ライフサイエンスコーパス: Schaffer collateral

1 forming synapses with the impinging axons of Schaffer collateral.

2 mostly representing excitatory terminals of Schaffer collaterals.

3 approximately 80% of the synaptic input from Schaffer collaterals.

4 sociated with the axons and terminals of the Schaffer collaterals.

5 re also evident after tetanic stimulation of Schaffer collaterals.

6 rea CA1 following tetanic stimulation of the Schaffer collaterals.

7 tion spike by orthodromic stimulation of the Schaffer collaterals.

8 ippocampus in response to stimulation of the Schaffer collaterals.

9 in response to electrical stimulation of the Schaffer collaterals.

10 tential-evoked [Ca(2+)](i) in the boutons of Schaffer collaterals.

11 Stimulation of the Schaffer collaterals (50 Hz, 2 sec) resulted in increase

12 utamate released by repetitive activation of Schaffer collaterals activates group I metabotropic glut

13 theta cycle interval by proximally targeted Schaffer collateral activity, temporoammonic EPSPs propa

14 ired synaptic inputs from entorhinal but not Schaffer-collateral afferents.

15 in hippocampal slices by stimulation of the Schaffer collaterals, an excitatory fibre tract that pro

16 ur results demonstrate that, like LTP in the Schaffer collateral and mossy fiber pathways, MPP LTP al

17 on activity-dependent synaptic plasticity at Schaffer collateral and perforant path synapses in the h

18 ctionally important interactions between the Schaffer collateral and perforant pathways have been hyp

19 One week after transection of the Schaffer collateral and temporoammonic afferents to CA1

20 rons depends on the coincident activation of Schaffer collateral and temporoammonic inputs at the dis

21 al synapses requires integration of both the Schaffer collateral and temporoammonic pathways.

22 for long-term potentiation (LTP) in both the Schaffer collateral and the mossy fibers of the hippocam

23 amatergic or GABA-ergic transmission between Schaffer collaterals and CA1 neurons in rat hippocampal

24 cilitated glutamatergic transmission between Schaffer collaterals and CA1 neurons indicated that in a

25 on markedly impairs synaptic potentiation of Schaffer collaterals and commissural inputs to the CA1 a

26 s in brain slices by transecting hippocampal Schaffer collaterals and cortical layer I axons.

27 paired-pulse stimulation was applied to the Schaffer collaterals and population spikes were monitore

28 CA or sham-operated rats by stimulating the Schaffer collaterals and recording in the CA1 pyramidal

29 implanted with stimulating electrodes in the Schaffer collaterals and with recording electrodes in th

30 LTP) evoked by high-frequency stimulation of Schaffer collaterals, and that CN2097 attenuates this LT

31 nerated mainly by the entorhinal input, CA3 (Schaffer) collaterals, and voltage-dependent Ca(2+) curr

32 of the normal CA3 outflow tract through the Schaffer collaterals are well known, their aberrant reor

33 (basket cells) or dendritically projecting (Schaffer collateral-associated cells) interneurons and p

34 LTP is not elicited in CCK-expressing Schaffer collateral-associated cells, which lack CP-AMPA

35 ket cells and dendritically projecting, CCK+ Schaffer collateral-associated interneurons.

36 terneuron, the so-called 'basket cells' and 'Schaffer collateral-associated' cells, which innervate s

37 ium to block synaptic transmission, allowing Schaffer-collateral axon fiber volleys to be recorded fr

38 -type potassium channels that are present in Schaffer collateral axons and terminals.

39 Schaffer collateral axons form excitatory synapses that

40 e reported conflicting results as to whether Schaffer collateral axons have target-cell specific shor

41 ombine optogenetic stimulation of identified Schaffer collateral axons with two-photon imaging of pos

42 eduction of O-GlcNAc levels had no effect on Schaffer collateral CA1 basal hippocampal synaptic trans

43 forms of long-term potentiation (LTP) at the Schaffer collateral CA1 synapse require stimulation of b

44 ects of this hypothesis were examined at the Schaffer collateral CA1 synapse, where both long-term de

45 phase of NMDAR-dependent LTP at hippocampal Schaffer collateral-CA1 (Sch-CA1) synapses.

