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1 and two gram negative pathogens (E. coli and Salmonella).
2 ling of the tagged amplicon from E. coli and Salmonella.
3 with host adaptation and systemic disease in Salmonella.
4 mples that have been achieved up to diagnose salmonella.
5 ween antimicrobial susceptible and resistant Salmonella.
6 n of zoonotic bacterial pathogens especially Salmonella.
7 d post-translational modification studies in Salmonella.
8 tion and promoting sustained colonization by Salmonella.
9 ion (LOD) down to 10CFUmL(-1) of E. coli and Salmonella.
10 ive technologies for real-time monitoring of salmonella.
11 umber in the infected cells, is derived from Salmonella 5'-leader of the ribosomal RNA transcript and
12                       We now report that the Salmonella adaptor ClpS binds to the N terminus of the r
13 3% and 10 to 16% of mice with Klebsiella and Salmonella administrations, respectively.
14  PhoPQ-dependent Mg(2+) homeostasis protects Salmonella against nitrooxidative stress.
15                Traditional methodologies for salmonella analysis provide high reliability and very lo
16 f FlgK differs from that of its orthologs in Salmonella and Burkholderia, whose structures have previ
17  found in only 16 to 29% and 0% of mice with Salmonella and Klebsiella administrations, respectively.
18 ompete for the preferred nutrient sources of Salmonella and thus prevent or treat infection.
19 ntial pathogens (e.g. Clostridium difficile, Salmonella, and Escherichia coli) that do not produce si
20 an efficient carrier for nasal delivery of a Salmonella antigen that results in protection upon activ
21 hus, this is the first to demonstrate use of Salmonella aptamers for development of the colorimetric
22                                Non-typhoidal Salmonella are associated with gastrointestinal disease
23                      We found that wild-type Salmonella are capable of replicating within infected ho
24 stone genes from the entire GH complement of Salmonella are required to degrade glycans to change inf
25 patients infected with an invasive pathogen, Salmonella, are considered, based on recent studies in a
26      Here, we use the duplicate tuf genes in Salmonella as a quantitatively tractable model system fo
27         The host glycans were altered during Salmonella association via the induction of N-glycan bio
28                                              Salmonella bacteremia was found in only 16 to 29% and 0%
29 epleting cellular Sal-1 strongly renders the Salmonella bacteria less resistant to the host defenses
30         The reduction in the amount of fecal Salmonella bacteria with Lactobacillus treatment was dem
31                                              Salmonella behaved similarly.
32 gainst Salmonella colitis and showed reduced Salmonella burdens in viscera, suggesting that adenosine
33 n against systemic infections by E. coli and Salmonella by directly coating bacteria to promote killi
34 oduce this technique into the human pathogen Salmonella by incorporating p-azido-phenylalanine, benzo
35 abrogated the fitness advantage conferred on Salmonella by lactate utilization.
36 on of potentially pathogenic Escherichia and Salmonella by reducing the bioavailability of respirator
37 wo-component systems of Escherichia coli and Salmonella can also perceive an osmotic upshift, another
38           In the present study, we show that Salmonella can exploit mammalian cell non-classical micr
39                                              Salmonella can hijack host atypical miRNA processing mac
40  Anaerobic bacteria, such as Clostridium and Salmonella, can selectively invade and colonize in tumor
41                      Approximately 97% lower Salmonella carriage is measured in a treated group, 14 d
42                                              Salmonella causes over a million foodborne illnesses per
43 espite the increased luminal colonization by Salmonella, CD73(f/f)Villin(Cre) mice were protected aga
44 is measured in a treated group, 14 days post-Salmonella challenge.
45 tigen, which mediated significant killing of Salmonella Choleraesuis and provided full protection aga
46 n 83% or 50% heterologous protection against Salmonella Choleraesuis challenge, respectively.
47 t expression of heterologous O-antigens from Salmonella Choleraesuis in Salmonella Typhimurium.
