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1 3 CD8 significantly correlated with OTU1348 (Staphylococcus).
2                    Staphylococcus hyicus and Staphylococcus agnetis are two coagulase-variable staphy
3 aracterized by increases in the abundance of Staphylococcus and/or Streptococcus.
4 ts with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%] vs 4 [12.9%]; P = .02)
5                   Aspergillus spp. (17%) and Staphylococcus aureus (10.7%) were the commonest pathoge
6                                              Staphylococcus aureus (12.9%) and Pseudomonas aeruginosa
7 cocci (21%, 10/48) and methicillin-sensitive Staphylococcus aureus (19%, 9/48).
8  positive bacterial pathogens was (88%), and Staphylococcus aureus (50.3%) was the predominantly isol
9   Community-associated methicillin-resistant Staphylococcus aureus (CA-MRSA) are the cause of a sever
10   Community-associated methicillin-resistant Staphylococcus aureus (CA-MRSA) causes infections associ
11 e community-associated methicillin-resistant Staphylococcus aureus (CA-MRSA) epidemic in the United S
12 t, community-acquired, methicillin-resistant Staphylococcus aureus (CA-MRSA) with specific molecular
13 terococcus faecalis, Pseudomonas aeruginosa, Staphylococcus aureus (including clinical isolates of MR
14   Livestock-associated methicillin-resistant Staphylococcus aureus (LA-MRSA) is an emerging problem i
15  control practices for methicillin-resistant Staphylococcus aureus (MRSA) and multidrug-resistant Aci
16 erminal peptide of the methicillin-resistant staphylococcus aureus (MRSA) and self-assembles to form
17 luding the "superbugs" methicillin-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant En
18 iotic activity against methicillin-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant En
19 zed projects involving methicillin-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant en
20  the treatment of both methicillin resistant Staphylococcus aureus (MRSA) and vancomycin-resistant st
21 ere is limited data on methicillin-resistant Staphylococcus aureus (MRSA) carriage in dental clinics.
22                        Methicillin-resistant Staphylococcus aureus (MRSA) caused 57% of the ISIs.
23 was targeted to reduce methicillin-resistant Staphylococcus aureus (MRSA) growth.
24 e rate of infection by methicillin-resistant Staphylococcus aureus (MRSA) has declined over the past
25 e community-associated methicillin-resistant Staphylococcus aureus (MRSA) incidence in the United Sta
26                        Methicillin-resistant Staphylococcus aureus (MRSA) infection is a serious thre
27      The prevalence of methicillin-resistant Staphylococcus aureus (MRSA) infection was 24% and multi
28 Health care-associated methicillin-resistant Staphylococcus aureus (MRSA) infections are a burden on
29 talizations related to methicillin-resistant Staphylococcus aureus (MRSA) infections between 2010 and
30 ch to the treatment of methicillin-resistant Staphylococcus aureus (MRSA) infections by demonstrating
31                        Methicillin-resistant Staphylococcus aureus (MRSA) is a bacterium that causes
32                        Methicillin-resistant Staphylococcus aureus (MRSA) is an emerging cause of cat
33                        Methicillin-resistant Staphylococcus aureus (MRSA) is responsible for large nu
34                        Methicillin-resistant Staphylococcus aureus (MRSA) is the most common healthca
35 f community-associated methicillin-resistant Staphylococcus aureus (MRSA) is unclear.
36 ital and identified 81 methicillin-resistant Staphylococcus aureus (MRSA) isolates.
37 g epidemic lineages of methicillin-resistant Staphylococcus aureus (MRSA) over sensitive isolates (me
38                        Methicillin-resistant Staphylococcus aureus (MRSA) represents a major contribu
39  at increased risk for methicillin-resistant Staphylococcus aureus (MRSA) skin and soft tissue infect
40 ts score, preoperative methicillin-resistant Staphylococcus aureus (MRSA) status, and receipt of mupi
41                        Methicillin-resistant Staphylococcus aureus (MRSA) was first observed in 1960,
42                         Meticillin-resistant Staphylococcus aureus (MRSA) was first reported in 1998
43 ococcus aureus (MSSA), methicillin-resistant Staphylococcus aureus (MRSA), Listeria monocytogenes and
44  with the emergence of methicillin-resistant Staphylococcus aureus (MRSA).