46 n in L-LTP in hippocampal slices in both the Schaffer collateral-CA1 and mossy fiber-CA3 pathways.

47 sion and long-term potentiation (LTP) in the Schaffer Collateral-CA1 and the mossy fiber-CA3 pathways

48 n the strength of excitatory transmission at Schaffer collateral-CA1 cell synapses of the hippocampus

49 Eliprodil markedly improved the recovery of Schaffer collateral-CA1 excitatory postsynaptic potentia

50 y visualize presynaptic vesicular release at Schaffer collateral-CA1 excitatory synapses in hippocamp

51 as enhanced paired-pulse facilitation in the Schaffer Collateral-CA1 glutamatergic synapses of the cb

52 to modulate long-term potentiation (LTP) at Schaffer collateral-CA1 hippocampal synapses.

53 The basal synaptic transmission of Schaffer collateral-CA1 neurons was normal but long-term

54 he efficacy of neurotransmission between the Schaffer collateral-CA1 pathway in the rat transverse hi

55 m modulation of synaptic transmission in the Schaffer collateral-CA1 pathway of the hippocampus.

56 late phase of long-term potentiation in the Schaffer collateral-CA1 pathway.

57 d depression of synaptic transmission at the Schaffer collateral-CA1 pyramidal cell synapse through a

58 LTP at Schaffer collateral-CA1 pyramidal cell synapses remained

59 NMDA receptor-dependent LTP was enhanced at Schaffer collateral-CA1 pyramidal cell synapses.

60 Through electrophysiological analysis of the Schaffer collateral-CA1 synapse in dorsal hippocampus, w

61 F-dependent long-lasting potentiation at the Schaffer collateral-CA1 synapse in hippocampus.

62 iation (LTP) of synaptic transmission at the Schaffer collateral-CA1 synapse in the hippocampus is su

63 Long-term potentiation (LTP) at the Schaffer collateral-CA1 synapse involves interacting sig

64 e to block agonist-mediated responses at the Schaffer collateral-CA1 synapse, a location at which neu

65 nhibition of group III mGluR function at the Schaffer collateral-CA1 synapse.

66 ficits in basal synaptic transmission at the Schaffer collateral-CA1 synapse.

67 s of long-term depression at the hippocampal Schaffer collateral-CA1 synapse.

68 This occurred both at Schaffer collateral-CA1 synapses and at CA3-CA3 synapses

69 ly disrupted, revealing hyperexcitability at Schaffer collateral-CA1 synapses and depression of mossy

70 erm depression (LTD) of synaptic strength at Schaffer collateral-CA1 synapses by simultaneously eleva

71 II mGlu receptors to synaptic plasticity at Schaffer collateral-CA1 synapses in acute slices of adul

72 ssion and synaptic plasticity at hippocampal Schaffer collateral-CA1 synapses in adult and juvenile m

73 also is required for the induction of LTD at Schaffer collateral-CA1 synapses in hippocampal slices.

74 We examined this question at the Schaffer collateral-CA1 synapses in slices of mouse hipp

75 At Schaffer collateral-CA1 synapses in the hippocampus, pre

76 e magnitude of NMDAR-NR2B conductance at rat Schaffer collateral-CA1 synapses in vitro.

77 we show that long-term potentiation (LTP) at Schaffer collateral-CA1 synapses is greatly enhanced in

78 nylglycine (DHPG) was markedly attenuated at Schaffer collateral-CA1 synapses of mice lacking caveoli

79 Physiological studies at Schaffer collateral-CA1 synapses uncovered a synaptic fu

80 path-dentate gyrus granule cell, CA3-CA3 and Schaffer collateral-CA1 synapses without effects at moss

81 iated excitation at TA-CA1 synapses, but not Schaffer collateral-CA1 synapses, after CUS, with a corr

82 show normal synaptic transmission and LTP at Schaffer collateral-CA1 synapses, and have no deficits i

83 z/1 s) in control slices elicited LTP at the Schaffer collateral-CA1 synapses, but neither LTP nor LT

84 at the CA3 recurrent collateral synapses and Schaffer collateral-CA1 synapses, but not at the mossy f

85 At hippocampal Schaffer collateral-CA1 synapses, LTD was significantly

86 of synaptic transmission was not observed at Schaffer collateral-CA1 synapses, while the submaximal t

87 -term depression of synaptic transmission at Schaffer collateral-CA1 synapses.