48 ombinant Asd(+) plasmid pCZ1 with the cloned Salmonella Choleraesuis O-antigen gene cluster was intro
49                                     IMS anti-Salmonella coated magnetic beads were applied to capture
50 (f/f)Villin(Cre) mice were protected against Salmonella colitis and showed reduced Salmonella burdens
51                                  Analysis of Salmonella colonization in IEC-specific CD73 knockout mi
52 ment inside infected host cells known as the Salmonella-containing vacuole.
53 -component regulatory system to exist inside Salmonella-containing vacuoles in the macrophage, as wel
54                                              Salmonella contains 47 glycosyl hydrolases (GHs) that ma
55  demonstrated that the 5' leader mRNA of the Salmonella corA gene can adopt two mutually exclusive co
56 approaches in electrochemical biosensors for Salmonella detection are presented and a critical analys
57 t challenges towards a complete solution for Salmonella detection in microbial food control based on
58 the commercially available rapid methods for Salmonella detection is provided along with a critical d
59        Available incidence data for invasive salmonella disease in sub-Saharan Africa are scarce.
60 it is a major cause of invasive nontyphoidal Salmonella disease, associated with high case fatality.
61 ed plasma membrane cholesterol, facilitating Salmonella docking and invasion.
62 to monitor recruitment of endogenous LC3C to Salmonella during xenophagy, as well as to mitochondria
63      By reconstituting the activities of the Salmonella effector SopE, we recapitulated Rho GTPase-dr
64  Ag(+) resistance was initially found on the Salmonella enetrica serovar Typhimurium multi-resistance
65 -propanediol utilization microcompartment of Salmonella enterica and use it to analyze the function o
66                                              Salmonella enterica are invasive intracellular pathogens
67 at viable Francisella tularensis, as well as Salmonella enterica bacteria transferred from infected c
68                                  Serovars of Salmonella enterica cause both gastrointestinal and syst
69            Recent papers have shown that the Salmonella enterica FinO-domain protein ProQ binds a lar
70 infection with Burkholderia pseudomallei and Salmonella enterica HMBA treatment was also associated w
71 tructures of GusRs from Escherichia coli and Salmonella enterica in complexes with a glucuronide liga
72 d in the EnvZ-OmpR two-component system from Salmonella enterica in vitro and in vivo, which directly
73                                    EutT from Salmonella enterica is a member of a class of enzymes te
74 Between March 1, 2010, and Jan 31, 2014, 135 Salmonella enterica serotype Typhi (S Typhi) and 94 iNTS
75 o develop the first human challenge model of Salmonella enterica serovar Paratyphi A infection.
76 ting protein, to influence susceptibility to Salmonella enterica serovar Typhi (S Typhi) infection.
77                                              Salmonella enterica serovar Typhi (S Typhi) is responsib
78                                              Salmonella enterica serovar Typhi causes the systemic di
79                                              Salmonella enterica serovar Typhi is a human-restricted
80        We report a typhoid fever case with a Salmonella enterica serovar Typhi isolate showing extend
81                                              Salmonella enterica serovar Typhi, the causative agent o
82 ly showed that l-asparaginase II produced by Salmonella enterica serovar Typhimurium (S Typhimurium)
83 BA would be more resistant to infection with Salmonella enterica serovar Typhimurium (S Typhimurium).
84 wo intracellular [Listeria monocytogenes and Salmonella enterica serovar Typhimurium (S.
