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1 was sporadically positive in a patient with Stevens-Johnson syndrome.
2 ocular cicatricial pemphigoid>chemical burns>Stevens-Johnson syndrome.
5 ty and HLA-B*57:01 and carbamazepine-induced Stevens-Johnson syndrome and HLA-B*15:02 have been imple
7 oat (ENT) lesions are frequently involved in Stevens-Johnson syndrome and toxic epidermal necrolysis
10 valuated for the life-threatening dermatoses Stevens-Johnson syndrome and toxic epidermal necrolysis.
11 erum levels and the outcome of patients with Stevens-Johnson syndrome and toxic epidermal necrolysis.
12 mmune mechanisms in the pathogenesis of both Stevens-Johnson syndrome and vanishing bile duct syndrom
13 keratopathy, lattice and Avellino dystrophy, Stevens-Johnson syndrome, and chemical/thermal injury.
14 are bilaterally blind from diseases such as Stevens-Johnson syndrome, and ocular pemphigoid have lit
15 mediated, or type I, reactions, anaphylaxis, Stevens-Johnson syndrome, and toxic epidermal necrolysis
16 disorders such as graft-versus-host disease, Stevens-Johnson syndrome, and toxic epidermal necrolysis
18 iously to corneal diseases, astigmatism, and Stevens-Johnson syndrome fall within corneal epithelial
19 The most common indications for surgery were Stevens-Johnson syndrome in 41.7% (20 of 48 eyes) and mu
22 e vanishing bile duct syndrome shortly after Stevens-Johnson syndrome is described; this was temporal
23 tric oxide in toxic epidermal necrolysis and Stevens-Johnson syndrome may cause the epidermal apoptos
24 patients with bronchiolitis obliterans from Stevens-Johnson syndrome often have progressive disease
25 rom seven patients with actively progressing Stevens-Johnson syndrome or toxic epidermal necrolysis.
26 notypes, including the blistering conditions Stevens-Johnson syndrome (SJS) and toxic epidermal necro
27 nce of a relationship between HLA-B*1502 and Stevens-Johnson syndrome (SJS) and toxic epidermal necro
32 ffective ophthalmologic treatments for acute Stevens-Johnson syndrome (SJS) as well as the emerging t
35 tion of drug-induced liver injury (DILI) and Stevens-Johnson syndrome (SJS) or toxic epidermal necros
37 tively analyzed 74 cases of SCARs, including Stevens-Johnson syndrome (SJS), toxic epidermal necrolys
39 vity reactions (DHR) induced by PPI, such as Stevens-Johnson syndrome (SJS), toxic epidermal necrolys
40 redisposition to carbamazepine (CBZ)-induced Stevens-Johnson syndrome (SJS)/toxic epidermal necrolysi
41 nced a mucocutaneous reaction described as a Stevens-Johnson syndrome that was attributed to chemothe
43 nical ventilation is required in one of four Stevens-Johnson syndrome/toxic epidermal necrolysis pati
45 nophilia and systemic symptoms syndrome, and Stevens-Johnson syndrome/toxic epidermal necrolysis, can
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