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1 odium azide, 37 degrees C) and in vivo (male Sprague-Dawley rats).
2 ive taste memory on taste preference in male Sprague Dawley rats.
3 endritic release of CCK in the brain in male Sprague Dawley rats.
4 e post-retrieval extinction paradigm in male Sprague Dawley rats.
5 al ischemia-reperfusion injury (IRI) in male Sprague Dawley rats.
6 tegmental area (VTA) of adolescent mice and Sprague Dawley rats.
7 enates and cultured hippocampal neurons from Sprague Dawley rats.
8 One hundred sixteen male Sprague Dawley rats.
9 Twenty-seven healthy Sprague Dawley rats.
10 learned helplessness (cNLH); and (3) control Sprague Dawley rats.
11 seeking, an animal model of relapse, in male Sprague Dawley rats.
12 into separate WAT depots in male and female Sprague Dawley rats.
13 rotid artery occlusion (BCCAO) in adult male Sprague Dawley rats.
14 capable, uncontrollable tail shocks (ISs) in Sprague Dawley rats.
15 uced seizures in urethane anesthetized, male Sprague Dawley rats.
16 ular, and imaging approaches in adult female Sprague Dawley rats.
17 vector and to knock down VAN GLP-1Rs in male Sprague Dawley rats.
18 growth, and phenotype of OMECs cultured from Sprague-Dawley rats.
19 d high CNS permeability in C57Bl/6s mice and Sprague-Dawley rats.
20 e superior colliculus and globus pallidus of Sprague-Dawley rats.
21 owing its intravenous administration to male Sprague-Dawley rats.
22 both autistic-like behavior and epilepsy in Sprague-Dawley rats.
23 nds were created on the palatal mucosa of 54 Sprague-Dawley rats.
24 entration and AQP2 protein in the kidneys of Sprague-Dawley rats.
25 y, with greater effects in BN versus control Sprague-Dawley rats.
26 T ) were recorded in 28 conscious adult male Sprague-Dawley rats.
27 signaling and hedonic expression in 21 male Sprague-Dawley rats.
28 in O/W emulsions after oral gastric feeding Sprague-Dawley rats.
29 AL did not affect CO2 sensitivity in control Sprague-Dawley rats.
30 of this study was to test this hypothesis in Sprague-Dawley rats.
31 enovirus-mediated overexpression of sFlt1 in Sprague-Dawley rats.
32 was induced by cortical contusion injury in Sprague-Dawley rats.
33 ar changes were seen after 6 weeks of VWR in Sprague-Dawley rats.
34 Lewis rats but were significantly altered in Sprague-Dawley rats.
35 lorinated, and provided as drinking water to Sprague-Dawley rats.
36 vivo in DMBA induced mammary tumor in female Sprague-Dawley rats.
37 Agents were administered in diets to female Sprague-Dawley rats.
38 GDER was induced in 6- to 8-week-old male Sprague-Dawley rats.
39 m DORA-22, an analog of the DORA MK-6096, in Sprague-Dawley rats.
40 Male Sprague-Dawley rats.
41 y sustained testosterone suppression in male Sprague-Dawley rats.
42 al day 5 (P5), 14 (P14), and adults (P72) in Sprague-Dawley rats.
43 atial memory for a well-learned task in male Sprague-Dawley rats.
44 behaviors and drug consumption compared with Sprague-Dawley rats.
45 Thirty-two adult male Sprague-Dawley rats.
46 r mandibular molar roots in 65 mature female Sprague-Dawley rats.
47 induced by cecal ligation and perforation in Sprague-Dawley rats.
48 ed liver samples was examined using eighteen Sprague-Dawley rats.
49 cardium 1 week after ischemia-reperfusion in Sprague-Dawley rats.
50 olliphor P 407 gels (KP 407) intranasally in Sprague-Dawley rats.
51 bicin were conducted in male BALB/c mice and Sprague-Dawley rats.
52 adult-born hippocampal neurons in adult male Sprague-Dawley rats.
53 cision in the anterior edentulous maxilla of Sprague-Dawley rats.