85                                              Salmonella enterica serovar Typhimurium (S.
86                                              Salmonella enterica serovar Typhimurium can inject effec
87                                  Conversely, Salmonella enterica serovar Typhimurium causes gastroent
88 l compartments and a reduced ability to kill Salmonella enterica serovar Typhimurium compared to that
89                        The ST313 pathovar of Salmonella enterica serovar Typhimurium contributes to a
90                 We also demonstrate that the Salmonella enterica serovar Typhimurium core promoter is
91 toxified outer membrane vesicles (OMVs) from Salmonella enterica serovar Typhimurium displaying the v
92                                              Salmonella enterica serovar Typhimurium exploits the hos
93                  We found that intracellular Salmonella enterica serovar Typhimurium induced the binu
94 n simultaneously in pathogen and host during Salmonella enterica serovar Typhimurium infection and re
95 alis, Escherichia coli K12, E. coli O157:H7, Salmonella enterica serovar Typhimurium LT2, Staphylococ
96 ed a metabolically competent, but avirulent, Salmonella enterica serovar Typhimurium mutant for its a
97 we present the structure of the prototypical Salmonella enterica serovar Typhimurium pathogenicity is
98  antigen from Mycobacterium tuberculosis, in Salmonella enterica serovar Typhimurium strain SL3261.
99 sine harbors bacteriostatic activity against Salmonella enterica serovar Typhimurium that is not shar
100 egulatory system coordinates the response of Salmonella enterica serovar Typhimurium to diverse envir
101 he current study, we examined the ability of Salmonella enterica serovar Typhimurium to infect the ce
102  bound in vivo by the CspA family members of Salmonella enterica serovar Typhimurium to link the cons
103                Listeria monocytogenes V7 and Salmonella enterica serovar Typhimurium were used as mod
104 ic bacteria, either Klebsiella pneumoniae or Salmonella enterica serovar Typhimurium, enhanced transl
105 nterohemorrhagic Escherichia coli (EHEC) and Salmonella enterica serovar Typhimurium, or the surrogat
106 create FLIM-phasor maps of Escherichia coli, Salmonella enterica serovar Typhimurium, Pseudomonas aer
107  To examine individual functions, strains of Salmonella enterica serovar Typhimurium, the murine mode
108  that TcpB protein can efficiently attenuate Salmonella enterica serovar Typhimurium-induced pyroptos
109 t intracellular states of the model pathogen Salmonella enterica serovar Typhimurium.
110 helial oxygenation, and aerobic expansion of Salmonella enterica serovar Typhimurium.
111 en Vibrio cholerae and the invasive pathogen Salmonella enterica serovar Typhimurium.
112 hlortetracycline on the temporal dynamics of Salmonella enterica spp. enterica in feedlot cattle.
113  (EPEC), and Citrobacter rodentium Moreover, Salmonella enterica strains encode up to three NleB orth
114  S. bongori, S. enterica subsp. salamae, and Salmonella enterica subsp. arizonae The beta-lactamase T
115 aced this domain with a nuclease domain from Salmonella enterica subsp. arizonae This modified V. cho
116                        Human infections with Salmonella enterica subspecies enterica serovar Senftenb
117          We report a traveler who acquired a Salmonella enterica subspecies enterica serovar Typhi st
118       Within host cells such as macrophages, Salmonella enterica translocates virulence (effector) pr
119 onlethal gastric infections of Gram-negative Salmonella enterica Typhimurium (ST), a major source of
120 in bacterial cells, particularly E. coli and Salmonella enterica Typhimurium.
121 bserved PPI, including Bacillus subtilis and Salmonella enterica which are predicted to have up to 18
122 rimers specific for E. coli eaeA (151bp) and Salmonella enterica yfiR (375bp) genes.
123 aMN):DMB phosphoribosyltransferases (CobT in Salmonella enterica), in a reaction that is considered t
124                                   Strains of Salmonella enterica, and other organisms lacking RidA, h
125  the foodborne pathogens E. coli O157:H7 and Salmonella enterica, in detail a nucleic acid lateral fl
126 ccus pneumoniae, Mycobacterium tuberculosis, Salmonella enterica, Klebsiella pneumoniae, and Escheric
127 ted microorganisms such as Escherichia coli, Salmonella enterica, Listeria innocua, Mycobacterium par
128                        Two major serovars of Salmonella enterica, Typhi and Typhimurium, have evolved
129 leic acid, a major lipid in E. coli Last, in Salmonella enterica, ubiK was required for proliferation
130 of the essential endoribonuclease RNase E in Salmonella enterica.