45 cal bacteria including methicillin-resistant Staphylococcus aureus (MRSA).
46 and drug resistance in methicillin-resistant Staphylococcus aureus (MRSA).
47 ng to the phenotype of methicillin-resistant Staphylococcus aureus (MRSA).
48 ng domains specific to methicillin-resistant Staphylococcus aureus (MRSA).
49 iotic activity against methicillin-resistant Staphylococcus aureus (MRSA).
50  treat patients with methicillin-susceptible Staphylococcus aureus (MSSA) infections, beta-lactams ar
51 ive bacteria, namely methicillin-susceptible Staphylococcus aureus (MSSA), methicillin-resistant Stap
52 ignificantly higher in methicillin-resistant Staphylococcus aureus (OR, 2.80; 95% CI, 1.65-4.74) and
53 ome of house dust mites (HDM) has shown that Staphylococcus aureus (S. aureus) and Escherichia coli (
54 that MCL played an anti-inflammatory role in Staphylococcus aureus (S. aureus) and methicillin-resist
55                                              Staphylococcus aureus (S. aureus) carriage and sensitiza
56  handling and consumption in the transfer of Staphylococcus aureus (S. aureus) from livestock to cons
57                                              Staphylococcus aureus (S. aureus) infections are among t
58                  Colonization of the skin by Staphylococcus aureus (S. aureus) is increased in atopic
59                                              Staphylococcus aureus (S. aureus) is one of the most com
60                             Correspondingly, Staphylococcus aureus (S. aureus) isolates from lesional
61 etection of Lactobacillus species (spp.) and Staphylococcus aureus (S. aureus) using gold nanoparticl
62 wed by Streptococci (Strep) species (20.8%), Staphylococcus aureus (SA) (10.2%), other Gram-positive
63 ife-threatening infectious pathogens such as Staphylococcus aureus (SA) and Mycobacterium tuberculosi
64 lights on the surface of the human pathogens Staphylococcus aureus (SaEf-Tu) and Mycoplasma pneumonia
65 only occurred between siblings suggests that Staphylococcus aureus acquisition in our CF population o
66 ium-dependent antimicrobial activity against Staphylococcus aureus and Bacillus subtilis with MICs ra
67 ainst key Gram-positive pathogens (including Staphylococcus aureus and enterococci), a mode of action
68  hospital acquired infections: gram-positive Staphylococcus aureus and gram-negative Pseudomonas aeru
69 n sequester Ni(II) from two human pathogens, Staphylococcus aureus and Klebsiella pneumoniae, that ut
70                                              Staphylococcus aureus and P. jirovecii had higher densit
71 nic wounds are typically polymicrobial, with Staphylococcus aureus and Pseudomonas aeruginosa being t
72                      Chronic coinfections of Staphylococcus aureus and Pseudomonas aeruginosa frequen
73  strains (Bacillus cereus, Escherichia coli, Staphylococcus aureus and Pseudomonas aeruginosa) and in
74 d be observed for pathogenic bacteria (e.g., Staphylococcus aureus and Pseudomonas aeruginosa).
75  2.6 log reductions of methicillin-resistant Staphylococcus aureus and Pseudomonas aeruginosa, respec
76 t lawn biofilms of bioluminescent strains of Staphylococcus aureus and Pseudomonas aeruginosa.
77 ia Escherichia coli, Listeria monocytogenes, Staphylococcus aureus and Salmonella enteritidis.
78 or-stimulating bacteria in the upper airway (Staphylococcus aureus and Staphylococcus epidermidis) an
79 t Escherichia coli, Mycobacterium smegmatis, Staphylococcus aureus and Staphylococcus simulans.
