ライフサイエンスコーパス: Sin Nombre virus

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1 ouse (Peromyscus maniculatus), infected with Sin Nombre virus.

2 other HPS-associated hantaviruses, including Sin Nombre virus.

3 cessfully passaged a mouse-adapted strain of Sin Nombre virus from deer mice (Peromyscus maniculatus)

4 ombinant PCMV was constructed that contained Sin Nombre virus glycoprotein G1 (SNV-G1) fused in frame

5 d to clarify the involvement of Cdc42 in the Sin Nombre virus internalization and the signaling pathw

6 of virus assembly was recently described for Sin Nombre virus, it is likely that all of the new Ameri

7 In contrast, the assembly of Sin Nombre virus N protein-HTNV vRNA complexes was inhib

8 d RNA binding by using bacterially expressed Sin Nombre virus N protein.

9 The K(d) values for HTNV and Sin Nombre virus N proteins were nearly identical for th

10 served nucleotides of the panhandle, whereas Sin Nombre virus N requires the first 23 nucleotides for

11 ection with pathogenic hantaviruses, such as Sin Nombre virus or Andes virus (ANDV).

12 he Andes, Puumala, Prospect Hill, Seoul, and Sin Nombre viruses recognize their individual homologous

13 ibition of host autophagy machinery inhibits Sin Nombre virus replication in cells, suggesting that a

14 e deer mouse with its homologous hantavirus, Sin Nombre virus, results in low levels of immune gene e

15 rdiopulmonary syndrome (HCPS), we quantified Sin Nombre virus S segment viral RNA in plasma samples f

16 New York 1 virus (NY-1) and Sin Nombre virus (SN) are associated with hantavirus pul

17 f the core domains of NP (NPcore) encoded by Sin Nombre virus (SNV) and Andes virus (ANDV), which are

18 Sin Nombre virus (SNV) and Hantaan virus (HTN) infect en

19 used to investigate vertical transmission of Sin Nombre virus (SNV) and SNV-specific antibody.

20 Andes virus (ANDV) and Sin Nombre virus (SNV) are rodent-borne hantaviruses tha

21 sid (N) proteins of hantaviruses such as the Sin Nombre virus (SNV) bind to membranes and viral RNAs,

22 Sin Nombre virus (SNV) causes the zoonotic disease hanta

23 a zoonotic disease caused by transmission of Sin Nombre virus (SNV) from chronically infected deer mi

24 , a laboratory animal model for the study of Sin Nombre virus (SNV) infection or associated disease h

25 olved in developing capillary leakage during Sin Nombre virus (SNV) infection.

26 Sin Nombre virus (SNV) is a highly pathogenic New World

27 Sin Nombre virus (SNV) is a rodent-borne hantavirus that

28 The Sin Nombre virus (SNV) is the etiologic agent of hantavi

29 Sin Nombre virus (SNV) is the primary cause of hantaviru

30 Sin Nombre virus (SNV) is thought to establish a persist

31 e sites solubilized in envelope membranes of Sin Nombre virus (SNV) particles.

32 ing of inactivated and fluorescently labeled Sin Nombre virus (SNV) to the integrin PSI domain stimul

33 Sin Nombre virus (SNV), a member of the Hantavirus genus

34 n with HPS-associated hantaviruses, NY-1 and Sin Nombre virus (SNV), is inhibited by antibodies to be

35 s maniculatus) are the natural reservoirs of Sin Nombre virus (SNV), the etiologic agent of most HCPS

36 e we show that rhesus macaques infected with Sin Nombre virus (SNV), the primary etiological agent of

37 igate early events in the innate response to Sin Nombre virus (SNV), the principal etiologic agent of

38 cases of HCPS in North America are caused by Sin Nombre virus (SNV), which is carried asymptomaticall

39 ar responses in both Andes virus (ANDV)- and Sin Nombre virus (SNV)-infected A549 and Huh7-TLR3 cells

40 antavirus cardiopulmonary syndrome caused by Sin Nombre virus (SNV).

41 of Peromyscus maniculatus, the reservoir of Sin Nombre virus (SNV).

42 a rare but acute fulminant disease caused by Sin Nombre virus (SNV).

43 er mice are the principal reservoir hosts of Sin Nombre virus, the etiologic agent of most hantavirus

44 eer mice can be experimentally infected with Sin Nombre virus, which now allows provocative examinati

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