131 m impacted by the metabolic stress of 2AA in Salmonella enterica.
132 ilament among B. subtilis, P. aeruginosa and Salmonella enterica.
133          We report on a technology to reduce Salmonella enteritidis in poultry.
134 ons of people each year and among pathogens, Salmonella Enteritidis is most widely found bacteria cau
135 domonas aeruginosa, Pseudomonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escheric
136 ria monocytogenes, Staphylococcus aureus and Salmonella enteritidis.
137                       The proportions of non-Salmonella Enterobacteriaceae with extended spectrum bet
138            Thus, Nramp1 is not essential for Salmonella entry into the CNS or neuroinflammation, but
139                   There was no occurrence of Salmonella, Escherichia coli or psychotropic bacteria.
140 this study provides an explanation as to how Salmonella evades activation of autophagy mechanisms as
141 clusion, we demonstrate a novel strategy for Salmonella evading the host immune clearance, in which S
142     Taken together, these data indicate that Salmonella flagellin has unique adjuvant properties that
143 g of human TLR5 and a secreted derivative of Salmonella flagellin structurally analogous to a clinica
144                       At a site early in the Salmonella flgM gene, the effects on translation of repl
145 ner to manipulate host cells into becoming a Salmonella-friendly zone.
146 the proportion of multi-drug resistant (MDR) Salmonella from day 4 through day 26, which was the last
147 results reveal a means whereby intracellular Salmonella gain access to the host cell cytosol from wit
148 trols iron export from vacuoles and inhibits Salmonella growth in macrophages.
149 reseeable future trends for onsite detecting salmonella have been summarized.
150                                              Salmonella have developed a sophisticated machinery to e
151 antimicrobials reduced overall prevalence of Salmonella; however, these treatments increased the prop
152 ated to determine the periodic prevalence of Salmonella in a population of dogs and cats in the Unite
153              The overall study prevalence of Salmonella in cats (3 of 542) was <1%.
154 s defective transepithelial translocation by Salmonella In conclusion, we define a novel antimicrobia
155 cipitation was developed for the analysis of Salmonella in dairy products.
156 xpression promotes intracellular survival of Salmonella in macrophages, and contributes to the resist
157 re virulent and less able to protect against Salmonella in some instances.
158 e deletion of cspC and cspE fully attenuates Salmonella in systemic mouse infection.
159                      Additionally, growth of Salmonella in the presence of histone deacetylases inhib
160 helminth infection increased colonization by Salmonella independently of T regulatory or Th2 cells.
161                                       During Salmonella-induced gastroenteritis, mucosal inflammation
162                              Within the gut, Salmonella-infected enterocytes are expelled into the lu
163                 First, depletion of Sal-1 in Salmonella-infected epithelial cells significantly incre
164 stinct microbiota members prevent intestinal Salmonella infection by enhancing antibacterial IFNgamma
165 n of specific gut bacteria that protect from Salmonella infection by priming host IFN-gamma responses
166 espond to bacterial and helminth infections: Salmonella infection caused an increase in the abundance
167 e show that a chronic systemic non-typhoidal Salmonella infection in an immunocompromised human patie
168   We found that T cell clonotypes in a mouse Salmonella infection model span early activated CD4(+) T
169 regulate a maximal innate immune response to Salmonella infection, allowing a sustained inflammatory
170 ption of raw food as a major risk factor for Salmonella infection.
171 erform lactate fermentation, thus supporting Salmonella infection.
