ライフサイエンスコーパス: Streptococcus

1 es in the abundance of Staphylococcus and/or Streptococcus.

2 187, nigericin, Candida albicans and Group B Streptococcus.

3 d over 11 y ago showed the highest levels of Streptococcus (18.4%), Haemophilus (12.7%) and Neisseria

4 The most frequent species was Streptococcus agalactiae (34%), and 52% of all cases wer

5 Streptococcus agalactiae (GBS) is the leading cause worl

6 Streptococcus agalactiae (group B Streptococcus [GBS]) c

7 Streptococcus agalactiae (group B Streptococcus [GBS]) h

8 Streptococcus agalactiae (group B Streptococcus [GBS]) i

9 Streptococcus agalactiae (group B Streptococcus, GBS) is

10 tans (MetR), Streptococcus iniae (CpsY), and Streptococcus agalactiae (MtaR) that regulate methionine

11 ometric biosensor for the rapid detection of Streptococcus agalactiae (S. agalactiae).

12 GacI homologs perform a similar function in Streptococcus agalactiae and Enterococcus faecalis In co

13 Group B Streptococcus (GBS) or Streptococcus agalactiae are beta-hemolytic gram-positiv

14 Streptococcus agalactiae can cause urinary tract infecti

15 The burden of disease caused by Streptococcus agalactiae has increased significantly amo

16 splenic infarction as rare complications of Streptococcus agalactiae infective endocarditis.

17 Group B streptococcus (GBS) or Streptococcus agalactiae is a beta-hemolytic, Gram-posit

18 eria meningitidis, Streptococcus pneumoniae, Streptococcus agalactiae, cytomegalovirus, enterovirus,

19 ra included Staphlyococcus, Pseudomonas, and Streptococcus, all of which have proinflammatory and pat

20 a and other typical early colonisers such as Streptococcus and Enterobacteriaceae, iii) domination of

21 Group B streptococcus and respiratory syncytial virus are leadin

22 l immunisation strategies to prevent group B streptococcus and respiratory syncytial virus infections

23 nown about immune protection against group B streptococcus and respiratory syncytial virus, identifie

24 of the healthy steady state (eg, Prevotella, Streptococcus, and Veillonella).

25 gram-positive bacteria, such as Bacillus and Streptococcus, are small, linear peptides secreted from

26 h media to have antibacterial action against Streptococcus bacteria.

27 se correlated with an increased abundance of Streptococcus bovis and a decreased abundance of the Bac

28 Typhimurium LT2, Staphylococcus aureus, and Streptococcus bovis).

29 serogroup C (MenC) or Gram-positive group B Streptococcus, capsular type III (GBS-III) bacteria resu

30 015, that reported the prevalence of group B streptococcus colonisation in pregnant women.

31 did a systematic review of maternal group B streptococcus colonisation studies by searching MEDLINE,

32 ODs and TLRs, whereas increased abundance of Streptococcus correlated with increased NOD-like recepto

33 enrichment of either Haemophilus influenzae, Streptococcus, Corynebacterium, Moraxella, or Staphyloco

34 eported an antagonistic relationship between Streptococcus cristatus and P. gingivalis, and identifie

35 d that an 11-mer peptide (SAPP) derived from Streptococcus cristatus arginine deiminase (ArcA) was ab

36 abundance of Fusobacterium increased whereas Streptococcus decreased in both primary and metastatic s

37 Furthermore, Group A Streptococcus did not survive better on the skin after w

38 inst both early-onset and late-onset group B streptococcus disease.

39 within the homodimer interface of Rgg2 from Streptococcus dysgalactiae (Rgg2Sd).

40 ted from the cell envelop of bovine mastitis Streptococcus dysgalactiae 2023 is reported for the firs

41 t results for trimethoprim-sulfadiazine with Streptococcus equi subspecies are interpreted based on h

42 ts, belonging to Fusobacterium, Pseudomonas, Streptococcus, Filifactor, and Parvimonas.

