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1 reduced the abundance of the basal resource (Serratia).
2 strains of the opportunistic human pathogen, Serratia.
3 years; Nocardia, 0.81 per 100 patient-years; Serratia, 0.98 per 100 patient-years, and severe Staphyl
4 y transcriptional activator CarR(39006) from Serratia 39006 has no detectable affinity for cognate AH
5 toxin, showing a remarkable predominance of Serratia and Clostridium species, which switched from as
8 ers of two genera of Gram-negative bacteria, Serratia and Erwinia, produce a beta-lactam antibiotic,
9 convenient spectrophotometric assay, by the Serratia and Pseudomonas approximately 50-kDa extracellu
10 s such as the non-specific endonuclease from Serratia and the sequence-specific His-Cys box homing en
11 . subtilis colonies, swarming by Proteus and Serratia, and spatially organized interspecific metaboli
16 However, estimated rates of encounter with Serratia based on these modifications were higher for in
18 cosmid containing approximately 35 kb of the Serratia chromosome encodes synthesis of the pigment in
19 We report the isolation of a third locus in Serratia, containing convergently transcribed genes, sma
21 tom are conserved in nucleases homologous to Serratia endonuclease, suggesting that the water cluster
24 rom other insect-associated bacteria such as Serratia entomophila, an insect pathogen, and Yersinia p
26 ures of the class A carbapenemase SFC-1 from Serratia fonticola and of complexes of its Ser70 Ala (Mi
27 comprised of (i) a bacterial basal resource (Serratia fonticola), (ii) an intermediate consumer (Para
28 monella, Escherichia, Bacillus, Pseudomonas, Serratia, Hafnia, Enterobacter, Citrobacter, and Lactoba
29 al ABC iron transporters that include Sfu of Serratia, Hit of Haemophilus, and Yfu of Yersinia entero
30 espite a poorly conserved sequence, which in Serratia includes a cysteine bridge thought to play a re
33 ble protease Ser2 is secreted by the species Serratia liquefaciens, a psychrotrophic bacteria frequen
36 r cloacae (9.1%), Acinetobacter spp. (6.2%), Serratia marcescens (5.5%), Enterobacter aerogenes (4.4%
39 east histone acetyltransferase 1) and SmAAT (Serratia marcescens aminoglycoside 3-N-acetyltransferase
40 luding yeast histone acetyltransferase 1 and Serratia marcescens aminoglycoside 3-N-acetyltransferase
41 cal GCN5-related N-acetyltransferase (GNAT), Serratia marcescens aminoglycoside 3-N-acetyltransferase
42 sequences of the cheA loci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrat
43 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli differ in both
44 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli have distinct a
45 entical to the natural product isolated from Serratia marcescens and from overexpression of the biosy
46 sfu and hit operons previously reported for Serratia marcescens and Haemophilus influenzae, respecti
47 ence time of TLM on the ecFabB homologues in Serratia marcescens and Klebsiella pneumonia is an impor
48 Ds accumulate in midgut cells in response to Serratia marcescens and Sindbis virus or when the native
49 nces were noted among Acinetobacter spp. and Serratia marcescens and, to a lesser extent, with Strept
52 ybrid microswimmer system driven by multiple Serratia marcescens bacteria, we quantify the chemotacti
54 idguts after they fed on the insect pathogen Serratia marcescens but not after feeding on the Leishma
55 functional antibiotic resistance enzyme from Serratia marcescens catalyzes adenylation and acetylatio
56 egative bacterium and opportunistic pathogen Serratia marcescens causes ocular infections in healthy
59 , sensitive detection of Escherichia coli or Serratia marcescens cultures from 1 to 10(3) CFU mL(-1).