54 One dose of amphetamine was injected into Sprague-Dawley rats.
57 tering HUP-A i.p. or intrathecally to female Sprague-Dawley rats (200-235 g body weight) after modera
60 xperiments were conducted using anesthetized Sprague-Dawley rats 3 weeks after treatment with either
61 cordings of phrenic nerve activity in female Sprague Dawley rats (3-4 months) revealed a direct corre
66 ibrillary acidic protein promoter in 62 male Sprague Dawley rats, 4 dominant-negative soluble N-ethyl
69 ted 4 different hemorrhage models using male Sprague-Dawley rats (6 rats/model), aged 10 to 14 weeks
71 Tregs, we studied selected groups of newborn Sprague-Dawley rats according to feeding plan: dam+/-LR1
72 s were administered in an i.v. bolus to male Sprague Dawley rats after starting a s.c. infusion of ME
74 ed Geniposide-induced hepatic injury in male Sprague-Dawley rats after 3-day intragastric administrat
76 to a pharmacokinetic study performed on male Sprague-Dawley rats after administration of a single dos
78 sensitization to cocaine was established in Sprague Dawley rats and was measured by locomotion and b
81 ehaviorally sensitive, low alcohol-consuming Sprague-Dawley rats and DBA/2 mice and behaviorally inse
82 using microdosing of the unlabeled ligand in Sprague-Dawley rats and in wild-type and KOR knockout mi
83 is and/or beta-AR levels in diabetic hearts, Sprague-Dawley rats and miR-133a transgenic (miR-133aTg)
86 Sprague-Dawley rats, SU5416/hypoxia-treated Sprague-Dawley rats, and SU5416/hypoxia-treated C57BL/6
88 g to AKAPs in the nucleus accumbens shell of Sprague-Dawley rats attenuates reinstatement induced by
95 8)F- AMC20: was evaluated in brain slices of Sprague-Dawley rats by in vitro autoradiography and in l
96 TPase (RhoAV14) in the AH outflow pathway in Sprague-Dawley rats by using lentiviral vector-based gen
97 tic dysfunction and hypertension in pregnant Sprague-Dawley rats challenged with lipopolysaccharide (
99 ed behaviors occurred in both Long-Evans and Sprague-Dawley rats despite the fact that the 6-week HFD
100 onverges on the prosurvival role of MEF2D in Sprague Dawley rat embryonic (E18) hippocampal neurons.
103 iver, and fat were collected from adult male Sprague-Dawley rats exposed to increasing doses of the P
104 selectively bred Crl:OP[CD] rats and outbred Sprague-Dawley rats fed an HE diet showing high levels o
111 oltage oscillations at 100 Hz in dissociated Sprague Dawley rat hippocampal neurons in single trial r
113 t that acutely increasing O-GlcNAcylation in Sprague Dawley rat hippocampal slices induces an NMDA re
114 prelimbic region of the mPFC of adult, male, Sprague Dawley rats impaired the acquisition and reconso
117 ion in awake and spontaneously behaving male Sprague Dawley rats interacting with a female, I tested
123 Maxillary second molars were extracted in Sprague Dawley rats (n = 30), and either bisphosphonate
130 ut function from the (18)F-FDG PET images in Sprague-Dawley rats (n = 4) and C57BL/6 mice (n = 5), us
136 neurons from the glabrous skin of adult male Sprague-Dawley rats, NCX activity, as assessed with fura
137 is issue, we transduced primary neurons from Sprague-Dawley rats or APP(-/-) mice (B6.129S7-App(tm1Db
138 analyzed hepatic transcriptional dynamics in Sprague-Dawley rats over a period of 24 hours to assess
141 ein 1) is administered at the spinal cord of Sprague Dawley rats, priming is detected at the peripher
143 ion brain injury or sham surgery, adult male Sprague Dawley rats received vehicle or rolipram (0.03 m
151 ted and high-fat, high-carbohydrate diet-fed Sprague-Dawley rats, respectively, using intravenous adm
153 le burst of high-intensity exercise) in male Sprague-Dawley rats restores the defective hypoglycemia
154 that spinal nerve ligation (SNL, L5) in male Sprague Dawley rats resulted in behavioral allodynia, wh