43 Colonization by Streptococcus gallolyticus subsp. gallolyticus (SGG) is

44 Group A Streptococcus (GAS) acquires mutations of the virulence

45 rface-associated virulence factor of group A Streptococcus (GAS) and an antigenically variable target

46 Hypervirulent group A streptococcus (GAS) can inhibit neutrophil recruitment a

47 Hypervirulent disease due to group A Streptococcus (GAS) can result from strains with mutatio

48 We compared group A Streptococcus (GAS) culture with a rapid helicase-depend

49 Studies of the human pathogen group A Streptococcus (GAS) define the carrier phenotype to be a

50 Group A Streptococcus (GAS) has acquired an arsenal of virulence

51 with a prevalence as high as that of group A Streptococcus (GAS) in adolescents and young adults.

52 Rheumatic heart disease (RHD) after group A streptococcus (GAS) infections is heritable and prevalen

53 Group A Streptococcus (GAS) is a human-only pathogen that causes

54 Group A Streptococcus (GAS) is a major human pathogen that cause

55 Group A Streptococcus (GAS) is among the top ten causes of infec

56 isease outbreaks in humans caused by group A Streptococcus (GAS) is an on-going public health threat.

57 necrotizing fasciitis (NF) caused by group A Streptococcus (GAS) or Staphylococcus aureus.

58 Group A Streptococcus (GAS) skin infections are particularly pre

59 This is true for the group A Streptococcus (GAS), a pathogen that causes both invasiv

60 tance by one leading human pathogen, group A Streptococcus (GAS).

61 The Group A Streptococcus (GAS, Streptococcus pyogenes) is a Gram-po

62 ing the human bacterial pathogen the group A Streptococcus (GAS; S. pyogenes) as a model organism, we

63 athogen, Streptococcus pyogenes (the group A streptococcus [GAS]) has specifically adapted to evade h

64 ted pathogen Streptococcus pyogenes (Group A Streptococcus, GAS) has long focused on invasive illness

65 Infections such as group B Streptococcus (GBS) are an important cause of maternal s

66 antial progress in the prevention of group B Streptococcus (GBS) disease with the introduction of int

67 Group B streptococcus (GBS) infection is a leading cause of deat

68 Group B Streptococcus (GBS) is an encapsulated, gram-positive pa

69 Group B Streptococcus (GBS) is an important perinatal pathogen,

70 ernal rectovaginal colonization with group B Streptococcus (GBS) is the most common pathway for GBS d

71 died the population structure of 102 group B Streptococcus (GBS) isolates prospectively sampled in 20

72 Studies have suggested that group B Streptococcus (GBS) maternal rectovaginal colonization d

73 Maternal vaccination against group B Streptococcus (GBS) might provide protection against inv

74 Group B Streptococcus (GBS) or Streptococcus agalactiae are beta

75 Group B streptococcus (GBS) or Streptococcus agalactiae is a bet

76 Group B Streptococcus (GBS) remains a leading cause of neonatal

77 ntal antibody transfer specific to 8 group B Streptococcus (GBS) surface proteins among 81 HIV-uninfe

78 ho are rectovaginally colonized with group B Streptococcus (GBS), but the risk of EOGBS from vertical

79 rehensive estimates of the burden of group B Streptococcus (GBS), including invasive disease in pregn

80 Streptococcus agalactiae (group B Streptococcus [GBS]) causes serious infections in neonat

81 Streptococcus agalactiae (group B Streptococcus [GBS]) has not been described as a foodbor

82 Streptococcus agalactiae (group B Streptococcus [GBS]) is an important neonatal pathogen a

83 Streptococcus agalactiae (group B Streptococcus, GBS) is a leading cause of invasive bacte

84 taxa at high relative abundance and reduced Streptococcus, Gemella, and Porphyromonas taxa relative

85 Streptococcus genus probe 4 and Neisseria genus probe 2

86 could detect 65 +/- 10 muM H2O2 produced by Streptococcus gordonii (Sg) in a simulated biofilm at 50

87 nge between two bacterial species, commensal Streptococcus gordonii and pathogenic Streptococcus muta

