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1 ighest activity (IC(5)(0): 0.3 muM) on adult Schistosoma mansoni .
2 tal in transmission of the human blood fluke Schistosoma mansoni.
3 aneous infections with the helminth parasite Schistosoma mansoni.
4 Th2 response against the parasitic helminth Schistosoma mansoni.
5 characterized eIF4E from the human parasite Schistosoma mansoni.
6 , intermediate host of the human blood fluke Schistosoma mansoni.
7 e hard tick Ixodes ricinus, and the flatworm Schistosoma mansoni.
8 igmosomoides polygyrus, Trichuris muris, and Schistosoma mansoni.
9 tissues of mice infected with the trematode Schistosoma mansoni.
10 ing cells when these mice were infected with Schistosoma mansoni.
11 e identified in the Platyhelminth trematode, Schistosoma mansoni.
12 potent activity against pathogenic trematode Schistosoma mansoni.
13 exacerbated in Batf3(-/-) mice infected with Schistosoma mansoni.
14 nt intermediate hosts of the human pathogen, Schistosoma mansoni.
15 rganization of the cathepsin D gene locus of Schistosoma mansoni.
16 infection with the type 2-promoting pathogen Schistosoma mansoni.
17 in-13 overexpression or after infection with Schistosoma mansoni.
18 ed a cDNA encoding PHM in the human parasite Schistosoma mansoni.
19 t baseline and in response to infection with Schistosoma mansoni.
20 e fibrosis induced by the parasitic helminth Schistosoma mansoni.
21 me released by the cercarial larvae stage of Schistosoma mansoni.
22 sponse development to the parasitic helminth Schistosoma mansoni.
23 in the life cycle of the trematode pathogen Schistosoma mansoni.
24 ilk and on the Th2 driving helminth parasite Schistosoma mansoni.
25 hemokines in experimental mouse models using Schistosoma mansoni.
26 mice to survive infection with the parasite Schistosoma mansoni.
27 l intermediate host of the human blood fluke Schistosoma mansoni.
28 ria glabrata to infection with the trematode Schistosoma mansoni.
29 sensitization with soluble egg Ags (SEA) of Schistosoma mansoni.
30 ting a protein library of the human parasite Schistosoma mansoni.
31 chronic infection with the helminth parasite Schistosoma mansoni.
32 ve collagen deposition during infection with Schistosoma mansoni.
33 (+/-) mice were infected percutaneously with Schistosoma mansoni.
34 rom the smooth muscles of the human parasite Schistosoma mansoni.
35 transferase (SULT) in the parasitic flatworm Schistosoma mansoni.
36 on of liver granulomas in mice infected with Schistosoma mansoni.
37 e hosts for the digenetic trematode parasite Schistosoma mansoni.
38 tion of neoblast-like cells in the trematode Schistosoma mansoni.
39 ll trafficking in response to challenge with Schistosoma mansoni.
40 anctuary staff, were naturally infected with Schistosoma mansoni.
41 against the blood-feeding trematode parasite Schistosoma mansoni.
42 ced exclusively by the eggs of the trematode Schistosoma mansoni.
43 llowing infection with the helminth parasite Schistosoma mansoni.
44 e hookworm (45%), Mansonella perstans (21%), Schistosoma mansoni (18%), and Plasmodium falciparum (11
47 uence tag data base of the helminth parasite Schistosoma mansoni, a causative agent of schistosomiasi
49 g the role of B. glabrata in transmission of Schistosoma mansoni, a digenean parasite that infects ne
50 ized a new member of the cyclase family from Schistosoma mansoni, a member of the Platyhelminthes phy
53 During infection with the helminth parasite Schistosoma mansoni, Ab regulates hepatic inflammation,
55 ic disease caused by the parasitic trematode Schistosoma mansoni after deposition of eggs in the live
58 glycoprotein (VSVG) for the transduction of Schistosoma mansoni and delivery of reporter transgenes
60 Two populations with differing histories of Schistosoma mansoni and hepatitis C infection were compa
61 on, a cohort of 163 Ugandans coinfected with Schistosoma mansoni and HIV-1 was treated with praziquan
64 We show that Ugandan adults coinfected with Schistosoma mansoni and human immunodeficiency virus typ
65 co-infection model in C57BL/6 mice involving Schistosoma mansoni and Leishmania donovani, two importa
66 wed much less mortality after infection with Schistosoma mansoni and much more susceptibility to Nipp
67 -21R-/- mice with the Th2-inducing pathogens Schistosoma mansoni and Nippostrongylus brasiliensis and
68 in mice infected with the helminth parasite Schistosoma mansoni and observed an upregulation of CD14
69 d two Ca(2+) channel beta subunits, one from Schistosoma mansoni and one from Schistosoma japonicum.