157 d a multimodal approach, with five groups of Sprague-Dawley rats studied for 8 months: control rats,
158 PH was studied in monocrotaline-treated Sprague-Dawley rats, SU5416/hypoxia-treated Sprague-Dawl
160 nction compared with reconsolidation in male Sprague-Dawley rats that had been trained to self-admini
161 study in chronically catheterized awake male Sprague-Dawley rats that received an acute VMH microinje
162 A controlled laboratory study of 3y male Sprague-Dawley rats that were randomized to 4 groups of
163 we show that in conscious, unrestrained male Sprague Dawley rats the infusion of insulin into the thi
167 ced by 2,4,6-trinitrobenzenesulfonic acid in Sprague Dawley rats to identify inflammation-induced cha
168 cordings in an in vitro slice preparation of Sprague Dawley rats to investigate the effects of physio
169 we used activity-guided optogenetics in male Sprague Dawley rats to silence IL pyramidal neurons opti
170 he pharmacokinetic studies were performed in Sprague-Dawley rats to assess the feasibility of transde
171 nerve as a model of neuropathic pain in male Sprague-Dawley rats to assess the time-dependent changes
172 ime course study was performed exposing male Sprague-Dawley rats to CB11 via nose-only inhalation wit
177 ways and their postsynaptic targets in adult Sprague-Dawley rats to understand how these striatal cir
178 udal extent of the ventrolateral medulla, in Sprague Dawley rats treated with hydralazine or saline.
179 anastomosis rat and sham-operated, pair-fed Sprague-Dawley rats treated with ammonia-lowering therap
180 next generation sequencing of the livers of Sprague-Dawley rats treated with TAA at three doses (4.5
192 Therefore, to address this question, male Sprague-Dawley rats underwent surgeries for implantation
194 n, the extent of covalent protein binding in Sprague-Dawley rats upon exposure to 8:2 FTOH and the 6:
196 s after the initial ischemic injury, in male Sprague-Dawley rats via intraspinal injections of chondr
197 l function for both strains; however, in the Sprague-Dawley rats, VWR exacerbated falls in GFR and RP
207 illation was electrically induced in 30 male Sprague-Dawley rats weighing between 450 and 550 g.
221 RMTg in punished reward seeking, adult male Sprague Dawley rats were tested in several cost-benefit
223 lth in two generations of offspring, GC-eGFP Sprague Dawley rats were weaned onto HFD (45% fat) or Co
225 es of medial prefrontal cortex (mPFC) of six Sprague-Dawley rats were acquired with a transmission el
227 lt (4-6 months) and aged (20-24 months) male Sprague-Dawley rats were anesthetized with isoflurane, p
236 Using a validated ischemic wound model, Sprague-Dawley rats were divided into four groups for da
247 institutional animal care committee, 60 male Sprague-Dawley rats were fed either a standard chow for
286 learning and retention compared with outbred Sprague Dawley rats, whereas bLRs show reduced extinctio
287 C450Y CCT4 was identified in a stock of Sprague-Dawley rats, whereas H147R CCT5 was found in a h
288 frying under vacuum (9.9kPa), after feeding Sprague-Dawley rats, while also understanding its relati
289 vical ganglion (SCG) neurons from adult male Sprague Dawley rats with or without added NGF and compar
292 ) into the dorsal medial NTS (dmNTS) of male Sprague-Dawley rats with coronary artery ligation-induce
294 ation in sympathetic neurons from adult male Sprague-Dawley rats with electrophysiological and optica
296 t maternal inflammation (modeled by pregnant Sprague-Dawley rats with lipopolysaccharides (LPS) chall
297 ry-olfactory artery (ACA/OA) bifurcations in Sprague-Dawley rats with or without ECFCs transfusion.
298 In this study, intrarectal inoculations of Sprague-Dawley rats with predatory bacteria were perform
300 cemic (10-15 mg/dL) clamps were performed in Sprague-Dawley rats with simultaneous electrocardiogram
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