88 Streptococcus gordonii and Streptococcus sanguinis are p

89 Studies revealed that S. oralis, like Streptococcus gordonii and Streptococcus sanguinis, bind

90 igher, respectively, in the S-ECC group, and Streptococcus gordonii and Streptococcus sanguinis, whic

91 omonas gingivalis and the accessory pathogen Streptococcus gordonii interact to form communities in v

92 an important determinant of colonization by Streptococcus gordonii, an oral commensal and opportunis

93 icroorganisms, e.g., Veillonella parvula and Streptococcus gordonii, stimulated higher levels of ROS

94 In the gram-positive Streptococcus gordonii, the ability to form disulfide bo

95 erobes, including aerotolerant ones, such as Streptococcus gordonii, use pyruvate dehydrogenase to de

96 commensal species Streptococcus sanguinis or Streptococcus gordonii.

97 An emm32.2 invasive group A streptococcus (iGAS) outbreak occurred in Liverpool from

98 isease samples revealed greater abundance of Streptococcus in benign vocal fold disease suggesting th

99 Group B streptococcus infection can be associated with invasive

100 rysms of visceral arteries caused by Group B streptococcus infection.

101 l regulators of Streptococcus mutans (MetR), Streptococcus iniae (CpsY), and Streptococcus agalactiae

102 Streptococcus intermedius is an opportunistic bacterial

103 the LXG polymorphic toxin family present in Streptococcus intermedius mediate cell contact- and Esx

104 culture found Porphyromonas endodontalis and Streptococcus intermedius, and specific culture found Me

105 were greater proportions of Actinomyces and Streptococcus-like species and lower proportions of Veil

106 disease suggesting that mucosal dominance by Streptococcus may be a factor in disease etiology.

107 Oral taxon 44, while Streptococcus mitis, Rothia mucilaginosa and Haemophilus

108 omologous with transcriptional regulators of Streptococcus mutans (MetR), Streptococcus iniae (CpsY),

109 ported identification of two Spx proteins in Streptococcus mutans - SpxA1 was the primary activator o

110 the oral cavity with the cariogenic pathogen Streptococcus mutans and other bacteria.

111 n reaction (PCR)/quantitative PCR (qPCR) for Streptococcus mutans and Streptococcus sobrinus.

112 Streptococcus mutans biofilm was disrupted from sandblas

113 The Streptococcus mutans Cid/Lrg system represents an ideal

114 Streptococcus mutans employs a key virulence factor, thr

115 frequently detected with heavy infection of Streptococcus mutans in plaque-biofilms from children af

116 Streptococcus mutans is a biofilm-forming oral pathogen

117 The cnm gene in Streptococcus mutans isolated from saliva was screened u

118 The cariogenic bacterial species Streptococcus mutans metabolizes sucrose to initiate bio

119 ace and competes with oral pathogens such as Streptococcus mutans to maintain oral health.

120 Extremely high levels of Streptococcus mutans were detected in the S-ECC group.

121 significant antibiofilm bioactivity against Streptococcus mutans, a causative agent of human dental

122 In subjects positive for Streptococcus mutans, collagen binding activity was posi

123 ll-known acidogenic/aciduric species such as Streptococcus mutans, Scardovia wiggsiae, Parascardovia

124 In Streptococcus mutans, Streptococcus suis, and species of

125 Among these is Streptococcus mutans, the causative agent of dental cari

126 However, the function of this molecule in Streptococcus mutans, the primary aetiological agent of

127 mensal Streptococcus gordonii and pathogenic Streptococcus mutans.

128 Streptococcus oralis is a leading cause of subacute infe

129 Actinomyces naeslundi and Streptococcus oralis levels were significantly higher in

130 d in an exaggerated inflammatory response to Streptococcus pneumonia, with increases in neutrophil mo

131 Streptococcus pneumoniae (pneumococcus) is a major human

132 Streptococcus pneumoniae (pneumococcus) is an oval-shape

133 Streptococcus pneumoniae (pneumococcus) produces many ca

134 e-wall like) peptidoglycan (PG) synthesis in Streptococcus pneumoniae (pneumococcus); yet, mechanisms

135 Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae (S. pneumoniae), and Haemophilu

136 Populations of Streptococcus pneumoniae (SP) are typically structured i

137 Streptococcus pneumoniae (SP) is a pathogenic bacterium

138 ly cover only 13 of the over 90 serotypes of Streptococcus pneumoniae (Sp), so nonvaccine serotypes a

139 ulence of important human pathogens, such as Streptococcus pneumoniae (Sp).