88 remental long-term potentiation (LTP) at the Schaffer collateral-CA1 synapses.

89 tors induces LTD of synaptic transmission at Schaffer collateral-CA1 synapses.

90 stores each prevent the induction of LTD at Schaffer collateral-CA1 synapses.

91 els of long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

92 paired long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

93 on and long-term potentiation at hippocampal Schaffer collateral-CA1 synapses.

94 m potentiation (LTP) when locally applied to Schaffer collateral-CA1 synapses.

95 uates activity-driven synaptic plasticity at Schaffer collateral-CA1 synapses.

96 riods of hypoxia increases the resistance of Schaffer collateral-CA1 synaptic potentials to further,

97 In presynaptic Schaffer collateral-CA1 terminals of control hippocampal

98 Schaffer collateral-CA1 tetanic long-term potentiation d

99 erm depression of synaptic strength (LTD) at Schaffer collateral-CA1, commissural/associational-CA3 a

100 synaptic plasticity was investigated at the Schaffer-collateral-CA1 pyramidal cell synapse of mouse

101 Our findings show that at hippocampal Schaffer collateral CA3-CA1 synapses, theta-burst LTP re

102 ed before (control) and after tetanizing the Schaffer/collateral commissural pathway.

103 0 mV, paired-pulse stimulation of one set of Schaffer collateral-commissural fibres resulted in homos

104 was tested with use of one such pathway, the Schaffer collateral-commissural projection to CA1 pyrami

105 Long-term potentiation (LTP) at the Schaffer collateral-commissural synapses in the CA1 area

106 ing up the majority of glutamate released at Schaffer collateral-commissural synapses in the hippocam

107 ave been shown to potentiate transmission at Schaffer collateral-commissural synapses on CA1 pyramida

108 Proline enhanced Schaffer collateral-commissural synaptic transmission ev

109 est that the proline-induced potentiation of Schaffer collateral-commissural synaptic transmission pr

110 either concentration of proline potentiated Schaffer collateral-commissural synaptic transmission.

111 LTP in mossy fiber (MF)-CA3 pathway and the Schaffer collateral/commissural (SC)-CA1 pathway differ

112 have measured evoked glutamate release from Schaffer collateral/commissural fiber terminals in CA1 b

113 late-phase long-term potentiation (L-LTP) at Schaffer collateral/commissural fiber-CA1 synapses in ra

114 idylinositol 3-kinase (PI3-kinase) in LTP at Schaffer collateral/commissural fiber-CA1 synapses in ra

115 f excitatory postsynaptic field potential at Schaffer collateral/commissural fiber-CA1 synapses showe

116 We stimulated the Schaffer collateral/commissural fibers in acute hippocam

117 We find that stimulation of Schaffer collateral/commissural fibers in hippocampal sl

118 ices in which the cell bodies of presynaptic Schaffer collateral/commissural fibres are removed.

119 idal cells co-aligned with the glutamatergic Schaffer collateral/commissural input.

120 y ramifying in the pyramidal cell layer; (2) Schaffer collateral/commissural pathway-associated inter

121 tion of various stimulation protocols to the Schaffer collateral/commissural projections in freely mo

122 release properties of zinc-positive CA3-CA1 Schaffer collateral/commissural synapses in the stratum

123 the amygdala and examining stress effects on Schaffer collateral/commissural-CA1 LTP and spatial memo

124 ivity at various times after tetanization of Schaffer collateral/commissural-CA1 synapses.