172 lular proliferation within host cells during Salmonella infections, although none have been found to
173                                              Salmonella infectious diseases spreading every day throu
174                     To establish infections, Salmonella injects virulence effectors that hijack the h
175  of typhoid fever and invasive non-typhoidal salmonella (iNTS) disease in sub-Saharan Africa, and the
176 stic dissection revealed how VAC14 regulates Salmonella invasion and typhoid fever susceptibility and
177                                              Salmonella invasion protein A (SipA) is a dual-function
178  revealed a key role for myosin VI (MYO6) in Salmonella invasion.
179 his regulator is required for Pat to control Salmonella invasion.
180                                              Salmonella is an intracellular pathogen infecting a wide
181 l mechanism used by macrophages to eradicate Salmonella is production of reactive oxygen species.
182 he Escherichia coli genome, and reveals that Salmonella is remarkably amenable to genome-scale modifi
183          According to the recent statistics, Salmonella is still an important public health issue in
184   By screening 73 clinical and environmental Salmonella isolates, we identified EspJ homologues in S.
185 ite its potential importance in Escherichia, Salmonella, Klebsiella, Shigella, and Yersinia opportuni
186 entation, increasing both lactate levels and Salmonella lactate utilization.
187 und the protective efficacy of broad-ranging Salmonella lipopolysaccharide conjugate vaccines.
188                                              Salmonella more rapidly clear the ceca of birds administ
189  samples, and urinary mutagenicity using the Salmonella mutagenicity assay (Ames test).
190  Furthermore, by visualizing this machine in Salmonella mutants we obtained major insights into the m
191 , rotavirus (n = 15), sapovirus (n = 9), and Salmonella (n = 8).
192 ifferentially regulates the E. coli K-12 and Salmonella nrf operons.
193 s employed to control foodborne nontyphoidal Salmonella (NTS), infections have not declined in decade
194                                          How Salmonella obtains nutrients for growth within this intr
195 oactive compounds which inhibit migration of Salmonella on wet surfaces.
196                           Risk estimates for Salmonella or EHEC-related AGI were most sensitive to th
197  of the protein LC3B following engulfment of Salmonella or treatment with autophagy-inducing rapamyci
198 6 (10%) positive cultures for Campylobacter, Salmonella, or Shigella entero-pathogens in traditional
199 nteric fever, caused by Salmonella Typhi and Salmonella Paratyphi A, is the leading cause of bacteria
200 d aptamers based biosensors for detection of salmonella pathogen.
201 are sufficient to protect C. elegans against Salmonella pathogenesis in a tol-1-dependent manner.
202 ch directly enhanced bacterial expression of Salmonella pathogenicity island 1 (SPI-1) genes and incr
203 his TRAF6 ubiquitination is triggered by the Salmonella pathogenicity island 1 (SPI-1) T3SS effectors
204       S. enterica subsp. salamae encodes the Salmonella pathogenicity island 1 (SPI-1), SPI-2, and th
205                Here, starting with the 35 kb Salmonella pathogenicity island 1 (SPI-1), we eliminated
206  polymorphonuclear leukocytes, SipA or other Salmonella pathogenicity island 1 effectors had no effec
207 d that the effect was dependent on an intact Salmonella pathogenicity island 2 (SPI-2) type 3 secreti
208 hagocytosis, a significant proportion of the Salmonella population forms non-growing persisters throu
209       Rural dogs were also more likely to be Salmonella positive than urban (P = 0.002) or suburban (
210           Of note is that almost half of the Salmonella-positive animals were clinically nondiarrheic
211 suggests an overall decline in prevalence of Salmonella-positive dogs and cats over the last decades
212                                              Salmonella-positive dogs were significantly more likely
213 brogate the cytotoxicity of RNS against phoQ Salmonella, presumably by limiting the formation of pero
214                               On day 0, mean Salmonella prevalence was 75.0% and the vast majority of
215 ate count (APC) and Campylobacter as well as Salmonella prevalence.