140 Streptococcus pneumoniae (the pneumococcus) is a human p

141 Streptococcus pneumoniae (the pneumococcus) is the leadi

142 Even in the vaccine era, Streptococcus pneumoniae (the pneumococcus) remains a le

143 s is a prerequisite for the human pathobiont Streptococcus pneumoniae (the pneumococcus) to cause sev

144 Catabolism of galactose by Streptococcus pneumoniae alters the microbe's metabolism

145 ureus in keratitis; Streptococcus viridians, Streptococcus pneumoniae and Coagulase negative Staphylo

146 States for instance, Staphylococcus aureus, Streptococcus pneumoniae and Haemophilus influenzae are

147 R events in the gram-positive human pathogen Streptococcus pneumoniae and in the human genome.

148 atory infection by the major human pathogens Streptococcus pneumoniae and Klebsiella pneumoniae.

149 us aureus, Coagulase negative Staphylococci, Streptococcus pneumoniae and Pseudomonas aeruginosa are

150 ng affects both susceptibility to subsequent Streptococcus pneumoniae and Staphylococcus aureus infec

151 Respiratory viruses and Streptococcus pneumoniae are known to be copathogens in

152 terococcus faecalis ATCC 29212 (broth only), Streptococcus pneumoniae ATCC 49619 (disk and broth), an

153 In mice, Streptococcus pneumoniae can invade the myocardium, indu

154 Before 1945, Streptococcus pneumoniae caused more than 90% of cases o

155 Streptococcus pneumoniae commonly inhabits the nasophary

156 this study, we use glycoconjugates of type 3 Streptococcus pneumoniae CPS (Pn3P) to assess whether th

157 peptidoglycan from the midcells of dividing Streptococcus pneumoniae D39 background cells.

158 The respiratory pathogen Streptococcus pneumoniae deploys type IV pili to take up

159 higher in a Type I diabetic rabbit model of Streptococcus pneumoniae endophthalmitis.

160 Streptococcus pneumoniae expresses capsular polysacchari

161 In addition, in vivo infection with Streptococcus pneumoniae failed to induce PU.1 expressio

162 Reduction in yield of Streptococcus pneumoniae from NP culture was approximate

163 est and MALDI-TOF for the differentiation of Streptococcus pneumoniae from other mitis group streptoc

164 Streptococcus pneumoniae has demonstrated a remarkable a

165 d transmission of the opportunistic pathogen Streptococcus pneumoniae in an infant mouse model.

166 g the efficacy of geOMVs as vaccines against Streptococcus pneumoniae in mice, and against Campylobac

167 ing of the spleen, we identify a tropism for Streptococcus pneumoniae in this organ mediated by tissu

168 the mechanistic effects of these factors on Streptococcus pneumoniae infection in mice.

169 es in cerebrospinal fluid from children with Streptococcus pneumoniae infection, compared with childr

170 Streptococcus pneumoniae infections arising in hospitali

171 The Streptococcus pneumoniae Invasive Disease network (SpIDn

172 Streptococcus pneumoniae is a common colonizer of the hu

173 Streptococcus pneumoniae is a leading cause of invasive

174 Serotype-specific protection against Streptococcus pneumoniae is an important limitation of t

175 Pulmonary infection by Streptococcus pneumoniae is characterized by a robust al

176 tend our knowledge on the mechanisms whereby Streptococcus pneumoniae is cleared by the spleen.