125 bserved that stress reduced the magnitude of Schaffer collateral/commissural-Cornu Ammonis field 1 lo

126 Hz) was delivered to one of two independent Schaffer-collateral/commissural projections, while the s

127 CRS reduced long-term potentiation (LTP) of Schaffer collateral/commisural-CA1 pathway, phospho-alph

128 egulating synaptic plasticity at hippocampal Schaffer collateral-cornu ammonis 1 (SC-CA1) synapses ha

129 otentiation of long-term potentiation at the Schaffer collateral/cornu ammonis 1 synapse in the dorsa

130 y stimulation (LFS) (900 pulses/1 Hz) of the Schaffer collaterals depressed the initial slope of the

131 in two separate afferent pathways among the Schaffer collaterals during intracellular recording of C

132 n excitatory inputs, the perforant path, and Schaffer collaterals during theta and non-theta behavior

133 Brief stimulation of the Schaffer collaterals elicited an alkaline pH(e) transien

134 egion of the hippocampus, stimulation of the Schaffer collaterals elicits an alkaline pH(e) transient

135 g of back-propagating action potentials with Schaffer collateral EPSPs was accompanied by an overall

136 ected animals after 5 months showed that the Schaffer collateral-evoked EPSP was attenuated, the effe

137 tial strength and the short-term dynamics of Schaffer collateral excitatory synapses are regulated by

138 stigate in detail the short-term dynamics of Schaffer collateral excitatory synapses onto CA1 stratum

139 Although CB1Rs are also expressed at Schaffer-collateral excitatory terminals, long-term plas

140 slices, we find that short trains (2-3 s) of Schaffer collateral fiber stimulation delivered at 5 Hz

141 gh CA3 pyramidal cells give rise to both the Schaffer collateral fiber synapses in CA1 and the assoc-

142 Glutamate spillover is observed between Schaffer collateral fiber synapses onto CA1 pyramidal ce

143 mpal slices, long-term potentiation (LTP) at Schaffer collateral fiber synapses onto CA1 pyramidal ce

144 ey provide a measure of axonal conduction in Schaffer-collateral fibers.

145 We found that orthodromic stimulation of the Schaffer collaterals for 10 min reduces the amplitude of

146 In hippocampus, activation of the Schaffer collaterals generates an extracellular alkaline

147 tate gyrus as well as decremental LTP at the Schaffer collateral-->CA1 pyramidal cell synapse.

148 erm potentiation (LTP) can be induced in the Schaffer collateral-->CA1 synapse of hippocampus by stim

149 PG-induced inhibition of transmission at the Schaffer collateral-->CA1 synapse.

150 long-term potentiation (LTP) at hippocampal Schaffer collateral-->CA1 synapses in an activity- and t

151 SCs) evoked by electrical stimulation of the Schaffer collaterals in CA1 hippocampal pyramidal cells.

152 Schaffer collaterals in each slice were stimulated at 40

153 e dendrites were unlabeled, mossy fibers and Schaffer collaterals in the hippocampal formation, baske

154 induction of inflammation revealed enhanced Schaffer collateral-induced excitatory field potentials

155 gh and low levels of nAChR activation in the Schaffer collaterals inhibit and facilitate, respectivel

156 dendrites that receive a uniform density of Schaffer collateral input (approximately 100-250 microme

157 amate receptor (mGluR) stimulation either by Schaffer collateral input to CA1 neurones in brain slice

158 receptors modulate synaptic function at the Schaffer collateral input to CA1 pyramidal cells, thereb

159 ross the range of apical dendrites receiving Schaffer collateral input.

160 tion (LTP) induced by theta-burst pairing of Schaffer collateral inputs and postsynaptic firing is as

161 udies revealed that the amplitude of unitary Schaffer collateral inputs increases with distance from

162 induction of long-term potentiation (LTP) of Schaffer collateral inputs to hippocampal CA1 pyramidal

163 at perforant path synapses is lower than at Schaffer collateral inputs.

164 PSPs evoked by electrical stimulation of the Schaffer collateral inputs.

165 as little effect on LTP at the more proximal Schaffer collateral inputs.

166 nged in epileptic animals, and input via the Schaffer collaterals is actually decreased despite reduc

167 Schaffer-collateral long-term potentiation (LTP) was imp

168 Moreover, TRPV1 knockouts have reduced Schaffer collateral LTP, which is rescued by activating

169 pecifically and quickly to affect LTP in the Schaffer collaterals of the hippocampus.

170 TP elicited by either tetanic stimulation of Schaffer collaterals or a pairing protocol, and persiste

171 no expression within the perforant path, the Schaffer collaterals, or neuronal cell bodies.