216  evading the host immune clearance, in which Salmonella produce microRNA-like functional RNA fragment
217 etyl-lysine, and phosphoserine into selected Salmonella proteins including a microcompartment shell p
218             Here, we show that intracellular Salmonella recruit the host proteins LAMP-2A and Hsc73,
219    Preventing intracellular acidification of Salmonella renders it avirulent, suggesting that acid st
220 himurium yfgA mutant lost the characteristic Salmonella rod-shaped appearance, exhibited increased se
221                                              Salmonella serotype Reading isolates were extensively MD
222     S. Enteritidis is a further example of a Salmonella serotype that displays niche plasticity, with
223                                     Only six Salmonella serotypes were detected.
224 The mucosal inflammatory response induced by Salmonella serovar Typhimurium creates a favorable niche
225           To investigate the distribution of Salmonella serovars in MCL and their products, a total o
226  produced by both nontyphoidal and typhoidal Salmonella serovars.
227 biopsies from patients with IBS, addition of Salmonella significantly reduced levels of occludin; sub
228 te the simultaneous detection of E. coli and Salmonella sp. in hamburger sample using a multichannel
229 specificity was low ( approximately 60%) for Salmonella species.
230 mation about antigenic targets of protective Salmonella-specific T and B cells.
231                                              Salmonella spp accounted for 33% or more of all bacteria
232 ns of pathogenic bacteria (Escherichia coli, Salmonella spp and Vibrio cholerae), with 8 strains of e
233 le for community-acquired infections such as Salmonella spp, Campylobacter spp, N gonorrhoeae, and H
234 nterohemorrhagic Escherichia coli (EHEC), or Salmonella spp.
235 ons in P. ruminicola 23, whereas E. coli and Salmonella spp. responses to excess nitrogen involve onl
236  studies were conducted by testing Shigella, Salmonella spp., Salmonella typhimurium and Staphylococc
237 his article, we report that simply combining Salmonella SseB with flagellin substantially enhances pr
238 ikely to explain the additive phenotype of a Salmonella strain lacking both SseK1 and SseK3.
239                          We report here that Salmonella strains with mutations of phoPQ are hypersens
240 22-nt RNA fragment, Sal-1, which facilitates Salmonella survival in the infected host.
241                  Finally, Sal-1 facilitating Salmonella survival through suppressing iNOS induction w
242   The mechanism through which Sal-1 promotes Salmonella survival, however, remains unknown.
243 ntimutagenic activity than zerumbone against Salmonella tester strains.
244  much greater effect to inhibit Listeria and Salmonella than non-emulsion, aqueous formulations.
245 nt for its ability to compete with wild-type Salmonella The modified Nissle strain became more virule
246 tribution toward the intracellular growth of Salmonella The results reveal a means whereby intracellu
247  toxin-producing Escherichia coli (STEC) and Salmonella" The document says CDC and its public health
248 s response was sufficient to protect against Salmonella tissue invasion and involved a previously rep
249 an salmonellosis, yet the strategies used by Salmonella to colonize chickens are poorly understood.
250        Therefore, we assessed the ability of Salmonella to disseminate to the central nervous system
251         The increased susceptibility of phoQ Salmonella to RNS requires molecular O2 and coincides wi
252 he visualization and characterization of the Salmonella type III secretion machine in live bacteria b
253 ctor of the human-adapted bacterial pathogen Salmonella Typhi (6,7) , the cause of typhoid fever in h
254  Typhoid toxin, a unique virulence factor of Salmonella Typhi (the cause of typhoid fever), recapitul
255                     Enteric fever, caused by Salmonella Typhi and Salmonella Paratyphi A, is the lead
256                          Shigella sonnei and Salmonella Typhi cause significant morbidity and mortali
257  gene and did not belong to the dominant H58 Salmonella Typhi clade.
258 ampylobacter spp, Neisseria gonorrhoeae, and Salmonella typhi were included in the high-priority tier
259 B. subtilis, Enterococcus, P. aeruginosa and Salmonella typhi) to antibiotics such as ampicillin and
260  related AB5 toxin encoded by the broad-host Salmonella Typhimurium (15) .