70 exposed EC2 domain from Sm-TSP-2, a TSP from Schistosoma mansoni and one of the better prospects for
74 omologues from two other schistosome species-Schistosoma mansoni and Schistosoma bovis, which are imp
75 nt humoral responses in humans infected with Schistosoma mansoni and Schistosoma haematobium and cons
76 nevitably influence both the distribution of Schistosoma mansoni and Schistosoma haematobium and the
80 strated that mice concurrently infected with Schistosoma mansoni and Toxoplasma gondii undergo accele
83 nyl PZQ derivatives was tested against adult Schistosoma mansoni, and values in the micromolar range
86 nsiveness (AHR) and tissue eosinophilia in a Schistosoma mansoni antigen-sensitized and airway-challe
87 humoral immune response to crude and defined Schistosoma mansoni antigens aggregates within families.
88 f double-stranded RNA interference (RNAi) in Schistosoma mansoni, appraises delivery systems for tran
89 e, we demonstrate that somatic stem cells in Schistosoma mansoni are biased towards generating a popu
91 o infective larvae of the parasitic helminth Schistosoma mansoni are poorly understood, but here for
93 The blood flukes Schistosoma japonicum and Schistosoma mansoni are the first major human platyhelmi
95 Infections with helminth parasites, such as Schistosoma mansoni, are often chronic and characterized
97 in a Ca(v)beta subunit of the human parasite Schistosoma mansoni (beta(Sm)), a motif that does not oc
98 y parasite ova during natural infection with Schistosoma mansoni, but the role of TGF-beta is less cl
103 to excretory/secretory products released by Schistosoma mansoni cercariae rapidly produce IL-10 as a
104 vy and quantifiable occupational exposure to Schistosoma mansoni cercariae revealed that some individ
105 t exposure of mice to repeated doses (4x) of Schistosoma mansoni cercariae, compared to a single dose
109 nts with acute HCV hepatitis with or without Schistosoma mansoni coinfection, a parasitic infection w
111 ucture of a hammerhead ribozyme derived from Schistosoma mansoni containing the rate-enhancing periph
113 that B cell-deficient (muMT) mice exposed to Schistosoma mansoni develop augmented tissue pathology a
115 to determine whether children infected with Schistosoma mansoni develop protection-related immune re
117 C57BL/6 mice infected with the helminth Schistosoma mansoni develop small hepatic granulomas aro
119 weight, although a recent treatment trial in Schistosoma mansoni did not detect this association.
120 nized with radiation-attenuated cercariae of Schistosoma mansoni display resistance to challenge infe
121 g and the adult worm developmental stages of Schistosoma mansoni during chronic infections with the p
122 ted, pathogen-derived Ag (soluble extract of Schistosoma mansoni egg [SEA]) that induces type 2 immun
124 onstrated that induction of Th2 responses by Schistosoma mansoni egg Ag is largely due to carbohydrat
126 egulated in response to the helminth soluble Schistosoma mansoni egg Ag, which conditions DCs to indu
127 nulomas elicited with Mycobacterium bovis or Schistosoma mansoni egg Ag-coated beads, respectively.
