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1 3% versus 69%; Fusarium, 84% versus 42%; and Scedosporium, 94% versus 18%, respectively).
2 ecies of Acremonium, Fusarium, Paecilomyces, Scedosporium, and Blastoschizomyces is suggested as a me
3 abase for the identification of Aspergillus, Scedosporium, and Fusarium species (n = 28) by matrix-as
4 portant fungi (CIF), defined as Aspergillus, Scedosporium, and Trichosporon species and Exophiala der
5 osely related fungi Pseudallescheria boydii, Scedosporium apiospermum and S. aurantiacum in the Pseud
6 ium oxysporum, Scedosporium prolificans, and Scedosporium apiospermum hyphae after caspofungin exposu
7          Respiratory tract colonization with Scedosporium apiospermum in patients with chronic suppur
8 olani, and Paecilomyces variotii and for two Scedosporium apiospermum isolates.
9 voriconazole geometric mean (G mean) MFC for Scedosporium apiospermum was lower (2.52 microg/ml) than
10 ith an invasive soft tissue infection due to Scedosporium apiospermum was successfully treated with v
11 iliforme, and F. solani; and two isolates of Scedosporium apiospermum) by the CLSI reference broth mi
12 pergillus terreus, Scedosporium prolificans, Scedosporium apiospermum, Fusarium oxysporum/Fusarium so
13 losely related fungi in the Pseudallescheria-Scedosporium complex or with a wide range of mould and y
14 m and S. aurantiacum in the Pseudallescheria-Scedosporium complex, is a contributing aetiology to tsu
15                 It does not amplify Candida, Scedosporium, Fusarium or Rhizopus species and its clini
16                               Unfortunately, Scedosporium infections are generally resistant to ampho
17 n for lung transplantation candidates, since Scedosporium infections occurring posttransplantation ar
18 rgillus, 3 (7%) had Fusarium, and 2 (4%) had Scedosporium infections.
19 ium isolates, 5 Trichosporon isolates, and 5 Scedosporium isolates.
20 nding our respiratory medicine unit have had Scedosporium organisms isolated from sputum samples.
21 nce of direct patient-to-patient spread, and Scedosporium organisms were not isolated from dust, soil
22 wn for Fusarium spp. (2 to >8 microg/ml) and Scedosporium prolificans (>8 microg/ml) by the three age
23                                              Scedosporium prolificans is a soil saprophyte that is as
24          The dematiaceous (melanized) fungus Scedosporium prolificans is an emerging and frequently f
25 e generally resistant to amphotericin B, and Scedosporium prolificans strains are particularly resist
26 oryzae, Fusarium solani, Fusarium oxysporum, Scedosporium prolificans, and Scedosporium apiospermum h
27  Pseudallescheria boydii, Rhizopus arrhizus, Scedosporium prolificans, and Sporothrix schenckii were
28 vus, Aspergillus niger, Aspergillus terreus, Scedosporium prolificans, Scedosporium apiospermum, Fusa
29 ta, Fusarium solani, Paecilomyces lilacinus, Scedosporium prolificans, Trichoderma longibrachiatum, a
30 s lilacinus, Rhizopus sp. (two species), and Scedosporium sp. (two species): (i) two media (supplemen
31 s isolates, 6 Rhizopus arrhizus isolates, 23 Scedosporium sp. isolates, 23 dematiaceous fungi, and 5
32 ng 1 Fusarium species (F. moniliforme) and 2 Scedosporium species (S. apiospermum and S. prolificans)
33                                              Scedosporium species are increasingly isolated from immu
34  osteomyelitis or septic arthritis caused by Scedosporium species in immunocompetent patients.
35           The prevalences of Aspergillus and Scedosporium species were 40.8% and 5.2%, respectively,
36  Mucor, Paecilomyces, Penicillium, Rhizopus, Scedosporium, Sporothrix, or other aspergilli tested.
37 ainst A. fumigatus, limited activity against Scedosporium spp., and no activity against zygomycetes o
38  Candida tropicalis), Aspergillus fumigatus, Scedosporium spp., Fusarium spp., and zygomycetes (inclu
39  those with other mold infections (Fusarium, scedosporium, zygomycosis, etc.), those with candidemia,

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