261                                           In Salmonella typhimurium and Escherichia coli, the virulen
262 ta and IL-18 in response to NLRC4 activators Salmonella Typhimurium and flagellin, canonical or non-c
263 ducted by testing Shigella, Salmonella spp., Salmonella typhimurium and Staphylococcus aureus on E. c
264 tremely susceptible to systemic infection by Salmonella Typhimurium because of loss-of-function mutat
265                                              Salmonella Typhimurium causes a self-limiting gastroente
266 luster was introduced into three constructed Salmonella Typhimurium Deltaasd mutants: SLT11 (Deltarfb
267                                              Salmonella typhimurium infection is reported to activate
268  that severity of disease induced by enteric Salmonella Typhimurium infection is strongly modulated b
269                           Here, we show that Salmonella Typhimurium infection was accompanied by dysb
270                                       During Salmonella Typhimurium infection, intestinal CX3CR1(+) c
271  sub-ng/muL standard DNA and 10(1) copies of Salmonella typhimurium InvA gene sequences (cloned in E.
272                      We recode 200 kb of the Salmonella typhimurium LT2 genome through a process we t
273                  Flagellin isolated from the Salmonella Typhimurium SJW1660 strain, which differs by
274 able co-culture of metabolically competitive Salmonella typhimurium strains in microfluidic devices.
275 ere, we show that human NAIP also senses the Salmonella Typhimurium T3SS inner rod protein PrgJ and t
276 luorescently labeled Escherichia coli HS and Salmonella typhimurium that passed through from the muco
277 nced cytokine expression during infection by Salmonella typhimurium This occurred in the first 3 d of
278 t a high-resolution in situ structure of the Salmonella Typhimurium type III secretion machine obtain
279  Roseburia intestinalis, Ruminococcus obeum, Salmonella typhimurium, and Clostridium difficile) to qu
280 Three bacterial pathogens (Escherichia coli, Salmonella typhimurium, and methicillin-resistant Staphy
281 ts, including latex beads, Escherichia coli, Salmonella typhimurium, and Mycobacterium tuberculosis i
282 domonas fluorescens, Salmonella Enteritidis, Salmonella Typhimurium, Escherichia coli).
283 Here, we report that an intestinal pathogen, Salmonella Typhimurium, inhibits anorexia by manipulatin
284 minths induce IgG1, whereas Th1 Ags, such as Salmonella Typhimurium, predominantly induce IgG2a.
285                            Here, by studying Salmonella Typhimurium, we show that the E3 ligase LUBAC
286 chnique was used to capture a food pathogen, Salmonella typhimurium, with starting concentrations as
287  an animal model of sepsis, we observed that Salmonella typhimurium-infected mice exhibited simultane
288 s O-antigens from Salmonella Choleraesuis in Salmonella Typhimurium.
289  macrophages with the intracellular pathogen Salmonella typhimurium.
290 lla pneumophila, Pseudomonas aeruginosa, and Salmonella typhimurium.
291 l specificity of Zur, ZntR, RcnR and FrmR in Salmonella Typhimurium.
292 mmunomagnetic separation (IMS) for detecting Salmonella typhimurium.
293            The use of recombinant attenuated Salmonella vaccine (RASV) strains is a promising strateg
294                 The development of a subunit Salmonella vaccine has been hindered by the absence of d
295  strategy for the development of multivalent Salmonella vaccines.
296                                              Salmonella virulence depends in part on its pathogenicit
297 effector activities work together to promote Salmonella virulence.
298                                              Salmonella was isolated from faecal samples and antimicr
299  and 4 controls were mounted in chambers and Salmonella were added; we studied passage routes through
300                    Using SIRCAS, we create a Salmonella with 1557 synonymous leucine codon replacemen

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