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1       One example is serratin, isolated from Serratia marcescens.
2 ypocrea jecorina and the chitinase ChiA from Serratia marcescens.
3 ost resistance test using the live bacterium Serratia marcescens.
4  controls, including Clostridium species and Serratia marcescens.
5  T6SS from the opportunistic human pathogen, Serratia marcescens.
6 y 18 nonprocessive endochitinase, ChiC, from Serratia marcescens.
7  display a TagJ homologue as shown here with Serratia marcescens.
8 nce to infection with the bacterial pathogen Serratia marcescens.
9 infection by a Gram-negative entomopathogen, Serratia marcescens.
10 acter freundii group, Enterobacter spp., and Serratia marcescens.
11 em receptors from Pseudomonas aeruginosa and Serratia marcescens.
12  study of the processive chitinase ChiA from Serratia marcescens.
13  the clearance of a bacterial infection with Serratia marcescens, 3 Acps significantly reduced the ba
14 r cloacae (9.1%), Acinetobacter spp. (6.2%), Serratia marcescens (5.5%), Enterobacter aerogenes (4.4%
15                       Secretomes from 95% of Serratia marcescens, 71% of Pseudomonas aeruginosa, 29%
16                                              Serratia marcescens, a member of the carbapenem-resistan
17                                              Serratia marcescens, a member of the Enterobacteriaceae
18 structure of anthranilate synthase (AS) from Serratia marcescens, a mesophilic bacterium, has been so
19 east histone acetyltransferase 1) and SmAAT (Serratia marcescens aminoglycoside 3-N-acetyltransferase
20 luding yeast histone acetyltransferase 1 and Serratia marcescens aminoglycoside 3-N-acetyltransferase
21 cal GCN5-related N-acetyltransferase (GNAT), Serratia marcescens aminoglycoside 3-N-acetyltransferase
22  sequences of the cheA loci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrat
23 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli differ in both
24 the aspartate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli have distinct a
25 entical to the natural product isolated from Serratia marcescens and from overexpression of the biosy
26  sfu and hit operons previously reported for Serratia marcescens and Haemophilus influenzae, respecti
27 ence time of TLM on the ecFabB homologues in Serratia marcescens and Klebsiella pneumonia is an impor
28 Ds accumulate in midgut cells in response to Serratia marcescens and Sindbis virus or when the native
29 nces were noted among Acinetobacter spp. and Serratia marcescens and, to a lesser extent, with Strept
30 e, Proteus spp., Pseudomonas aeruginosa, and Serratia marcescens) and 6 antimicrobial resistance dete
31 roPhenoloxidase activity, resistance against Serratia marcescens), and for the life history traits, a
32  no known homologues, a homologue of OmpF of Serratia marcescens, and a locus (designated rscBAC) wit
33 revealed eradication of Pseudomonas species, Serratia marcescens, and Enterobacter aerogenes in most
34 trobacter freundii, Yersinia enterocolitica, Serratia marcescens, and Morganella morganii) and two no
35 (Neisseria gonorrhoeae and N. meningitidis), Serratia marcescens, and other gram-negative bacteria ut
36 ve gram-negative bacteria (Escherichia coli, Serratia marcescens, and Pseudomonas aeruginosa).
37 rium tumefaciens, Agrobacterium radiobacter, Serratia marcescens, and Pseudomonas aureofaciens) and f
38 st similar to biotin synthases from E. coli, Serratia marcescens, and Saccharomyces cerevisiae (about
39 inst Burkholderia cepacia, Escherichia coli, Serratia marcescens, and Stenotrophomonas maltophilia is
40                                        Using Serratia marcescens as a model organism, we identify her
41  we identify a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox.
42 l intensive care unit of a hospital acquired Serratia marcescens bacteremia.
43 ybrid microswimmer system driven by multiple Serratia marcescens bacteria, we quantify the chemotacti
44                               In March 2011, Serratia marcescens bloodstream infections (BSIs) were i
45 idguts after they fed on the insect pathogen Serratia marcescens but not after feeding on the Leishma
46 ngs were observed with another CGD pathogen, Serratia marcescens, but not with Escherichia coli.
47 functional antibiotic resistance enzyme from Serratia marcescens catalyzes adenylation and acetylatio
48 egative bacterium and opportunistic pathogen Serratia marcescens causes ocular infections in healthy
49 of an alpha + beta domain similar to that of Serratia marcescens chitinases A and B.
50                                              Serratia marcescens culture filtrates have been reported
51 , sensitive detection of Escherichia coli or Serratia marcescens cultures from 1 to 10(3) CFU mL(-1).