177 at the activation of macrophage NF-kappaB by Streptococcus pneumoniae is highly diverse, with a prepo

178 Streptococcus pneumoniae is responsible for diseases cau

179 Streptococcus pneumoniae is the main cause of bacterial

180 We analyzed whole genome sequences of 1,680 Streptococcus pneumoniae isolates from four independent

181 An increase in Streptococcus pneumoniae nasopharynx (NP) colonization d

182 ls were stimulated in vitro with heat-killed Streptococcus pneumoniae or CD3/CD28 antibodies and stai

183 al [CI] = 3.27-5.37; n = 2432 participants), Streptococcus pneumoniae otitis media (OR = 2.51; 95% CI

184 ediated hemolysis of ES PspCN, a CFH-binding Streptococcus pneumoniae protein domain, binds CFH tight

185 Several Streptococcus pneumoniae proteins play a role in pathoge

186 Streptococcus pneumoniae serotype 1 is one of the leadin

187 Using the Streptococcus pneumoniae serotype 2 CPS, which is synthe

188 Streptococcus pneumoniae serotype 3 strains emerge frequ

189 Streptococcus pneumoniae serotype 35B is a nonvaccine se

190 eak was due to multiple pathogens, including Streptococcus pneumoniae serotype 5 and influenza viruse

191 ting invasive pneumococcal disease caused by Streptococcus pneumoniae Some components of the S. pneum

192 related to the capsular polysaccharide from Streptococcus pneumoniae type 37, which consists of a be

193 he commensal genus Neisseria and the species Streptococcus pneumoniae was associated with lower EAC r

194 beta-lactam and co-trimoxazole resistance in Streptococcus pneumoniae with accuracies ranging from 88

195 Pneumococcus (Streptococcus pneumoniae) remains a significant cause of

196 re used to test 10 Staphylococcus aureus, 10 Streptococcus pneumoniae, 10 Haemophilus influenzae, and

197 ryngeal infection by S. pyogenes, but not by Streptococcus pneumoniae, a bacterium that does not prod

198 Natural genetic transformation of Streptococcus pneumoniae, an important human pathogen, m

199 stillation of MS-WF, mice were infected with Streptococcus pneumoniae, and bronchoalveolar lavage flu

200 coli derived lipopolysaccharide, heat-killed Streptococcus pneumoniae, and Mycobacterium tuberculosis

201 lla catarrhalis, Haemophilus influenzae, and Streptococcus pneumoniae, but not other bacterial pathog

202 of pneumolysin, the main virulence factor of Streptococcus pneumoniae, by cryoEM.

203 In Streptococcus pneumoniae, competence develops transientl

204 ding proteins that Staphylococcus aureus and Streptococcus pneumoniae, Gram-positive bacterial pathog

205 and Acanthamoeba), six bacterial pathogens (Streptococcus pneumoniae, Haemophilus influenzae, Neisse

206 ncoding the only PP2C Ser/Thr phosphatase in Streptococcus pneumoniae, indicating that GpsB plays a k

207 cial cell wall constituent of the pathobiont Streptococcus pneumoniae, is bound to peptidoglycan (wal

208 phis infected with the common lung pathogens Streptococcus pneumoniae, Legionella pneumophila, or Myc

209 Streptococcus pneumoniae, like many other naturally tran

210 se bacterial species: Staphylococcus aureus, Streptococcus pneumoniae, Mycobacterium tuberculosis, Sa

211 intratracheally with Klebsiella pneumoniae, Streptococcus pneumoniae, or lipopolysaccharide.

212 teria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus agalactiae, cyto

213 In a number of bacterial species, including Streptococcus pneumoniae, the prevalence of resistance h

214 ative data for the pattern of disease due to Streptococcus pneumoniae, trends in the serotype of inva

215 In Streptococcus pneumoniae, we find a genomically and ecol

216 esults were demonstrated in a mouse model of Streptococcus pneumoniae-induced empyema.

217 PD and challenged with opsonized serotype 14 Streptococcus pneumoniae.

218 so abrogating the virulence of the pathogen Streptococcus pneumoniae.

219 Th) 17 cells are important in the control of Streptococcus pneumoniae.

220 concept of this approach for 2 serotypes of Streptococcus pneumoniae.

221 owth of the major human respiratory pathogen Streptococcus pneumoniae.