64 alf-site pairs of the trpEDCBA operator from Serratia marcescens indicated an obligate hierarchy of s
65 eudo-outbreaks of Pseudomonas aeruginosa and Serratia marcescens infections associated with bronchosc
77 olates, 6 Pseudomonas aeruginosa isolates, 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isol
79 to 0.5 nM alpha-thrombin by only 10% whereas Serratia marcescens metalloprotease reduced the Ca2+ res
80 ion of extracellular nuclease (Nuc) from the Serratia marcescens nucA chromosomal locus is inhibited
84 e that is located upstream of NucC-dependent Serratia marcescens promoters and the late promoters of
85 sented here in complex with chitinase B from Serratia marcescens provide further insight into the mec
86 and hhdB, which, based on their homology to Serratia marcescens shlA and shlB genes, are believed to
87 ith exogenous bacteria (Enterobacter sp. and Serratia marcescens strain Db11) and parasitic African t
92 lated data and experimental Tn-Seq data from Serratia marcescens transposon mutant library used to id
93 ce of infection with Burkholderia cepacia or Serratia marcescens was caused by a new strain in 9 of 1
94 ely 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (IC(50), >10,0
95 e, Proteus spp., Pseudomonas aeruginosa, and Serratia marcescens) and 6 antimicrobial resistance dete
96 that coevolution with a bacterial pathogen (Serratia marcescens) resulted in significantly more outc
97 roPhenoloxidase activity, resistance against Serratia marcescens), and for the life history traits, a
98 the clearance of a bacterial infection with Serratia marcescens, 3 Acps significantly reduced the ba
102 structure of anthranilate synthase (AS) from Serratia marcescens, a mesophilic bacterium, has been so
103 no known homologues, a homologue of OmpF of Serratia marcescens, and a locus (designated rscBAC) wit
104 revealed eradication of Pseudomonas species, Serratia marcescens, and Enterobacter aerogenes in most
105 trobacter freundii, Yersinia enterocolitica, Serratia marcescens, and Morganella morganii) and two no
106 (Neisseria gonorrhoeae and N. meningitidis), Serratia marcescens, and other gram-negative bacteria ut
108 rium tumefaciens, Agrobacterium radiobacter, Serratia marcescens, and Pseudomonas aureofaciens) and f
109 st similar to biotin synthases from E. coli, Serratia marcescens, and Saccharomyces cerevisiae (about
110 inst Burkholderia cepacia, Escherichia coli, Serratia marcescens, and Stenotrophomonas maltophilia is
111 we identify a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox.
112 ngs were observed with another CGD pathogen, Serratia marcescens, but not with Escherichia coli.
113 of the enterobacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
114 the enteric bacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
115 nt algorithms, especially in differentiating Serratia marcescens, Escherichia coli, and Yersinia ente
116 phore secreted by the Gram-negative bacteria Serratia marcescens, extracts heme from host hemoprotein
117 film formation in the opportunistic pathogen Serratia marcescens, mutations in an oxyR homolog and pr
118 that contained group B Streptococcus (GBS), Serratia marcescens, or Escherichia coli before their se
119 is (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneumonia, and
120 monas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interfering with their
121 gens (Escherichia coli, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulg
122 li, Salmonella enterica serovar Typhimurium, Serratia marcescens, Shigella flexneri, Enterobacter aer
123 ebsiella pneumoniae, Pseudomonas aeruginosa, Serratia marcescens, Staphylococcus aureus, and Stenotro
124 caused by ingesting the pathogenic bacteria Serratia marcescens, suggesting that subdued has novel f
125 d the same set of D. melanogaster lines with Serratia marcescens, the bacterium used in the previous
126 coli, Pseudomonas spp., Salmonella enterica, Serratia marcescens, Vibrio vulnificus and Enterobacter
127 t the lipases produced by P. fluorescens and Serratia marcescens, which comprise a second sequence fa
128 e describe the structure of chitinase B from Serratia marcescens, which consists of a catalytic domai
129 FLP analysis except for Escherichia coli and Serratia marcescens, which could not be interdifferentia
130 ana, we isolated the Gram-negative bacterium Serratia marcescens, which is a potent entomopathogen th
144 se structures suggests that the magnesium of Serratia nuclease participates in catalysis via an inner
145 analog for which structural data exist, the Serratia nuclease, indicates several interesting differe
146 On the genus level, FF mice had increased Serratia (P < 0.001) and Lactococcus (P < 0.05) whereas
148 irulent bacteriophage (PhiMAM1) that infects Serratia plymuthica was isolated from the natural enviro
149 hin a community dominated by a nearly clonal Serratia population and harboring a lower abundance Ente
153 ble to protease degradation and suggest that Serratia protease is able to differentiate the GPIb-medi
157 a high-resolution crystal structure for the Serratia sp. ATCC 39006 carbapenem resistance protein Ca
161 enterobacterium Erwinia (Pectobacterium) and Serratia sp. ATCC 39006, intrinsic resistance to the car
164 ponsible for prodigiosin biosynthesis in two Serratia sp. In this article we report the creation of i
165 gmented antibiotic, prodigiosin, produced by Serratia sp. is known to involve separate pathways for t
167 ic activity is essential for cytotoxicity in Serratia sp. SCBI and that its regulation appears to be
168 ion of predicted protease genes in wild-type Serratia sp. SCBI, the highest mRNA levels for the alkal
170 ntial for extracellular protease activity in Serratia sp. strain SCBI and to determine what role prot
172 host-associated Gammaproteobacteria species (Serratia sp.) that was absent from soil yet observed in
176 nal fluid cultures growing Enterobacter spp, Serratia spp, or Citrobacter spp were evaluated using th
177 uginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Stenotrophomonas maltophilia (n
178 terococcus spp., Pseudomonas aeruginosa, and Serratia spp. were recovered from infected devices, whil
179 ingle patient isolates of Enterobacter spp., Serratia spp., Citrobacter spp., and Pseudomonas aerugin
180 differentiate Klebsiella, Enterobacter, and Serratia spp., enteric pathogens were identified only by
185 catalytic residue corresponding to Arg57 in Serratia, the structure determined here indicates that A
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