80                    Regarding microorganisms, Staphylococcus aureus and streptococci slightly declined
81 ween CP and two solute-binding proteins that Staphylococcus aureus and Streptococcus pneumoniae, Gram
82 ture had direct bactericidal effects against Staphylococcus aureus and Streptococcus pyogenes and pro
83 ly monitor the growth of both gram-positive (Staphylococcus aureus and Streptococcus pyogenes) and gr
84 oded CRISPR/associated protein 9 (Cas9) from Staphylococcus aureus and Streptococcus pyogenes, and re
85 pecies commonly associated with PJI, namely, Staphylococcus aureus and Streptococcus pyogenes.
86 ning, we employ orthogonal Cas9 enzymes from Staphylococcus aureus and Streptococcus pyogenes.
87 gens (SAgs) are potent exotoxins secreted by Staphylococcus aureus and Streptococcus pyogenes.
88 erial pathogens, including Escherichia coli, Staphylococcus aureus and Vibrio cholera, identified a n
89 loferrin A (SA) and staphyloferrin B (SB) of Staphylococcus aureus are essential for iron acquisition
90  Coagulase-negative staphylococci (CoNS) and Staphylococcus aureus are part of the natural flora of h
91 um of activity of nafithromycin were tested: Staphylococcus aureus ATCC 25923 (disk only), S. aureus
92                                              Staphylococcus aureus bacteraemia is a common cause of s
93 mulates interleukin 10 (IL-10) production in Staphylococcus aureus bacteremia (SaB) animal models, bu
94 have been shown to improve the management of Staphylococcus aureus bacteremia (SAB).
95 ic infection is an important complication of Staphylococcus aureus bacteremia (SAB).
96 -adhering pathogens, such as Shiga toxin and Staphylococcus aureus bacteria.
97 ine, oxacillin and rifampicin) in preventing Staphylococcus aureus biofilms was investigated using Mi
98 r disparities existed in hospital-onset (HO) Staphylococcus aureus bloodstream infections (BSIs) and
99 fflux pump is involved in internalization of Staphylococcus aureus by A549 lung epithelial cells.
100                                              Staphylococcus aureus causes very serious infections of
101                                          The Staphylococcus aureus cell surface contains cell wall-an
102               By contrast, the Gram-positive Staphylococcus aureus cells appeared to be protected fro
103 igate this, we tracked spacer acquisition in Staphylococcus aureus cells harbouring a type II CRISPR-
104 totypic housekeeping sortase A (SaSrtA) from Staphylococcus aureus cleaves LPXTG-containing proteins
105   Livestock-associated methicillin-resistant Staphylococcus aureus clonal complex 398 (LA-MRSA CC398)
106 ed the highest efficiency when P. aeruginosa/Staphylococcus aureus co-culture RNA samples were tested
107 k et al. (2017) find that a commensal blocks Staphylococcus aureus colonization by producing a signal
108                                              Staphylococcus aureus colonization contributes to skin i
109                                              Staphylococcus aureus colonization levels inversely corr
110 asal polyps and asthma, show increased nasal Staphylococcus aureus colonization.
111                                              Staphylococcus aureus commonly colonizes the epidermis,
112                                              Staphylococcus aureus displays a clonal population struc
113 ted with a low-diversity skin microbiota and Staphylococcus aureus dominance.
114 n and the presence of IgE antibodies against Staphylococcus aureus enterotoxins (SAEs).
115                                              Staphylococcus aureus expresses a panel of cell wall-anc
116                                              Staphylococcus aureus forms biofilms on indwelling medic
117 seful single-test adjunct for distinguishing Staphylococcus aureus from S. delphini and other members
118                                              Staphylococcus aureus from these 2 groups were introduce
119  we observed that IFN-beta can directly kill Staphylococcus aureus Further, a mutant S. aureus that i
120     We investigated de novo mutation in 1163 Staphylococcus aureus genomes from 105 infected patients
121 lular nonsiderophilic Y enterocolitica O8 or Staphylococcus aureus Hepcidin analogs may be useful for
122                            Mice with chronic Staphylococcus aureus implant infections were treated by
123 ococcus was isolated in 39 of 63 (62%) eyes, Staphylococcus aureus in 7 of 63 (11%) eyes, and Strepto
124 ve Staphylococci, Pseudomonas aeruginosa and Staphylococcus aureus in keratitis; Streptococcus viridi
125 tyrosine kinase (SYK) activity and uptake of Staphylococcus aureus in microglial cell line BV-2 in a
126 ature myeloid cells, expanded during chronic Staphylococcus aureus infection and promoted bacterial p
127 y to subsequent Streptococcus pneumoniae and Staphylococcus aureus infection as well as the intensity
128 unctionally contribute to protection against Staphylococcus aureus infection.