128 cobacteria bovis protein Ags and helminthic, Schistosoma mansoni egg Ags elicited multiple chemokines
129 cteria bovis purified protein derivative and Schistosoma mansoni egg antigen challenge indicating an
130 pe (induced by the pulmonary embolization of Schistosoma mansoni egg antigen-coated beads in mice sen
134 nt were confirmed by in vivo studies using a Schistosoma mansoni egg-challenged mouse model, a well-s
135 obese mice with recombinant helminth-derived Schistosoma mansoni egg-derived omega1 (omega1), a poten
136 signaling participates in the development of Schistosoma mansoni egg-induced CD4(+) Th2 responses, it
143 osylated T2 ribonuclease (RNase) secreted by Schistosoma mansoni eggs and abundantly present in solub
144 model, mice are sensitized with inactivated Schistosoma mansoni eggs and are subsequently challenged
147 sensitized and subsequently challenged with Schistosoma mansoni eggs developed pulmonary hypertensio
148 responses against Trichuris muris worms and Schistosoma mansoni eggs do not develop in mice with IRF
149 ch the mice were sensitized with inactivated Schistosoma mansoni eggs followed by S. mansoni egg Ag c
150 d by the intrapulmonary embolization of live Schistosoma mansoni eggs into S. mansoni-sensitized mice
153 nsitivity pulmonary granulomas by embolizing Schistosoma mansoni eggs to the lungs of osteopontin-def
154 condary pulmonary granulomata in response to Schistosoma mansoni eggs was blunted in MCP-1(-/-) mice,
155 le egg antigens (SEA; a soluble extract from Schistosoma mansoni eggs) inhibit the activation of DCs
157 optimal Th2 responses following exposure to Schistosoma mansoni eggs, a potent and natural Th2-induc
158 After exposure to bleomycin (BLM), but not Schistosoma mansoni eggs, IL-17A produced by CD4(+) and
171 ties of PGE(2) were produced by cercariae of Schistosoma mansoni following incubation with linoleic a
172 onal study, examining children infected with Schistosoma mansoni from 6 schools in Uganda that had pr
174 ta, a natural host for the human blood fluke Schistosoma mansoni Granulins are growth factors that dr
175 esolution crystal structure of a full-length Schistosoma mansoni hammerhead ribozyme that permits us
177 ferring immunity to the intestinal trematode Schistosoma mansoni Here, we report that abrogation of I
178 amates were prepared as potent inhibitors of Schistosoma mansoni histone deacetylase 8 (smHDAC8).
186 fection of mammals by the parasitic helminth Schistosoma mansoni induces antibodies to glycan antigen
187 hal forms of liver pathology that develop in Schistosoma mansoni infected mice polarized for type-1 a
188 mune reactions after praziquantel therapy in Schistosoma mansoni-infected fishermen working in an are
189 s derived from the spleens and granulomas of Schistosoma mansoni-infected mice during the course of t
190 is both necessary and sufficient to prevent Schistosoma mansoni-infected mice from developing severe
192 inflammation was analyzed in offspring from Schistosoma mansoni-infected mothers mated during the TH
196 mmunopathological characteristics, caused by Schistosoma mansoni infection in IL-4 receptor alpha-def
199 t helminthic parasites using the established Schistosoma mansoni infection model in 2 novel mouse mod
201 aecal slides over three consecutive days for Schistosoma mansoni infection simultaneously by age grou
203 IL-13) characterize the host response after Schistosoma mansoni infection, and recent studies have i
205 thin cDCs impaired Th2 cell responses during Schistosoma mansoni infection, Schistosoma egg antigen (
216 h coinfection of hepatitis B virus (HBV) and Schistosoma mansoni is a frequent event in humans, littl
224 ence of interleukin-4 (IL-4), infection with Schistosoma mansoni leads to a severe fatal disease rath
227 a glabrata is an intermediate snail host for Schistosoma mansoni, one of the important schistosomes i
228 resistance of B. glabrata to infection with Schistosoma mansoni or Echinostoma paraensei, and functi
231 as evident in DCs responding to the helminth Schistosoma mansoni or the allergen house dust mite (HDM
232 th either the Th2 response-inducing parasite Schistosoma mansoni or with the Th1 response-inducing pa
236 oma haematobium-Plasmodium falciparum versus Schistosoma mansoni-P. falciparum) has produced conflict
238 fraction S3) prepared from immature (4-week) Schistosoma mansoni parasites can induce partial, serum-
241 PCK was identified, and the full recombinant Schistosoma mansoni PEPCK (rSm-PEPCK) of 626 amino acids
242 Here, we express SMDR2, a Pgp homologue from Schistosoma mansoni (Platyhelminthes), in Chinese hamste
243 h2-polarizing Ag (soluble egg Ag (SEA)) from Schistosoma mansoni potently stimulate Th2 responses in
245 larvae (cercariae) of the trematode parasite Schistosoma mansoni rapidly penetrate human skin by degr
248 ome worm antigens were associated with lower Schistosoma mansoni reinfection intensity, while no asso
250 inflammation in infection with the helminth Schistosoma mansoni represents a cellular hypersensitivi
255 encoding a second full-length member of the Schistosoma mansoni RXR family (SmRXR-2) was identified.