172 P16-CREB, enhanced in vivo LTP evoked in the Schaffer collateral pathway and caused significant chang

173 Here we show that tetanic stimulation of the Schaffer collateral pathway causes an increase in the co

174 Long-term potentiation (LTP) in the Schaffer collateral pathway from the CA3 to the CA1 regi

175 report similar changes in plasticity at the Schaffer collateral pathway in alert behaving mice.

176 paired cAMP-dependent LTP in the hippocampal Schaffer collateral pathway induced by either forskolin

177 We suggest that tetanic stimulation of the Schaffer collateral pathway may induce new synthesis of

178 Neuronal stimulation in the Schaffer collateral pathway promotes pS857 dephosphoryla

179 The Schaffer collateral pathway provides hippocampal CA1 pyr

180 Using a computer model of the hippocampal Schaffer collateral pathway that incorporates evidence f

181 rine) on input-output (I-O) relations in the Schaffer collateral pathway to CA1 (SC-CA1) and mossy fi

182 opment, basic synaptic transmission from the Schaffer collateral pathway to CA1 pyramidal neurons rem

183 were determined in acute brain sections; the Schaffer collateral pathway was stimulated and the field

184 y-phase LTP to late-phase LTP (L-LTP) in the Schaffer collateral pathway, likely as a result of incre

185 ircuit-specific and were not observed in the Schaffer collateral pathway-associated inhibitory synaps

186 circuit-specific and are not observed in the Schaffer collateral pathway-associated inhibitory synaps

187 e dentate perforant path, but not in the CA1 Schaffer collateral pathway.

188 ase of long-term potentiation (L-LTP) in the Schaffer collateral pathway.

189 ase of long-term potentiation (L-LTP) in the Schaffer collateral pathway.

190 tratum radiatum following stimulation of the Schaffer collateral pathway.

191 hippocampal slices after tetanization of the Schaffer collateral pathway.

192 ional knockout (cKO) mice in the hippocampal Schaffer collateral pathway.

193 y processes or long-term potentiation of the Schaffer collateral pathway.

194 requency electrical stimulation (LFS) of the Schaffer collateral pathway.

195 phase of long-term potentiation (LTP) in the Schaffer collateral pathway.

196 imulation in the hippocampal mossy fiber and Schaffer collateral pathways.

197 ium Green AM to measure Ca(2+) transients in Schaffer collateral presynaptic terminals simultaneously

198 We provide the first evidence of novel CA3 Schaffer collateral projection to the entorhinal cortex.

199 and slowly developing heterosynaptic LTD, of Schaffer collateral-pyramidal cell synapses in adult rat

200 Variance-mean analyses of Schaffer collateral release probability (P(r)) at varyin

201 CB1Rs are located on Schaffer collateral (Sc) axon terminals in the hippocamp

202 gic and has a larger NMDA component than the Schaffer collateral (sc) input.

203 torhinal perforant path (PP) and hippocampal Schaffer collateral (SC) inputs to CA1 pyramidal cells s

204 d activation of entorhinal cortical (EC) and Schaffer collateral (SC) inputs to hippocampal CA1 pyram

205 oammonic (TA) pathway and indirectly via the Schaffer collateral (SC) pathway from CA3.

206 rences in short- and long-term plasticity at Schaffer collateral (SC) synapses in the dorsal and vent

207 mission and plasticity at dorsal and ventral Schaffer collateral (SC) synapses in the mouse hippocamp

208 has been studied extensively at hippocampal Schaffer collateral (SC) synapses.

209 mory and long-term potentiation (LTP) at CA1 Schaffer collateral (SC) synapses.

210 oach, induced different types of hippocampal Schaffer collateral (SC) to CA1 synaptic plasticity, dep

211 ly mGlu7 is expressed presynaptically at the Schaffer collateral (SC)-CA1 synapse in the hippocampus

212 nt long-term depression (LTD) at hippocampal Schaffer collateral (SC)-CA1 synapses.