129 uenza (21 days) did not exacerbate secondary Staphylococcus aureus infection.
130 Clostridium difficile, methicillin-resistant Staphylococcus aureus infections and vancomycin-resistan
131          Multiple candidate vaccines against Staphylococcus aureus infections have failed in clinical
132                                              Staphylococcus aureus infections of the skin and soft ti
133  cells are the etiologic agents of recurrent Staphylococcus aureus infections.
134 promoted the association and phagocytosis of Staphylococcus aureus into macrophages.
135                                              Staphylococcus aureus is a highly successful human patho
136                                              Staphylococcus aureus is a human commensal but also has
137                                              Staphylococcus aureus is a leading cause of both nosocom
138                                              Staphylococcus aureus is a major cause of skin and soft
139                                The bacterium Staphylococcus aureus is a major human pathogen for whic
140                                              Staphylococcus aureus is a medically important pathogen
141      The type VII secretion system (T7SS) of Staphylococcus aureus is a multiprotein complex dedicate
142                                              Staphylococcus aureus is a serious human pathogen with r
143                                              Staphylococcus aureus is an AD-associated pathogen produ
144                                              Staphylococcus aureus is an important opportunistic path
145                                              Staphylococcus aureus is an opportunistic human pathogen
146                                              Staphylococcus aureus is an opportunistic pathogen and v
147                         Skin colonization by Staphylococcus aureus is associated with severity of ato
148                                              Staphylococcus aureus is highly adapted to its host and
149                                              Staphylococcus aureus is in competition for colonization
150                                              Staphylococcus aureus is the leading cause of infection
151                                              Staphylococcus aureus is the leading cause of skin and s
152                                              Staphylococcus aureus is the leading cause of skin and s
153                                              Staphylococcus aureus is the most common cause of skin a
154                                              Staphylococcus aureus is the most common infectious agen
155 r differentiating S. hyicus, S. agnetis, and Staphylococcus aureus Isolates (n = 62) were selected fr
156 elatedness and assess population dynamics of Staphylococcus aureus isolates from a cohort of CF patie
157  approximately 85% of all relaxases found in Staphylococcus aureus isolates.
158 tect mecC-mediated beta-lactam resistance in Staphylococcus aureus Kriegeskorte and colleagues report
159                                              Staphylococcus aureus NOS (saNOS) has previously been sh
160 ional structures of ribosomal particles from Staphylococcus aureus obtained by X-ray crystallography
161 lity of phosphate for all forms of life, how Staphylococcus aureus obtains this nutrient during infec
162 onic biofilm-associated infections caused by Staphylococcus aureus often lead to significant increase
163 t of diseases such as tuberculosis, malaria, Staphylococcus aureus or gonorrhea has led to rapidly in
164  It is tougher to treat than most strains of Staphylococcus aureus or staph, because it is resistant
165 t affect phagocytosis of Escherichia coli or Staphylococcus aureus or their intracellular killing.
166  and local epidemiological investigations of Staphylococcus aureus outbreaks.
167 ificant selectivity for PrkA relative to the Staphylococcus aureus PASTA kinase Stk1.