222 y erm(B) confer most macrolide resistance in Streptococcus pneumoniae.

223 es was correlated with relative abundance of Streptococcus pneumoniae.

224 e the agglutination of specific serotypes of Streptococcus pneumoniae.

225 ia, including the major respiratory pathogen Streptococcus pneumoniae.

226 cell wall biosynthesis and cell division of Streptococcus pneumoniae.

227 The Streptococcus pnuenomae pilus island 1 pili are composed

228 luding differentiation of S. pneumoniae from Streptococcus pseudopneumoniae.

229 block the endonuclease activity of type II-A Streptococcus pyogene Cas9 (SpyCas9).

230 s aureus (10 [43.5%] vs 4 [12.9%]; P = .02), Streptococcus pyogenes (2 [8.7%] vs 19 [61.3%]; P < .001

231 to the surface of the human pathogen group A Streptococcus pyogenes (GAS) and subsequent hPg activati

232 Dimeric M-proteins (M-Prt) in group A Streptococcus pyogenes (GAS) are surface-expressed virul

233 the Gram-positive human-restricted pathogen Streptococcus pyogenes (Group A Streptococcus, GAS) has

234 The IgG-degrading enzyme derived from Streptococcus pyogenes (IdeS), an endopeptidase, cleaves

235 A reactivity against IgG-degrading enzyme of Streptococcus pyogenes (IdeS)- or pepsin-generated F(ab'

236 en was used to identify mutations in rgg2 of Streptococcus pyogenes (rgg2Sp ) that conferred pheromon

237 The RNA-guided CRISPR-Cas9 nuclease from Streptococcus pyogenes (SpCas9) has been widely repurpos

238 In vitro assays demonstrate that Cas9 from Streptococcus pyogenes (SpCas9) is more active in creati

239 (CRISPR)-associated endonuclease (Cas)9 from Streptococcus pyogenes (SpCas9) is used to deplete VEGFR

240 As an exclusively human pathogen, Streptococcus pyogenes (the group A streptococcus [GAS])

241 tudy, we created an RNase III null mutant of Streptococcus pyogenes and its RNA sequencing (RNA-Seq)

242 ce against lethal soft tissue infection with Streptococcus pyogenes and prevented bacterial dissemina

243 al effects against Staphylococcus aureus and Streptococcus pyogenes and protected against staphylococ

244 ive clinical diagnose include Staphylococci, Streptococcus pyogenes and Pseudomonas aeruginosa in ble

245 ustom DNA-binding modules, the nuclease-dead Streptococcus pyogenes Cas9 (dCas9) protein, which recog

246 The success of Streptococcus pyogenes Cas9 (SpCas9) has led to the disc

247 ss and efficiency as previously reported for Streptococcus pyogenes Cas9 (SpCas9).

248 o 70 degrees C, compared to 45 degrees C for Streptococcus pyogenes Cas9 (SpyCas9), which expands the

249 hese inhibitors also blocked the widely used Streptococcus pyogenes Cas9 when assayed in Escherichia

250 protein containing a catalytically defective Streptococcus pyogenes Cas9, a cytidine deaminase, and a

251 However, in M. tuberculosis, the existing Streptococcus pyogenes Cas9-based CRISPRi system is of l

252 d expression and activity of SLO, DNase, and Streptococcus pyogenes cell envelope protease in vitro.

253 ected mutagenesis of an endoglycosidase from Streptococcus pyogenes of serotype M49 (Endo-S2) and the

254 re, and clears infection in a mouse model of Streptococcus pyogenes peritonitis.