213 Field recordings in the hippocampal Schaffer collaterals (SC) pathway revealed significantly

214 ding NMDAR-mediated responses at hippocampal Schaffer collaterals (SC)-CA1 and medial perforant path-

215 the perforant path (PP) and from CA3 through Schaffer collaterals (SC).

216 e perforant path (PP) and indirectly through Schaffer collaterals (SC).

217 ther increase or decrease the probability of Schaffer-collateral (SC)-evoked CA1 spikes.

218 ase across a range of Pr at synapses between Schaffer collaterals (SCs) and CA1 pyramidal cells in ac

219 t path (PP) of the entorhinal cortex and the Schaffer collaterals (SCs) from individual CA3 pyramidal

220 Evoked responses of CA1 neurons to Schaffer collateral stimulation changed over the cycle,

221 inhibits the population spike (PS) evoked by Schaffer collateral stimulation in hippocampal slices.

222 atio onto CA1 pyramidal cells in response to Schaffer collateral stimulation in slices from young adu

223 ine (10 microM) enhanced NMDAR currents with Schaffer collateral stimulation in WT mice but not HZ mi

224 amidal neurones revealed that high frequency Schaffer collateral stimulation resulted in a prolonged

225 ked in CA1 minislices by sub-threshold 35 Hz Schaffer collateral stimulation that activated fast-spik

226 WT mice, but not in Prnp(0/0) mice, pairing Schaffer collateral stimulation with depolarization of C

227 The present findings show that, using Schaffer collateral stimulation, the effects of SNX and

228 c currents, evoked in CA1 principal cells by Schaffer collateral stimulation, were detected in hippoc

229 duced the amplitude of responses elicited by Schaffer collateral stimulation, without influencing mem

230 ratio of CA1 pyramidal cells in response to Schaffer collateral stimulation.

231 utamate, than the excitatory LTP produced by Schaffer collateral stimulation.

232 n the functional dynamics of the hippocampal Schaffer collateral synapse by using data-driven nonpara

233 icited by such heterosynaptic pairing at the Schaffer collateral synapse in mice.

234 ng-term synaptic potentiation (L-LTP) at the Schaffer collateral synapse of the hippocampus is an exp

235 activity induces synaptic plasticity at the Schaffer collateral synapse onto CA1 pyramidal neurones.

236 tential diversity of nAChR influences at the Schaffer collateral synapse onto CA1 pyramidal neurons.

237 voltage and calcium signals in spines after Schaffer collateral synapse stimulation.

238 esynaptic and postsynaptic components of the Schaffer collateral synapse.

239 were obtained with regard to the hippocampal Schaffer collateral synapse.

240 ession and facilitation were apparent in the Schaffer collateral synapse.

241 ates the induction of synaptic plasticity at Schaffer collateral synapses and hippocampal-dependent l

242 d that deletion of Cdc42 impaired LTP in the Schaffer collateral synapses and postsynaptic structural

243 lasticity in corticocortical connections and Schaffer collateral synapses beyond the critical period.

244 nd chemically induced NMDAR-dependent LTD at Schaffer collateral synapses but did not affect potentia

245 be related to impairment of the E-LTP in the Schaffer collateral synapses but not of that of the perf

246 m potentiation (LTP) of synaptic strength at Schaffer collateral synapses has largely been attributed

247 and neurotransmitter release at hippocampal Schaffer collateral synapses in both tottering (tg, alph

248 r a potential aspartate-mediated response at Schaffer collateral synapses in organotypic hippocampal

249 (LTP) induced by theta burst stimulation at Schaffer collateral synapses in the hippocampus.

250 ansmission and spine density specifically at Schaffer collateral synapses in the stratum radiatum (SR

251 arget cell-specific short-term plasticity at Schaffer collateral synapses in which the activation of

252 production of distance-dependent scaling of Schaffer collateral synapses is an elevated postsynaptic

253 Schaffer collateral synapses on CA2 neurons are distinct

254 To counteract this amplitude filtering, Schaffer collateral synapses onto CA1 pyramidal cells co

255 eurons containing somatostatin, we show that Schaffer collateral synapses onto the EGFP-expressing so

256 n, whereas the release of NPY that modulates Schaffer collateral synapses requires integration of bot

257 in the fundamental presynaptic properties of Schaffer collateral synapses that could account for dist

258 lectively required for those forms of LTP at Schaffer collateral synapses that recruit a presynaptic

259 contrast, we show that at mature hippocampal Schaffer collateral synapses the magnitudes of Ca2+ tran

260 s long-term potentiation at the CA1 proximal Schaffer collateral synapses when the two inputs are pai

261 eurons, deficits in synaptic transmission at Schaffer collateral synapses, and blunted plasticity and

262 l CA1 neurons through the perforant path and Schaffer collateral synapses, respectively.