52              The hemophore protein HasA from Serratia marcescens cycles between two states as follows
53 of the enterobacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
54  the enteric bacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vul
55 nt algorithms, especially in differentiating Serratia marcescens, Escherichia coli, and Yersinia ente
56                                          The Serratia marcescens extracellular nuclease gene, nucA, i
57       A family of mutants overexpressing the Serratia marcescens extracellular nuclease has been know
58                                          The Serratia marcescens extracellular nuclease is a secreted
59 phore secreted by the Gram-negative bacteria Serratia marcescens, extracts heme from host hemoprotein
60                              Hemophores from Serratia marcescens (HasA(sm)) and Pseudomonas aeruginos
61 alf-site pairs of the trpEDCBA operator from Serratia marcescens indicated an obligate hierarchy of s
62 eudo-outbreaks of Pseudomonas aeruginosa and Serratia marcescens infections associated with bronchosc
63 a California hospital acquired postoperative Serratia marcescens infections, and 1 died.
64                      Chitinase B (ChiB) from Serratia marcescens is a family 18 exo-chitinase whose c
65                                              Serratia marcescens is a gram-negative environmental bac
66                                              Serratia marcescens is a soil- and water-derived bacteri
67                                              Serratia marcescens is a well-known cause of nosocomial
68                                              Serratia marcescens is an extremely rare cause of necrot
69                        The enteric bacterium Serratia marcescens is an opportunistic human pathogen.
70                                              Serratia marcescens is an opportunistic pathogen associa
71                                              Serratia marcescens is frequently isolated from lenses o
72                The extracellular nuclease of Serratia marcescens is one of a wide variety of enzymes
73                                            A Serratia marcescens isolate was particularly efficient i
74 olates, 6 Pseudomonas aeruginosa isolates, 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isol
75                      Three bla(SME)-carrying Serratia marcescens isolates and one bla(NDM-1) carrying
76 to 0.5 nM alpha-thrombin by only 10% whereas Serratia marcescens metalloprotease reduced the Ca2+ res
77 film formation in the opportunistic pathogen Serratia marcescens, mutations in an oxyR homolog and pr
78 ion of extracellular nuclease (Nuc) from the Serratia marcescens nucA chromosomal locus is inhibited
79                                          The Serratia marcescens NucC protein is structurally and fun
80                   Extracellular secretion of Serratia marcescens nuclease occurs as a two-step proces
81 immune priming during infections with either Serratia marcescens or with Escherichia coli.
82  that contained group B Streptococcus (GBS), Serratia marcescens, or Escherichia coli before their se
83 is (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneumonia, and
84 monas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interfering with their
85 e that is located upstream of NucC-dependent Serratia marcescens promoters and the late promoters of
86 gens (Escherichia coli, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulg
87 sented here in complex with chitinase B from Serratia marcescens provide further insight into the mec
88  that coevolution with a bacterial pathogen (Serratia marcescens) resulted in significantly more outc
89                            R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenc
90 li, Salmonella enterica serovar Typhimurium, Serratia marcescens, Shigella flexneri, Enterobacter aer
91  and hhdB, which, based on their homology to Serratia marcescens shlA and shlB genes, are believed to
92 ebsiella pneumoniae, Pseudomonas aeruginosa, Serratia marcescens, Staphylococcus aureus, and Stenotro
93 ith exogenous bacteria (Enterobacter sp. and Serratia marcescens strain Db11) and parasitic African t
94  the RNA-binding protein, RsmA, in Ecc71 and Serratia marcescens strain SM274.
95 ed to the chromosome of carbapenem-resistant Serratia marcescens strains.
96  caused by ingesting the pathogenic bacteria Serratia marcescens, suggesting that subdued has novel f
97 d the same set of D. melanogaster lines with Serratia marcescens, the bacterium used in the previous
98 ing medium on prodigiosin (PG) production by Serratia marcescens TKU011 is examined.
99                 We adsorbed swarmer cells of Serratia marcescens to polydimethylsiloxane or polystyre
100 lated data and experimental Tn-Seq data from Serratia marcescens transposon mutant library used to id
101 coli, Pseudomonas spp., Salmonella enterica, Serratia marcescens, Vibrio vulnificus and Enterobacter
102 ce of infection with Burkholderia cepacia or Serratia marcescens was caused by a new strain in 9 of 1
103 ely 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (IC(50), >10,0
104 t the lipases produced by P. fluorescens and Serratia marcescens, which comprise a second sequence fa
105 e describe the structure of chitinase B from Serratia marcescens, which consists of a catalytic domai
106 FLP analysis except for Escherichia coli and Serratia marcescens, which could not be interdifferentia
107 ana, we isolated the Gram-negative bacterium Serratia marcescens, which is a potent entomopathogen th

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