255 Streptococcus pyogenes secretes many toxins that facilit

256 The globally prominent pathogen Streptococcus pyogenes secretes potent immunomodulatory

257 identifying variation, we pooled DNA of 100 Streptococcus pyogenes strains of different emm types in

258 protein against the Gram-positive bacterium Streptococcus pyogenes This protein is composed of two d

259 The Cas9 protein from Streptococcus pyogenes was pre-complexed with a single g

260 oth gram-positive (Staphylococcus aureus and Streptococcus pyogenes) and gram-negative bacteria (Pseu

261 The Group A Streptococcus (GAS, Streptococcus pyogenes) is a Gram-positive human pathoge

262 In Streptococcus pyogenes, a common and potentially deadly

263 tein 9 (Cas9) from Staphylococcus aureus and Streptococcus pyogenes, and recombinant Cas9 and develop

264 resent in pathogenic streptococci, including Streptococcus pyogenes, S. agalactiae, S. pneumoniae, an

265 The important human pathogen, Streptococcus pyogenes, synthesizes a key antigenic surf

266 Studying the pilus tip adhesin Spy0125 of Streptococcus pyogenes, we developed a single molecule a

267 elical peptide epitope from the M protein of Streptococcus pyogenes, were designed by exchanging one

268 with PJI, namely, Staphylococcus aureus and Streptococcus pyogenes.

269 Cas9 enzymes from Staphylococcus aureus and Streptococcus pyogenes.

270 toxins secreted by Staphylococcus aureus and Streptococcus pyogenes.

271 ween ADs in general (that is, not limited to Streptococcus-related conditions) and both OCD and TD/CT

272 al maternal immunisation initiatives-group B streptococcus, respiratory syncytial virus, pertussis, a

273 alysis provide evidence for coinfection with Streptococcus (S.) pneumoniae.

274 Streptococcus gordonii and Streptococcus sanguinis are pioneer colonizers of dental

275 Streptococcus sanguinis is an early colonizer of the too

276 rse biofilms formed by the commensal species Streptococcus sanguinis or Streptococcus gordonii.

277 Streptococcus sanguinis, a naturally competent opportuni

278 t S. oralis, like Streptococcus gordonii and Streptococcus sanguinis, binds platelets via terminal si

279 To enhance genetic manipulation of Streptococcus sanguinis, we have developed a cloning-ind

280 d taxa across the plaque groups, followed by Streptococcus sanguinis, which was highly abundant in CF

281 S-ECC group, and Streptococcus gordonii and Streptococcus sanguinis, which were 5- and 2-fold higher

282 ing in the formation of a fragile biofilm in Streptococcus sanguinis.

283 tive PCR (qPCR) for Streptococcus mutans and Streptococcus sobrinus.

284 with early nasopharyngeal colonization with Streptococcus species and age of first febrile lower res

285 orkflow by predicting essential genes in six Streptococcus species and mapping the essential genes to

286 hylococcus aureus in 7 of 63 (11%) eyes, and Streptococcus species in 7 of 63 (11%) eyes.

287 l reactions in a way that was unique to each Streptococcus species, leading to species-specific outco

288 y can differentiate S. pneumoniae from other Streptococcus species.

289 ncreased abundance of Enterobacteriaceae and Streptococcus spp. and, functionally, in the potential f

290 Streptococcus suis is a zoonotic swine pathogen and a ma

291 trast, alpha-mannosyldiacylglycerol found in Streptococcus suis or alpha-mannosylceramide demonstrate

292 In Streptococcus mutans, Streptococcus suis, and species of the bovis, salivarius

293 t genus level Escherichia, Faecalibacterium, Streptococcus, Sutterella and Veillonella were increased

294 ous of these proteins, the CRISPR1 Cas9 from Streptococcus thermophilus (dCas9Sth1), typically achiev

295 PepX aminopeptidase from Streptococcus thermophilus ACA DC 0022, used in Greek Fe

296 extracted from Pleurotus eryngii (PEPS) and Streptococcus thermophilus ASCC 1275 (EPS) were sulphona

297 ysine-tryptophan bond has been identified in Streptococcus thermophilus, and a reaction mechanism has

298 fied an unrelated Acr in a virulent phage of Streptococcus thermophilus.

299 t with disseminated Mycobacterium abscessus, Streptococcus viridians bacteremia, and cytomegalovirus

300 nosa and Staphylococcus aureus in keratitis; Streptococcus viridians, Streptococcus pneumoniae and Co

301 ria (such as Lactobacillus, Turicibacter and Streptococcus) were found in the stomach and small intes