168         Remarkably, the clinically important Staphylococcus aureus pathogenicity islands (SaPIs) use
169                                              Staphylococcus aureus pathogenicity islands (SaPIs), suc
170 lose sequence relationship between PBP 3 and Staphylococcus aureus PBP 2A, which is responsible for m
171                                              Staphylococcus aureus plays an important role in sepsis,
172              Antibiotic-resistant strains of Staphylococcus aureus pose a major threat to human healt
173                       Airway-colonization by Staphylococcus aureus predisposes to the development of
174 ress response as seen, for example, with the Staphylococcus aureus PRF or sulfide oxidation and dispo
175 2017) define a pathway by which epicutaneous Staphylococcus aureus promotes skin inflammation and may
176 trated that this protease rapidly hydrolyzes Staphylococcus aureus protein A, an important S. aureus
177    To enable high-accuracy quantification of Staphylococcus aureus proteins, we have developed a glob
178 dulin alpha3 (PSMalpha3) peptide secreted by Staphylococcus aureus PSMalpha3 formed elongated fibrils
179 lar microbes, such as the bacterial pathogen Staphylococcus aureus Recruitment and activation of neut
180 do not usually develop effective immunity to Staphylococcus aureus reinfection.
181                                              Staphylococcus aureus remains a causative agent for morb
182 em into groups, including tandem repeats and Staphylococcus aureus repeat (STAR)-like elements.
183 luate the effect of route of administration, Staphylococcus aureus skin colonization, and disease sev
184  precluded primary outcome meta-analysis for Staphylococcus aureus skin or soft-tissue infections.
185                            Here, we describe Staphylococcus aureus sortase A-mediated crosslinking of
186 ed bacteria including a multi-drug resistant Staphylococcus aureus strain Y5 and ampicillin resistant
187              However, superantigen-producing Staphylococcus aureus strains are often part of the huma
188 ma-primed MCs guide activation of T cells by Staphylococcus aureus superantigen and, when preincubate
189 y was to test the ability of WLBU2 to remove Staphylococcus aureus surgical implant biofilms.
190 2-component leukotoxin LukAB is critical for Staphylococcus aureus targeting and killing of human neu
191                       Phenotypic variants of Staphylococcus aureus that display small colonies, reduc
192 tor on the surface of the bacterial pathogen Staphylococcus aureus that extracts heme from hemoglobin
193 e followed 3 wk later by an i.v. exposure to Staphylococcus aureus This procedure resulted in a marke
194 d secretion in response to nigericin and the Staphylococcus aureus toxin leukocidin AB (LukAB).
195 ome-wide screen and identified the essential Staphylococcus aureus tRNA m(1)G37 methyltransferase enz
196          Finally, a modular synthesis of the Staphylococcus aureus type 5 capsular polysaccharide rep
197              Small-colony variants (SCVs) of Staphylococcus aureus typically lack a functional electr
198  al. (2017) show that the bacterial pathogen Staphylococcus aureus unexpectedly secretes and repurpos
199                   The Gram-positive pathogen Staphylococcus aureus uses one primary resistance mechan
200                 The bacterial human pathogen Staphylococcus aureus uses oxygen as a terminal electron
201  activity against Pseudomonas aeruginosa and Staphylococcus aureus was assessed by microdilution assa
202 aused by Escherichia coli, Enterococcus, and Staphylococcus aureus we observed that cocolonization wi
203           Using this method 80.3 +/- 5.6% of Staphylococcus aureus with a starting concentration of
204 ssociation between Clostridium neonatale and Staphylococcus aureus with NEC (P = 0.001 and P = 0.002)
205 L and 95.4 +/- 1.0% of Methicillin-resistant Staphylococcus aureus with starting concentration of 10
206 d short palindromic repeat-associated 9 from Staphylococcus aureus) and guide RNA constructs into an
207  increased susceptibility to both bacterial (Staphylococcus aureus) and viral (murine CMV) infection
208 nella typhimurium, and methicillin-resistant Staphylococcus aureus) were successfully isolated and de
209 tions of those methods, were used to test 10 Staphylococcus aureus, 10 Streptococcus pneumoniae, 10 H
210 rgic reactions to the gram-positive pathogen Staphylococcus aureus, a frequent colonizer of the upper
211 the outcome of systemic infections caused by Staphylococcus aureus, a leading cause of bacterial endo
212                                              Staphylococcus aureus, a metabolically flexible gram-pos
213 g on NorA, the most important efflux pump of Staphylococcus aureus, an efflux pump inhibitors (EPIs)
214 nk between EET formation and the presence of Staphylococcus aureus, an organism frequently colonizing
215 eir ability to internalize Escherichia coli, Staphylococcus aureus, and Bacillus anthracis particles.
