed to control foodborne nontyphoidal Salmonella (NTS), infections have not declined in decade
197 of the protein LC3B following engulfment of Salmonella or treatment with autophagy-inducing rapamyci
198 6 (10%) positive cultures for Campylobacter, Salmonella, or Shigella entero-pathogens in traditional
199 nteric fever, caused by Salmonella Typhi and Salmonella Paratyphi A, is the leading cause of bacteria
201 are sufficient to protect C. elegans against Salmonella pathogenesis in a tol-1-dependent manner.
202 ch directly enhanced bacterial expression of Salmonella pathogenicity island 1 (SPI-1) genes and incr
203 his TRAF6 ubiquitination is triggered by the Salmonella pathogenicity island 1 (SPI-1) T3SS effectors
206 polymorphonuclear leukocytes, SipA or other Salmonella pathogenicity island 1 effectors had no effec
207 d that the effect was dependent on an intact Salmonella pathogenicity island 2 (SPI-2) type 3 secreti
208 hagocytosis, a significant proportion of the Salmonella population forms non-growing persisters throu
211 suggests an overall decline in prevalence of Salmonella-positive dogs and cats over the last decades
213 brogate the cytotoxicity of RNS against phoQ Salmonella, presumably by limiting the formation of pero
216 evading the host immune clearance, in which Salmonella produce microRNA-like functional RNA fragment
217 etyl-lysine, and phosphoserine into selected Salmonella proteins including a microcompartment shell p
219 Preventing intracellular acidification of Salmonella renders it avirulent, suggesting that acid st
220 himurium yfgA mutant lost the characteristic Salmonella rod-shaped appearance, exhibited increased se
222 S. Enteritidis is a further example of a Salmonella serotype that displays niche plasticity, with
224 The mucosal inflammatory response induced by Salmonella serovar Typhimurium creates a favorable niche
227 biopsies from patients with IBS, addition of Salmonella significantly reduced levels of occludin; sub
228 te the simultaneous detection of E. coli and Salmonella sp. in hamburger sample using a multichannel
232 ns of pathogenic bacteria (Escherichia coli, Salmonella spp and Vibrio cholerae), with 8 strains of e
233 le for community-acquired infections such as Salmonella spp, Campylobacter spp, N gonorrhoeae, and H
235 ons in P. ruminicola 23, whereas E. coli and Salmonella spp. responses to excess nitrogen involve onl
236 studies were conducted by testing Shigella, Salmonella spp., Salmonella typhimurium and Staphylococc
237 his article, we report that simply combining Salmonella SseB with flagellin substantially enhances pr
245 nt for its ability to compete with wild-type Salmonella The modified Nissle strain became more virule
246 tribution toward the intracellular growth of Salmonella The results reveal a means whereby intracellu
247 toxin-producing Escherichia coli (STEC) and Salmonella" The document says CDC and its public health
248 s response was sufficient to protect against Salmonella tissue invasion and involved a previously rep
249 an salmonellosis, yet the strategies used by Salmonella to colonize chickens are poorly understood.
252 he visualization and characterization of the Salmonella type III secretion machine in live bacteria b
253 ctor of the human-adapted bacterial pathogen Salmonella Typhi (6,7) , the cause of typhoid fever in h
254 Typhoid toxin, a unique virulence factor of Salmonella Typhi (the cause of typhoid fever), recapitul
258 ampylobacter spp, Neisseria gonorrhoeae, and Salmonella typhi were included in the high-priority tier
259 B. subtilis, Enterococcus, P. aeruginosa and Salmonella typhi) to antibiotics such as ampicillin and
262 ta and IL-18 in response to NLRC4 activators Salmonella Typhimurium and flagellin, canonical or non-c
263 ducted by testing Shigella, Salmonella spp., Salmonella typhimurium and Staphylococcus aureus on E. c
264 tremely susceptible to systemic infection by Salmonella Typhimurium because of loss-of-function mutat
266 luster was introduced into three constructed Salmonella Typhimurium Deltaasd mutants: SLT11 (Deltarfb
268 that severity of disease induced by enteric Salmonella Typhimurium infection is strongly modulated b
271 sub-ng/muL standard DNA and 10(1) copies of Salmonella typhimurium InvA gene sequences (cloned in E.
274 able co-culture of metabolically competitive Salmonella typhimurium strains in microfluidic devices.
275 ere, we show that human NAIP also senses the Salmonella Typhimurium T3SS inner rod protein PrgJ and t
276 luorescently labeled Escherichia coli HS and Salmonella typhimurium that passed through from the muco
277 nced cytokine expression during infection by Salmonella typhimurium This occurred in the first 3 d of
278 t a high-resolution in situ structure of the Salmonella Typhimurium type III secretion machine obtain
279 Roseburia intestinalis, Ruminococcus obeum, Salmonella typhimurium, and Clostridium difficile) to qu
280 Three bacterial pathogens (Escherichia coli, Salmonella typhimurium, and methicillin-resistant Staphy
281 ts, including latex beads, Escherichia coli, Salmonella typhimurium, and Mycobacterium tuberculosis i
283 Here, we report that an intestinal pathogen, Salmonella Typhimurium, inhibits anorexia by manipulatin
284 minths induce IgG1, whereas Th1 Ags, such as Salmonella Typhimurium, predominantly induce IgG2a.
286 chnique was used to capture a food pathogen, Salmonella typhimurium, with starting concentrations as
287 an animal model of sepsis, we observed that Salmonella typhimurium-infected mice exhibited simultane
299 and 4 controls were mounted in chambers and Salmonella were added; we studied passage routes through
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