263 At Schaffer collateral synapses, the induction of long-term

264 pocampal slices after tetanic stimulation of Schaffer collateral synapses.

265 d the induction of long term potentiation at Schaffer collateral synapses.

266 s of both AMPAR and NMDAR currents evoked at Schaffer collateral synapses.

267 e of cleft glutamate concentrations found at Schaffer collateral synapses.

268 nt role in the distance-dependent scaling of Schaffer collateral synapses.

269 LTP are reversible modifications of the same Schaffer collateral synapses.

270 ptors (5-HT(1B)Rs), without affecting nearby Schaffer collateral synapses.

271 issural and mossy fiber synapses but not CA1 Schaffer collateral synapses.

272 dendritic atrophy and LTP attenuation in CA1 Schaffer collateral synapses.

273 ippocampus of symptomatic Mecp2(308/Y) mice, Schaffer-collateral synapses exhibited enhanced basal sy

274 anced induction of long term potentiation at Schaffer-collateral synapses in area CA1 of the hippocam

275 B, we measured long-term depression (LTD) of Schaffer-collateral synapses in the hippocampus of c-Rel

276 t can be facilitated by modest activation of Schaffer-collateral synapses in the upper apical dendrit

277 In contrast, AMPAR-mediated input at local Schaffer-collateral synapses on neurogliaform cells rema

278 These results suggest that part of the Schaffer collateral synaptic response has pharmacologica

279 The presence of a significant Schaffer collateral system from the pyramidal neurons in

280 ial fiber system is a component of the hilar Schaffer collateral system in humans.

281 we measure this replenishment rate at active Schaffer collateral terminals by determining the maximum

282 the rapidly recycling vesicle pool (RRP) at Schaffer collateral terminals in field CA1 of rat hippoc

283 denosine receptors at neighboring excitatory Schaffer collateral terminals, which could counteract ef

284 N-type voltage-gated Ca(2+) channels at the Schaffer collateral than perforant path synapses.

285 of estradiol on the functional integrity of Schaffer collateral to CA1 (Sch-CA1) pyramidal cell syna

286 llel fiber to Purkinje cell synapse, and the Schaffer collateral to CA1 pyramidal cell synapse.

287 h that under intact pharmacology, LTP of the Schaffer collateral to CA1 pyramidal neuron synapses wil

288 c deletion of TRPA1 channels affected LTP of Schaffer collateral to CA1 pyramidal neuron synapses.

289 GluR-dependent long-term depression (LTD) at Schaffer collateral to CA1 pyramidal synapses of the hip

290 (AMPA)-type glutamate receptors (AMPARs) at Schaffer collateral to CA1 synapses in postischemic hipp

291 ficits and specific synaptic dysfunctions at Schaffer collateral to CA1 synapses.

292 and -independent forms of LTP induced in the Schaffer collateral to CA1 synapses.

293 ds preceding the seizure without bursts (cut Schaffer collateral tract) and in comparison with bursts

294 n with bursts preceding the seizures (intact Schaffer collateral tract).

295 at hippocampal long-term potentiation in the Schaffer collaterals was identical in homozygous, hetero

296 ease during low-frequency stimulation of the Schaffer collaterals were altered in scrapie-infected mi

297 astrocytes to electrical stimulation of the Schaffer collaterals were monitored by confocal microsco

298 The projections from CA3 to CA1 (Schaffer collaterals), which terminate in the strata rad

299 in CA3 pyramidal neurons and its efferents - Schaffer collateral, which causes the depolarization, ac

300 Prolonged 1 Hz stimulation (900 pulses) of Schaffer collaterals, which normally depresses CA3/CA1 s