216 ebsiella pneumoniae, Pseudomonas aeruginosa, Staphylococcus aureus, and coagulase-negative staphyloco
217 igh-density populations of Escherichia coli, Staphylococcus aureus, and Mycobacterium smegmatis to qu
218 emophilus influenzae, Moraxella catarrhalis, Staphylococcus aureus, and Pneumocystis jirovecii.
219 at suppress the growth of a common pathogen, Staphylococcus aureus, and predicted which genera were a
220 y RNAs (sRNA) in P. aeruginosa as well as in Staphylococcus aureus, another important human pathogen
221 monas aeruginosa, Moraxella catarrhalis, and Staphylococcus aureus, bacteria that occasionally coloni
222 ae after challenge with Escherichia coli and Staphylococcus aureus, but had no significant effect aft
223 e been implicated in nosocomial outbreaks of Staphylococcus aureus, but the dearth of evidence from n
224                       Gram-negative bacilli, Staphylococcus aureus, Chlamydia, Mycoplasma, and Legion
225                                           In Staphylococcus aureus, ClpP associates to the substrate
226                       As far as this review, Staphylococcus aureus, Coagulase negative Staphylococci,
227 l presence, including the frequent colonizer Staphylococcus aureus, contributes to inhibition of heal
228 t Gram-positive and Gram-negative pathogens (Staphylococcus aureus, Enterobacteriaceae, Pseudomonas a
229  increased uptake in vitro in live bacteria (Staphylococcus aureus, Escherichia coli, and Pseudomonas
230 y in vitro uptake in representative bacteria-Staphylococcus aureus, Escherichia coli, Pseudomonas aer
231 he growth of gram-positive (Bacillus cereus, Staphylococcus aureus, Listeria monocytogenes, Geobacill
232 apid and simultaneous identification (ID) of Staphylococcus aureus, Staphylococcus lugdunensis, and S
233 he world, in the United States for instance, Staphylococcus aureus, Streptococcus pneumoniae and Haem
234 ata sets from six diverse bacterial species: Staphylococcus aureus, Streptococcus pneumoniae, Mycobac
235                                 However, for Staphylococcus aureus, the accumulation of within-host d
236                In the Gram-positive pathogen Staphylococcus aureus, the membrane-bound ATP-dependent
237                                              Staphylococcus aureus, the most common pathogen in bone
238 on and infection due to meticillin-resistant Staphylococcus aureus, vancomycin-resistant enterococci,
239  infecting the clinically relevant bacterium Staphylococcus aureus, we demonstrate for the first time
240   By examining secreted virulence factors of Staphylococcus aureus, we determine that the bacterial l
241 decision in the opportunistic human pathogen Staphylococcus aureus, which generates the phenotypic bi
242                                              Staphylococcus aureus, which has become the predominant
243 ns are a family of potent toxins secreted by Staphylococcus aureus, which target white blood cells pr
244 ed honeys against Pseudomonas aeruginosa and Staphylococcus aureus, with a particular focus on two ma
245 infection, specifically Escherichia coli and Staphylococcus aureus, with differences summarized throu
246  patients presented with significantly lower Staphylococcus aureus-specific serum IgG compared to cys
247 119 targets from Acinetobacter baumannii and Staphylococcus aureus.
248                 We probe this strategy using Staphylococcus aureus.
249 nas aeruginosa, Acinetobacter baumannii, and Staphylococcus aureus.
250 lly, naturally and persistently colonised by Staphylococcus aureus.
251  the emergence of antibiotic-resistant (ABR) Staphylococcus aureus.
252 NF) caused by group A Streptococcus (GAS) or Staphylococcus aureus.
253 topic dermatitis are frequently colonized by Staphylococcus aureus.
254  detachment process for the bacterial strain Staphylococcus aureus.
255 ganisms on the in vivo 'essential' genome of Staphylococcus aureus.
256 stic microorganisms such as enteric rods and Staphylococcus aureus.
257  are unable to complement an L27 deletion in Staphylococcus aureus.