256 e Ags from the eggs of the helminth parasite Schistosoma mansoni (schistosome egg Ag (SEA)) leads to
257 cules from the eggs of the helminth parasite Schistosoma mansoni (SEA) suppress LPS-induced activatio
258 crystal structures of the GTPase domain of a Schistosoma mansoni septin (SmSEPT10), one bound to GDP
260 opeptidases (legumains) from the bloodfluke, Schistosoma mansoni (SmAE), and the hard tick, Ixodes ri
262 The taurocyamine kinase from the blood fluke Schistosoma mansoni (SmTK) belongs to the phosphagen kin
263 ilonRI coaggregation mediated by HIVgp120 or Schistosoma mansoni soluble egg Ag accelerated maximal C
266 onses in vivo in CCR8(-/)- mice in models of Schistosoma mansoni soluble egg antigen (SEA)-induced gr
267 -wide preventive chemotherapy strategies for Schistosoma mansoni, spatial scan statistics were used t
268 velop intestinal inflammation, and a control Schistosoma mansoni-specific Th1 clone did not induce co
270 -term continuously proliferative cultures of Schistosoma mansoni sporocysts capable of generating cer
271 earch identified Schistosoma haematobium and Schistosoma mansoni surveys done in, respectively, 9318
273 tigens, including the allergen-like proteins Schistosoma mansoni tegumental-allergen-like 1 protein (
276 ant public health problem and infection with Schistosoma mansoni the major cause of liver disease.
277 t work we assessed the lncRNAs complement of Schistosoma mansoni, the blood fluke that causes schisto
278 In infection with the trematode helminth Schistosoma mansoni, the severity of CD4 T cell-mediated
279 egrated QSAR-based virtual screening (VS) of Schistosoma mansoni thioredoxin glutathione reductase (S
280 ova in mice infected with the human parasite Schistosoma mansoni through mechanisms that are currentl
282 levels in 177 Ugandans (aged 7-50) in a high Schistosoma mansoni transmission area, both before and 7
285 ociation was observed between infection with Schistosoma mansoni, Trichuris, or Strongyloides species
286 ucture of a hammerhead ribozyme derived from Schistosoma mansoni under conditions that permit detaile
289 irst description of an SCP/TAPS gene family (Schistosoma mansoni venom allergen-like (SmVALs)) in the
290 development after the injection of eggs from Schistosoma mansoni was investigated using wild-type (WT
291 dent folding of the hammerhead ribozyme from Schistosoma mansoni was monitored with double electron-e
293 ed by relatively recent empirical studies on Schistosoma mansoni, we use a mathematical model to inve
294 aboons with primary or secondary exposure to Schistosoma mansoni were compared with each other over a
296 T) mice infected with the parasitic helminth Schistosoma mansoni, were found to be severely impaired
297 d antibodies in sera from mice infected with Schistosoma mansoni, which revealed the presence of both
299 we challenge one paradigm by targeting adult Schistosoma mansoni worms for immune elimination in an e
300 te the immune response in mice infected with Schistosoma mansoni, yet the specific contributions of I
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