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1 the surface of the human commensal bacterium Streptococcus gordonii.
2 re supernatants of Staphylococcus aureus and Streptococcus gordonii.
3 the presence of antecedent organisms such as Streptococcus gordonii.
4 s on rates of hydrogen peroxide synthesis by Streptococcus gordonii.
5 by adhering to early plaque bacteria such as Streptococcus gordonii.
6 commensal species Streptococcus sanguinis or Streptococcus gordonii.
7 with the antecedent oral biofilm constituent Streptococcus gordonii.
8 attaches and forms biofilms on substrata of Streptococcus gordonii.
9 ggregation receptor polysaccharides (RPS) of Streptococcus gordonii 38 and Streptococcus oralis J22 w
13 dominant salivary enzyme in humans, binds to Streptococcus gordonii, a primary colonizer of the tooth
14 oped in this study assessed a multi-species (Streptococcus gordonii, Actinobacillus actinomycetemcomi
17 an important determinant of colonization by Streptococcus gordonii, an oral commensal and opportunis
18 nvestigated its role in biofilm formation by Streptococcus gordonii, an organism that colonizes human
19 erence of early colonizing organisms such as Streptococcus gordonii and Actinomyces naeslundii to the
20 en paired with two other initial colonizers, Streptococcus gordonii and Actinomyces oris, as well as
21 Structural studies of the type-C PPases from Streptococcus gordonii and Bacillus subtilis reveal a ho
23 nge between two bacterial species, commensal Streptococcus gordonii and pathogenic Streptococcus muta
28 the human oral flora (Streptococcus mutans, Streptococcus gordonii and Streptococcus sanguinis) to d
30 igher, respectively, in the S-ECC group, and Streptococcus gordonii and Streptococcus sanguinis, whic
31 dulated by coculture with the oral commensal Streptococcus gordonii and the opportunistic commensal F
32 ction in vivo, mice were first infected with Streptococcus gordonii and then challenged with P. gingi
33 C, showed an increased ability to adhere to Streptococcus gordonii and to invade primary cultures of
36 l was synthesized, and Streptococcus mutans, Streptococcus gordonii, and Streptococcus sanguinis were
48 velops biofilm microcolonies on substrata of Streptococcus gordonii but not on Streptococcus mutans.
49 of PMNs or HL-60 cells abolished adhesion of Streptococcus gordonii but was required for adhesion of
50 pathogen Porphyromonas gingivalis adheres to Streptococcus gordonii by interacting with a specific re
56 a 585-bp gene was cloned and sequenced from Streptococcus gordonii Challis encoding a 20.5-kDa amyla
59 tococcus pyogenes) exposed on the surface of Streptococcus gordonii commensal bacterial vectors: (i)
60 produced by the glucosyltransferase (GTF) of Streptococcus gordonii confer a hard, cohesive phenotype
61 tion with P. gingivalis, whereas noninvasive Streptococcus gordonii did not have a significant effect
62 acid-containing receptors is associated with Streptococcus gordonii DL1 (Challis) but not with a spon
66 ageneric coaggregation, streptococci such as Streptococcus gordonii DL1 recognize receptor polysaccha
67 ne cluster and used to transform E. coli and Streptococcus gordonii DL1, a nonureolytic, dental plaqu
69 ating early colonizers of the tooth surface (Streptococcus gordonii DL1, Streptococcus oralis 34, and
70 he scaCBA operon in the human oral bacterium Streptococcus gordonii encodes the components of an ABC-
72 pV, a dipeptidase found in culture fluids of Streptococcus gordonii FSS2, was purified and characteri
73 polypeptide (259 kDa) in the oral bacterium Streptococcus gordonii, generates mutants that are marke
78 parasanguinis, Streptococcus sanguinis, and Streptococcus gordonii, inhibit the growth of P. aerugin
80 omonas gingivalis and the accessory pathogen Streptococcus gordonii interact to form communities in v