258 ciated infections like methicillin-resistant Staphylococcus aureus.
259 PR/Cas9 antimicrobial, broadly applicable to Staphylococcus aureus.
260 nterococcus faecium and meticillin-resistant Staphylococcus aureus.
261 found that spent media from the CoNS species Staphylococcus caprae can inhibit agr-mediated quorum se
262 r pyrrolidonyl arylamidase (PYR) activity in Staphylococcus delphini This study evaluated PYR activit
263 inetobacter and Moraxella species (type II), Staphylococcus epidermidis (type III), Porphyromonas and
264                                              Staphylococcus epidermidis harbours a Type III-A CRISPR-
265                                              Staphylococcus epidermidis has emerged as an important o
266 ding clinical isolates of MRSA and MSSA) and Staphylococcus epidermidis identified one candidate that
267 nd show that the common blood-borne pathogen Staphylococcus epidermidis influences this in vitro mode
268       The otherwise harmless skin inhabitant Staphylococcus epidermidis is a major cause of healthcar
269                                              Staphylococcus epidermidis is one of the primary bacteri
270 Similar proteolytic capacities were found in Staphylococcus epidermidis isolates but not in Staphyloc
271           Here we show that targeting by the Staphylococcus epidermidis type III-A CRISPR-Cas system
272                                              Staphylococcus epidermidis was used as the model target
273  the upper airway (Staphylococcus aureus and Staphylococcus epidermidis) and intestinal microbiota (L
274                                 We show that Staphylococcus epidermidis, a commensal bacterium in the
275                                              Staphylococcus epidermidis, a major component of skin fl
276 d on surfaces, and belong to species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseud
277                 The glucose transporter from Staphylococcus epidermidis, GlcPSe, is a homolog of the
278  a significant reduction in viable counts of Staphylococcus epidermidis, Saccharomyces cerevisiae, an
279 by severe sepsis with positive blood culture Staphylococcus haemolyticus, septic shock, multiple orga
280                                              Staphylococcus hyicus and Staphylococcus agnetis are two
281 us from S. delphini and other members of the Staphylococcus intermedius group.
282  from broth microdilution MIC testing of 993 Staphylococcus lugdunensis isolates recovered from patie
283 dentification (ID) of Staphylococcus aureus, Staphylococcus lugdunensis, and Staphylococcus species t
284 lum, and Corynebacterium, Acinetobacter, and Staphylococcus, often involved in mastitis cases, were t
285 es the B cell response through expression of staphylococcus protein A (SpA), a surface protein that d
286 a dominated by proinflammatory bacteria (eg, Staphylococcus, Pseudomonas, and Corynebacterium), anabo
287                            In the absence of Staphylococcus (S.) aureus in NPF, antibiotic therapy as
288 ys, inhibited IFN-beta and in the absence of Staphylococcus (S.) aureus, induced ST2 bearing cells in
289 aphylococcus epidermidis isolates but not in Staphylococcus saprophyticus Galectin-3-induced activati
290 pearance of the multidrug resistant bacteria Staphylococcus simulans in the fetal brain.
291 cterium smegmatis, Staphylococcus aureus and Staphylococcus simulans.
292                 A. actinomycetemcomitans and Staphylococcus species do not seem to play an important
293                               A total of 658 Staphylococcus species isolates (S. aureus, 211 isolates
294 ccus aureus, Staphylococcus lugdunensis, and Staphylococcus species to the genus level and the detect
295 of Aggregatibacter actinomycetemcomitans and Staphylococcus species was very low.
296 11 isolates; S. lugdunensis, 3 isolates; and Staphylococcus spp., 444 isolates) were recovered from m
297 ram positive bacteria (Enterococcus spp. and Staphylococcus spp., including two MRSA strains 0.3-8 mu
298  Cellulophaga lytica, Bacillus aquimaris and Staphylococcus warneri were undertaken.
299 mmensal isolates such as Microbacterium sp., Staphylococcus warneri, Flectobacillus major, Arthrobact
300                           Coagulase-negative Staphylococcus was isolated in 39 of 63 (62%) eyes, Stap

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