82 ed by early dental plaque colonizers such as Streptococcus gordonii interfere with the subsequent col
84 eptococcal coaggregation regulator (ScaR) of Streptococcus gordonii is a manganese-dependent transcri
95 ort, we show that the A regions from the two Streptococcus gordonii M5 antigen I/II proteins (SspA an
96 large cell-surface glycoprotein expressed by Streptococcus gordonii M99 that mediates binding of this
97 he cytosolic O-glycosyltransferase GtfA/B of Streptococcus gordonii modifies the Ser/Thr-rich repeats
100 ed with PAAP(+) S. sanguis than with PAAP(-) Streptococcus gordonii or type II collagen, suggesting a
102 p38 and then stimulated with oral commensal Streptococcus gordonii, oral pathogens Porphyromonas gin
103 r example, more Streptococcus sanguinis than Streptococcus gordonii organisms are consistently isolat
105 oral commensals Streptococcus sanguinis and Streptococcus gordonii release DNA in a process induced
107 nalling between Porphyromonas gingivalis and Streptococcus gordonii serves to constrain development o
108 ion of hydrogen peroxide in solution above a Streptococcus gordonii (Sg) bacterial biofilm was studie
109 could detect 65 +/- 10 muM H2O2 produced by Streptococcus gordonii (Sg) in a simulated biofilm at 50
113 uence motif within the C-terminal portion of Streptococcus gordonii SspB (AgI/II) is bound by Porphyr
114 tion between Streptococcus mutans AgI/II and Streptococcus gordonii SspB in their interaction with th
115 icroorganisms, e.g., Veillonella parvula and Streptococcus gordonii, stimulated higher levels of ROS
122 ologous serine-rich surface glycoproteins of Streptococcus gordonii strains M99 and Challis, respecti
123 nces in virulence among seven representative Streptococcus gordonii strains were observed by using th
124 infantis group, Corynebacterium matruchotii, Streptococcus gordonii, Streptococcus cristatus, Capnocy
125 (Streptococcus mitis, Streptococcus oralis, Streptococcus gordonii, Streptococcus cristatus, Strepto
126 t AgI/II family polypeptides from strains of Streptococcus gordonii, Streptococcus intermedius and St
127 omologous enzymes in Streptococcus pyogenes, Streptococcus gordonii, Streptococcus mutans, Staphyloco
128 phylococcus aureus, Streptococcus sanguinis, Streptococcus gordonii, Streptococcus oralis, and Strept
129 nt understanding of accessory Sec systems in Streptococcus gordonii, Streptococcus parasanguinis, Myc
130 racterized and closely related oral species, Streptococcus gordonii, Streptococcus sanguinis, and car
131 lizes lactate produced by the oral bacterium Streptococcus gordonii, suggesting the potential for cro
132 Hsa are homologous surface glycoproteins of Streptococcus gordonii that bind sialic acid moieties on
133 (30-day) colonization of smooth surfaces by Streptococcus gordonii that incorporates the nutrient fl
134 spB is a serine-rich glycoprotein adhesin of Streptococcus gordonii that is exported to the bacterial
137 ies have shown that P. gingivalis adheres to Streptococcus gordonii through interaction of the minor
138 SspB (antigen I/II family proteins) can bind Streptococcus gordonii to other oral bacteria and also t
142 erobes, including aerotolerant ones, such as Streptococcus gordonii, use pyruvate dehydrogenase to de
144 The amylase-binding protein A (AbpA) of Streptococcus gordonii was found to be undetectable in s
145 tA gene in the human oral commensal organism Streptococcus gordonii was insertionally inactivated.
146 tococcus oralis, Streptococcus sanguinis, or Streptococcus gordonii was investigated using flow cell
147 gnated atlS, encoding a major autolysin from Streptococcus gordonii, was identified and characterized
148 To investigate disulfide bond formation in Streptococcus gordonii, we identified five putative TDOR
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